Search results for "EOs"

showing 10 items of 2714 documents

Sng1 associates with Nce102 to regulate the yeast Pkh–Ypk signalling module in response to sphingolipid status

2016

International audience; All cells are delimited by biological membranes, which are consequently a primary target of stress-induced damage. Cold alters membrane functionality by decreasing lipid fluidity and the activity of membrane proteins. In Saccharomyces cerevisiae, evidence links sphingolipid homeostasis and membrane phospholipid asymmetry to the activity of the Ypk1/2 proteins, the yeast orthologous of the mammalian SGK1-3 kinases. Their regulation is mediated by different protein kinases, including the PDK1 orthologous Pkh1/2p, and requires the function of protein effectors, among them Nce102p, a component of the sphingolipid sensor machinery. Nevertheless, the mechanisms and the act…

0301 basic medicineMyriocinOrm2Saccharomyces-cerevisiaeMembrane propertiesFatty Acids MonounsaturatedGlycogen Synthase Kinase 3Bacteriocins[SDV.IDA]Life Sciences [q-bio]/Food engineeringHomeostasisPhosphorylationMicroscopy ConfocalbiologyEffectorPlasma-membraneActin cytoskeleton[ SDV.IDA ] Life Sciences [q-bio]/Food engineeringPhospholipid translocationTransmembrane proteinCell biologyCold TemperatureBiochemistryP-type atpasesSignal transductionCold stressCell-wall integrityProtein BindingSignal TransductionProteins slm1Saccharomyces cerevisiae ProteinsPhospholipid translocationHigh-pressureSaccharomyces cerevisiaeImmunoblottingFluorescence PolarizationSaccharomyces cerevisiaeSignallingModels Biological3-Phosphoinositide-Dependent Protein Kinases03 medical and health sciencesBudding yeastMolecular BiologySphingolipids030102 biochemistry & molecular biologyTryptophan permeasePhospholipid flippingMembrane ProteinsCell Biologybiology.organism_classificationActin cytoskeletonSphingolipidYeast030104 developmental biologyMembrane proteinMutationPeptidesReactive Oxygen Species
researchProduct

Nrf2 expression driven by Foxp3 specific deletion of Keap1 results in loss of immune tolerance in mice

2020

European journal of immunology 50(4), 515-524 (2020). doi:10.1002/eji.201948285

0301 basic medicineNF-E2-Related Factor 2T cellImmunologyAutoimmunitychemical and pharmacologic phenomenaBiologyLymphocyte ActivationT-Lymphocytes Regulatorydigestive systemenvironment and public healthImmune toleranceImmunomodulationMice03 medical and health sciences0302 clinical medicineImmune systemImmune TolerancemedicineAnimalsHomeostasisImmunology and AllergyTranscription factorPI3K/AKT/mTOR pathwayInflammationMice KnockoutKelch-Like ECH-Associated Protein 1ChimeraEffectorTOR Serine-Threonine KinasesPeripheral toleranceFOXP3Forkhead Transcription Factorshemic and immune systemsrespiratory systemCell biologyMice Inbred C57BLOxidative Stress030104 developmental biologymedicine.anatomical_structure030215 immunology
researchProduct

Alignment Free Dissimilarities for Nucleosome Classification

2016

Epigenetic mechanisms such as nucleosome positioning, histone modifications and DNA methylation play an important role in the regulation of cell type-specific gene activities, yet how epigenetic patterns are established and maintained remains poorly understood. Recent studies have shown a role of DNA sequences in recruitment of epigenetic regulators. For this reason, the use of more suitable similarities or dissimilarity between DNA sequences could help in the context of epigenetic studies. In particular, alignment-free dissimilarities have already been successfully applied to identify distinct sequence features that are associated with epigenetic patterns and to predict epigenomic profiles…

0301 basic medicineNearest neighbour classifiersKnn classifierSettore INF/01 - Informatica030102 biochemistry & molecular biologybiologyComputer scienceSpeech recognitionEpigeneticContext (language use)Computational biologyL-tuples03 medical and health sciences030104 developmental biologyHistoneSimilarity (network science)DNA methylationbiology.proteinNucleosomeEpigeneticsAlignment free DNA sequence dissimilaritiesk-mersNucleosome classificationEpigenomics
researchProduct

Chronic intestinal inflammation in mice expressing viral Flip in epithelial cells

2018

Viruses are present in the intestinal microflora and are currently discussed as a potential causative mechanism for the development of inflammatory bowel disease. A number of viruses, such as Human Herpesvirus-8, express homologs to cellular FLIPs, which are major contributors for the regulation of epithelial cell death. In this study we analyzed the consequences of constitutive expression of HHV8-viral FLIP in intestinal epithelial cells (IECs) in mice. Surprisingly, expression of vFlip disrupts tissue homeostasis and induces severe intestinal inflammation. Moreover vFlip(IEC-tg) mice showed reduced Paneth cell numbers, associated with excessive necrotic cell death. On a molecular level vF…

0301 basic medicineNecrosisTransgeneImmunologyInflammationMice TransgenicBiologydigestive systemArticle03 medical and health sciencesMiceNecrosisViral ProteinsmedicineImmunology and AllergyAnimalsHomeostasisHumansTissue homeostasisCells CulturedRegulation of gene expressionMice KnockoutNF-kappa BHerpesviridae InfectionsInflammatory Bowel DiseasesEpitheliumCell biologyI-kappa B KinaseIntestines030104 developmental biologymedicine.anatomical_structureEnterocytesGene Expression RegulationFlipPaneth cellHerpesvirus 8 Humanmedicine.symptom
researchProduct

Cholinergic signaling controls immune functions and promotes homeostasis

2020

Abstract Acetylcholine (ACh) was created by nature as one of the first signaling molecules, expressed already in procaryotes. Based on the positively charged nitrogen, ACh could initially mediate signaling in the absence of receptors. When evolution established more and more complex organisms the new emerging organs systems, like the smooth and skeletal muscle systems, energy-generating systems, sexual reproductive system, immune system and the nervous system have further optimized the cholinergic signaling machinery. Thus, it is not surprising that ACh and the cholinergic system are expressed in the vast majority of cells. Consequently, multiple common interfaces exist, for example, betwee…

0301 basic medicineNervous systemCell signalingImmunologyCholinergic AgentsBiology03 medical and health sciences0302 clinical medicineImmune systemMemorymedicineAnimalsHomeostasisHumansImmunologic FactorsLearningImmunology and AllergyReceptorOrganismPharmacologyImmunity030104 developmental biologymedicine.anatomical_structureImmune System030220 oncology & carcinogenesisCholinergicNeuroscienceHomeostasisAcetylcholineSignal Transductionmedicine.drugInternational Immunopharmacology
researchProduct

Homeostatic interplay between electrical activity and neuronal apoptosis in the developing neocortex

2017

An intriguing feature of nervous system development in most animal species is that the initial number of generated neurons is higher than the number of neurons incorporated into mature circuits. A substantial portion of neurons is indeed eliminated via apoptosis during a short time window - in rodents the first two postnatal weeks. While it is well established that neurotrophic factors play a central role in controlling neuronal survival and apoptosis in the peripheral nervous system (PNS), the situation is less clear in the central nervous system (CNS). In postnatal rodent neocortex, the peak of apoptosis coincides with the occurrence of spontaneous, synchronous activity patterns. In this …

0301 basic medicineNervous systemCentral nervous systemApoptosisNeocortexBiologyMembrane Potentials03 medical and health sciences0302 clinical medicineNeurotrophic factorsmedicineAnimalsHumansNeuronsNeocortexGeneral Neuroscience030104 developmental biologymedicine.anatomical_structurenervous systemApoptosisCerebral cortexPeripheral nervous systemSynapsesCalciumNeuroscience030217 neurology & neurosurgeryHomeostasisNeuroscience
researchProduct

Tuning neural circuits by turning the interneuron knob

2017

Interneurons play a critical role in sculpting neuronal circuit activity and their dysfunction can result in neurological and neuropsychiatric disorders. To temporally structure and balance neuronal activity in the adult brain interneurons display a remarkable degree of subclass-specific plasticity, of which the underlying molecular mechanisms have recently begun to be elucidated. Grafting new interneurons to pre-existing neuronal networks allows for amelioration of circuit dysfunction in rodent models of neurological disease and can reopen critical windows for circuit plasticity. The crucial contribution of specific classes of interneurons to circuit homeostasis and plasticity in health an…

0301 basic medicineNeuronal PlasticityInterneurongenetic structuresGeneral NeurosciencefungiBiology03 medical and health sciences030104 developmental biology0302 clinical medicinemedicine.anatomical_structurenervous systemInterneuronsmedicineBiological neural networkPremovement neuronal activityAnimalsHomeostasisHumansNervous System DiseasesReprogrammingNeuroscience030217 neurology & neurosurgery
researchProduct

Impact of Cholesterol Metabolism in Immune Cell Function and Atherosclerosis

2020

Cholesterol, the most important sterol in mammals, helps maintain plasma membrane fluidity and is a precursor of bile acids, oxysterols, and steroid hormones. Cholesterol in the body is obtained from the diet or can be de novo synthetized. Cholesterol homeostasis is mainly regulated by the liver, where cholesterol is packed in lipoproteins for transport through a tightly regulated process. Changes in circulating lipoprotein cholesterol levels lead to atherosclerosis development, which is initiated by an accumulation of modified lipoproteins in the subendothelial space; this induces significant changes in immune cell differentiation and function. Beyond lesions, cholesterol levels also play …

0301 basic medicineNeutrophilsLipoproteinsT-LymphocytesT cellInflammationlcsh:TX341-641Review030204 cardiovascular system & hematologyMonocytesMice03 medical and health scienceschemistry.chemical_compound0302 clinical medicineImmune systemimmune cellsmedicineAnimalsHomeostasisHumansCell ProliferationImmunity CellularNutrition and DieteticsChemistryCholesterolMacrophagesMonocytecholesterolLipid MetabolismSterolhematopoiesisCell biology030104 developmental biologymedicine.anatomical_structureLiverinflammationlipids (amino acids peptides and proteins)medicine.symptomatherosclerosismetabolismlcsh:Nutrition. Foods and food supplyIntracellularFood ScienceHormoneNutrients
researchProduct

Sucralose and Cardiometabolic Health: Current Understanding from Receptors to Clinical Investigations

2021

International audience; The excess consumption of added sugar is consistently found to be associated with weight gain, and a higher risk of type 2 diabetes mellitus, coronary heart disease, and stroke. In an effort to reduce the risk of cardiometabolic disease, sugar is frequently replaced by low- and null-calorie sweeteners (LCSs). Alarmingly, though, emerging evidence indicates that the consumption of LCSs is associated with an increase in cardiovascular mortality risk that is amplified in those who are overweight or obese. Sucralose, a null-caloric high-intensity sweetener, is the most commonly used LCS worldwide, which is regularly consumed by healthy individuals and patients with metab…

0301 basic medicineNon-Nutritive SweetenersSucroseSucraloseCalorieglucose metabolismMedicine (miscellaneous)030209 endocrinology & metabolismReviewOverweightGut floraAdded sugar03 medical and health scienceschemistry.chemical_compound0302 clinical medicineEnvironmental healthmedicineHumansGlucose homeostasis2. Zero hungerNutrition and Dieteticsbiologybusiness.industrysweet and bitter taste receptorType 2 Diabetes Mellitussucralosetaste signaling cascadecardiovascular healthbiology.organism_classification3. Good health030104 developmental biologyDiabetes Mellitus Type 2chemistryCardiovascular Diseaseslow-calorie sweetenermedicine.symptombusiness[SDV.AEN]Life Sciences [q-bio]/Food and NutritionWeight gainFood ScienceAdvances in Nutrition
researchProduct

Subtracting the sequence bias from partially digested MNase-seq data reveals a general contribution of TFIIS to nucleosome positioning.

2017

Background TFIIS stimulates RNA cleavage by RNA polymerase II and promotes the resolution of backtracking events. TFIIS acts in the chromatin context, but its contribution to the chromatin landscape has not yet been investigated. Co-transcriptional chromatin alterations include subtle changes in nucleosome positioning, like those expected to be elicited by TFIIS, which are elusive to detect. The most popular method to map nucleosomes involves intensive chromatin digestion by micrococcal nuclease (MNase). Maps based on these exhaustively digested samples miss any MNase-sensitive nucleosomes caused by transcription. In contrast, partial digestion approaches preserve such nucleosomes, but intr…

0301 basic medicineNucleosome mappinglcsh:QH426-470MNase-sensitive nucleosomesRNA polymerase IIComputational biologySaccharomyces cerevisiaeReal-Time Polymerase Chain ReactionBiotecnologia03 medical and health sciencesTranscription (biology)Gene expressionGeneticsNucleosomeMNase-seqMicrococcal NucleaseMolecular BiologyGenebiologyMethodologyHigh-Throughput Nucleotide SequencingPromoterChromatinNucleosomeslcsh:Genetics030104 developmental biologyNucleosomal fuzzinessSubtraction TechniqueTFIISbiology.proteinTranscriptional Elongation FactorsGenèticaMicrococcal nuclease
researchProduct