Search results for "Endothelium."

showing 10 items of 748 documents

Ultrastructural study on human lung in alveolitis versus pulmonary fibrosis

1993

Lung specimens of 21 patients with diffuse interstitial lung disease were examined. The present ultrastructural study outlines the topography and distribution of inflammatory changes in the interstitium, endothelium, and in pneumocytes and phagocytes. Alveolitis is characterized by marked regenerative activity of type II pneumocytes (cuboid metaplasia), intraluminal macrophage accumulation, endothelial swelling, multilamination of the endothelial basement membrane, pericapillary edema, and primarily by cellular infiltrates in the interstitial space. The most prominent feature of the interstitium in pulmonary fibrosis is the lack of immunoinflammatory cells. In some areas there is a marked a…

AdultMalePathologymedicine.medical_specialtyEndotheliumPulmonary FibrosisEpitheliumInterstitial spaceEdemaMacrophages AlveolarDrug DiscoveryPulmonary fibrosismedicineHumansLymphocytesLungGenetics (clinical)InflammationBasement membraneMetaplasiaLungbusiness.industryPneumonia PneumocystisType-II PneumocytesInterstitial lung diseaseGeneral MedicineMiddle Agedrespiratory systemmedicine.diseaseCapillariesmedicine.anatomical_structureImmunologyMolecular MedicineFemalemedicine.symptombusinessAlveolitis Extrinsic AllergicThe Clinical Investigator
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Intravascular papillary endothelial hyperplasia: report of 4 cases with immunohistochemical findings.

2008

Intravascular papillary endothelial hyperplasia (IPEH) is a benign endothelial proliferation, usually intravascular, that may mimic angiosarcoma. In this report, four new cases of IPEH involving the oral region are described. The affected sites were the lower lip, labial comissure and the submandibular region. After clinical evaluation, the complete removal of the lesions showed a circumscribed and soft mass. Histologically, the major feature was a reactive proliferation of endothelial cells composed of small papillary structures with hypocellular and hyalinized cores arising in an organized thrombus. Immunohistochemical staining for CD34 was strongly positive in endothelial cells. Vimentin…

AdultMalePathologymedicine.medical_specialtyProliferative indexEndotheliumCD34VimentinmedicineHumansAngiosarcomaGeneral DentistryAgedHyperplasiabiologybusiness.industryHyperplasiaMiddle Aged:CIENCIAS MÉDICAS [UNESCO]medicine.diseaseImmunohistochemistrymedicine.anatomical_structureOtorhinolaryngologyIntravascular papillary endothelial hyperplasiaUNESCO::CIENCIAS MÉDICASbiology.proteinImmunohistochemistrySurgeryFemaleEndothelium VascularbusinessMouth DiseasesMedicina oral, patologia oral y cirugia bucal
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Morphological Alterations and Stress Protein Variations in Lung Biopsies Obtained from Autopsies of COVID-19 Subjects

2021

Molecular chaperones, many of which are heat shock proteins, play a role in cell stress response and regulate the immune system in various ways, such as in inflammatory/autoimmune reactions. It would be interesting to study the involvement of these molecules in the damage done to COVID-19-infected lungs. In our study, we performed a histological analysis and an immunomorphological evaluation on lung samples from subjects who succumbed to COVID-19 and subjects who died from other causes. We also assessed Hsp60 and Hsp90 distribution in lung samples to determine their location and post-translational modifications. We found histological alterations that could be considered pathognomonic for CO…

AdultMalePathologymedicine.medical_specialtyendotheliumEndotheliumQH301-705.5Hsp90InflammationArticleImmune systemCOVID-19EndotheliumHsp60Hsp90InflammationSARS-CoV-2Heat shock proteinSARS-CoV-2; COVID-19; Hsp60; Hsp90; endothelium; inflammationmedicineHumansBiology (General)LungHeat-Shock ProteinsAgedInflammationLungSARS-CoV-2Settore BIO/16 - Anatomia Umanabusiness.industryCOVID-19Endothelial CellsColocalizationGeneral MedicineMiddle AgedHsp60medicine.anatomical_structureImmunohistochemistryFemaleHSP60Autopsymedicine.symptombusinessCells; Volume 10; Issue 11; Pages: 3136
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Cyclooxygenase 2-selective and nonselective nonsteroidal anti-inflammatory drugs induce oxidative stress by up-regulating vascular NADPH oxidases.

2008

Cyclooxygenase 2-selective inhibitors (coxibs) and nonselective nonsteroidal anti-inflammatory drugs (NSAIDs) are associated with an increase in cardiovascular events. The current study was designed to test the effect of coxibs and nonselective NSAIDs on vascular superoxide and nitric oxide (NO) production. mRNA expression of endothelial NO synthase (eNOS) and of the vascular NADPH oxidases was studied in spontaneously hypertensive rats (SHR) and in human endothelial cells. The expression of Nox1, Nox2, Nox4, and p22phox was increased markedly by the nonselective NSAIDs diclofenac or naproxen and moderately by rofecoxib or celecoxib in the aorta and heart of SHR. The up-regulation of NADPH …

AdultMalePharmacologychemistry.chemical_compoundEnosRats Inbred SHRAnimalsHumansPharmacologyNADPH oxidasebiologyCyclooxygenase 2 InhibitorsNitrotyrosineAnti-Inflammatory Agents Non-SteroidalNOX4NADPH Oxidasesbiology.organism_classificationRatsUp-RegulationOxidative StresschemistryCyclooxygenase 2NOX1Apocynincardiovascular systembiology.proteinMolecular MedicineFemaleP22phoxEndothelium VascularPeroxynitriteThe Journal of pharmacology and experimental therapeutics
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Iris Claw Phakic Intraocular Lens for High Myopia

1997

ABSTRACT BACKGROUND: The implantation of a WorstFechner iris claw intraocular lens (IOL) is one of the surgical procedures used for the correction of high myopia. This technique reduces myopia with stähle refractive results; however, its potential long-term risks have not been evaluated. We report results in 94 eyes with a minimum follow -up of 3 years. METHODS: We studied 94 eyes of 62 patients with myopia 5= -7.00 diopters (D) who underwent Worst-Fechner IOL implantation. Lens decentration, permeability of the blood-aqueous barrier by iris angiography, and changes in corneal endothelial density were analyzed. RESULTS: Mean follow-up time was 48.9 months (range 36 to 72 mo). Three years af…

AdultMaleRefractive errormedicine.medical_specialtyTime FactorsVisual acuitygenetic structuresEye diseasemedicine.medical_treatmentVisual AcuityIrisEmmetropiaIntraocular lensRefraction OcularPhakic intraocular lensVision disorderPostoperative ComplicationsOphthalmologyMyopiamedicineHumansDioptreAgedRetrospective StudiesLenses Intraocularbusiness.industryEndothelium CornealMiddle Agedmedicine.diseaseeye diseasesOphthalmologyTreatment OutcomeFemaleSurgerysense organsmedicine.symptombusinessFollow-Up StudiesJournal of Refractive Surgery
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Endothelial study of iris-claw phakic lens: four year follow-up.

1998

Abstract Purpose: To study quantitative and morphometric endothelial changes in phakic eyes implanted with the Worst iris-claw lens to correct high myopia. Setting: Department of Ophthalmology, University Hospital “La Fe”, Valencia, Spain. Material and Methods: This retrospective study involved 111 phakic eyes (73 patients) implanted with the Worst iris-claw lens. Noncontact specular microscopy and computer-assisted analysis was performed preoperatively and 6 months and 1, 2, 3, and 4 years postoperatively. Results: The mean cell loss was 3.85% at 6 months, 6.59% at 1 year, 9.22% at 2 years, 11.68% at 3 years, and 13.42% at 4 years. At 2 years, the hexagonality and coefficient variation in …

AdultMaleReoperationmedicine.medical_specialtyRefractive errorVisual acuitygenetic structuresAnterior Chambermedicine.medical_treatmentEye diseaseCorneal TouchVisual AcuityIrisIntraocular lensCell CountPhakic intraocular lensCorneal DiseasesVision disorderPostoperative ComplicationsLens Implantation IntraocularOphthalmologyCorneaMyopiaMedicineHumansCell SizeRetrospective Studiesbusiness.industryEndothelium CornealMiddle Agedmedicine.diseaseeye diseasesSensory SystemsSurgeryOphthalmologymedicine.anatomical_structureSurgeryFemalesense organsmedicine.symptombusinessFollow-Up StudiesJournal of cataract and refractive surgery
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Loss of the preconditioning effect of rosuvastatin during sustained therapy: a human in vivo study

2011

Studies have demonstrated that the acute administration of 3-hydroxy-3 methylglutaryl coenzyme A (HMG-CoA) reductase inhibitors has protective effects in the setting of ischemia-reperfusion (IR). Previously, we demonstrated that a single dose of rosuvastatin prevented IR-induced endothelial dysfunction in humans through a cyclooxygenase-2-dependent mechanism. Whether the chronic administration of HMG-CoA reductase inhibitors provides similar protection remains controversial and is unknown in humans. Eighteen male volunteers were randomized to receive a single dose of rosuvastatin (20 mg) or placebo. Twenty-four hours later, endothelium-dependent, radial artery flow-mediated dilation (FMD) w…

AdultMaleTime FactorsAdolescentEndotheliumPhysiologyCoenzyme AHyperemiaPharmacologyReductaseDrug Administration ScheduleYoung Adultchemistry.chemical_compoundDouble-Blind MethodIschemiaIn vivoPhysiology (medical)medicineHumansRosuvastatinRosuvastatin CalciumOntarioAnalysis of VarianceSulfonamidesCyclooxygenase 2 Inhibitorsbiologybusiness.industryFluorobenzenesVasodilationRosuvastatin CalciumPyrimidinesmedicine.anatomical_structurechemistryCelecoxibRegional Blood FlowReperfusion InjuryRadial ArteryHMG-CoA reductasebiology.proteinCelecoxibPyrazolesHydroxymethylglutaryl-CoA Reductase InhibitorsCardiology and Cardiovascular MedicinebusinessBlood Flow Velocitymedicine.drugAmerican Journal of Physiology-Heart and Circulatory Physiology
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Continuous therapy with transdermal nitroglycerin does not affect biomarkers of vascular inflammation and injury in healthy volunteers.

2009

Continuous exposure to nitroglycerin (GTN) results in development of tolerance and is associated with increased free radical production and abnormal endothelial function. Elevated plasma biomarkers of inflammation have been shown to be associated with endothelial dysfunction in most cardiovascular conditions. It remains unclear whether exposure to GTN is also associated with increased biomarkers of endothelial and vascular injury or vascular inflammation. In an investigator-blind study, a total of 28 healthy volunteers were randomized to continuous therapy with GTN (0.6 mg/h 24 h/day for 7 days) or no therapy. Venous blood was collected on day 0 and day 7. Plasma levels of markers such as …

AdultMaleVasculitisTime FactorsEndotheliumAdolescentPhysiologyVasodilator AgentsInflammationPharmacologyAdministration CutaneousArginineLesionNitroglycerinYoung AdultPhysiology (medical)MedicineHumansEndothelial dysfunctionTransdermalPharmacologyDose-Response Relationship Drugbusiness.industryInterleukin-6Tumor Necrosis Factor-alphaGeneral MedicineVenous bloodmedicine.diseaseLipoproteins LDLDose–response relationshipP-Selectinmedicine.anatomical_structureAnesthesiacardiovascular systemTumor necrosis factor alphaEndothelium Vascularmedicine.symptombusinessCell Adhesion MoleculesBiomarkerscirculatory and respiratory physiologyCanadian journal of physiology and pharmacology
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Pentaerythrityl Tetranitrate and Nitroglycerin, but not Isosorbide Mononitrate, Prevent Endothelial Dysfunction Induced by Ischemia and Reperfusion

2007

Background— Short term exposure to nitroglycerin (GTN) has protective properties that are similar to ischemic preconditioning. Whether other organic nitrates such as pentaerithrityl tetranitrate (PETN) and isosorbide mononitrate (ISMN) have similar protective effects has not been explored. Methods and Results— In a randomized, parallel, double blind, controlled trial, 37 healthy young volunteers received no therapy (n=10), transdermal GTN 1.2 mg for 2 hours (n=9), PETN 80 mg (n=9), or ISMN 40 mg (n=9). Twenty-four hours later, endothelium-dependent flow-mediated vasodilation (FMD) was measured before and after local exposure to ischemia and reperfusion (IR). In the no therapy group, IR blu…

AdultMaleVasodilator AgentsIschemiaVasodilationPentaerythritol tetranitrateIsosorbide DinitratePharmacologyNitroglycerinchemistry.chemical_compoundDouble-Blind MethodmedicineIsosorbide mononitrateHumansPentaerythritol TetranitrateEndothelial dysfunctionIschemic PreconditioningChemistrymedicine.diseaseReperfusion InjuryAnesthesiacardiovascular systemIschemic preconditioningEndothelium VascularIsosorbide dinitrateCardiology and Cardiovascular MedicineReperfusion injurymedicine.drugArteriosclerosis, Thrombosis, and Vascular Biology
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Contractile responses of human thyroid arteries to vasopressin

2013

Abstract Aims In the present study we investigated the intervention of nitric oxide and prostacyclin in the responses to vasopressin of isolated thyroid arteries obtained from multi-organ donors. Main methods Paired artery rings from glandular branches of the superior thyroid artery, one normal and the other deendothelised, were mounted in organ baths for isometric recording of tension. Concentration–response curves to vasopressin were determined in the absence and in the presence of either the vasopressin V 1 receptor antagonist d(CH 2 ) 5 Tyr(Me)AVP (10 − 8  M), the nitric oxide synthase inhibitor N G -monomethyl- l -arginine ( L -NMMA, 10 − 4  M), or the inhibitor of prostaglandins indom…

AdultMaleVasopressinmedicine.medical_specialtyEndotheliumVasopressinsIndomethacinThyroid GlandProstacyclinIn Vitro TechniquesGeneral Biochemistry Genetics and Molecular BiologyNitric oxideSuperior thyroid arterychemistry.chemical_compoundmedicine.arteryInternal medicinemedicineHumansDrug InteractionsGeneral Pharmacology Toxicology and PharmaceuticsAgedVasopressin receptoromega-N-MethylarginineDose-Response Relationship DrugbiologyChemistryThyroidArteriesGeneral MedicineMiddle AgedArginine VasopressinNitric oxide synthaseEndocrinologymedicine.anatomical_structureVasoconstrictionbiology.proteinFemalehormones hormone substitutes and hormone antagonistsmedicine.drugLife Sciences
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