Search results for "Ectopic expression"

showing 10 items of 41 documents

Involvement of the transcription factor FoxM1 in contact inhibition

2012

Contact inhibition is a crucial mechanism regulating proliferation in vitro and in vivo. Although it is generally accepted that contact inhibition plays a pivotal role in maintaining tissue homeostasis, the molecular mechanisms of contact inhibition are still not fully understood. FoxM1 is known as a proliferation-associated transcription factor and is upregulated in many cancer types. Vice versa, anti-proliferative signals, such as TGF-β and differentiation signals decrease FoxM1 expression. Here we investigated the role of FoxM1 in contact inhibition in fibroblasts. We show that protein expression of FoxM1 is severely and rapidly downregulated upon contact inhibition, probably by inhibiti…

MAPK/ERK pathwayCyclin ABiophysicsDown-RegulationCell Cycle ProteinsCyclin AProtein Serine-Threonine KinasesBiochemistryMiceDownregulation and upregulationProto-Oncogene ProteinsAnimalsPhosphorylationRNA Small InterferingExtracellular Signal-Regulated MAP KinasesMolecular BiologyTranscription factorTissue homeostasisbiologyContact InhibitionKinaseForkhead Box Protein M1Contact inhibitionForkhead Transcription FactorsCell BiologyG1 Phase Cell Cycle CheckpointsCell biologyNIH 3T3 Cellsbiology.proteinEctopic expressionBiochemical and Biophysical Research Communications
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Selective targeting of IRF4 by synthetic microRNA-125b-5p mimics induces anti-multiple myeloma activity in vitro and in vivo

2015

Interferon regulatory factor 4 (IRF4) is an attractive therapeutic target in multiple myeloma (MM). We here report that expression of IRF4 mRNA inversely correlates with microRNA (miR)-125b in MM patients. Moreover, we provide evidence that miR-125b is downregulated in TC2/3 molecular MM subgroups and in established cell lines. Importantly, constitutive expression of miR-125b-5p by lentiviral vectors or transfection with synthetic mimics impaired growth and survival of MM cells and overcame the protective role of bone marrow stromal cells in vitro. Apoptotic and autophagy-associated cell death were triggered in MM cells on miR-125b-5p ectopic expression. Importantly, we found that the anti-…

MaleCancer ResearchStromal cellApoptosisBiologyMiceRNA interferenceDownregulation and upregulationIn vivoIRF4Cell Line TumormicroRNAAutophagymedicineAnimalsHumansGenes Tumor SuppressorCell ProliferationmicroRNACell growthHematologyTransfectionMolecular biologymultiple myelomaMicroRNAsmedicine.anatomical_structureOncologyInterferon Regulatory FactorsCancer researchOriginal ArticleEctopic expressionBone marrow
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The LIM Homeodomain Factor Lhx2 Is Required for Hypothalamic Tanycyte Specification and Differentiation

2014

Hypothalamic tanycytes, a radial glial-like ependymal cell population that expresses numerous genes selectively enriched in embryonic hypothalamic progenitors and adult neural stem cells, have recently been observed to serve as a source of adult-born neurons in the mammalian brain. The genetic mechanisms that regulate the specification and maintenance of tanycyte identity are unknown, but are critical for understanding how these cells can act as adult neural progenitor cells. We observe that LIM (Lin-11, Isl-1, Mec-3)-homeodomain geneLhx2is selectively expressed in hypothalamic progenitor cells and tanycytes. To test the function ofLhx2in tanycyte development, we used an intersectional gene…

MaleCell typeEpendymal CellCellular differentiationNeurogenesisEpendymoglial CellsLIM-Homeodomain Proteinsradial gliaHypothalamusMice TransgenicBiologytanycytesMicemedicineAnimalshypothalamustranscription factorGeneticsTanycyteGeneral NeuroscienceNeurogenesisependymal cellsCell DifferentiationArticlesNeural stem cellCell biologyNeuroepithelial cellmedicine.anatomical_structureembryonic structuresEctopic expressionFemalemetabolismTranscription Factors
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Control of apterous by vestigial drives indirect flight muscle development in drosophila

2003

0012-1606 (Print) Journal Article Research Support, Non-U.S. Gov't; Drosophila thoracic muscles are comprised of both direct flight muscles (DFMs) and indirect flight muscles (IFMs). The IFMs can be further subdivided into dorsolongitudinal muscles (DLMs) and dorsoventral muscles (DVMs). The correct patterning of each category of muscles requires the coordination of specific executive regulatory programs. DFM development requires key regulatory genes such as cut (ct) and apterous (ap), whereas IFM development requires vestigial (vg). Using a new vg(null) mutant, we report that a total absence of vg leads to DLM degeneration through an apoptotic process and to a total absence of DVMs in the …

MaleNerve Tissue Proteins/genetics/metabolismMuscle Fibers SkeletalMutantTranscription Factors/genetics/*metabolismmedicine.disease_causeMyoblastsTwist transcription factorMyoblasts/physiologyDrosophila ProteinsWings AnimalDevelopmentalCells CulturedRegulator geneRegulation of gene expressionWing/growth & development/physiologyMutationCulturedMusclesGene Expression Regulation DevelopmentalNuclear ProteinsDrosophila Proteins/genetics/*metabolismAnatomyMuscle degenerationCell biologytwistDrosophilacutMuscles/metabolism/pathology/*physiologyIndirect flight musclesCellsLIM-Homeodomain ProteinsMuscle Fibers/pathology/physiologyNerve Tissue ProteinsBiologyvestigialNuclear Proteins/genetics/*metabolismmedicineHomeodomain Proteins/genetics/*metabolismAnimalsDrosophila/*growth & developmentDrosophilaMolecular BiologyHomeodomain ProteinsTwist-Related Protein 1Cell Biologybiology.organism_classificationapterousTwist Transcription FactorGene Expression RegulationMutationEctopic expressionTranscription FactorsDevelopmental BiologyDevelopmental Biology
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PINK1 displays tissue-specific subcellular location and regulates apoptosis and cell growth in breast cancer cells.

2010

The PINK1 gene is mutated in the germ line of patients with hereditary early-onset Parkinson disease, and PINK1 prosurvival function at neuronal mitochondria has been related with the etiology of this disease. However, the expression and function of PINK1 protein in nonneuronal tissues has not been determined yet. Here, we have analyzed PINK1 protein expression and subcellular distribution in normal and neoplastic human tissues and investigated the function of PINK1 in breast carcinoma cells. PINK1 protein, as stained by a specific anti-PINK1 monoclonal antibody, was widely expressed in human tissues, displaying high expression in epithelial tissues and in the central nervous system and low…

MaleProgrammed cell deathLung NeoplasmsApoptosisBreast NeoplasmsBiologymedicine.disease_causePathology and Forensic MedicineMiceCell Line TumormedicineAnimalsHumansTissue DistributionCell ProliferationCell growthCancermedicine.diseaseSquamous carcinomaCancer researchCarcinoma Squamous CellEctopic expressionFemaleBreast diseaseCarcinogenesisBreast carcinomaProtein KinasesHuman pathology
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Mandibular-pelvic-patellar syndrome (mpp) is a novel pitx1-related disorder due to alteration of pitx1 transactivation ability

2020

International audience; PITX1 is a homeobox transcription factor essential for hindlimb morphogenesis. Two PITX1-related human disorders have been reported to date: PITX1 ectopic expression causes Liebenberg syndrome, characterized by malformation of upper limbs showing a "lower limb" appearance; PITX1 deletions or missense variation cause a syndromic picture including clubfoot, tibial hemimelia, and preaxial polydactyly. We report two novel PITX1 missense variants, altering PITX1 transactivation ability, in three individuals from two unrelated families showing a distinct recognizable autosomal dominant syndrome, including first branchial arch, pelvic, patellar, and male genital abnormaliti…

MaleTranscriptional ActivationPathologymedicine.medical_specialtyHindlimb morphogenesis[SDV]Life Sciences [q-bio]Mutation MissensepelvisBiologyPierre-Robin03 medical and health sciencesTransactivationGeneticsmedicineMissense mutationAnimalsHumansPaired Box Transcription FactorsChildPITX1Genetics (clinical)030304 developmental biologyMice Knockoutcleft palate0303 health sciencesBone Diseases Developmental030305 genetics & heredityPreaxial polydactylyInfant NewbornLiebenberg syndromemedicine.disease3. Good healthgenitalpatella[SDV.GEN.GH]Life Sciences [q-bio]/Genetics/Human geneticsChild PreschoolHomeoboxEctopic expressionHaploinsufficiency
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G protein-coupled odorant receptors underlie mechanosensitivity in mammalian olfactory sensory neurons

2014

Mechanosensitive cells are essential for organisms to sense the external and internal environments, and a variety of molecules have been implicated as mechanical sensors. Here we report that odorant receptors (ORs), a large family of G protein-coupled receptors, underlie the responses to both chemical and mechanical stimuli in mouse olfactory sensory neurons (OSNs). Genetic ablation of key signaling proteins in odor transduction or disruption of OR–G protein coupling eliminates mechanical responses. Curiously, OSNs expressing different OR types display significantly different responses to mechanical stimuli. Genetic swap of putatively mechanosensitive ORs abolishes or reduces mechanical res…

Mice 129 StrainPatch-Clamp TechniquesG protein[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionSensory systemMice Transgenicodorant receptorsBiologyReceptors OdorantMechanotransduction CellularOlfactory Receptor NeuronsMiceg protein-coupled receptorsAnimalsHumansCalcium SignalingMechanotransductionReceptorG protein-coupled receptormechanotransductionMice KnockoutMultidisciplinaryheterologous expressionBiological SciencesRecombinant ProteinsMice Inbred C57BLHEK293 CellsMice Inbred DBA[ SDV.NEU ] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Mutagenesis Site-DirectedEctopic expressionMechanosensitive channels[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]NeuroscienceTransduction (physiology)Mechanoreceptors[SDV.AEN]Life Sciences [q-bio]/Food and Nutritionmechanical sensorsSignal Transduction
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Combined Targeting of the Menin-MLL1 Chromatin Complex and FLT3 As a Novel Therapeutic Concept Against NPM1 Mutant or MLL-Rearranged AML with Mutated…

2019

NPM1mutant (NPM1mut) and MLL1-rearranged (MLL-r) acute myeloid leukemias (AMLs) exhibit aberrant expression of HOX and MEIS1 transcription factors and commonly harbor an activating mutation in the receptor tyrosine kinase FLT3. Pharmacologic inhibition of the menin-MLL1 complex reverses leukemogenic gene expression including MEIS1 and FLT3 and represents a therapeutic opportunity for the treatment of these leukemias. Here, we investigate the contribution of the menin-MLL1 complex to leukemic FLT3 signaling and assess the therapeutic potential of dual menin-MLL1 and FLT3 targeting. First, we performed RNA sequencing to delineate transcriptional changes associated with menin-MLL1 inhibition (…

NPM1ImmunologyPonatinibCell BiologyHematologyBiologymedicine.diseaseBiochemistrychemistry.chemical_compoundLeukemiachemistryhemic and lymphatic diseasesGene expressionCancer researchmedicineEctopic expressionGrowth inhibitionCrenolanibQuizartinibBlood
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Secretory Pathway Research: The More Experimental Systems the Better

2012

Transient gene expression, in plant protoplasts or specific plant tissues, is a key technique in plant molecular cell biology, aimed at exploring gene products and their modifications to examine functional subdomains, their interactions with other biomolecules, and their subcellular localization. Here, we highlight some of the major advantages and potential pitfalls of the most commonly used transient gene expression models and illustrate how ectopic expression and the use of dominant mutants can provide insights into protein function.

Protein functionMolecular cell biologySecretory PathwayProtoplastsResearchfungiMutantfood and beveragesBiological TransportCell BiologyPlant ScienceBiologySubcellular localizationCell biologyPlant LeavesPerspectiveGene expressionEctopic expressionGeneSecretory pathwayFluorescent DyesThe Plant Cell
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APOBEC4 Enhances the Replication of HIV-1

2016

APOBEC4 (A4) is a member of the AID/APOBEC family of cytidine deaminases. In this study we found a high mRNA expression of A4 in human testis. In contrast, there were only low levels of A4 mRNA detectable in 293T, HeLa, Jurkat or A3.01 cells. Ectopic expression of A4 in HeLa cells resulted in mostly cytoplasmic localization of the protein. To test whether A4 has antiviral activity similar to that of proteins of the APOBEC3 (A3) subfamily, A4 was co-expressed in 293T cells with wild type HIV-1 and HIV-1 luciferase reporter viruses. We found that A4 did not inhibit the replication of HIV-1 but instead enhanced the production of HIV-1 in a dose-dependent manner and seemed to act on the viral L…

RNA virusesMale0301 basic medicineMolecular biologylcsh:MedicineArtificial Gene Amplification and ExtensionCytidinePathology and Laboratory MedicineVirus ReplicationBiochemistryPolymerase Chain ReactionJurkat cellschemistry.chemical_compoundCytidine deaminationImmunodeficiency VirusesTranscription (biology)TestisMedicine and Health Scienceslcsh:SciencePromoter Regions GeneticMultidisciplinaryCytidineTransfectionEnzymesImmunoblot AnalysisMedical MicrobiologyDeaminationViral PathogensViruses293T cellsCell linesPathogensOxidoreductasesBiological culturesLuciferaseResearch ArticleMolecular Probe TechniquesDNA constructionBiologyMicrobiologyCell Line03 medical and health sciencesCytidine DeaminaseRetrovirusesHumansMicrobial PathogensHIV Long Terminal Repeat030102 biochemistry & molecular biologylcsh:RLentivirusHEK 293 cellsOrganismsBiology and Life SciencesHIVProteinsPromoterMolecular biologyResearch and analysis methodsMolecular biology techniques030104 developmental biologychemistryPlasmid ConstructionHIV-1Enzymologylcsh:QEctopic expressionCloningPLOS ONE
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