Search results for "Endothelial Cell"

showing 10 items of 497 documents

Intraovarian regulation of gonadotropin-dependent folliculogenesis depends on notch receptor signaling pathways not involving Delta-like ligand 4 (Dl…

2013

Background In-situ hybridisation studies demonstrate that Notch receptors and ligands are expressed in granulosa cells (GCs) and in the theca layer vasculature of growing follicles. Notch signaling involves cell-to-cell interaction mediated by transmembrane receptors and ligands. This signaling pathway may represent a novel intraovarian regulator of gonadotropin-dependent follicular development to the preovulatory stage. We hypothesized that blocking Notch pathways would disrupt follicular maturation in the mouse ovary. Methods Hypophysectomized CD21 female mice were administered pregnant mare serum gonadotropin (PMSG) for 3 days to stimulate follicular development. In one experiment, a pan…

Gonadotropins EquineYW152FMiceEndocrinologyOvarian FolliclePregnancyFollicular phaseReceptor Notch2Receptor Notch1Receptor Notch4Receptor Notch3education.field_of_studyReceptors NotchIntracellular Signaling Peptides and ProteinsObstetrics and GynecologyImmunohistochemistryFolliculogenesisObstetricsmedicine.anatomical_structureNotch proteinsThecaTheca Cellscardiovascular systemFemaleJaggedFolliculogenesisSignal Transductionendocrine systemmedicine.medical_specialtyNotchGamma-secretase inhibitorNotch signaling pathwayDll4BiologyProto-Oncogene ProteinsInternal medicineGeneticsmedicineAnimalsHorsesOvarian follicleAntibodies BlockingeducationGranulosa CellsDelta-like ligand 4ResearchOvaryEndothelial CellsMembrane ProteinsMuscle SmoothEstradiol secretionEndocrinologyReproductive MedicineGynecologyFOS: Biological sciencesDevelopmental BiologyReproductive Biology and Endocrinology
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Protective role of the complement regulatory protein human CD-55 in cardiac xenograft: a descriptive study and a revision of the literature.

2002

The limited and inadequate availability of organs from human donors has resulted in the utilisation of xenografts as an alternative tool. Nevertheless, hyperacute rejection (HAR) following xenograft determines the loss of the transplanted organ. The “primum movens” is the activation of the complement pathway mediated by the binding of natural xenogenic antibodies to the endothelium of the graft, followed by the lysis of the endothelial cells with subsequent oedema, thrombosis and necrosis of the transplanted organ. In this work we describe morphological and biomolecular observations of isolated human-decay accelerating factor (h-DAF, CD55) transgenic pig hearts, after perfusion for four hou…

Graft RejectionHistologyCD55 AntigensSwineEndothelial cells2734Blotting WesternTransplantation HeterologousComplementCell BiologyOrgan SizeImmunohistochemistryMicroscopy ElectronEndothelial cellMembrane glycoproteinCoronary CirculationGenetic engineering:6 - Ciencias aplicadas::61 - Medicina::611 - Anatomía [CDU]AnimalsHeart TransplantationHumansXenotransplantationAnatomyComplement; Endothelial cells; Genetic engineering; Membrane glycoproteins; Xenotransplantation; Anatomy; 2734; Histology; Cell Biology
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Postoperative Repositioning of Inverted Descemet Membrane Endothelial Keratoplasty Grafts

2019

Item does not contain fulltext PURPOSE: To present 4 cases of postoperative repositioning of inverted grafts after Descemet membrane endothelial keratoplasty (DMEK). METHODS: Retrospective case reports of 4 patients presenting with subtotal to total graft detachment after DMEK, most probably owing to upside-down graft positioning. Graft repositioning was performed 1 to 14 days after initial DMEK surgery. In 3 cases (cases 1, 3, and 4), repositioning was performed without removing the graft from the anterior chamber (AC), whereas for case 2, the graft had to be removed from the AC to attain correct graft orientation. RESULTS: In all 4 cases, the initially upside-down DMEK grafts could be suc…

Graft RejectionMaleReoperationmedicine.medical_specialtyDescemet membraneClear corneasSensory disorders Donders Center for Medical Neuroscience [Radboudumc 12]Corneal Diseases03 medical and health sciences0302 clinical medicinePostoperative ComplicationsCorneal edemaOphthalmologyCorneamedicineHumansDescemet MembraneAgedRetrospective StudiesAged 80 and overbusiness.industryMiddle AgedEndothelial cell densityOphthalmologymedicine.anatomical_structuresurgical procedures operative030221 ophthalmology & optometryFemalebusiness030217 neurology & neurosurgeryDescemet Stripping Endothelial KeratoplastyCornea. the Journal of Cornea and External Disease
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The gut microbiota - a modulator of endothelial cell function and a contributing environmental factor to arterial thrombosis.

2019

Introduction: There is emerging evidence linking the commensal gut microbiota with the development of cardiovascular disease and arterial thrombosis. In immunothrombosis, the host clotting system protects against the dissemination of invading microbes, not considering the huge number of microbes that interact with host physiology in a mutualistic fashion. Areas covered: Interestingly, recent research revealed that colonizing gut microbes profoundly influence host innate immune pathways that support arterial thrombus growth. The gut microbiota promotes arterial thrombus formation by enhancing the pro-adhesive capacity of the vascular endothelium, triggering hepatic von Willebrand factor synt…

Gut floraEnvironment03 medical and health sciences0302 clinical medicineVon Willebrand factorCell AdhesionMedicineAnimalsHumansPlateletPlatelet activationImmunologic SurveillanceToll-like receptorInnate immune systembiologybusiness.industryEndothelial CellsThrombosisHematologyArteriesmedicine.diseasebiology.organism_classificationThrombosisGastrointestinal MicrobiomeEndothelial stem cell030220 oncology & carcinogenesisImmunologybiology.proteinDisease SusceptibilityEndothelium VascularbusinessBiomarkers030215 immunologyExpert review of hematology
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Role of endothelial cell stress in the pathogenesis of chronic heart failure.

2009

Endothelial cells are key modulators of diverse physiological processes, and their impaired function is a cause of numerous cardiovascular diseases. Under physiologic condition, the reactive oxygen and nitrogen mediators in endothelia lead to the signal propagation of the initial stimulus, by forming molecules with a longer half-life like hydrogen peroxide. Hydrogen peroxide is the focus of growing attention in endothelial biology, and consequently the enzymes involved in its generation and clearance are viewed as novel mediators of great importance. In particular, among peroxidases, myeloperoxidase is recognized as a key enzyme, capable of impairing intracellular NO reservoirs as well as p…

Heart FailureEndotheliumbiologyEndothelial cells Myeloperoxidase Hydrogen Peroxide Oxidative Stress Enos Nitric Oxide Superoxide ROS RNS 3-Chlorotyrosine 3-Nitrotyrosine Nitrosylaton ReviewSuperoxideSettore BIO/16 - Anatomia UmanaOxidative phosphorylationmedicine.disease_causeNitric oxideCell biologyEndothelial stem cellchemistry.chemical_compoundOxidative Stressmedicine.anatomical_structurechemistryMyeloperoxidaseChronic Diseasemedicinebiology.proteinHumansEndothelium VascularReactive Oxygen SpeciesOxidative stressIntracellularPeroxidaseFrontiers in bioscience (Landmark edition)
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Cellular and tissue expression of DAPIT, a phylogenetically conserved peptide

2011

DAPIT (Diabetes Associated Protein in Insulin-sensitive Tissues) is a small, phylogenetically conserved, 58 amino acid peptide that was previously shown to be down-regulated at mRNA level in insulin-sensitive tissues of type 1 diabetes rats. In this study we characterize a custom made antibody against DAPIT and confirm the mitochondrial presence of DAPIT on cellular level. We also show that DAPIT is localized in lysosomes of HUVEC and HEK 293T cells. In addition, we describe the histological expression of DAPIT in several tissues of rat and man and show that it is highly expressed especially in cells with high aerobic metabolism and epithelial cells related to active transport of nutrients …

HistologyCellular respirationProtein subunitBiophysicsPeptideV-ATPaseBiologyMitochondrionAntibodiesMitochondrial ProteinsHuman Umbilical Vein Endothelial CellsV-ATPaseAnimalsHumansmitochondrionta315lcsh:QH301-705.5PhylogenyDAPIT mitochondrion V-ATPase type 1 diabeteschemistry.chemical_classificationRegulation of gene expressionOriginal Papertype 1 diabetes.HEK 293 cellsMembrane ProteinsCell BiologyProton PumpsCell biologyMitochondriaRatsHEK293 CellsMembrane proteinchemistryBiochemistryGene Expression Regulationlcsh:Biology (General)Organ SpecificityLysosomesDAPITEuropean Journal of Histochemistry
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Measuring (biological) materials mechanics with atomic force microscopy. 2. Influence of the loading rate and applied force (colloidal particles)

2020

Atomic force microscopy (AFM) is the most often used tool to study the mechanical properties of eukaryotic cells. Due to their complex assembly, cells show viscoelastic properties. When performing experiments, one has to consider the influence of both loading rate and maximum load on the measured mechanical properties. Here, we employed colloidal particles of various sizes (from 2 to 20 μm diameter) to perform force spectroscopy measurements on endothelial cells at loading rates varying from 0.1 to 50 μm/s, and maximum loads ranging from 1 to 25 nN. We were able to determine the non-linear dependence of cell viscoelastic properties on the loading rate which followed a weak power law. In add…

HistologyMaterials scienceWork (physics)Force spectroscopyEndothelial CellsYoung's modulus030206 dentistry02 engineering and technologyMechanicsMicroscopy Atomic Force021001 nanoscience & nanotechnologyPower lawViscoelasticityStiffening03 medical and health sciencesMedical Laboratory Technologysymbols.namesake0302 clinical medicineColloidal particlesymbolsLoading rateAnatomy0210 nano-technologyInstrumentationMechanical PhenomenaMicroscopy Research and Technique
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Mild Heat Stress Enhances Angiogenesis in a Co-culture System Consisting of Primary Human Osteoblasts and Outgrowth Endothelial Cells

2013

The repair and regeneration of large bone defects, including the formation of functional vasculature, represents a highly challenging task for tissue engineering and regenerative medicine. Recent studies have shown that vascularization and ossification can be stimulated by mild heat stress (MHS), which would offer the option to enhance the bone regeneration process by relatively simple means. However, the mechanisms of MHS-enhanced angiogenesis and osteogenesis, as well as potential risks for the treated cells are unclear. We have investigated the direct effect of MHS on angiogenesis and osteogenesis in a co-culture system of human outgrowth endothelial cells (OECs) and primary osteoblasts …

Hot TemperatureCell SurvivalAngiogenesisCellular differentiationBiomedical EngineeringNeovascularization PhysiologicMedicine (miscellaneous)ApoptosisBioengineeringBiologyRegenerative medicineArticleTissue engineeringOsteogenesisHeat shock proteinHumansRNA MessengerHeat shockBone regenerationCells CulturedCaspase 7OsteoblastsCaspase 3Regeneration (biology)Endothelial CellsCell DifferentiationCoculture TechniquesCapillariesUp-RegulationCell biologyImmunologyHeat-Shock ResponseTissue Engineering Part C: Methods
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Shedding light on the elusive role of endothelial cells in cytomegalovirus dissemination.

2011

Cytomegalovirus (CMV) is frequently transmitted by solid organ transplantation and is associated with graft failure. By forming the boundary between circulation and organ parenchyma, endothelial cells (EC) are suited for bidirectional virus spread from and to the transplant. We applied Cre/loxP-mediated green-fluorescence-tagging of EC-derived murine CMV (MCMV) to quantify the role of infected EC in transplantation-associated CMV dissemination in the mouse model. Both EC- and non-EC-derived virus originating from infected Tie2-cre + heart and kidney transplants were readily transmitted to MCMV-naïve recipients by primary viremia. In contrast, when a Tie2-cre + transplant was infected by pri…

Human cytomegalovirusMuromegalovirusmedicine.medical_treatmentKidneyMicelcsh:QH301-705.5Kidney transplantationHeart transplantationbiologyvirus diseasesHeartAnimal ModelsHost-Pathogen InteractionInfectious Diseasessurgical procedures operativemedicine.anatomical_structureOncologyMedical MicrobiologyCytomegalovirus InfectionsMedicineResearch Articlelcsh:Immunologic diseases. AllergyEndotheliumImmunologyCongenital cytomegalovirus infection610ViremiaMice TransgenicMicrobiologyVirusModel OrganismsMuromegalovirusVirologyGeneticsmedicineAnimalsViremiaBiologyMolecular BiologyEndothelial Cellsmedicine.diseasebiology.organism_classificationVirologyKidney Transplantationlcsh:Biology (General)ImmunologyHeart TransplantationSurgeryParasitologyEndothelium Vascularlcsh:RC581-607PLoS pathogens
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Candida albicans adhesin Als3p is dispensable for virulence in the mouse model of disseminated candidiasis

2011

The presence of specific proteins, including Ece1p, Hwp1p and Als3p, distinguishes theCandida albicanshyphal cell wall from that of yeast-form cells. These proteins are thought to be important for the ability ofC. albicanscells to adhere to living and non-living surfaces and for the cell-to-cell adhesion necessary for biofilm formation, and also to be pivotal in mediatingC. albicansinteractions with endothelial cells. Using anin vitroflow adhesion assay, we previously observed that yeast cells bind in greater numbers to human microvascular endothelial cells than do hyphal or pseudohyphal cells. This is consistent with previous observations that, in a murine model of disseminated candidiasis…

HyphaeMicrobiologyMicrobiologyMicrobial PathogenicityFungal ProteinsMiceCandida albicansCell AdhesionAnimalsHumansCandida albicansCell adhesionFungal proteinMice Inbred BALB CbiologyVirulenceBiofilmCandidiasisEndothelial Cellsbiology.organism_classificationDisseminated CandidiasisCorpus albicansBacterial adhesinDisease Models AnimalFemaleHyphal cell wallGene Deletion
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