Search results for "ERYTHROPOIETIN"

showing 10 items of 113 documents

The Effect of Erythropoietin on Tumor Oxygenation in Normal and Anemic Rats

1994

Anemia associated with malignancy is a common clinical problem. Its etiology is varied and includes nutritional causes, hemorrhage, hemolysis, bone marrow metastasis and hypoplasia, paraneoplastic syndromes, and chemotherapy1, with many patients presenting with anemia even before they receive cytotoxic therapy and even if their bone marrow is not invaded by tumor cells2. The response of tumors to standard radiotherapy and oxygen-dependent chemotherapy in these patients is often less satisfactory than in subjects with normal hemoglobin levels3. This is presumed to be due to the worsening of tumor oxygenation as a result of the decreased oxygen-carrying capacity of the blood in these anemic t…

medicine.medical_specialtyChemotherapymedicine.diagnostic_testbusiness.industryAnemiamedicine.medical_treatmentTumor OxygenationHematocritmedicine.diseaseMalignancyGastroenterologymedicine.anatomical_structureErythropoietinInternal medicinemedicineBone marrowSarcomabusinessmedicine.drug
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Erythropoietin in Traumatic Brain Injury: An Answer Will Come Soon

2015

Traumatic brain injury (TBI) is a major cause of morbidity and mortality in the United States. It is estimated that each year TBIs are associated with 1.1 million emergency department visits, 235,000 hospitalizations, and 50,000 deaths (1). Despite improvements in medical interventions, there are still no neuroprotective agents available to counteract secondary or delayed damage to the traumatically injured human brain or to promote its repair. TBI encompasses heterogeneous etiologic, anatomical, and molecular patterns of injury that exhibit different propensities to cause cerebral damage. Without careful consideration of individual injuries, the results of therapeutic trials remain difficu…

medicine.medical_specialtyClinical Trials as Topicbusiness.industryTraumatic brain injurySettore MED/27 - NeurochirurgiaMedicine (all)Neuroprotective AgentRecombinant Proteinmedicine.diseaseRecombinant ProteinsEpoetin AlfaNeuroprotective AgentsErythropoietinBrain InjuriesBrain InjurieReceptors ErythropoietinMedicineHumansSurgeryNeurology (clinical)businessIntensive care medicineErythropoietinmedicine.drugHuman
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Neurodegenerative changes are prevented by Erythropoietin in the pmn model of motoneuron degeneration

2014

Motoneuron diseases are fatal neurodegenerative disorders characterized by a progressive loss of motoneurons, muscle weakness and premature death. The progressive motor neuronopathy (pmn) mutant mouse has been considered a good model for the autosomal recessive childhood form of spinal muscular atrophy (SMA). Here, we investigated the therapeutic potential of Erythropoietin (Epo) on this mutant mouse. Symptomatic or pre-symptomatic treatment with Epo significantly prolongs lifespan by 84.6% or 87.2% respectively. Epo preserves muscle strength and significantly attenuates behavioural motor deficits of mutant pmn mice. Histological and metabolic changes in the spinal cord evaluated by immunoh…

medicine.medical_specialtyMutantMotor ActivitySpinal Muscular Atrophies of ChildhoodMiceCellular and Molecular NeuroscienceWestern blotInternal medicineReceptors ErythropoietinmedicineAnimalsErythropoietinMotor NeuronsPharmacologymedicine.diagnostic_testbusiness.industryMuscle weaknessSpinal muscular atrophymedicine.diseaseSpinal cordSMA*Mice Mutant StrainsDisease Models Animalmedicine.anatomical_structureEndocrinologySpinal CordErythropoietinImmunohistochemistrymedicine.symptombusinessNeurosciencemedicine.drugNeuropharmacology
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Erythropoietin treatment of the anaemia of myelofibrosis with myeloid metaplasia: results in 20 patients and review of the literature

2004

Recombinant human erythropoietin (rHuEPO) is an effective treatment for the anaemia that occurs secondary to various conditions, but its role in myelofibrosis with myeloid metaplasia (MMM) is not well established. rHuEPO, at an initial dose of 10 000 U thrice a week, was given to 20 patients with MMM and anaemia. Complete response (CR) was defined as transfusion cessation with normal haemoglobin (Hb) levels and partial response (PR) as a transfusion decrease > or =50% and Hb > 10 g/dl maintained for at least 8 weeks. Nine patients (45%) showed a favourable response to treatment, including four CR and five PR, four of whom have maintained their response at a median follow-up of 12.5 months (…

medicine.medical_specialtyMyeloidHematologybusiness.industryAnemiaHematologymedicine.diseaseResponse to treatmentGastroenterologymedicine.anatomical_structureErythropoietinTransfusion requirementMetaplasiaInternal medicineImmunologyMedicinemedicine.symptombusinessMyelofibrosismedicine.drugBritish Journal of Haematology
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Apoptotic role of Fas/Fas ligand system in the regulation of erythropoiesis

1999

Abstract The possible involvement of Fas and Fas ligand (FasL) in the regulation of erythropoiesis was evaluated. Immunohistochemistry of normal bone marrow specimens revealed that several immature erythroblasts undergo apoptosis in vivo. Analysis of bone marrow erythroblasts and purified progenitors undergoing unilineage erythroid differentiation showed that Fas is rapidly upregulated in early erythroblasts and expressed at high levels through terminal maturation. However, Fas crosslinking was effective only in less mature erythroblasts, particularly at basophilic level, where it induced apoptosis antagonized by high levels of erythropoietin (Epo). In contrast, FasL was selectively induced…

medicine.medical_specialtyPopulationImmunologyBiologyBiochemistryFas ligandReticulocyteSettore MED/04 - PATOLOGIA GENERALEInternal medicinehemic and lymphatic diseasesmedicineCytotoxic T celleducationeducation.field_of_studyhemic and immune systemsCell BiologyHematologyCell biologyHaematopoiesismedicine.anatomical_structureEndocrinologyErythropoietinApoptosisErythropoiesismedicine.drugcirculatory and respiratory physiology
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An overview of new pharmacological treatments for cerebrovascular dysfunction after experimental subarachnoid hemorrhage

2004

Cerebral vasospasm and the resulting cerebral ischemia occurring after subarachnoid hemorrhage (SAH) are still responsible for the considerable morbidity and mortality in patients affected by cerebral aneurysms. Mechanisms contributing to the development of vasospasm, abnormal reactivity of cerebral arteries and cerebral ischemia after SAH have been intensively investigated in recent years. It has been suggested that the pathogenesis of vasospasm is related to a number of pathological processes, including endothelial damage, smooth muscle cell contraction resulting from spasmogenic substances generated during lyses of subarachnoid blood clots, changes in vascular responsiveness and inflamma…

medicine.medical_specialtyPotassium ChannelsSubarachnoid hemorrhageCerebral arteriesIschemiaNeuroprotectionCerebral vasospasmInternal medicinemedicineAnimalsHumansNitric Oxide Donorscardiovascular diseasesErythropoietinVascular diseasebusiness.industryEndothelinsGeneral NeuroscienceVasospasmSubarachnoid Hemorrhagemedicine.diseaseRecombinant Proteinsnervous system diseasesCerebrovascular DisordersErythropoietinAnesthesiaCardiologyNeurology (clinical)businessmedicine.drugBrain Research Reviews
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Rapid hemodilution induced by desmopressin after erythropoietin administration in humans

2011

We have shown that treatment with desmopressin has a very effective hemodilution effect in healthy humans. These results led us to suggest the possible role of desmopressin to mask blood doping in sports. Based on our results, the World Anti-Doping Agency included the desmopressin in the 2011 List of Prohibited Substances and Methods. On this occasion, the aim of our study was to test the desmopressin-induced hemodilution after rHuEpo administration in humans. This was an intra-subject, crossover study in which five physically active males acted as their own controls. A basal blood sample was taken on their first visit to the laboratory. The next day, the subjects began the treatment. They …

medicine.medical_specialtyReticulocytesPLASMA VOLUME EXPANDERS[SDV]Life Sciences [q-bio]Physical Therapy Sports Therapy and RehabilitationHematocrit01 natural sciences03 medical and health sciences0302 clinical medicineBlood dopingInternal medicinemedicineEducación Física y DeportivaHaematocritHemoglobinSTIMULATION INDEXlcsh:Sports medicineDesmopressinHEMOGLOBINmedicine.diagnostic_testbusiness.industry010401 analytical chemistryPlasma volume expanders030229 sport sciencesCrossover study3. Good health0104 chemical sciencesStimulation index and plasma volume expandersEndocrinologyBasal (medicine)HematocritErythropoietinAnesthesiaHemoglobinHEMATOCRITHaemoglobinbusinesslcsh:RC1200-1245hormones hormone substitutes and hormone antagonistsmedicine.drugJournal of Human Sport and Exercise
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Erythropoietin and the heart: physiological effects and the therapeutic perspective.

2014

Erythropoietin (Epo) has been thought to act exclusively on erythroid progenitor cells. The identification of Epo receptor (EpoR) in non-haematopoietic cells and tissues including neurons, astrocytes, microglia, immune cells, cancer cell lines, endothelial cells, bone marrow stromal cells, as well as cells of myocardium, reproductive system, gastrointestinal tract, kidney, pancreas and skeletal muscle indicates that Epo has pleiotropic actions. Epo shows signals through protein kinases, anti-apoptotic proteins and transcription factors. In light of interest of administering recombinant human erythropoietin (rhEpo) and its analogues for limiting infarct size and left ventricular (LV) remodel…

medicine.medical_specialtyStromal cellCardiotonic AgentsAngiogenesisNeovascularization PhysiologicInflammationerythroid progenitor cellshemic and lymphatic diseasesInternal medicineEpo receptorReceptors ErythropoietinMedicineHumansErythropoietinCardioprotectionMicrogliabusiness.industryHeartErythropoietin receptorErythropoietin; Epo receptor; erythroid progenitor cellsEndocrinologymedicine.anatomical_structureErythropoietinCancer researchAirway RemodelingBone marrowmedicine.symptomCardiology and Cardiovascular Medicinebusinessmedicine.drugInternational journal of cardiology
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Erythropoietin for Subarachnoid Hemorrhage: Is There a Reason for Hope?

2011

I b S a r a S ubarachnoid hemorrhage (SAH) associated with a ruptured cerebral aneurysm remains a source of significant morbidity and mortality, not only from the initial hemorrhage but lso from the delayed complications, such as cerebral vasospasm ften severe enough to induce irreversible changes in cerebral erfusion. Several drugs have been developed that have the otential to limit cerebral vasospasm and delayed ischemic eurologic deficit, thus improving outcome for patients. Howver, although numerous agents that can prevent arterial narrowng and/or block the excitatory cascade of events leading to schemic neuronal death in experimental conditions, there is still o pharmacologic agent tha…

medicine.medical_specialtySubarachnoid hemorrhageErythropoietin subarachnoid hemorrhageBlood volumeAneurysm RupturedNeuroprotectionAneurysmCerebral vasospasmInternal medicinemedicineErythropoietin; Neuroprotection; Subarachnoid hemorrhageHumansSubarachnoid hemorrhageErythropoietinSettore MED/27 - Neurochirurgiabusiness.industryEpoetin alfaPharmacologic Agentmedicine.diseaseRecombinant ProteinsNeuroprotectionEpoetin AlfaNeuroprotective AgentsErythropoietinCardiologySurgeryNeurology (clinical)businessmedicine.drug
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The Role of Erythropoietin in Aneurysmal Subarachnoid Haemorrhage: From Bench to Bedside

2014

Subarachnoid haemorrhage (SAH) caused by a ruptured aneurysm accounts for only 5 % of strokes, but occurs at a fairly young age and carries a poor prognosis. Delayed cerebral ischaemia (DCI) is an important cause of death and dependence after aneurysmal SAH. The current mainstay of preventing DCI is nimodipine and maintenance of normovolemia, but even with this strategy DCI occurs in a considerable proportion of patients.

medicine.medical_specialtySubarachnoid hemorrhagebusiness.industryVasospasmmedicine.diseaseBench to bedsidenervous system diseasesAneurysmErythropoietinInternal medicinemedicineCardiologySubarachnoid haemorrhagecardiovascular diseasesbusinessNimodipinemedicine.drugCause of death
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