Search results for "Enalaprilat"

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Biowaiver Monographs for Immediate-Release Solid Oral Dosage Forms: Enalapril

2018

Literature data relevant to the decision to allow a waiver of in vivo bioequivalence testing for the marketing authorization of immediate-release, solid oral dosage forms containing enalapril maleate are reviewed. Enalapril, a prodrug, is hydrolyzed by carboxylesterases to the active angiotensin-converting enzyme inhibitor enalaprilat. Enalapril as the maleate salt is shown to be highly soluble, but only 60%-70% of an orally administered dose of enalapril is absorbed from the gastrointestinal tract into the enterocytes. Consequently, enalapril maleate is a Biopharmaceutics Classification System class III substance. Because in situ conversion of the maleate salt to the sodium salt is sometim…

DrugEnalaprilatmedia_common.quotation_subjectAdministration OralPharmaceutical ScienceAngiotensin-Converting Enzyme InhibitorsBioequivalencePharmacology030226 pharmacology & pharmacyPermeabilityDosage form03 medical and health sciences0302 clinical medicineDrug StabilityEnalaprilmedicineHumansProdrugsEnalaprilmedia_commonChromatographyChemistryProdrugBiopharmaceutics Classification SystemIntestinal AbsorptionSolubilityTherapeutic EquivalencyEnalapril Maleate030220 oncology & carcinogenesisTabletsmedicine.drugJournal of Pharmaceutical Sciences
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ACE inhibitor potentiation of bradykinin-induced venoconstriction

1997

1. Angiotensin-converting enzyme (ACE) inhibitors exert their cardiovascular effects not only by preventing the formation of angiotensin II (AII), but also by promoting the accumulation of bradykinin in or at the vessel wall. In addition, certain ACE inhibitors have been shown to augment the vasodilator response to bradykinin, presumably by an interaction at the level of the B2 receptor. We have investigated whether this is a specific effect of the ACE inhibitor class of compounds in isolated endothelium-denuded segments of the rabbit jugular vein where bradykinin elicits a constrictor response which is exclusively mediated by activation of the B2 receptor. 2. Moexiprilat and ramiprilat (< …

PharmacologyRamiprilmedicine.medical_specialtybiologyEnalaprilatBradykininAngiotensin-converting enzymeCaptoprilchemistry.chemical_compoundEndocrinologychemistryInternal medicineACE inhibitorcardiovascular systemmedicinebiology.proteinBradykinin receptorRamiprilatmedicine.drugBritish Journal of Pharmacology
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