Search results for "EXPRESSION"

showing 10 items of 5168 documents

Nicotine-induced fibroblast growth factor-2 restores the age-related decline of precursor cell proliferation in the subventricular zone of rat brain.

2007

Precursor cell proliferation is present in the subventricular zone (SVZ) of the lateral ventricles and the subgranular zone (SGZ) of the dentate gyrus of the hippocampus of adult rat and persists during aging although at reduced levels. Previous studies have shown that acute intermittent nicotine treatment significantly increases fibroblast growth factor-2 (FGF-2) expression in several brain regions of aged rats. The aim of the present investigation was to test the hypothesis that nicotine-induced expression of FGF-2 may restore the age-related decline of precursor cell proliferation. It was first demonstrated that nicotine treatment increases both mRNA and protein FGF-2 in the SVZ of aged …

Malemedicine.medical_specialtyAgingNicotineBasic fibroblast growth factorSubventricular zoneCell CountNerve Tissue ProteinsBiologyFibroblast growth factorSettore BIO/09 - FisiologiaAntibodiesSubgranular zoneNestinchemistry.chemical_compoundIntermediate Filament ProteinsInternal medicinePrecursor cellLateral VentriclesGlial Fibrillary Acidic ProteinmedicineAnimalsNicotinic AgonistsRats WistarMolecular BiologyCell ProliferationAnalysis of VarianceCell growthGeneral NeuroscienceDentate gyrusFibroblast growth factor receptor 1BrainPrecursor proliferationFGF-2 FGFR1 SVZ Nicotine AgedRatsAdult Stem CellsEndocrinologymedicine.anatomical_structurenervous systemchemistryBromodeoxyuridineGene Expression RegulationFibroblast Growth Factor 2Neurology (clinical)Developmental BiologyBrain research
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Behavioural parameters in aged rats are related to LTP and gene expression of ChAT and NMDA-NR2 subunits in the striatum.

2004

Striatal parameters were assessed for their relevance to age-related behavioural decline. Forty aged rats (28-30 months) were tested in the water maze and open field. Of these, seven superior and seven inferior learners were compared with each other in terms of levels of in vitro short- and long-term potentiation (STP and LTP), and gene expression of choline acetyltransferase (ChAT) as well as of the NMDA-NR2A-C subunits assessed by quantitative RT-PCR. Results revealed that the superior as compared with the inferior learners had higher levels of ChAT mRNA in the striatum. For the superior group, ChAT mRNA was correlated with escape on to the cued platform in the water maze, whereas level o…

Malemedicine.medical_specialtyAgingeducationLong-Term PotentiationStriatumWater mazeReceptors N-Methyl-D-AspartateOpen fieldCholine O-AcetyltransferaseInternal medicineGene expressionmedicineAnimalsRNA MessengerRats WistarMaze LearningGeneral NeuroscienceLong-term potentiationCholine acetyltransferaseCorpus StriatumRatsEndocrinologynervous systemGene Expression RegulationSynaptic plasticityExploratory BehaviorNMDA receptorPsychologyNeuroscienceThe European journal of neuroscience
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Effects of experimental type 1 diabetes and exercise training on angiogenic gene expression and capillarization in skeletal muscle.

2006

Diabetes alters microvascular structure and function and is a major risk factor for cardiovascular diseases. In diabetic skeletal muscle, impaired angiogenesis and reduced VEGF-A expression have been observed, whereas in healthy muscle exercise is known to have opposite effects. We studied the effects of type 1 diabetes and combined exercise training on angiogenic mRNA expression and capillarization in mouse skeletal muscle. Microarray and real-time PCR analyses showed that diabetes altered the expression of several genes involved in angiogenesis. For example, levels of proangiogenic VEGF-A, VEGF-B, neuropilin-1, VEGFR-1, and VEGFR-2 were reduced and the levels of antiangiogenic thrombospon…

Malemedicine.medical_specialtyAngiogenesisNeovascularization PhysiologicMice Inbred StrainsBiologyBiochemistryDiabetes Mellitus ExperimentalNeovascularizationMiceInternal medicineDiabetes mellitusPhysical Conditioning AnimalGene expressionGeneticsmedicineAnimalsMuscle SkeletalMolecular BiologyRegulation of gene expressionType 1 diabetesNeovascularization PathologicSkeletal muscleRibonuclease Pancreaticmedicine.diseaseCapillariesDisease Models Animalmedicine.anatomical_structureEndocrinologyDiabetes Mellitus Type 1Gene Expression RegulationAngiogenesis Inducing Agentsmedicine.symptomAngiogenesis Inducing AgentsBiotechnologyFASEB journal : official publication of the Federation of American Societies for Experimental Biology
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Cigarette smoking affects specific sperm oxidative defenses but does not cause oxidative DNA damage in infertile men

2008

Objective To evaluate the effects of tobacco consumption on the oxidative defenses of sperm, the glutathione system (GS), and sperm DNA oxidation. Design Double-blind experimental study. Setting Andrology laboratory in a university-affiliated private setting. Patient(s) One hundred seventeen semen samples from infertile males. Intervention(s) None. Main Outcome Measure(s) (a) sperm GS enzymatic activity with respect to glutathione peroxidase isoforms GPx-1 and GPx-4, glutathione reductase (GR), and cellular glutathione (GSH) content (n = 29); (b) GPx-1, GPx-4, and GR m RNA expression analysis (n = 33); (c) oxidative DNA damage quantification using OXIDNA assay kit (n = 55). Two groups were …

Malemedicine.medical_specialtyAntioxidantDNA damagemedicine.medical_treatmentGlutathione reductaseBiologymedicine.disease_causeGene Expression Regulation Enzymologicchemistry.chemical_compoundGlutathione Peroxidase GPX1Internal medicinemedicineHumansRNA MessengerPhospholipid-hydroperoxide glutathione peroxidaseInfertility Malechemistry.chemical_classificationGlutathione PeroxidaseGlutathione peroxidaseSmokingObstetrics and GynecologyGlutathionePhospholipid Hydroperoxide Glutathione PeroxidaseGlutathioneSpermatozoaSpermOxidative StressGlutathione ReductaseEndocrinologyReproductive MedicinechemistryOxidative stressDNA DamageFertility and Sterility
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Mitochondria from females exhibit higher antioxidant gene expression and lower oxidative damage than males

2003

We have investigated the differential mitochondrial oxidative stress between males and females to understand the molecular mechanisms enabling females to live longer than males. Mitochondria are a major source of free radicals in cells. Those from female rats generate half the amount of peroxides than those of males. This does not occur in ovariectomized animals. Estrogen replacement therapy prevents the effect of ovariectomy. Mitochondria from females have higher levels of reduced glutathione than those from males. Those from ovariectomized rats have similar levels to males, and estrogen therapy prevents the fall in glutathione levels that occurs in ovariectomized animals. Oxidative damage…

Malemedicine.medical_specialtyAntioxidantOvariectomymedicine.medical_treatmentMitochondria LiverMitochondrionBiologymedicine.disease_causeDNA MitochondrialBiochemistryAntioxidantsGene Expression Regulation EnzymologicSuperoxide dismutasechemistry.chemical_compoundRNA Ribosomal 16SPhysiology (medical)Internal medicinemedicineAnimalsRats WistarDNA Primerschemistry.chemical_classificationGlutathione PeroxidaseReverse Transcriptase Polymerase Chain ReactionSuperoxide DismutaseGlutathione peroxidaseEstrogensGlutathioneGlutathionePeroxidesRatsOxygenOxidative StressEndocrinologychemistryOvariectomized ratbiology.proteinRNAFemaleDismutaseReactive Oxygen SpeciesOxidation-ReductionOxidative stressFree Radical Biology and Medicine
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Decreased cell proliferation and higher oxidative stress in fibroblasts from Down Syndrome fetuses. Preliminary study

2013

Abstract Down Syndrome is the most common chromosomal disease and is also known for its decreased incidence of solid tumors and its progeroid phenotype. Cellular and systemic oxidative stress has been considered as one of the Down Syndrome phenotype causes. We correlated, in a preliminary study, the fibroblast proliferation rate and different cell proliferation key regulators, like Rcan1 and the telomere length from Down Syndrome fetuses, with their oxidative stress profile and the Ribonucleic acid and protein expression of the main antioxidant enzymes together with their activity. Increased oxidized glutathione/glutathione ratio and high peroxide production were found in our cell model. Th…

Malemedicine.medical_specialtyAntioxidantmedicine.medical_treatmentPrimary Cell CultureSuperoxide dismutasemedicine.disease_causeSuperoxide dismutasechemistry.chemical_compoundFetusSuperoxide Dismutase-1ThioredoxinsInternal medicineGlutaredoxinmedicineHumansThioredoxinMolecular BiologyGlutaredoxinsCell ProliferationSkinchemistry.chemical_classificationReactive oxygen speciesGlutathione PeroxidaseTelomere lengthbiologyGlutathione peroxidaseTelomere HomeostasisGlutathioneRcan1FibroblastsTelomereCatalaseGlutathioneProgeroidOxidative StressEndocrinologychemistryBiochemistryGene Expression Regulationbiology.proteinMolecular MedicineFemaleThioredoxinDown SyndromeOxidative stressSignal TransductionBiochimica et Biophysica Acta (BBA) - Molecular Basis of Disease
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Mesenchymal Stem Cells Improve Motor Functions and Decrease Neurodegeneration in Ataxic Mice

2014

The main objective of this work is to demonstrate the feasibility of using bone marrow-derived stem cells in treating a neurodegenerative disorder such as Friedreich's ataxia. In this disease, the dorsal root ganglia of the spinal cord are the first to degenerate. Two groups of mice were injected intrathecally with mesenchymal stem cells isolated from either wild-type or Fxntm1Mkn/Tg(FXN)YG8Pook (YG8) mice. As a result, both groups presented improved motor skills compared to nontreated mice. Also, frataxin expression was increased in the dorsal root ganglia of the treated groups, along with lower expression of the apoptotic markers analyzed. Furthermore, the injected stem cells expressed th…

Malemedicine.medical_specialtyAtaxiaCellular differentiationGene ExpressionBone Marrow CellsMice TransgenicMotor ActivityMesenchymal Stem Cell TransplantationTransplantation AutologousMiceGlutathione Peroxidase GPX1Neurotrophin 3Internal medicineGanglia SpinalIron-Binding ProteinsDrug DiscoverymedicineGeneticsAnimalsTransplantation HomologousNerve Growth FactorsMolecular BiologyInjections SpinalPharmacologyGlutathione PeroxidasebiologyBrain-Derived Neurotrophic FactorMesenchymal stem cellCell DifferentiationMesenchymal Stem CellsAnatomySpinal cordCatalaseDisease Models AnimalEndocrinologymedicine.anatomical_structureFriedreich AtaxiaFrataxinbiology.proteinMolecular MedicineOriginal ArticleFemaleBone marrowmedicine.symptomStem cellAdult stem cell
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Antagonistic effects of hypertrehalosemic neuropeptide on the activities of 6-phosphofructo-1-kinase and fructose-1,6-bisphosphatase in cockroach fat…

2001

Hypertrehalosemic neuropeptides from the corpora cardiaca such as the decapeptide Bld HrTH bring about a profound switch in the metabolic activity of cockroach fat body during which production of the blood sugar trehalose is stimulated while the catabolism of carbohydrate (glycolysis) is inhibited. The mechanisms of the metabolic switch are not fully understood. Incubation of isolated fat body from the cockroach Blaptica dubia with 10(-8) M Bld HrTH, for 10-60 min, stimulated glycogen breakdown and increased the content of the substrates of both the glycolytic enzyme 6-phosphofructo-1-kinase (PFK, EC 2.7.1.11) and the gluconeogenic enzyme fructose-1,6-bisphosphatase (FBPase, EC 3.1.3.11) in…

Malemedicine.medical_specialtyBlaptica dubiaPhosphofructokinase-1Fat BodyFructose 16-bisphosphataseCockroachesIn Vitro TechniquesBiologyBiochemistryGene Expression Regulation Enzymologicchemistry.chemical_compoundInternal medicineFructosediphosphatesmedicineAnimalsGlycolysisPhosphofructokinase 1Molecular BiologyCatabolismNeuropeptidesTrehaloseFructosebiology.organism_classificationAdenosine MonophosphateFructose-BisphosphataseKineticsEndocrinologyFructose 26-bisphosphatechemistryBiochemistryInsect HormonesInsect Sciencebiology.proteinGlycogenPhosphofructokinaseInsect Biochemistry and Molecular Biology
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Upregulation of Phospholipase D Expression and Activation in Ventricular Pressure-Overload Hypertrophy

2005

Evidence for a role of phospholipase D (PLD) in cellular proliferation and differentiation is accumulating. We studied PLD activity and expression in normal and hypertrophic rat and human hearts. In rat heart, abdominal aortic banding (constriction to 50% of original lumen) caused hypertrophy in the left ventricle (as shown by weight index and ANP expression) by about 15% after 30 days without histological evidence of fibrosis or signs of decompensation and in the right ventricle after 100 days. The hypertrophy was accompanied by small increases of basal PLD activity and strong potentiation of stimulated PLD activity caused by 4β-phorbol-12β,13α-dibutyrate (PDB) and by phenylephrine. The mR…

Malemedicine.medical_specialtyBlotting WesternCardiomegalyBiologyGene Expression Regulation EnzymologicMuscle hypertrophyRats Sprague-DawleyDownregulation and upregulationInternal medicinePhospholipase DVentricular PressuremedicineAnimalsHumansRNA MessengerPhenylephrineProtein Kinase CProtein kinase CPharmacologyReverse Transcriptase Polymerase Chain ReactionPhospholipase DPLD2lcsh:RM1-950Body WeightRatsReceptors AdrenergicUp-RegulationEnzyme ActivationIsoenzymeslcsh:Therapeutics. Pharmacologymedicine.anatomical_structureEndocrinologyVentricleVentricular pressureMolecular Medicinelipids (amino acids peptides and proteins)Signal Transductionmedicine.drugJournal of Pharmacological Sciences
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The selective cyclooxygenase-1 inhibitor SC-560 suppresses cell proliferation and induces apoptosis in human hepatocellular carcinoma cells

2006

Two isoforms of cyclooxygenase (COX) are known, and to date most studies have implicated COX-2 in the development and progression of various human cancers. Increasing evidence suggests that COX-1 may also play a similar role. Indeed, we have recently observed that the dual COX-1/COX-2 inhibitor indomethacin induces apoptosis in human hepatocellular carcinoma (HCC) cell lines more effectively than the selective COX-2 inhibitors, possibly implicating COX-1 in HCC. In this study we investigated the expression of COX-1 in non-tumor and malignant human liver tissues, as well as the effects of the highly selective COX-1 inhibitor SC-560 on cell growth and apoptosis in human HCC cell lines. Expres…

Malemedicine.medical_specialtyCarcinoma HepatocellularCellApoptosisBiologyGene Expression Regulation EnzymologicCell Line TumorInternal medicineSurvivinGeneticsmedicineHumansCyclooxygenase InhibitorCyclooxygenase InhibitorsRNA MessengerAgedCell ProliferationOncogeneCell growthApoptosiGeneral MedicineMiddle AgedCell cycleImmunohistochemistryXIAPGene Expression Regulation NeoplasticEndocrinologymedicine.anatomical_structureCyclooxygenase 2ApoptosisCell culturePyrazoleCyclooxygenase 1Cancer researchPyrazolesFemaleHumanInternational Journal of Molecular Medicine
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