Search results for "Enzyme Inhibitor"

showing 10 items of 627 documents

Chronic Therapy With Isosorbide-5-Mononitrate Causes Endothelial Dysfunction, Oxidative Stress and a Marked Increase in Vascular Endothelin-1 Express…

2011

Aims Isosorbide-5-mononitrate (ISMN) is one of the most frequently used compounds in the treatment of coronary artery disease predominantly in the USA. However, ISMN was reported to induce endothelial dysfunction, which was corrected by vitamin C pointing to a crucial role of reactive oxygen species (ROS) in causing this phenomenon. We sought to elucidate the mechanism how ISMN causes endothelial dysfunction and oxidative stress in vascular tissue. Methods and results Male Wistar rats ( n = 69 in total) were treated with ISMN (75 mg/kg/day) or placebo for 7 days. Endothelin (ET) expression was determined by immunohistochemistry in aortic sections. Isosorbide-5-mononitrate infusion caused si…

Malemedicine.medical_specialtyNitric Oxide Synthase Type IIIIsosorbide DinitratePharmacologymedicine.disease_causeBiochemistryMicechemistry.chemical_compoundSuperoxidesEnosInternal medicinePhysiology (medical)medicineAnimalsNitric Oxide DonorsEnzyme InhibitorsRats WistarEndothelial dysfunctionCyclic GMPAortaMice KnockoutNADPH oxidaseEndothelin-1biologybusiness.industryNADPH Oxidasesmedicine.diseasebiology.organism_classificationEndothelin 1BosentanRatsNitric oxide synthaseEndothelial stem cellOxidative StressNG-Nitroarginine Methyl EsterEndocrinologychemistryApocyninbiology.proteinEndothelium VascularCardiology and Cardiovascular MedicineEndothelin receptorbusinessOxidative stressSignal Transductionmedicine.drugFree Radical Biology and Medicine
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GABAergic inhibition of nitric oxide-mediated relaxation of guinea-pig ileum

1999

The effects of GABA receptor agonists were investigated on guinea-pig isolated ileum longitudinal muscle with intact myenteric plexus. Electrical field stimulation (1 Hz, 10 s) of the histamine (1 microM)-precontracted preparation caused a contraction followed by a relaxation. Relaxations were inhibited by L-N(G)-nitroarginine (L-NA; EC50 3 microM) in a concentration-dependent manner. The inhibitory action of 10 microM L-NA was blocked by 10 microM L-arginine but not by D-arginine, which indicates that the relaxation was largely mediated by endogenous nitric oxide (NO). Tetrodotoxin (1 microM) reduced the relaxation only by about 50%. GABA and the GABA(B) agonist, baclofen, inhibited the fi…

Malemedicine.medical_specialtyNitric Oxide Synthase Type IIIMuscle RelaxationGuinea PigsTetrodotoxinIn Vitro TechniquesGABAB receptorBicucullineNitric OxideNitroargininechemistry.chemical_compoundGABA receptorIleumInternal medicinemedicineAnimalsGABA-A Receptor AgonistsEnzyme InhibitorsGABA Agonistsgamma-Aminobutyric AcidMyenteric plexusPharmacologyMuscimolGABAA receptorMuscle SmoothGeneral MedicineBicucullineElectric StimulationBaclofenEndocrinologynervous systemchemistryMuscimolGABA-B Receptor AgonistsSaclofenBiophysicsFemaleNitric Oxide SynthaseHistaminemedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
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Modulation by NO of acetylcholine release in the ileum of wild-type and NOS gene knockout mice.

2002

Nitric oxide (NO) inhibits the release of acetylcholine and cholinergic contractions in the small intestine of several species, but no information is available about the mouse ileum. This study examines the effects of NO on the electrically evoked release of [3H]acetylcholine and smooth muscle contraction in myenteric plexus-longitudinal muscle preparations of wild-type mice and of neuronal NO synthase (nNOS) and endothelial NOS (eNOS) knockout mice. The NOS inhibitor N G-nitro-l-arginine (l-NNA) and the guanylyl cyclase inhibitor 1 H-[1,2,4]oxadiazolo[4,3-α]quinoxalin-1-one (ODQ) concentration dependently increased the evoked [3H]acetylcholine release and cholinergic contractions in prepa…

Malemedicine.medical_specialtyNitric Oxide Synthase Type IIIPhysiologyNitric Oxide Synthase Type IIIleumNitric Oxide Synthase Type IBiologyIn Vitro TechniquesNitric OxideNitroarginineNitric oxidechemistry.chemical_compoundMiceIleumPhysiology (medical)Internal medicineQuinoxalinesmedicineAnimalsNitric Oxide DonorsEnzyme InhibitorsGene knockoutMice KnockoutOxadiazolesHepatologyPenicillamineGastroenterologyNitric Oxide Synthase Type IIISmall intestineAcetylcholineElectric StimulationNitric oxide synthaseEndocrinologymedicine.anatomical_structurechemistrybiology.proteinCholinergicNitric Oxide SynthaseGastrointestinal MotilityAcetylcholinemedicine.drugAmerican journal of physiology. Gastrointestinal and liver physiology
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Inhibition of gastric acid secretion by stress: A protective reflex mediated by cerebral nitric oxide

1996

Moderate somatic stress inhibits gastric acid secretion. We have investigated the role of endogenously released NO in this phenomenon. Elevation of body temperature by 3°C or a reduction of 35 mmHg (1 mmHg = 133 Pa) in blood pressure for 10 min produced a rapid and long-lasting reduction of distension-stimulated acid secretion in the rat perfused stomach in vivo . A similar inhibitory effect on acid secretion was produced by the intracisternal (i.c.) administration of oxytocin, a peptide known to be released during stress. Intracisternal administration of the NO-synthase inhibitor, N G -nitro- l -arginine methyl ester ( l -NAME) reversed the antisecretory effect induced by all these stimul…

Malemedicine.medical_specialtyNitric OxideNitric oxideGastric Acidchemistry.chemical_compoundStress PhysiologicalInternal medicinemedicineAnimalsEnzyme InhibitorsRats WistarMicroinjectionMultidisciplinarybiologyBrainBiological SciencesImmunohistochemistryRatsVagus nerveNitric oxide synthaseNG-Nitroarginine Methyl EsterEndocrinologyDorsal motor nucleuschemistryOxytocinbiology.proteinReflexGastric acidFemaleNitric Oxide Synthasemedicine.drugProceedings of the National Academy of Sciences
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Cardiac effects of isoliquiritigenin

1997

The effects of isoliquiritigenin on force of contraction (Fc), L-type Ca2+ current (I(Ca)) and intracellular Ca2+ concentration ([Ca2+]i) were investigated in rat ventricular heart muscle. Isoliquiritigenin increased Fc and I(Ca) and, after longer exposure times, resting tension and [Ca2+]i. The effect of isoliquiritigenin (100 microM) on I(Ca) was diminished by Rp-cAMPS (30 microM). 1H-[1,2,4]oxa- diazolo[4,3-a]quinoxalin-1-one (50 microM) did not influence the effects of isoliquiritigenin on Fc and I(Ca). The positive inotropic effects of isoprenaline and forskolin, but not of 3-isobutyl-1-methylxanthine, were potentiated by isoliquiritigenin (100 microM). In the presence of milrinone (10…

Malemedicine.medical_specialtyPatch-Clamp TechniquesFura-2In Vitro TechniquesMembrane PotentialsRats Sprague-Dawleychemistry.chemical_compoundChalconeChalconesAldehyde ReductaseInternal medicineIsoprenalinemedicineAnimalsDrug InteractionsEnzyme InhibitorsCyclic GMPPharmacologyPlants MedicinalForskolinMyocardiumPhosphodiesteraseHeartCyclic AMP-Dependent Protein KinasesMyocardial ContractionRatsElectrophysiologyEndocrinologychemistryGuanylate CyclaseMilrinoneCalciumFemalemedicine.symptomSoluble guanylyl cyclaseIsoliquiritigeninMuscle contractionmedicine.drugEuropean Journal of Pharmacology
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Inhibition of mechanical activity by neurotensin in rat proximal colon: involvement of nitric oxide.

1997

The aim of the present study was to define the nature of inhibitory action of neurotensin in rat proximal colon. Mechanical activity was detected as changes of intraluminal pressure. Neurotensin (10(-10) to 10(-7) M), in the presence of atropine (10(-6) M), guanethidine (10(-6) M), and nifedipine (10(-8) M), induced a tetrodotoxin-insensitive inhibitory effect characterized by the complete disappearance of the spontaneous phasic contractions. The inhibitory effect of neurotensin (10(-7) M) was abolished by scorpion venom (Leiurus quinquestriatus hebraeus) (10(-6) g/ml) or high K+ (40 mM KCl), whereas it persisted in the presence of omega-conotoxin GVIA, (10(-7) M). N omega-nitro-L-arginine…

Malemedicine.medical_specialtyPhysiologyColonNeuropeptideScorpion VenomsTetrodotoxinIn Vitro TechniquesInhibitory postsynaptic potentialNitric Oxidecomplex mixturesNitric oxidechemistry.chemical_compoundomega-Conotoxin GVIAPhysiology (medical)Internal medicinemedicineAnimalsOmega-Conotoxin GVIAEnzyme InhibitorsRats WistarGuanethidineNeurotensinHepatologybiologyGastroenterologyRatsNitric oxide synthaseEndocrinologyNG-Nitroarginine Methyl EsterMechanism of actionchemistrybiology.proteinPotassiumFemalemedicine.symptomGastrointestinal MotilityPeptidesmedicine.drugNeurotensinThe American journal of physiology
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Glucagon-like peptide-2 relaxes mouse stomach through vasoactive intestinal peptide release.

2009

Glucagon-like peptide-2 (GLP-2) influences different aspects of the gastrointestinal function, including epithelial growth, digestion, absorption, motility, and blood flow. Intraluminal pressure from isolated mouse stomach was recorded to investigate whether GLP-2 affects gastric tone and to analyze its mechanism of action. Regional differences between diverse parts of the stomach were also examined using circular muscular strips from fundus and antrum. In the whole stomach, GLP-2 (0.3–100 nM) produced concentration-dependent relaxation with a maximum that was about 75% of relaxation to 1 μM isoproterenol (IC50 = 2.5 nM). This effect was virtually abolished by desensitization of GLP-2 rece…

Malemedicine.medical_specialtyPhysiologyVasoactive intestinal peptideGastric motilityMotilityTetrodotoxinIn Vitro TechniquesPeptide hormoneBiologySettore BIO/09 - FisiologiaMiceenteric nervous systemPhysiology (medical)Internal medicineGlucagon-Like Peptide 2Pyloric AntrummedicineAnimalsChymotrypsingastric motilityGastric FundusEnzyme InhibitorsSympathomimeticsHepatologyStomachdigestive oral and skin physiologyIsoproterenolGastroenterologygastrointestinal hormoneGlucagon-like peptide-2Mice Inbred C57BLVIPNG-Nitroarginine Methyl EsterEndocrinologymedicine.anatomical_structureGastric EmptyingGastrointestinal hormoneGastrointestinal functionhormones hormone substitutes and hormone antagonistsSodium Channel BlockersVasoactive Intestinal Peptide
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A randomized multicentre trial to compare revascularization with optimal medical therapy for the treatment of chronic total coronary occlusions

2018

Aims: The clinical value of percutaneous coronary intervention (PCI) for chronic coronary total occlusions (CTOs) is not established by randomized trials. This study should compare the benefit of PCI vs. optimal medical therapy (OMT) on the health status in patients with at least one CTO.Method and results: Three hundred and ninety-six patients were enrolled in a prospective randomized, multicentre, open-label, and controlled clinical trial to compare the treatment by PCI with OMT with a 2:1 randomization ratio. The primary endpoint was the change in health status assessed by the Seattle angina questionnaire (SAQ) between baseline and 12 months follow-up. Fifty-two percent of patients have …

Malemedicine.medical_specialtyRandomizationmedicine.medical_treatmentAdrenergic beta-AntagonistsAngiotensin-Converting Enzyme Inhibitors030204 cardiovascular system & hematologyAngina Pectorislaw.inventionAngina03 medical and health sciences0302 clinical medicinePercutaneous Coronary InterventionRandomized controlled triallawInternal medicinemedicineClinical endpointHumansPercutaneous transluminal interventioncardiovascular diseases030212 general & internal medicineMyocardial infarctionOptimal medical therapyAgedNitratesbusiness.industryAngina PectoriPercutaneous coronary interventionAngiotensin-Converting Enzyme InhibitorMiddle Agedmedicine.diseaseClinical trialCoronary OcclusionChronic coronary occlusion; EQ-5D; Optimal medical therapy; Percutaneous transluminal intervention; Seattle angina questionnaire; Cardiology and Cardiovascular MedicineConventional PCIChronic DiseaseChronic coronary occlusionQuality of LifeSeattle angina questionnaireFemaleHydroxymethylglutaryl-CoA Reductase InhibitorsCardiology and Cardiovascular MedicinebusinessPlatelet Aggregation Inhibitors
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Adenosine A2A receptors in diffuse dermal fibrosis: pathogenic role in human dermal fibroblasts and in a murine model of scleroderma.

2006

Objective Adenosine regulates inflammation and tissue repair, and adenosine A2A receptors promote wound healing by stimulating collagen matrix production. We therefore examined whether adenosine A2A receptors contribute to the pathogenesis of dermal fibrosis. Methods Collagen production by primary human dermal fibroblasts was analyzed by real-time polymerase chain reaction, 14C-proline incorporation, and Sircol assay. Intracellular signaling for dermal collagen production was investigated using inhibitors of MEK-1 and by demonstration of ERK phosphorylation. In vivo effects were studied in a bleomycin-induced dermal fibrosis model using adenosine A2A receptor–deficient wild-type littermate …

Malemedicine.medical_specialtyReceptor Adenosine A2AImmunologyMAP Kinase Kinase 1Adenosine A2A receptorGene ExpressionBiologyMiceRheumatologyFibrosisInternal medicinemedicineImmunology and AllergyAnimalsHumansPharmacology (medical)RNA MessengerEnzyme InhibitorsReceptorCells CulturedMice Knockoutintegumentary systemTriazinesDermisPurinergic signallingFibroblastsTriazolesAdenosine A3 receptormedicine.diseaseAdenosineAdenosine receptorFibrosisMice Inbred C57BLDisease Models AnimalHydroxyprolineEndocrinologyScleroderma DiffuseCancer researchCollagenWound healingmedicine.drugArthritis and rheumatism
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Experimental diabetes induces hyperreactivity of rabbit renal artery to 5-hydroxytryptamine.

2002

Abstract The influence of diabetes on the response of isolated rabbit renal arteries to 5-hydroxytryptamine (5-HT) was examined. 5-HT induced a concentration-related contraction that was higher in arteries from diabetic rabbits than in arteries from control rabbits. Endothelium removal did not significantly modify 5-HT contractions in arteries from control rabbits but enhanced the response to 5-HT in arteries from diabetic rabbits. Incubation with N G -nitro- l -arginine ( l -NA) enhanced contractions to 5-HT in arteries from control and diabetic rabbits. In arteries with endothelium, this l -NA enhancement was lower in diabetic rabbits than in control rabbits. In arteries without endotheli…

Malemedicine.medical_specialtySerotoninContraction (grammar)EndotheliumIndomethacinIn Vitro TechniquesNitroarginineDiabetes Mellitus Experimentalchemistry.chemical_compoundNitroarginineRenal ArteryInternal medicinemedicine.arteryAlloxanmedicineAnimalsRenal arteryEnzyme InhibitorsPharmacologyDose-Response Relationship Drugbusiness.industryProstanoidDrug Synergismmedicine.anatomical_structureEndocrinologychemistryVasoconstrictionCirculatory systemRabbitsmedicine.symptomNitric Oxide SynthasebusinessVasoconstrictionBlood vesselEuropean journal of pharmacology
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