Search results for "Endothelial Cell"

showing 10 items of 497 documents

Bioresponsive poly(amidoamine)s designed for intracellular protein delivery.

2013

Poly(amidoamine)s with bioreducible disulfide linkages in the main chain (SS-PAAs) and pH-responsive, negatively charged citraconate groups in the sidechain have been designed for effective intracellular delivery and release of proteins with a net positive charge at neutral pH. Using lysozyme as a cationic model protein these water soluble polymers efficiently self-assemble into nanocomplexes by charge attraction. At pH 5 (the endosomal pH) the amide linkages connecting the citraconate groups in the sidechains of the SS-PAAs are hydrolyzed by intramolecular catalysis, resulting in expulsion of the negative citraconate groups and formation of protonated amine groups, resulting in charge reve…

StereochemistryBiomedical EngineeringBiochemistryBiomaterialsMETIS-302366chemistry.chemical_compoundNanocapsulesAmideIR-90177Materials TestingPolyaminesHumansMolecular BiologyCells CulturedCationic polymerizationEndothelial CellsProteinsGeneral MedicinePoly(amidoamine)CytosolMembranechemistryBiophysicsAmine gas treatingLysozymeIntracellularBiotechnologyActa biomaterialia
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Gold(I) Biscarbene Complexes Derived from Vascular-Disrupting Combretastatin A-4 Address Different Targets and Show Antimetastatic Potential

2014

Gold N-heterocyclic carbene (NHC) complexes are an emerging class of anticancer drugs. We present a series of gold(I) biscarbene complexes with NHC ligands derived from the plant metabolite combretastatin A-4 (CA-4) that retain its vascular-disrupting effect, yet address different cellular and protein targets. Unlike CA-4, these complexes did not interfere with tubulin, but with the actin cytoskeleton of endothelial and cancer cells. For the highly metastatic 518A2 melanoma cell line this effect was accompanied by a marked accumulation of cells in the G1 phase of the cell cycle and a suppression of active prometastatic matrix metalloproteinase-2. Despite these mechanistic differences the co…

StereochemistryNeovascularization PhysiologicAntineoplastic AgentsBiologyBiochemistryMicechemistry.chemical_compoundCoordination ComplexesTubulinCell Line TumorBibenzylsDrug DiscoveryHuman Umbilical Vein Endothelial CellsAnimalsHumansGeneral Pharmacology Toxicology and PharmaceuticsMelanomaCell ProliferationPharmacologyCombretastatin A-4Tube formationCombretastatinMice Inbred BALB COrganic ChemistryCell cycleActin cytoskeletonG2 Phase Cell Cycle CheckpointsActin CytoskeletonChorioallantoic membranechemistryDrug Resistance NeoplasmCell cultureCancer cellMCF-7 CellsCancer researchMolecular MedicineGoldHT29 CellsMethaneChemMedChem
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Endothelial function and pathogenesis of endothelial dysfunction

2018

Background: The crucial role of endothelium is due to the ability of endothelial cells to receive and concurrently respond to humoral and hemodynamic stimuli. The mechanisms that mediate these reactions are: the production of endothelium-derived factors and metabolizing enzymes; the expression of binding proteins and adhesive molecules; and the consequential shape changes. In fact, a wide range of anti-atherosclerotic action substances is produced by the endothelial cells with the objective of maintaining the balance between vasoconstriction and vasodilation, and inhibit or stimulate the proliferation and migration of smooth muscle cells, thrombogenesis and fibrinolysis. Smoke, age, hyperch…

StimuliPathologymedicine.medical_specialtyEndotheliumbusiness.industryImmunologymedicine.diseaseEndothelialPathogenesismedicine.anatomical_structureEndothelial cellDysfunctionPathogenesiImmunology and AllergyMedicineEndotheliumEndothelial dysfunctionbusinessFunction (biology)
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Permeability properties of a three-cell type in vitro model of blood-brain barrier.

2005

We previously found that RBE4.B brain capillary endothelial cells (BCECs) form a layer with blood‐brain barrier (BBB) properties if co‐cultured with neurons for at least one week. As astrocytes are known to modulate BBB functions, we further set a culture system that included RBE4.B BCECs, neurons and astrocytes. In order to test formation of BBB, we measured the amount of (3)H‐sucrose able to cross the BCEC layer in this three‐cell type model of BBB. Herein we report that both neurons and astrocytes induce a decrease in the permeability of the BCEC layer to sucrose. These effects are synergic as if BCECs are cultured with both neurons and astrocytes for 5 days, permeability to sucrose decr…

SucroseCell typeTime FactorsBlotting WesternVascular permeabilityBiologyBlood–brain barrierOccludinArticleCapillary PermeabilityOccludinmedicineAnimalsRats WistarCell Line TransformedNeuronsBrainEndothelial CellsMembrane ProteinsCell BiologyPermeationblood-brain barrier cortical neurons astrocytes brain capillary endothelial cells RBE4.B occludin.Coculture TechniquesRatsmedicine.anatomical_structurenervous systemMembrane proteinBiochemistryBlood-Brain BarrierPermeability (electromagnetism)Astrocytescardiovascular systembiology.proteinBiophysicsMolecular MedicineAntibody
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The effect of detergents on the basement membrane complex of a biologic scaffold material

2013

The basement membrane complex (BMC) is a critical component of the extracellular matrix (ECM) that supports and facilitates the growth of cells. This study investigates four detergents commonly used in the process of tissue decellularization and their effect upon the BMC. The BMC of porcine urinary bladder was subjected to 3% Triton-X 100, 8 mM 3-[(3-cholamidopropyl) dimethylammonio]-1-propanesulfonate (CHAPS), 4% sodium deoxycholate or 1% sodium dodecyl sulfate (SDS) for 24 h. The BMC structure for each treatment group was assessed by immunolabeling, scanning electron microscopy (SEM) and second harmonic generation (SHG) imaging of the fiber network. The composition was assessed by quantif…

Sus scrofaFluorescent Antibody TechniqueBiochemistryBasement MembraneGlycosaminoglycanExtracellular matrixImmunolabelingchemistry.chemical_compoundTissue ScaffoldChapsSodium dodecyl sulfateDecellularizationGlycosaminoglycansMicrovesselEndothelial CellDecellularizationTissue ScaffoldsIntegrin beta1Extracellular matrixGeneral Medicinemedicine.anatomical_structureCollagenHumanBiotechnologyDetergentMaterials scienceDetergentsBiomedical EngineeringArticleBiomaterialsImaging Three-DimensionalRe-endothelizationIn Situ Nick-End LabelingmedicineAnimalsHumansMolecular BiologyOrgan engineeringBasement membraneStaining and LabelingAnimalBiologic scaffoldAntigens CD29Endothelial CellsDNABiomaterialMolecular biologyKi-67 AntigenGlycosaminoglycanchemistryTissue DecellularizationMicrovesselsActa Biomaterialia
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Gold nanoparticle interactions with endothelial cells cultured under physiological conditions

2017

PEGylated gold nanoparticles (AuNPs) have an extended circulation time after intravenous injection in vivo and exhibit favorable properties for biosensing, diagnostic imaging, and cancer treatment. No impact of PEGylated AuNPs on the barrier forming properties of endothelial cells (ECs) has been reported, but recent studies demonstrated that unexpected effects on erythrocytes are observed. Almost all studies to date have been with static-cultured ECs. Herein, ECs maintained under physiological cyclic stretch and flow conditions and used to generate a blood-brain barrier model were exposed to 20 nm PEGylated AuNPs. An evaluation of toxic effects, cell stress, the release profile of pro-infla…

SwineBiomedical EngineeringNanoparticleNanotechnology02 engineering and technology010402 general chemistryBlood–brain barrier01 natural sciencesPolyethylene GlycolsIn vivoHuman Umbilical Vein Endothelial CellsMedicineAnimalsHumansGeneral Materials ScienceParticle SizeCells Culturedbusiness.industryEndothelial Cells021001 nanoscience & nanotechnologyQPR10104 chemical sciencesCancer treatmentCell stressmedicine.anatomical_structureColloidal goldBlood-Brain BarrierBiophysicsNanoparticlesCirculation timeGold0210 nano-technologybusinessBlood stream
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Cytotoxicity and inhibition of P-glycoprotein by selected medicinal plants from Thailand.

2014

Abstract Ethnopharmacological relevance Thai medicine has a long tradition of tonifying medicinal plants. In the present investigation, we studied the flower extracts of Jasminum sambac, Mammea siamensis, Mesua ferrea, Michelia alba, Mimusops elengi, and Nelumbo nucifera and speculated that these plants might influence metabolism and substance flow in the body. Materials and methods Isolation of porcine brain capillary endothelial cells (PBCECs) as well as multidrug-resistance CEM/ADR5000 leukemia cells, MDA-M;B-231 breast cancer, U-251 brain tumor, and HCT-116 colon cancer cells were used. The calcein-acetoxymethylester (AM) assay was used to measure inhibition of P-glycoprotein transport.…

SwineMesua ferreaMimusops elengiFlowersPharmacologyBlood–brain barrierchemistry.chemical_compoundCell Line TumorNeoplasmsDrug DiscoverymedicineAnimalsHumansATP Binding Cassette Transporter Subfamily B Member 1CytotoxicityP-glycoproteinPharmacologyMedicine East Asian TraditionalPlants MedicinalbiologyTraditional medicinePlant ExtractsMammeaBrainEndothelial Cellsmedicine.diseasebiology.organism_classificationThailandAntineoplastic Agents PhytogenicDrug Resistance MultipleLeukemiamedicine.anatomical_structurechemistryBlood-Brain BarrierDrug Resistance Neoplasmbiology.proteinEndothelium VascularGrowth inhibitionJournal of ethnopharmacology
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Liver-primed memory T cells generated under noninflammatory conditions provide anti-infectious immunity.

2013

SummaryDevelopment of CD8+ T cell (CTL) immunity or tolerance is linked to the conditions during T cell priming. Dendritic cells (DCs) matured during inflammation generate effector/memory T cells, whereas immature DCs cause T cell deletion/anergy. We identify a third outcome of T cell priming in absence of inflammation enabled by cross-presenting liver sinusoidal endothelial cells. Such priming generated memory T cells that were spared from deletion by immature DCs. Similar to central memory T cells, liver-primed T cells differentiated into effector CTLs upon antigen re-encounter on matured DCs even after prolonged absence of antigen. Their reactivation required combinatorial signaling thro…

T cellReceptors Antigen T-CellPriming (immunology)chemical and pharmacologic phenomenaBiologyCD8-Positive T-LymphocytesLymphocyte ActivationGeneral Biochemistry Genetics and Molecular BiologyMiceCross-PrimingAntigenCD28 AntigensmedicineAnimalslcsh:QH301-705.5Innate immune systemGene Expression ProfilingT-cell receptorReceptors Interleukin-12CD28Endothelial Cellshemic and immune systemsDendritic CellsAcquired immune systemListeria monocytogenesImmunity InnateNeuropilin-1Mice Inbred C57BLmedicine.anatomical_structurelcsh:Biology (General)LiverImmunologyImmunologic MemoryCD8Cell reports
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Type-II transmembrane prolyl dipeptidases and matrix metalloproteinases in membrane vesicles of active endothelial cells.

2006

Conclusions: Endothelia cells in sparse culture are migratory and increase the production of gelatinases of serine- and metallo-classes in membrane vesicles. Collectively, proteases associated with membrane vesicles degrade extracellular matrix components including type-I and type-IV collagens, laminin and fibronectin. Inhibitor studies suggest the existence of small gelatinases that were derived from these serine- and metallo-proteases. Thus, further studies are warranted to demonstrate the cooperative action of metallo- and serine proteases on cell surfaces and in extracellular vesicles during endothelial cell migration in 3D collagenous matrices, and potential proteolytic activation mech…

TUMOR-CELLSCell MembraneBREAST-CARCINOMA CELLSEndothelial CellsUP-REGULATIONANGIOGENESISMatrix MetalloproteinasesExtracellular MatrixACTIVATIONEnzyme ActivationNEUROPEPTIDE-YCell MovementSEPRASESettore BIO/10 - BiochimicaMETASTASISPEPTIDASE-IVHumansDipeptidyl-Peptidases and Tripeptidyl-PeptidasesINTEGRINCells CulturedAdvances in experimental medicine and biology
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A bacterial metabolite, trimethylamine N-oxide, disrupts the hemostasis balance in human primary endothelial cells but no coagulopathy in mice

2019

: The gut microbial metabolite, trimethylamine N-oxide (TMAO), was previously reported to induce platelet hypersensitivity, which leads to thrombotic risk. However, the molecular mechanism underlying the effects of TMAO on endothelial cells (EC), which is the primary vessel wall contact with the lumen, remains unclear. Here, we investigated the impact of TMAO on procoagulant activity (PCA) in EC and mice, for a possible link between microbiota and coagulation. To test the PCA of TMAO in EC, we performed one-stage clotting assays and converted into PCA. Antitissue factor (TF) antibody was used to test the TF role in PCA. Quantitative PCR was performed to measure the TF, thrombomodulin, IL-6,…

ThrombomodulinMetaboliteTrimethylamine N-oxide030204 cardiovascular system & hematologyPharmacologyThrombomodulinMethylaminesMice03 medical and health scienceschemistry.chemical_compound0302 clinical medicineAnimalsHumansPlateletProtein kinase ABlood CoagulationCells CulturedHemostasisMessenger RNANF-kappa BEndothelial CellsHematologyGeneral MedicineOxidantsReal-time polymerase chain reactionchemistryHemostasis030215 immunologyBlood Coagulation & Fibrinolysis
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