Search results for "Enzyme Inhibitor"

showing 10 items of 627 documents

Effects of canrenoate plus angiotensin-converting enzyme inhibitors versus angiotensin-converting enzyme inhibitors alone on systolic and diastolic f…

2004

Background Aldosterone (ALDO) exerts profibrotic effects, acting via the mineralocorticoid receptors in cardiovascular tissues. Aldosterone antagonism in combination with angiotensin-converting enzyme inhibition may better protect against the untoward effects of ALDO than angiotensin-converting enzyme inhibition alone. Methods In a double-blind randomized study, the tolerability and efficacy of canrenoate (25 mg/d) plus captopril versus captopril alone were evaluated in 510 patients with an acute anterior myocardial infarction (MI), a serum creatinine concentration 5.5 mEq/L and creatinine levels to >2.0 mg/L after 10 days of treatment were observed. At 180 days, the mitral E-wave–A-wave ra…

Malemedicine.medical_specialtyAngiotensin-Converting Enzyme Inhibitors Mineralocorticoid Receptor Antagonists/administration & dosage Myocardial Contraction/drug effects Myocardial Infarction/drug therapy Myocardial Infarction/physiopathologymedicine.drug_classMyocardial InfarctionDiastoleAngiotensin-Converting Enzyme InhibitorsPlacebochemistry.chemical_compoundDouble-Blind MethodInternal medicinemedicineHumansMineralocorticoid Receptor AntagonistsCreatinineAldosteronebiologybusiness.industryCaptoprilAngiotensin-converting enzymeMiddle AgedMyocardial ContractionchemistryTolerabilityMineralocorticoidCardiologybiology.proteinDrug Therapy CombinationFemaleCanrenoic AcidCardiology and Cardiovascular MedicinebusinessFollow-Up Studiesmedicine.drugAmerican Heart Journal
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Haploinsufficiency of ATP1A2 encoding the Na+/K+ pump alpha2 subunit associated with familial hemiplegic migraine type 2.

2003

Headache attacks and autonomic dysfunctions characterize migraine, a very common, disabling disorder with a prevalence of 12% in the general population of Western countries(1,2). About 20% of individuals affected with migraine experience aura, a visual or sensory-motor neurological dysfunction that usually precedes or accompanies the headache(3). Although the mode of transmission is controversial(4), population-based and twin studies have implicated genetic factors, especially in migraine with aura(5,6). Familial hemiplegic migraine is a hereditary form of migraine characterized by aura and some hemiparesis. Here we show that mutations in the gene ATP1A2 that encodes the alpha2 subunit of t…

Malemedicine.medical_specialtyAuraCell SurvivalPopulationMigraine with AuraMolecular Sequence DataDrug ResistanceBiologyHaploidyTransfectionATP1A2Internal medicineATP1A3Chlorocebus aethiopsGeneticsmedicineAnimalsHumansEnzyme InhibitorseducationOuabainFamilial hemiplegic migraineChromatography High Pressure LiquidGeneticseducation.field_of_studyBase Sequencemedicine.diseaseMigraine with auraPeptide FragmentsPedigreeEndocrinologyMigraineChromosomes Human Pair 1Case-Control StudiesCOS CellsMutationMutagenesis Site-DirectedFemaleCalcium Channelsmedicine.symptomSodium-Potassium-Exchanging ATPaseHaploinsufficiencyHeLa CellsNature genetics
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Captopril does not affect plasma endothelin-1 during thrombolysis and reperfusion.

1995

Studies showed that endothelin-1 (ET-1) was increased in the acute myocardial infarction (AMI). Experimental studies reported that captopril was able to reduce ET-1 secretion, and that ET-1 was increased during reperfusion. This study was aimed to verify if captopril was able to reduce plasma ET-1 during thrombolysis in AMI. Seventy-three patients, hospitalized for suspected AMI within 4 h from the onset of symptoms suitable for thrombolysis (1st episode), Killip class 1-2, were randomized (double blind) into two groups: group 1 (37 pts), 8 F/29 M, received captopril, 6.25 mg, orally 15 min before thrombolysis. Group 2: (36 pts) 8 F/28 M, received placebo before thrombolysis. All patients m…

Malemedicine.medical_specialtyCaptoprilTime Factorsmedicine.medical_treatmentMyocardial InfarctionAdministration OralAngiotensin-Converting Enzyme InhibitorsBlood PressureMyocardial ReperfusionPlaceboAnginaPlacebosElectrocardiographyDouble-Blind MethodHeart RateInternal medicineFibrinolysismedicineHumansThrombolytic TherapyMyocardial infarctionAngina UnstableCreatine KinaseKillip classbusiness.industryUnstable anginaEndothelinsCaptoprilThrombolysismedicine.diseaseRecombinant ProteinsSurgeryIsoenzymesTissue Plasminogen ActivatorCardiologyFemaleCardiology and Cardiovascular Medicinebusinessmedicine.drugInternational journal of cardiology
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The combination ace-inhibitors plus canreonate in patients with anterior myocardial infarction: safety and tolerability study.

2001

There is recent evidence that aldosterone (ALDO) exerts pro-fibrotic effects, acting via the mineral-corticoid receptors in cardiovascular tissues and partial aldosterone escape during ACE-inhibition treatment occurs.A double blind randomised study was performed to evaluate the feasibility, and tolerability of the administration of the 25 mg/day of canreonate plus captopril versus captopril alone in patients with anterior AMI unsuitable for thrombolysis and/or not receiving thrombolytic treatment, and unreperfused after thrombolysis. Fifty five patients hospitalised for anterior AMI,with a serum creatinine concentration2.0 mg/dl and a serum K concentration5.0 mmol per liter were randomised …

Malemedicine.medical_specialtyCaptoprilmedicine.medical_treatmentAldosterone escapeUrologyMyocardial InfarctionAngiotensin-Converting Enzyme Inhibitorschemistry.chemical_compoundDouble-Blind MethodmedicineHumanscardiovascular diseasesMyocardial infarctionAgedCreatinineE/A ratiobusiness.industryCaptoprilThrombolysisMiddle Agedmedicine.diseaseSurgerychemistryTolerabilityACE inhibitorFeasibility StudiesDrug Therapy CombinationFemaleCanrenoic AcidCardiology and Cardiovascular Medicinebusinessmedicine.drugInternational journal of cardiology
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Brain BDNF Levels Elevation Induced by Physical Training is Reduced after Unilateral Common Carotid Artery Occlusion in Rats

2014

International audience; We investigated the contribution of blood flow elevation in the cerebrovasculature to physical training-induced brain-derived neurotrophic factor (BDNF) levels elevation in the brain. Brain-derived neurotrophic factor protein levels were measured in the motor cortex 24 h after the last session of a forced treadmill walking (30 minutes a day, 18 m/minute for 7 consecutive days). Unilateral common carotid artery occlusion and modulation of exercise intensity (0 versus -10% inclination of the treadmill) were used as strategies to reduce the (normal) elevation of flow in the cerebrovasculature occurring during exercise. Administration of N-nitro-L-arginine methyl ester (…

Malemedicine.medical_specialtyCarotid Artery CommonINHIBITIONHemodynamicsArterial Occlusive DiseasesNitric OxideVascular occlusionNEUROTROPHIC FACTOR EXPRESSIONPhysical Conditioning AnimalRats Inbred SHRmedicine.arteryInternal medicineOcclusionmedicineAnimalsDYNAMIC EXERCISECommon carotid arteryEnzyme InhibitorsRats WistarTreadmillSPONTANEOUSLY HYPERTENSIVE-RATSBrain-derived neurotrophic factorNITRIC-OXIDEbusiness.industryBrain-Derived Neurotrophic FactorHemodynamicsBrainVELOCITYRatsNG-Nitroarginine Methyl EsterNeurologyCerebral blood flowHIPPOCAMPAL BDNFAnesthesiaHypertensionCEREBRAL-BLOOD-FLOWCardiologyExercise intensityOriginal ArticleEXERCISE-INDUCED INCREASESACTIVATION PATTERNS[ SCCO ] Cognitive scienceNeurology (clinical)medicine.symptomCardiology and Cardiovascular MedicinebusinessJournal of Cerebral Blood Flow & Metabolism
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Novel antihypertensive hexa- and heptapeptides with ACE-inhibiting properties: From the in vitro ACE assay to the spontaneously hypertensive rat

2011

Bioactive ACE inhibiting peptides are gaining interest in hypertension treatment. We have designed and screened six synthetic heptapeptides (PACEI48 to PACEI53) based on two hexapeptide leads (PACEI32 and PACEI34) to improve ACE inhibitory properties and assess their antihypertensive effects. ACE activity was assayed in vitro and ex vivo. Selected peptides were administered to spontaneously hypertensive rats (SHRs) and normotensive Wistar-Kyoto (WKY) rats. In vitro cytotoxicity was assessed with the MTT reduction test. The six heptapeptides at low micromolar concentration produced different degrees of in vitro inhibition of ACE activity using the synthetic substrate HHL or the natural subst…

Malemedicine.medical_specialtyCell SurvivalPhysiologyAdministration OralAngiotensin-Converting Enzyme InhibitorsBlood PressurePeptidyl-Dipeptidase APharmacologyRats Inbred WKYBiochemistryTissue Culture TechniquesMiceCellular and Molecular NeuroscienceEndocrinologySpontaneously hypertensive ratOral administrationRats Inbred SHRInternal medicineRenin–angiotensin systemmedicineAnimalsHumansInfusions IntravenousAntihypertensive AgentsbiologyChemistryAngiotensin-converting enzyme3T3 CellsHep G2 CellsIn vitroRatsCarotid ArteriesEndocrinologyVasoconstrictionHypertensionACE inhibitorbiology.proteinRabbitsAngiotensin Imedicine.symptomOligopeptidesVasoconstrictionEx vivomedicine.drug
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Modulation by nitric oxide of spontaneous mechanical activity in rat proximal colon.

1999

Summary 1 In order to examine the role of nitric oxide (NO) in the tonic neural inhibition in rat proximal colon, the effects of Nω-nitro- l-arginine methyl ester (L-NAME) were studied on the spontaneous contractions of circular muscle (monitored as intraluminal pressure changes) and of longitudinal muscle (detected as isometric tension changes). 2 L-NAME (3 × 10−−6–3 × 10−−4 m) caused a concentration-dependent increase in the amplitude of circular contractions, without affecting those of longitudinal muscle. This effect was prevented by l-arginine (1–5 × 10−−3 m), but not d-arginine. 3 In the presence of tetrodotoxin (10−−6 m), which per se induced increase of the pressure waves, L-NAME (1…

Malemedicine.medical_specialtyColonIsometric exerciseNeurotransmissionIn Vitro TechniquesInhibitory postsynaptic potentialNitric OxideNitric oxideTonic (physiology)chemistry.chemical_compoundInternal medicineIsometric ContractionmedicineAnimalsDrug InteractionsEnzyme InhibitorsRats WistarGuanethidinePharmacologyDose-Response Relationship DrugGeneral NeuroscienceMuscle SmoothRatsEndocrinologyNG-Nitroarginine Methyl EsterchemistryTetrodotoxinHexamethoniummedicine.drugMuscle ContractionJournal of autonomic pharmacology
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Influence of nitric oxide on neurogenic contraction and relaxation of the human gastroepiploic artery.

2003

Abstract Background The objective of this study was to characterize the neurogenic contraction and relaxation of the human gastroepiploic artery and to determine whether the responses are mediated by nitric oxide (NO) from neural or endothelial origin. Methods Rings of human gastroepiploic artery were obtained from 18 patients (12 men, 6 women) undergoing gastrectomy. The rings were suspended in organ baths for isometric recording of tension. We studied the contractile and relaxant responses to electrical field stimulation. Results In arteries under resting conditions, electrical field stimulation (2 to 8 Hz) caused frequency-dependent contractions that were of greater magnitude in arteries…

Malemedicine.medical_specialtyContraction (grammar)EndotheliumGastroepiploic ArteryTetrodotoxinIn Vitro TechniquesArginineAutonomic Nervous SystemNitric OxideNitric oxidechemistry.chemical_compoundInternal medicineInternal MedicinemedicineHumansAnesthetics LocalEnzyme InhibitorsGuanethidineAgedAged 80 and overomega-N-Methylargininebusiness.industryMiddle AgedVasodilationmedicine.anatomical_structureEndocrinologychemistryVasoconstrictionAnesthesiaCirculatory systemTetrodotoxinFemaleEndothelium Vascularmedicine.symptomNitric Oxide SynthasebusinessGastroepiploic ArteryMuscle contractionmedicine.drugAmerican journal of hypertension
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Modulation of adrenergic contraction of dog pulmonary arteries by nitric oxide and prostacyclin.

1999

Abstract The aim of this work was to investigate the influence of endothelium-derived nitric oxide and prostaglandins on the contractile responses of isolated dog pulmonary arteries to electrical field stimulation and noradrenaline. Electrical field stimulation (1–8 Hz, 20 v, 0.25 ms duration, for 30 s) produced frequency-dependent contractions that were abolished by tetrodotoxin, guanethidine and, prazosin (all at 10−6 M). Noradrenaline induced concentration-dependent contractions with an EC50 of 1.85 × 10−6 M. The increases in tension induced by electrical stimulation and noradrenaline were of greater magnitude in arteries denuded of endothelium. In segments with endothelium, NG-nitro- l …

Malemedicine.medical_specialtyEndotheliumArginineIndomethacinAdrenergicProstacyclinStimulationIn Vitro TechniquesPulmonary ArteryNitric OxideNitric oxidechemistry.chemical_compoundNorepinephrineDogsInternal medicinePrazosinmedicineAnimalsCyclooxygenase InhibitorsDrug InteractionsEnzyme InhibitorsGuanethidineAntihypertensive AgentsPharmacologyEpoprostenolElectric Stimulationmedicine.anatomical_structureEndocrinologyNG-Nitroarginine Methyl EsterchemistryVasoconstrictionProstaglandinsFemalemedicine.drugGeneral pharmacology
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Reversal of Endothelial Nitric Oxide Synthase Uncoupling and Up-Regulation of Endothelial Nitric Oxide Synthase Expression Lowers Blood Pressure in H…

2006

Objectives We sought to examine the hypothesis that a pharmacologic up-regulation of endothelial nitric oxide synthase (eNOS) combined with a reversal of eNOS uncoupling provides a protective effect against cardiovascular disease. Background Many cardiovascular diseases are associated with oxidant stress involving protein kinase C (PKC) and uncoupling of eNOS. Methods Messenger ribonucleic acid (mRNA) expression was analyzed with RNase protection assay or quantitative real-time polymerase chain reaction, vascular nitric oxide (NO) with spin trapping, and reactive oxygen species (ROS) with dihydroethidium fluorescence. Results Aortas of spontaneously hypertensive rats (SHR) showed an elevate…

Malemedicine.medical_specialtyEndotheliumRats Inbred WKYNitric oxidechemistry.chemical_compoundEnosInternal medicineRats Inbred SHRMedicineAnimalsMidostaurinEnzyme InhibitorsProtein Kinase Cbiologybusiness.industryNOX4biology.organism_classificationStaurosporineRatsUp-RegulationNitric oxide synthaseEndocrinologymedicine.anatomical_structurechemistryHypertensionbiology.proteincardiovascular systemP22phoxEndothelium VascularNitric Oxide SynthasebusinessCardiology and Cardiovascular MedicineNicotinamide adenine dinucleotide phosphateJournal of the American College of Cardiology
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