Search results for "Epigenetic"

showing 10 items of 632 documents

Gene expression changes after parental exposure to metals in the sea urchin affect timing of genetic programme of embryo development

2021

Simple Summary Intergenerational and transgenerational effects, in which exposure to stressors in a parental generation affects the phenotype of the offspring have been connected to anthropic impacts on biological systems. Therefore, environmental stress experienced inside a generation, particularly during gametogenesis, may lead to erroneous patterns in their offspring just emerging at early developmental stages. In this scenario, the sea urchin embryo represents a suitable model for integrating analyses of gene expression through embryogenesis with developmental alteration induced by environmental stressors. Herein we provide pieces of evidence for the alteration of the gene regulatory ne…

0301 basic medicineSea urchinanimal structuresOffspringIntergenerational effectsSettore BIO/11 - Biologia Molecolare010501 environmental sciencesEmbryo development01 natural sciencesArticleGeneral Biochemistry Genetics and Molecular BiologyAndrology03 medical and health sciencesbiology.animalGene expressionParental exposureEpigeneticslcsh:QH301-705.5GeneSea urchin0105 earth and related environmental sciencesGeneral Immunology and MicrobiologybiologyEmbryogenesisEmbryoPhenotype030104 developmental biologyGene expression profileslcsh:Biology (General)embryonic structuressea urchin; redox homeostasis; parental exposure; intergenerational effects; embryo development; gene expression profilesGeneral Agricultural and Biological SciencesEmbryo development Gene expression profiles Intergenerational effects Parental exposure Redox homeostasis Sea urchinRedox homeostasis
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Oxidative stress, autophagy, epigenetic changes and regulation by miRNAs as potential therapeutic targets in osteoarthritis

2015

Aging is a natural process characterized by the declining ability of the different organs and tissues to respond to stress, increasing homeostatic imbalance and risk of disease. Osteoarthritis (OA) is a multifactorial disease in which cartilage degradation is a central feature. Aging is the main risk factor for OA. In OA cartilage, a decrease in the number of chondrocytes and in their ability to regenerate the extracellular matrix and adequately respond to stress has been described. OA chondrocytes show a senescence secretory phenotype (SSP) consisting on the overproduction of cytokines (interleukins 1 and 6), growth factors (e.g., epidermal growth factor) and matrix metalloproteinases (MMP…

0301 basic medicineSenescenceMAPK/ERK pathwayAgingProgrammed cell deathDNA damageBiologymedicine.disease_causeBiochemistryChondrocyteEpigenesis Genetic03 medical and health sciencesChondrocytesOsteoarthritisAutophagymedicineAnimalsHumansMolecular Targeted TherapyEpigeneticsCellular SenescencePharmacologyAutophagyDNA MethylationCell biologyMicroRNAsOxidative Stress030104 developmental biologymedicine.anatomical_structureImmunologyReactive Oxygen SpeciesOxidative stressDNA DamageBiochemical Pharmacology
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Extracellular vesicles and redox modulation in aging

2019

Extracellular vesicles (EVs) are nowadays known to be mediators of cell-to-cell communication involved in physiological and pathological processes. The current expectation is their use as specific biomarkers and therapeutic tools due to their inner characteristics. However, several investigations still need to be done before we can use them in the clinic. First, their categorization is still under debate, although an accurate classification of EVs subtypes should be based on physical characteristics, biochemical composition or condition description of the cell of origin. Second, EVs carry lipids, proteins and nucleic acids that can induce epigenetic modifications on target cells. These carg…

0301 basic medicineSenescenceRedox modulationCell CommunicationOxidative phosphorylationBiologymedicine.disease_causeBiochemistryExtracellular vesiclesRedox statusCell biologyExtracellular Vesicles03 medical and health sciences030104 developmental biology0302 clinical medicinePhysiology (medical)medicineProspective StudiesEpigeneticsOxidation-Reduction030217 neurology & neurosurgeryBiogenesisOxidative stressFree Radical Biology and Medicine
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A Deep Learning Model for Epigenomic Studies

2016

Epigenetics is the study of heritable changes in gene expression that does not involve changes to the underlying DNA sequence, i.e. a change in phenotype not involved by a change in genotype. At least three main factor seems responsible for epigenetic change including DNA methylation, histone modification and non-coding RNA, each one sharing having the same property to affect the dynamic of the chromatin structure by acting on Nucleosomes posi- tion. A nucleosome is a DNA-histone complex, where around 150 base pairs of double-stranded DNA is wrapped. The role of nucleosomes is to pack the DNA into the nucleus of the Eukaryote cells, to form the Chromatin. Nucleosome positioning plays an imp…

0301 basic medicineSettore INF/01 - InformaticabiologyBase pairdeep learningGenomicsComputational biologyBioinformaticsChromatin03 medical and health sciences030104 developmental biologyHistoneclassificationDNA methylationbiology.proteinNucleosomeEpigeneticsnucleosome positioningEpigenomics2016 12th International Conference on Signal-Image Technology & Internet-Based Systems (SITIS)
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Recurrent Deep Neural Networks for Nucleosome Classification

2020

Nucleosomes are the fundamental repeating unit of chromatin. A nucleosome is an 8 histone proteins complex, in which approximately 147–150 pairs of DNA bases bind. Several biological studies have clearly stated that the regulation of cell type-specific gene activities are influenced by nucleosome positioning. Bioinformatic studies have improved those results showing proof of sequence specificity in nucleosomes’ DNA fragment. In this work, we present a recurrent neural network that uses nucleosome sequence features representation for their classification. In particular, we implement an architecture which stacks convolutional and long short-term memory layers, with the main purpose to avoid t…

0301 basic medicineSettore ING-INF/05 - Sistemi Di Elaborazione Delle InformazionibiologySettore INF/01 - InformaticaComputer scienceComputational biologyChromatin03 medical and health scienceschemistry.chemical_compound030104 developmental biologyHistoneRecurrent neural networkchemistryFragment (logic)biology.proteinNucleosomeNucleosome classification Epigenetic Deep learning networks Recurrent Neural NetworksGeneDNASequence (medicine)
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E4BP4/NFIL3 modulates the epigenetically repressed RAS effector RASSF8 function through histone methyltransferases

2018

RAS proteins are major human oncogenes, and most of the studies are focused on enzymatic RAS effectors. Recently, nonenzymatic RAS effectors (RASSF, RAS association domain family) have garnered special attention because of their tumor-suppressive properties in contrast to the oncogenic potential of the classical enzymatic RAS effectors. Whereas most members of RASSF family are deregulated by promoter hypermethylation, RASSF8 promoter remains unmethylated in many cancers but the mechanism(s) of its down-regulation remains unknown. Here, we unveil E4BP4 as a critical transcriptional modulator repressing RASSF8 expression through histone methyltransferases, G9a and SUV39H1. In line with these …

0301 basic medicineTumor suppressor geneBreast NeoplasmsBiologyBiochemistryEpigenesis Genetic03 medical and health sciences0302 clinical medicineHistocompatibility AntigensHistone methylationHumansEpigeneticsMolecular BiologySUV39H1EffectorTumor Suppressor ProteinsNFIL3Molecular Bases of DiseaseCell BiologyHistone-Lysine N-MethyltransferaseMethyltransferasesCell biologyNeoplasm ProteinsGene Expression Regulation NeoplasticRepressor Proteins030104 developmental biologyBasic-Leucine Zipper Transcription FactorsHEK293 Cells030220 oncology & carcinogenesisHistone methyltransferaseMCF-7 CellsFemaleFunction (biology)
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Combined HAT/EZH2 modulation leads to cancer-selective cell death

2018

Contains fulltext : 197351.pdf (Publisher’s version ) (Open Access) Epigenetic alterations have been associated with both pathogenesis and progression of cancer. By screening of library compounds, we identified a novel hybrid epi-drug MC2884, a HAT/EZH2 inhibitor, able to induce bona fide cancer-selective cell death in both solid and hematological cancers in vitro, ex vivo and in vivo xenograft models. Anticancer action was due to an epigenome modulation by H3K27me3, H3K27ac, H3K9/14ac decrease, and to caspase-dependent apoptosis induction. MC2884 triggered mitochondrial pathway apoptosis by up-regulation of cleaved-BID, and strong down-regulation of BCL2. Even aggressive models of cancer, …

0301 basic medicineacetylation; apoptosis; cancer; epigenetics; methylation; oncologyProgrammed cell death[SDV.CAN]Life Sciences [q-bio]/Cancer03 medical and health sciencesacetylation; apoptosis; cancer; epigenetics; methylationIn vivomedicinecancerMolecular Biologyacetylationepigeneticsbusiness.industryCancer; Epigenetics; Apoptosis; Acetylation; MethylationEZH2apoptosisApoptosiEpigeneticCancerEpigenomemedicine.disease3. Good healthLeukemia030104 developmental biologyOncologyApoptosisCancer researchmethylationbusinessEx vivoResearch PaperOncotarget
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An overview of the molecular mechanisms underlying development and progression of bicuspid aortic valve disease

2019

Bicuspid aortic valve (BAV) is a common congenital heart malformation frequently associated with the development of aortic valve diseases and severe aortopathy, such as aortic dilatation, aneurysm and dissection. To date, different genetic loci have been identified in syndromic and non- syndromic forms of BAV. Among these, genes involved in the regulation of extracellular matrix remodelling, epithelial to mesenchymal transition and nitric oxide metabolism appear to be the main contributors to BAV pathogenesis. However, no- single gene model explains BAV inheritance, suggesting that more factors are simultaneously involved. In this regard, characteristic epigenetic and immunological profiles…

0301 basic medicineaneurysm; aortic dilatation; aortic stenosis; aortopathy; bicuspid aortic valve; NOTCH1Aortic stenosibicuspid aortic valveHeart malformationAortic DiseasesHeart Valve Diseasesaortopathy610 Medicine & healthDisease030204 cardiovascular system & hematologyBioinformatics2705 Cardiology and Cardiovascular Medicine11459 Center for Molecular CardiologyPathogenesis03 medical and health sciences0302 clinical medicineAneurysmBicuspid aortic valveNOTCH1Bicuspid Aortic Valve Disease1312 Molecular BiologymedicineSettore MED/05 - Patologia ClinicaAnimalsHumansEpithelial–mesenchymal transitionEpigeneticsMolecular BiologyAortic dilatationbusiness.industryaortic stenosisaortic dilatationmedicine.disease030104 developmental biologyAortic ValveaneurysmDisease Progressioncardiovascular systemCardiology and Cardiovascular MedicinebusinessJournal of Molecular and Cellular Cardiology
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Co-regulation of paralog genes in the three-dimensional chromatin architecture.

2016

Paralog genes arise from gene duplication events during evolution, which often lead to similar proteins that cooperate in common pathways and in protein complexes. Consequently, paralogs show correlation in gene expression whereby the mechanisms of co-regulation remain unclear. In eukaryotes, genes are regulated in part by distal enhancer elements through looping interactions with gene promoters. These looping interactions can be measured by genome-wide chromatin conformation capture (Hi-C) experiments, which revealed self-interacting regions called topologically associating domains (TADs). We hypothesize that paralogs share common regulatory mechanisms to enable coordinated expression acco…

0301 basic medicineanimal structuresComputational biologyBiologyGenomeChromosome conformation capture03 medical and health sciencesMice0302 clinical medicineDogsGene DuplicationGene duplicationGeneticsAnimalsCluster AnalysisHumansPromoter Regions GeneticGeneChIA-PETGenomic organizationGeneticsRegulation of gene expressionGenomefungiGene regulation Chromatin and EpigeneticsComputational BiologyChromatin Assembly and DisassemblyBiological EvolutionChromatinChromatin030104 developmental biologyEnhancer Elements GeneticGene Expression Regulation030217 neurology & neurosurgeryNucleic acids research
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A new “sudden fright paradigm” to explore the role of (epi)genetic modulations of the DAT gene in fear-induced avoidance behavior

2020

Alterations in dopamine (DA) reuptake are involved in several psychiatric disorders whose symptoms can be investigated in knock out rats for the DA transporter (DAT-KO). Recent studies evidenced the role of epigenetic DAT modulation in depressive-like behavior. Accordingly, we used heterozygous (HET) rats born from both HET parents (termed MIX-HET), compared to HET rats born from WT-mother and KO-father (MAT-HET), implementing the role of maternal care on DAT modulation. We developed a "sudden fright" paradigm (based on dark-light test) to study reaction to fearful inputs in the DAT-KO, MAT-HET, MIX-HET, and WT groups. Rats could freely explore the whole 3-chambers apparatus; then, they wer…

0301 basic medicineanimal structuresEmotionsStimulus (physiology)Epigenesis GeneticReuptakechoice behavior03 medical and health sciencesBehavioral Neuroscience0302 clinical medicineDopamineDAT-KO ratAvoidance LearningGeneticsmedicineAnimalsFear conditioningEpigeneticsprefrontal cortex.Prefrontal cortexdopamine transporterDopamine transporterDopamine Plasma Membrane Transport ProteinsBehavior AnimalbiologyFearfear conditioningRatsDisease Models Animal030104 developmental biologyNeurologyAttention Deficit Disorder with Hyperactivitybiology.proteinSettore BIO/14 - Farmacologiaconditioned preferenceHistone deacetylaseNeuroscience030217 neurology & neurosurgerymedicine.drug
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