Search results for "Enhancer"

showing 10 items of 148 documents

Variants associated with HHIP expression have sex-differential effects on lung function

2021

Background: Lung function is highly heritable and differs between the sexes throughout life. However, little is known about sex-differential genetic effects on lung function. We aimed to conduct the first genome-wide genotype-by-sex interaction study on lung function to identify genetic effects that differ between males and females. Methods: We tested for interactions between 7,745,864 variants and sex on spirometry-based measures of lung function in UK Biobank (N=303,612), and sought replication in 75,696 independent individuals from the SpiroMeta consortium. Results: Five independent single-nucleotide polymorphisms (SNPs) showed genome-wide significant (P<5x10-8) interactions with sex …

0301 basic medicineSpirometrymedicine.medical_specialtyHHIPMedicine (miscellaneous)Expression ; Genome-wide Interaction Study ; Hhip ; Lung Function ; SexSingle-nucleotide polymorphismBiology3121 Internal medicineGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciences0302 clinical medicineExpression ; Genome-wide interaction study ; HHIP ; Lung function ; SexInternal medicineexpressionmedicinesexAlleleEnhancerGeneLung functionLunggenome-wide interaction studymedicine.diagnostic_test1184 Genetics developmental biology physiologylung functionALSPAC/dk/atira/pure/core/keywords/alspacDifferential effects030104 developmental biologyEndocrinologymedicine.anatomical_structure030228 respiratory system3121 General medicine internal medicine and other clinical medicine3111 Biomedicine
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Co-regulation of paralog genes in the three-dimensional chromatin architecture.

2016

Paralog genes arise from gene duplication events during evolution, which often lead to similar proteins that cooperate in common pathways and in protein complexes. Consequently, paralogs show correlation in gene expression whereby the mechanisms of co-regulation remain unclear. In eukaryotes, genes are regulated in part by distal enhancer elements through looping interactions with gene promoters. These looping interactions can be measured by genome-wide chromatin conformation capture (Hi-C) experiments, which revealed self-interacting regions called topologically associating domains (TADs). We hypothesize that paralogs share common regulatory mechanisms to enable coordinated expression acco…

0301 basic medicineanimal structuresComputational biologyBiologyGenomeChromosome conformation capture03 medical and health sciencesMice0302 clinical medicineDogsGene DuplicationGene duplicationGeneticsAnimalsCluster AnalysisHumansPromoter Regions GeneticGeneChIA-PETGenomic organizationGeneticsRegulation of gene expressionGenomefungiGene regulation Chromatin and EpigeneticsComputational BiologyChromatin Assembly and DisassemblyBiological EvolutionChromatinChromatin030104 developmental biologyEnhancer Elements GeneticGene Expression Regulation030217 neurology & neurosurgeryNucleic acids research
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Quantitative Assessment of Eye Phenotypes for Functional Genetic Studies Using Drosophila melanogaster

2016

AbstractAbout two-thirds of the vital genes in the Drosophila genome are involved in eye development, making the fly eye an excellent genetic system to study cellular function and development, neurodevelopment/degeneration, and complex diseases such as cancer and diabetes. We developed a novel computational method, implemented as Flynotyper software (http://flynotyper.sourceforge.net), to quantitatively assess the morphological defects in the Drosophila eye resulting from genetic alterations affecting basic cellular and developmental processes. Flynotyper utilizes a series of image processing operations to automatically detect the fly eye and the individual ommatidium, and calculates a phen…

0301 basic medicinegenetic structuresNeurogenesisComputational biologyInvestigationsQH426-470EyeAnimals Genetically Modified03 medical and health sciences0302 clinical medicineOmmatidiumGeneticsAnimalsDrosophila Proteinshuman disease modelsEnhancerMolecular BiologyGeneGenetics (clinical)Genetic Association StudiesGeneticsGene knockdownbiologyModels Geneticneurodevelopmental disordersReproducibility of Resultsbiology.organism_classificationommatidiaPhenotypeeye diseases030104 developmental biologyPhenotypeDrosophila melanogastermodifier screensrough eyeGene Knockdown TechniquesEye developmentsense organsDrosophila melanogaster030217 neurology & neurosurgeryDrosophila ProteinFunction (biology)AlgorithmsG3: Genes, Genomes, Genetics
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Clinical impact of GATA2 mutations in acute myeloid leukemia patients harboring CEBPA mutations: a study of the AML study group.

2016

Clinical impact of GATA2 mutations in acute myeloid leukemia patients harboring CEBPA mutations: a study of the AML study group

AdultMaleCancer Researchmedicine.medical_specialtyMyeloidAdolescentmedicine.disease_causeCohort Studies03 medical and health sciencesYoung Adult0302 clinical medicinehemic and lymphatic diseasesInternal medicineCEBPAmedicineHumansneoplasmsAgedMutationHematologybusiness.industryMyeloid leukemiaHematologyMiddle Agedmedicine.diseaseLymphomaGATA2 Transcription FactorHaematopoiesisLeukemiaLeukemia Myeloid Acutemedicine.anatomical_structureOncology030220 oncology & carcinogenesisMutationCancer researchCCAAT-Enhancer-Binding ProteinsFemalebusiness030215 immunologyLeukemia
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Association Between ABCB1 Genetic Variants and Persistent Chemotherapy-Induced Alopecia in Women With Breast Cancer

2020

Importance Persistent chemotherapy-induced alopecia (pCIA) has been recently described in patients with breast cancer and in its most severe form occurs in up to 10% of these patients. Genetic risk factors associated with pCIA have not been adequately explored. Objective To identify genetic variants associated with pCIA. Design, Setting, and Participants In this genetic association study, 215 women with breast cancer treated with docetaxel-based chemotherapy with a follow-up of 1.5 to 10 years after the end of the treatment were recruited retrospectively through 3 hospital oncology units across Spain between 2005 and 2018. Severe pCIA was defined as lack of scalp hair recovery (Common Termi…

AdultOncologymedicine.medical_specialtyATP Binding Cassette Transporter Subfamily BBiopsyBreast NeoplasmsGenome-wide association studyDocetaxelDermatologyPolymorphism Single Nucleotide030207 dermatology & venereal diseases03 medical and health sciences0302 clinical medicineBreast cancerRisk FactorsInternal medicineAntineoplastic Combined Chemotherapy ProtocolsmedicineHumansGenetic Predisposition to DiseasePromoter Regions GeneticAdverse effectRetrospective StudiesDose-Response Relationship Drugbusiness.industryAge FactorsCase-control studyAlopeciaCommon Terminology Criteria for Adverse EventsRetrospective cohort studyOdds ratioMiddle Agedmedicine.diseaseEnhancer Elements GeneticDocetaxelCase-Control Studies030220 oncology & carcinogenesisFemalebusinessHair FollicleFollow-Up StudiesGenome-Wide Association Studymedicine.drugJAMA Dermatology
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Hepatogenic differentiation of human mesenchymal stem cells from adipose tissue in comparison with bone marrow mesenchymal stem cells

2006

AIM: To investigate and compare the hepatogenic transdifferentiation of adipose tissue-derived stem cells (ADSC) and bone marrow-derived mesenchymal stem cells (BMSC) in vitro. Transdifferentiation of BMSC into hepatic cells in vivo has been described. Adipose tissue represents an accessible source of ADSC, with similar characteristics to BMSC. METHODS: BMSCs were obtained from patients undergoing total hip arthroplasty and ADSC from human adipose tissue obtained from lipectomy. Cells were grown in medium containing 15% human serum. Cultures were serum deprived for 2 d before cultivating under similar pro-hepatogenic conditions to those of liver development using a 2-step protocol with sequ…

AdultTranscriptional ActivationPathologymedicine.medical_specialtyCellular differentiationAdipose tissueBone Marrow CellsBiologyStem cell markerCytochrome P-450 Enzyme SystemClinical ResearchAlbuminsCell Line TumormedicineCytochrome P-450 CYP3AHumansCells CulturedAgedCCAAT-Enhancer-Binding Protein-betaRegeneration (biology)Mesenchymal stem cellTransdifferentiationGastroenterologyCell DifferentiationCytochrome P-450 CYP2E1Mesenchymal Stem CellsGeneral MedicineMiddle AgedPhenotypeAdipose TissueGene Expression RegulationHepatocyte Nuclear Factor 4HepatocytesHepatic stellate cellCancer researchThy-1 AntigensStem cellWorld Journal of Gastroenterology
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Negative Regulation of β Enolase Gene Transcription in Embryonic Muscle Is Dependent upon a Zinc Finger Factor That Binds to the G-rich Box within th…

1998

We have previously identified a muscle-specific enhancer within the first intron of the human beta enolase gene. Present in this enhancer are an A/T-rich box that binds MEF-2 protein(s) and a G-rich box (AGTGGGGGAGGGGGCTGCG) that interacts with ubiquitously expressed factors. Both elements are required for tissue-specific expression of the gene in skeletal muscle cells. Here, we report the identification and characterization of a Kruppel-like zinc finger protein, termed beta enolase repressor factor 1, that binds in a sequence-specific manner to the G-rich box and functions as a repressor of the beta enolase gene transcription in transient transfection assays. Using fusion polypeptides of b…

AgingTranscription GeneticMolecular Sequence DataDown-RegulationRepressorRegulatory Sequences Nucleic AcidBiologyBiochemistryDNA-binding proteinGene Expression Regulation EnzymologicMiceGene expressionAnimalsHumansAmino Acid SequenceCloning MolecularMuscle SkeletalEnhancerMolecular BiologyCell NucleusRegulation of gene expressionZinc fingerSp1 transcription factorBinding SitesSequence Homology Amino AcidZinc FingersCell BiologyMolecular biologyDNA-Binding ProteinsEnhancer Elements GeneticRegulatory sequencePhosphopyruvate HydrataseJournal of Biological Chemistry
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Enhancer trap infidelity in Drosophila optomotor-blind

2013

Reporter gene activity in enhancer trap lines is often implicitly assumed to mirror quite faithfully the endogenous expression of the "trapped" gene, even though there are numerous examples of enhancer trap infidelity. optomotor-blind (omb) is a 160 kb gene in which 16 independent P-element enhancer trap insertions of three different types have been mapped in a range of more than 60 kb. We have determined the expression pattern of these elements in wing, eye-antennal and leg imaginal discs as well as in the pupal tergites. We noted that one pGawB insertion (omb (P4) ) selectively failed to report parts of the omb pattern even though the missing pattern elements were apparent in all other 15…

Arthropod AntennaeNerve Tissue ProteinsEyeGenes ReporterEnhancer trapAnimalsDrosophila ProteinsWings AnimalDrosophila (subgenus)EnhancerPromoter Regions GeneticGeneGeneticsReporter genebiologyPupaChromosome MappingPromoterExtremitiesbiology.organism_classificationImaginal discMutagenesis InsertionalEnhancer Elements GeneticImaginal DiscsInsect ScienceDrosophilaT-Box Domain ProteinsDrosophila ProteinResearch Paper
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Gene Expression Analyses during Spontaneous Reversal of Cardiomyopathy in Mice with Repressed Nuclear CUG-BP, Elav-Like Family (CELF) Activity in Hea…

2015

CUG-BP, Elav-like family (CELF) proteins regulate cell type- and developmental stage-specific alternative splicing in the heart. Repression of CELF-mediated splicing activity via expression of a nuclear dominant negative CELF protein in heart muscle was previously shown to induce dysregulation of alternative splicing, cardiac dysfunction, cardiac hypertrophy, and dilated cardiomyopathy in MHC-CELFΔ transgenic mice. A “mild” line of MHC-CELFΔ mice that expresses a lower level of the dominant negative protein exhibits cardiac dysfunction and myopathy at a young age, but spontaneously recovers normal cardiac function and heart size with age despite the persistence of splicing defects. To the b…

CCAAT-Enhancer-Binding Protein-deltaMaleSerum Response FactorTranscription GeneticCardiomyopathylcsh:MedicineMice Transgenic030204 cardiovascular system & hematologyBiology03 medical and health sciencesMice0302 clinical medicineGene expressionSerum response factormedicineAnimalsHumansMyocytes Cardiaclcsh:Science030304 developmental biologyOligonucleotide Array Sequence AnalysisRegulation of gene expressionHemizygote0303 health sciencesMultidisciplinaryGene Expression ProfilingMyocardiumAlternative splicinglcsh:RGene targetingHeartmedicine.diseaseMolecular biologyCell biologyGene expression profilingAlternative SplicingGene Expression RegulationRNA splicinglcsh:QCalciumFemaleCardiomyopathiesResearch ArticlePLoS ONE
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Regulation of IL-12 p40 Promoter Activity in Primary Human Monocytes: Roles of NF-κB, CCAAT/Enhancer-Binding Protein β, and PU.1 and Identification o…

2001

Abstract Appropriate regulation of IL-12 expression is critical for cell-mediated immune responses. In the present study, we have analyzed the regulation of IL-12 p40 promoter activity in primary human monocytes in vivo. Accordingly, we analyzed the p40 promoter by in vivo footprinting in resting and activated primary human blood CD14+ monocytes. Interestingly, footprints at binding sites for trans-activating proteins such as C/EBP, NF-κB, and ETS were only found upon stimulation with LPS and IFN-γ. In contrast, a footprint over a purine-rich sequence at −155, termed GA-12 (GATA sequence in the IL-12 promoter), was observed in resting, but not activated, cells. Further characterization of t…

CD14ImmunologyDNA FootprintingLipopolysaccharide ReceptorsRepressorBiologyDinoprostoneMonocytesCell LineMicechemistry.chemical_compoundProto-Oncogene ProteinsGene expressionAnimalsHumansImmunology and AllergyBinding sitePromoter Regions GeneticPsychological repressionCells CulturedDNA PrimersBase SequenceCcaat-enhancer-binding proteinsCCAAT-Enhancer-Binding Protein-betaBinding proteinNF-kappa BNuclear ProteinsNF-κBInterleukin-12Molecular biologychemistryMutagenesis Site-DirectedTrans-ActivatorsInterleukin-4The Journal of Immunology
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