Search results for "ESTRADIOL"

showing 10 items of 203 documents

Associations of resting and peak fat oxidation with sex hormone profile and blood glucose control in middle-aged women.

2022

Background and Aims Menopause may reduce fat oxidation. We investigated whether sex hormone profile explains resting fat oxidation (RFO) or peak fat oxidation (PFO) during incremental cycling in middle-aged women. Secondarily, we studied associations of RFO and PFO with glucose regulation. Method and Results We measured RFO and PFO of 42 women (age 52–58 years) with indirect calorimetry. Seven participants were pre- or perimenopausal, 26 were postmenopausal, and nine were postmenopausal hormone therapy users. Serum estradiol (E2), follicle-stimulating hormone, progesterone, and testosterone levels were quantified with immunoassays. Insulin sensitivity (Matsuda index) and glucose tolerance (…

Blood GlucoseestradioliNutrition and Dieteticsvaihdevuodetglucose toleranceEndocrinology Diabetes and MetabolismhapettuminenmenopauseMedicine (miscellaneous)fat oxidationGlycemic ControlMiddle Agedinsuliiniresistenssirasva-aineenvaihduntahormonaaliset tekijätGlucoseglukoosi-intoleranssiestradiolBody Compositioninsulin sensitivityHumansFemaleInsulin ResistanceCardiology and Cardiovascular MedicineGonadal Steroid HormonesNutrition, metabolism, and cardiovascular diseases : NMCD
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Effects Of Combined Strength And Endurance Training On Physical Performance And Biomarkers Of Healthy Young Women

2018

Kyröläinen, H, Hackney, AC, Salminen, R, Repola, J, Häkkinen, K, and Haimi, J. Effects of combined strength and endurance training on physical performance and biomarkers of healthy young women. J Strength Cond Res 32(6): 1554– 1561, 2018—Cardiovascular fitness has decreased and obesity has increased in young adults worldwide during the last 10 years. Therefore, there is an urgent need to find out optimal exercise training programs for improving physical performance and health outcomes, especially among sedentary women. Subjects were 25- to 30-year-old women with a very low physical activity, and 65% of them were overweight (body mass index . 25). They performed endurance and strength traini…

Blood GlucosenaisetHydrocortisonekestävyysharjoitteluIsometric exerciseOverweightliikuntaBody Mass Indexsedentary womenHemoglobinsphysical training0302 clinical medicineSex Hormone-Binding GlobulinTestosteroneOrthopedics and Sports Medicine030212 general & internal medicineta315EstradiolVO2 maxGeneral MedicineLipoproteins LDLfyysinen kuntoCholesterolFemalevoimaharjoittelumedicine.symptomLipoproteins HDLAdultmedicine.medical_specialtyStrength trainingPhysical Therapy Sports Therapy and Rehabilitation03 medical and health sciencesOxygen ConsumptionEndurance trainingmedicineHumansMuscle StrengthExercise physiologyMuscle SkeletalExerciseTriglycerideskehonkoostumusnuoret aikuisetbody compositionhormonesblood lipidsbusiness.industryResistance Training030229 sport sciencesmedicine.diseaseObesityhormonitBicyclingPhysical Endurancephysical fitnessPhysical therapySedentary BehaviorbusinessBody mass indexBiomarkers
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The effect of hormone replacement therapy on Ca2+ mobilization and P-selectin (CD62P) expression in platelets examined under flow cytometry.

2004

A series of events, such as increase of cytoplasmic free calcium (Ca 2+ ) and expression of P-selectin (CD62P), an adhesion molecule, on the platelet surface, are significant indicators of platelet activation. We have used flow cytometry to examine Ca 2+ mobilization and CD62P expression in platelets in whole blood obtained in women prior to, and after, different forms of hormone replacement therapy. Thirty-two women completed a protocol consisting of two consecutive 1-month periods under oestradlol (E 2 ), administered orally (2 mg/day) or transdermally (50 μg/day) in random order, followed by a 4-week transdermal sequential regime, in which, during the last 14 days, either progesterone (3…

Blood Plateletsmedicine.medical_specialtyCytoplasmP-selectinHormone Replacement Therapychemistry.chemical_compoundInternal medicinemedicineMedroxyprogesterone acetateHumansPlateletPlatelet activationWhole bloodTransdermalEstradiolHematologyGeneral MedicineMiddle AgedPlatelet ActivationAdenosine diphosphateP-SelectinEndocrinologychemistryGene Expression RegulationCalciumFemaleMenopauseHormonemedicine.drugBlood coagulationfibrinolysis : an international journal in haemostasis and thrombosis
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The selective estrogen receptor modulator, bazedoxifene, reduces ischemic brain damage in male rat

2014

While the estrogen treatment of stroke is under debate, selective estrogen receptor modulators (SERMs) arise as a promising alternative. We hypothesize that bazedoxifene (acetate, BZA), a third generation SERM approved for the treatment of postmenopausal osteoporosis, reduces ischemic brain damage in a rat model of transient focal cerebral ischemia. For comparative purposes, the neuroprotective effect of 17β-estradiol (E2) has also been assessed. Male Wistar rats underwent 60min middle cerebral artery occlusion (intraluminal thread technique), and grouped according to treatment: vehicle-, E2- and BZA-treated rats. Optimal plasma concentrations of E2 (45.6±7.8pg/ml) and BZA (20.7±2.1ng/ml) w…

Brain InfarctionMaleSelective Estrogen Receptor Modulatorsmedicine.medical_specialtyIndolesmedicine.drug_classIschemiaHemodynamicsPostmenopausal osteoporosisNeuroprotectionBazedoxifeneIschemic brainInternal medicinemedicineAnimalsRats WistarEstradiolbusiness.industryGeneral NeuroscienceHemodynamicsBrainmedicine.diseaseNeuroprotective AgentsEndocrinologyIschemic Attack TransientSelective estrogen receptor modulatorEstrogenbusinessmedicine.drugNeuroscience Letters
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Stable expression of rat cytochrome P450IA2 cDNA and hydroxylation of 17β-estradiol and 2-aminofluorene in V79 Chinese hamster cells

1991

In continuation of our work toward the establishment of a working cell bank for metabolic and toxicological studies, V79 Chinese hamster cells were genetically engineered for stable expression of rat cytochrome P450IA2. Full-length cDNA encoding rat P450IA2 was obtained by searching a cDNA library made from Aroclor 1254-induced rat liver mRNA and by joining a small 5'-end fragment to a fragment containing the rest of the cDNA. The sequence of the cDNA was confirmed by DNA sequencing and comparison to a previously published cDNA sequence. The reconstructed full-length cDNA was inserted into a simian virus 40 early promoter-containing eukaryotic expression vector and cotransferred with the ne…

Cancer ResearchGenetic VectorsMolecular Sequence DataGene ExpressionIn Vitro TechniquesHydroxylationTransfectionChinese hamsterCell LineHydroxylationchemistry.chemical_compoundCricetulusCytochrome P-450 Enzyme SystemCytochrome P-450 CYP1A2CricetinaeComplementary DNAAnimalsAmino Acid SequenceCloning MolecularMolecular BiologyGeneSouthern blotFluorenesMessenger RNABase SequenceEstradiolbiologycDNA libraryDNABlotting Northernbiology.organism_classificationMolecular biologyRecombinant ProteinsRatsBlotBlotting SouthernchemistryOxidoreductasesMolecular Carcinogenesis
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Gonadotropin-releasing hormone agonist for the prevention of chemotherapy-induced ovarian failure in patients with lymphoma: 1-year follow-up of a pr…

2013

PURPOSE To assess the efficacy of gonadotropin-releasing hormone agonist (GnRHa) in preventing chemotherapy-induced ovarian failure in patients treated for Hodgkin or non-Hodgkin lymphoma within the setting of a multicenter, randomized, prospective trial. PATIENTS AND METHODS Patients age 18 to 45 years were randomly assigned to receive either the GnRHa triptorelin plus norethisterone (GnRHa group) or norethisterone alone (control group) concomitantly with alkylating agents containing chemotherapy. The primary end point was the premature ovarian failure (POF) rate (follicle-stimulating hormone [FSH] ≥ 40 IU/L) after 1 year of follow-up. Results Eighty-four of 129 randomly assigned patients …

Cancer ResearchTime FactorsLymphomamedicine.medical_treatmentGonadotropin-Releasing Hormone -- agonistsPrimary Ovarian InsufficiencyTriptorelin Pamoate -- therapeutic uselaw.inventionGonadotropin-Releasing HormoneGynécologieRandomized controlled triallawAntineoplastic Combined Chemotherapy ProtocolsClinical endpointBiological Markers -- bloodNorethindrone -- therapeutic useProspective StudiesTreatment FailureProspective cohort studyTriptorelin PamoateEstradiolLymphoma Non-HodgkinLymphoma -- drug therapyMiddle AgedTriptorelinHodgkin DiseasePremature ovarian failureLuteolytic AgentsOncologyHodgkin Disease -- drug therapyDrug Therapy CombinationFemaleEstradiol -- bloodmedicine.drugAdultAntineoplastic Combined Chemotherapy Protocols -- administration & dosage -- adverse effectsmedicine.medical_specialtyNorethisteronemedicine.drug_classUrologyFollicle Stimulating Hormone -- bloodGonadotropin-releasing hormone agonistmedicineHumansGynecologyChemotherapybusiness.industrymedicine.diseaseLuteolytic Agents -- therapeutic useCancérologieLymphoma Non-Hodgkin -- drug therapyPremenopausePrimary Ovarian Insufficiency -- blood -- chemically induced -- prevention & controlFollicle Stimulating HormoneNorethindronebusinessBiomarkersFollow-Up Studies
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3H-estradiol and3H-R5020 binding in cytosols of normal and neoplastic human ovarian tissue

1983

High-affinity cytoplasmic estrogen and progesterone receptors in normal and abnormal ovarian tissues were studied. Estradiol receptor was detectable in 65% and progesterone receptor in 36% of the malignant tumors; 39% of all malignant ovarian tissues were estradiol- as well as progesterone-receptor-positive. Tumors were said to be receptor-positive when the receptors bound greater than 5 fM steroid/mg cytosol protein. No correlations were found between receptor status and histopathological diagnosis. In normal ovarian tissues collected at various phases of the menstrual cycle no changes in [3H]-estradiol and [3H]-R5020 binding could be detected. Analysis of the receptor concentration for bo…

Cancer Researchmedicine.medical_specialtyReceptor StatusNorpregnadienesmedicine.drug_classmedia_common.quotation_subjectOvaryReceptors EstradiolBiologyTritiumPromegestoneCytosolOvarian carcinomaInternal medicineProgesterone receptormedicineHumansEstrogen Receptor StatusMenstrual cyclemedia_commonOvarian NeoplasmsTranscortinEstradiolOvaryEndocrinologymedicine.anatomical_structureReceptors EstrogenOncologyEstrogenFemaleReceptors Progesteronehormones hormone substitutes and hormone antagonistsTamoxifenmedicine.drugInternational Journal of Cancer
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Estradiol decreases xanthine dehydrogenase enzyme activity and protein expression innon-tumorigenicand malignant human mammary epithelial cells

2009

The retinoic acid deficiency in breast tumour epithelial cells has been ascribed to an insufficient expression of either the enzyme(s) involved in its biosynthesis or the cellular retinol binding protein (CRBP) or both. In an attempt to define the mechanisms underpinning retinoic acid deficiency in these cell model systems, we have investigated the potential regulatory effect of oestrogen (17β-estradiol) on one key player in retinoic acid biosynthesis, the xanthine dehydrogenase (XDH). This enzyme is consistently expressed and very active in non-malignant human mammary epithelial cells (HMEC), as opposed to tumour MDA-MB231 and MCF7 cells. In these latter two cell lines, as opposed to HMEC …

CellRetinoic acidTretinoinBiologyBiochemistryGene Expression Regulation Enzymologicchemistry.chemical_compoundCell Line TumorSettore BIO/10 - BiochimicaRETINOIC ACIDmedicineHumansRNA MessengerMammary Glands Humanskin and connective tissue diseasesXanthine oxidaseXANTHINE OXIDASEESTRADIOLMolecular BiologyRetinolEpithelial CellsCell BiologyMolecular biologyEnzyme assayGene Expression Regulation NeoplasticRetinoic acid receptormedicine.anatomical_structurechemistryXanthine dehydrogenaseCell culturebiology.proteinXANTHINE DEHYDROGENASEJournal of Cellular Biochemistry
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Molecular mechanisms involved in the hormonal prevention of aging in the rat.

2008

Previous data from our group have provided support for the role of GH, melatonin and estrogens in the prevention of aging of several physiological parameters from bone, liver metabolism, vascular activity, the central nervous system (CNS), the immune system and the skin. In the present work data on the molecular mechanisms involved are presented. A total of 140 male and female rats have been submitted to different treatments over 10 weeks, between 22 and 24 months of age. Males have been treated with GH and melatonin. Females were divided in two groups: intact and castrated at 12 months of age. The first group was treated with GH and melatonin and the second with the two latter compounds an…

Central Nervous SystemMalemedicine.medical_specialtyAgingmedicine.drug_classEndocrinology Diabetes and MetabolismOvariectomyClinical BiochemistryMitochondria LiverBiologymedicine.disease_causeNitric OxideBiochemistryMelatoninchemistry.chemical_compoundEndocrinologyCytosolInternal medicineSkin Physiological PhenomenamedicineAnimalsRats WistarMolecular BiologyMelatoninchemistry.chemical_classificationEstradiolGlutathione peroxidaseDentate gyrusNeurogenesisCytochromes cEstrogensCell BiologyGlutathioneIsoflavonesRatsEndocrinologychemistryLiverProto-Oncogene Proteins c-bcl-2EstrogenApoptosisGrowth HormoneMolecular MedicineFemaleOxidative stressmedicine.drugThe Journal of steroid biochemistry and molecular biology
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Finding the right dose of fulvestrant in breast cancer

2012

Fulvestrant is a selective estrogen receptor downregulator, behaving as a complete antagonist. It was initially approved, at a dose of 250 mg, to treat hormone dependant breast cancer in second line setting. However, a series of pharmacological and pre-clinical studies have suggested that a higher dose of 500 mg may be more effective. The present work summarizes and discusses clinical trials that have aimed to test the benefits of administering fulvestrant at a higher dose. The data support the use of a higher, and more possibly, effective dose of the agent.

Clinical Trials as TopicAntineoplastic Agents HormonalDose-Response Relationship DrugEstradiolFulvestrantbusiness.industryAntagonistEstrogen receptorBreast NeoplasmsGeneral MedicinePharmacologymedicine.diseaseEffective dose (pharmacology)Clinical trialSecond lineBreast cancerOncologymedicineHumansFemaleRadiology Nuclear Medicine and imagingbusinessFulvestrantmedicine.drugHormoneCancer Treatment Reviews
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