Search results for "Ectoderm"

showing 10 items of 100 documents

Highly restricted expression at the ectoderm–endoderm boundary of PIHbox 9, a sea urchin homeobox gene related to the human HB9 gene

1998

Abstract Characterisation of a sea urchin (P. lividus) homeobox gene PIHbox 9 is reported. The homeodomain of PIHbox9 is 95% identical to the homeodomain of the human HB9 gene, indicating that the two genes are highly related. Temporal expression analysis during sea urchin embryogenesis showed an absence of transcripts at early cleavage stages. At late gastrula stage, transcripts were barely detectable and reached the highest abundance at prism/early pluteus stages. By whole mount in situ hybridisation we observed a highly restricted expression in a few cells of the ectoderm–endoderm boundary of embryos at the prism stage. At pluteus stages, expression of PIHbox 9 was confined around the an…

EmbryologyEmbryo Nonmammaliananimal structuresEctodermParacentrotus lividusbiology.animalEctodermmedicineAnimalsPluteusSea urchinIn Situ HybridizationHomeodomain ProteinsGeneticsbiologyEndodermEmbryogenesisGene Expression Regulation DevelopmentalGastrulabiology.organism_classificationCell biologyGastrulationmedicine.anatomical_structureSea Urchinsembryonic structuresHomeoboxEndodermTranscription FactorsDevelopmental BiologyMechanisms of Development
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Up-to-date monitoring of childhood cancer long-term survival in Europe: central nervous system tumours.

2007

ABSTRACT Background Tumours of the central nervous system (CNS) account for 15–20% of all malignant childhood tumours in developed countries. Steady improvement of survival of children with CNS tumours has been reported for the past decades. However, these results, obtained by cohort analysis of survival, do not reflect the full extent of recent improvement. Methods Using selected registries from the database of the Automated Childhood Cancer Information System (ACCIS), we calculated period survival estimates for the years 1995–99 for children diagnosed with a malignant CNS tumour. Results The overall 10-year period survival estimate for the years 1995–99 was 59% for children with all CNS t…

EpendymomaPediatricsmedicine.medical_specialtyAdolescentCentral nervous systemAstrocytomaCentral Nervous System NeoplasmsmedicineHumansNeuroectodermal Tumors PrimitiveChildbusiness.industryAge FactorsInfant NewbornAstrocytomaCancerInfantHematologymedicine.diseasePrognosisEuropemedicine.anatomical_structureOncologyEl NiñoEpendymomaChild PreschoolCohortbusinessDeveloped countryCohort studyAnnals of oncology : official journal of the European Society for Medical Oncology
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The impact of next-generation sequencing technology on preimplantation genetic diagnosis and screening.

2013

Largely because of efforts required to complete the Human Genome Project, DNA sequencing has undergone a steady transformation with still-ongoing developments of high-throughput sequencing machines for which the cost per reaction is falling drastically. Similarly, the fast-changing landscape of reproductive technologies has been improved by genetic approaches. Preimplantation genetic diagnosis and screening were established more than two decades ago for selecting genetically normal embryos to avoid inherited diseases and to give the highest potential to achieve stable pregnancies. Most recent additions to the IVF practices (blastocyst/trophectoderm biopsy, embryo vitrification) and adoption…

GeneticsDNA Mutational AnalysisObstetrics and GynecologyComparative Genome HybridizationComputational biologyReproductive technologyFertilization in VitroBiologyPreimplantation genetic diagnosisDNA sequencingReproductive MedicineChromosome (genetic algorithm)PregnancyNew geneticsHuman Genome ProjectHumansHuman genomeFemalePreimplantation DiagnosisTrophectoderm biopsyFertility and sterility
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Yunis-Varón Syndrome Is Caused by Mutations in FIG4, Encoding a Phosphoinositide Phosphatase

2013

Yunis-Varón syndrome (YVS) is an autosomal-recessive disorder with cleidocranial dysplasia, digital anomalies, and severe neurological involvement. Enlarged vacuoles are found in neurons, muscle, and cartilage. By whole-exome sequencing, we identified frameshift and missense mutations of FIG4 in affected individuals from three unrelated families. FIG4 encodes a phosphoinositide phosphatase required for regulation of PI(3,5)P(2) levels, and thus endosomal trafficking and autophagy. In a functional assay, both missense substitutions failed to correct the vacuolar phenotype of Fig4-null mouse fibroblasts. Homozygous Fig4-null mice exhibit features of YVS, including neurodegeneration and enlarg…

GenotypePhosphataseMicrognathismMolecular Sequence DataLimb Deformities CongenitalMutation MissenseBiologyCompound heterozygositymedicine.disease_causeFrameshift mutation03 medical and health sciencesMice0302 clinical medicinePhosphatidylinositol PhosphatesEctodermal DysplasiaReportmedicineGeneticsMissense mutationAnimalsHumansExomeGenetic Predisposition to DiseaseGenetics(clinical)Yunis–Varon syndromeFrameshift MutationGenetics (clinical)030304 developmental biology0303 health sciencesMutationBone DevelopmentBase SequenceFlavoproteinsNeurodegenerationSequence Analysis DNAFibroblastsmedicine.diseaseMolecular biologyPhenotypePhosphoric Monoester HydrolasesCleidocranial Dysplasia030217 neurology & neurosurgeryThe American Journal of Human Genetics
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The ontogeny of the filter apparatus of anuran larvae (Amphibia, Anura)

1991

The pharynx ofBufo calamita, Rana temporaria andBombina variegata larvae (larval Types IV and III) changes considerably during the latter part of embryonic development. The entodermal regions between the visceral pockets flatten inward to form the anlagen of the filter plates. The ectoderm thrusts forward from the area of the persistent epidermal gills overlying the anlagen of the filter plates. The esophagus pushes dorsolaterally into the pharynx to give rise to the ciliary cushions. Comparison with the development ofXenopus laevis (larval Type I) reveals shared characters: (1) the filter plates are overlapped by the sensory layer of the epiderm and (2) the ciliary grooves are, like the ci…

Gillfood.ingredientbiologyOntogenyPipidaeEctodermAnatomybiology.organism_classificationMucusfoodmedicine.anatomical_structureRanoideaSalientiaYolkembryonic structuresmedicineAnimal Science and ZoologyDevelopmental BiologyZoomorphology
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Exome sequencing in a child with neurodevelopmental disorder and epilepsy: Variant analysis of the AHNAK2 gene

2022

Background The AHNAK2 gene encodes a large nucleoprotein expressed in several tissues, including brain, squamous epithelia, smooth muscle, and neuropil. Its role in calcium signaling has been suggested and to date, clear evidence about its involvement in the pathogenesis of clinical disorders is still lacking. Methods Here, we report a female 24-year-old patient diagnosed with a cardio-facio-cutaneous-like phenotype (CFC-like), characterized by epilepsy, psychomotor development delay, atopic dermatitis, congenital heart disease, hypotonia, and facial dysmorphism, who is compound heterozygote for two missense mutations in the AHNAK2 gene detected by exome sequencing. Results This patient had…

Heart Defects CongenitalAHNAK2 borderline intellectual functioning epilepsy facio-cardio-cutaneous-like phenotype NGS exomefacio-cardio-cutaneous-like phenotypeFaciesNGS exomeSettore MED/39 - Neuropsichiatria InfantileFailure to ThriveNucleoproteinsEctodermal DysplasiaNeurodevelopmental DisordersAHNAK2borderline intellectual functioningGeneticsHumansepilepsyExomeFemaleMolecular BiologyGenetics (clinical)Molecular Genetics & Genomic Medicine
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Cardio-facio-cutaneous syndrome and gastrointestinal defects: report on a newborn with 19p13.3 deletion including the MAP 2 K2 gene

2022

Abstract Background Cardio-facio-cutaneous syndrome (CFCS) belongs to RASopathies, a group of conditions caused by mutations in genes encoding proteins of the rat sarcoma/mitogen-activated protein kinase (RAS/MAPK) pathway. It is a rare syndrome, with about 300 patients reported. Main clinical manifestations include facial dysmorphisms, growth failure, heart defects, developmental delay, and ectodermal abnormalities. Mutations (mainly missense) of four genes (BRAF, MAP 2 K1, MAP 2 K2, and KRAS) have been associated to CFCS. However, whole gene deletions/duplications and chromosomal microdeletions have been also reported. Specifically, 19p13.3 deletion including MAP 2 K2 gene are responsible…

Heart Defects CongenitalComparative Genomic HybridizationEctodermal DysplasiaPregnancyCFCS RASopathies Contiguous gene syndrome Array-CGH Genotype-phenotype correlations HPS Case reportFaciesHumansFemaleSyndromeHernia UmbilicalFailure to ThriveItalian Journal of Pediatrics
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Immunohistochemical detection of EWS and FLI-1 proteinss in Ewing sarcoma and primitive neuroectodermal tumors: comparative analysis with CD99 (MIC-2…

2001

The molecular analysis of the t(11;22) rearrangement involving EWS/FLI-1 genes is likely to be of diagnostic value in Ewing sarcoma (ES) and primitive neuroectodermal tumors (PNET). The objective of the current study was to analyze the immunohistochemical expression of the EWS and FLI-1 proteins in a group of small round-cell tumors (SRCT) to determine their specificity and relevance in their differential diagnosis. Forty-eight cases-10 conventional ES, 4 large-cell ES, 5 PNET, 9 neuroblastomas (NB), 6 undifferentiated synovial sarcomas (SS), 5 rhabdomyosarcomas (RB), 5 non-Hodgkin lymphomas (NHL), 1 round-cell liposarcoma, and 3 mesenchymal chondrosarcomas-were analyzed. Immunocytochemistr…

HistologyImmunocytochemistryCD99Sarcoma EwingLiposarcomaBiology12E7 AntigenSensitivity and SpecificityHeterogeneous-Nuclear RibonucleoproteinsPathology and Forensic Medicinechemistry.chemical_compoundAntigenAntigens CDProto-Oncogene ProteinsmedicineHumansNeuroectodermal Tumors PrimitiveProto-Oncogene Protein c-fli-1medicine.diseaseImmunohistochemistryDNA-Binding ProteinsMedical Laboratory TechnologyAntigen retrievalchemistryRibonucleoproteinsCancer researchTrans-ActivatorsImmunohistochemistrySarcomaDifferential diagnosisRNA-Binding Protein EWSCell Adhesion MoleculesApplied immunohistochemistrymolecular morphology : AIMM
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Recognizable neonatal clinical features of aplasia cutis congenita

2020

Abstract Background Aplasia cutis congenita (ACC), classified in nine groups, is likely to be underreported, since milder isolated lesions in wellbeing newborns could often be undetected, and solitary lesions in the context of polymalformative syndromes could not always be reported. Regardless of form and cause, therapeutic options have in common the aim to restore the deficient mechanical and immunological cutaneous protection and to limit the risk of fluid leakage or rupture of the exposed organs. We aimed to review our institutional prevalence, comorbidities, treatment and outcome of newborns with ACC. Methods We conducted a retrospective study including all newborns affected by ACC and …

Male0301 basic medicinePediatricsmedicine.medical_specialtyMeningomyeloceleAbdominal compartment syndromeContext (language use)030105 genetics & heredityAplasia cutis congenita03 medical and health sciencesEctodermal DysplasiaPrevalenceHumansMedicineBody Weights and MeasuresRetrospective StudiesPregnancybusiness.industryResearchAbdominal wall defectInfant Newbornlcsh:RJ1-570Gestational agelcsh:PediatricsRetrospective cohort studyPrognosismedicine.diseaseSurvival RateAbdominal wall defectRetrospective study030104 developmental biologyItalyScalp defectFemaleApgar scoreJunctional epidermolysis bullosamedicine.symptombusiness
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First report of X-linked hypohidrotic ectodermal dysplasia with a hemizygous c.1142G > C in the EDA gene: variant of uncertain significance or new pa…

2021

AbstractBackgroundHypohidrotic Ectodermal Dysplasia (HED) is a genetic disorder which affects structures of ectodermal origin. X-linked hypohidrotic ectodermal dysplasia (XLHED) is the most common form of disease. XLHED is characterized by hypotrichosis, hypohydrosis and hypodontia. The cardinal features of classic HED become obvious during childhood.Identification of a hemizygous EDA pathogenic variant in an affected male confirms the diagnosis.Case presentationWe report on a male newborn with the main clinical characteristics of the X-linked HED including hypotrichosis, hypodontia and hypohidrosis. Gene panel sequencing identified a new hemizygous missense variant of uncertain significanc…

Male0301 basic medicineProbandMutation MissenseVariants of uncertain significance (VUS)Case ReportX-linked.030105 genetics & heredityPediatricsRJ1-57003 medical and health sciencesEDA geneHumansMedicineMissense mutationHypohidrotic ectodermal dysplasiaX chromosomeHemizygoteGeneticsX-linkedChromosomes Human XEctodermal Dysplasia 1 Anhidroticbusiness.industryInfant NewbornGenetic disorderGeneral MedicineEctodysplasinsmedicine.diseaseHypoidrotic ectodermal dysplasiaHypodontia030104 developmental biologyHypotrichosisEctodysplasin AbusinessItalian Journal of Pediatrics
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