Search results for "Estat"

showing 10 items of 1304 documents

Breastfeeding Practices Influence the Breast Milk Microbiota Depending on Pre-Gestational Maternal BMI and Weight Gain over Pregnancy

2021

Breastfeeding is critical for adequate neonatal microbial and immune system development affecting neonate health outcomes in the short and long term. There is a great interest in ascertaining which are the maternal factors contributing to the milk microbiota and the potential relevance for the developing infant. Thus, our study aimed to characterize the effect of mixed and exclusive breastfeeding practices on the milk microbiota and to determine the impact of pre-pregnancy body mass index (BMI) and weight gain over pregnancy on its composition. Breast milk samples from 136 healthy women were collected within the first month post-partum and milk microbiota profiling was analyzed by 16S rRNA …

0301 basic medicineAdultbreastfeeding030106 microbiologyBreastfeedingBreastfeedingbody mass indexBreast milkHealth outcomesArticle03 medical and health sciencesfluids and secretionsEnvironmental healthmedicineHumansTX341-641Body mass indexBifidobacterium2. Zero hungerPregnancyNutrition and DieteticsbiologyBacteriaMilk Humanbusiness.industryNutrition. Foods and food supplyMicrobiotabiology.organism_classificationmedicine.diseaseGestational Weight Gain3. Good health030104 developmental biologyBreast FeedingGestationFemalemedicine.symptombusinessBody mass indexWeight gainFood ScienceNutrients
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Characterization of NO-Induced Nitrosative Status in Human Placenta from Pregnant Women with Gestational Diabetes Mellitus

2017

Dysregulation of NO production is implicated in pregnancy-related diseases, including gestational diabetes mellitus (GDM). The role of NO and its placental targets in GDM pregnancies has yet to be determined. S-Nitrosylation is the NO-derived posttranslational protein modification that can modulate biological functions by forming NO-derived complexes with longer half-life, termed S-nitrosothiol (SNO). Our aim was to examine the presence of endogenous S-nitrosylated proteins in cysteine residues in relation to antioxidant defense, apoptosis, and cellular signal transduction in placental tissue from control (n=8) and GDM (n=8) pregnancies. S-Nitrosylation was measured using the biotin-switch …

0301 basic medicineAgingendocrine system diseasesPlacentaNitric Oxide Synthase Type IIExpressionApoptosisBiochemistryBody Mass Index0302 clinical medicineNitric-oxidePregnancyMitogen-Activated Protein Kinase 1Mitogen-Activated Protein Kinase 3biologyCaspase 3lcsh:CytologyNitrosylationP38General MedicineCatalaseCaspase 9TrophoblastsGestational diabetesmedicine.anatomical_structureCatalase030220 oncology & carcinogenesisFemaleResearch ArticleAdultmedicine.medical_specialtyArticle SubjectNitrosationNitric OxidePathophysiology03 medical and health sciencesErk1/2Internal medicinePlacentamedicineHumanslcsh:QH573-671Protein kinase BPregnancyFetusNitratesS-NitrosothiolsCesarean SectionCell BiologyPeroxiredoxinsmedicine.diseaseProtein s-nitrosylationDiabetes Gestational030104 developmental biologyEndocrinologyOxidative stressCase-Control Studiesbiology.proteinPeroxiredoxinProto-Oncogene Proteins c-aktOxidative Medicine and Cellular Longevity
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Maternal and fetal genetic contribution to gestational weight gain

2018

Background: Clinical recommendations to limit gestational weight gain (GWG) imply high GWG is causally related to adverse outcomes in mother or offspring, but GWG is the sum of several inter-related complex phenotypes (maternal fat deposition and vascular expansion, placenta, amniotic fluid and fetal growth). Understanding the genetic contribution to GWG could help clarify the potential effect of its different components on maternal and offspring health. Here we explore the genetic contribution to total, early and late GWG. Participants and methods: A genome-wide association study was used to identify maternal and fetal variants contributing to GWG in up to 10 543 mothers and 16 317 offspri…

0301 basic medicineAmniotic fluidEpidemiologyEndocrinology Diabetes and MetabolismEmbaràsMedicine (miscellaneous)Genome-wide association studyBLOOD-PRESSUREType 2 diabetes030204 cardiovascular system & hematology/dk/atira/pure/core/keywords/icepCOMMON SNPSGenètica mèdica0302 clinical medicinePregnancyWeight managementOFFSPRING ADIPOSITYMass index11 Medical and Health Sciences2. Zero hunger0303 health sciencesNutrition and DieteticsObstetricsHERITABILITYMedical geneticsta3141ASSOCIATIONGestational Weight Gainddc:3. Good healthGestational diabetesCHILDREN ALSPACmedicine.anatomical_structurePREGNANCYOBESITYMENDELIAN RANDOMIZATIONGestationOriginal ArticleFemaleICEPmedicine.symptomLife Sciences & Biomedicine13 EducationTRAITSmedicine.medical_specialtyOffspringBirth weightPes corporalDevelopmentBiology03 medical and health sciencesEndocrinology & MetabolismFetusPlacentaInternal medicinemedicineJournal ArticleHumans030304 developmental biologyFetusPregnancyScience & TechnologyNutrition & Dieteticsbusiness.industryta3121Body weightmedicine.diseaseta3123BIRTH-WEIGHTBODY-MASS INDEX030104 developmental biologyEndocrinologybusinessBody mass indexWeight gainHUMAN HEIGHTGenome-Wide Association Study
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Immaturity for gestational age of microvasculature and placental barrier in term placentas with high weight

2017

Abstract Objective Villous immaturity for gestational age is a multifactorial developmental deviation associated with unexpected placental insufficiency, fetal hypoxia and term fetal death. In our previous work we have shown that immature CD15+/CD31+/CD34+ endothelial cells were an important indicator of placental villous immaturity and chronic insufficiency. The aim of this study was to perform a comparative analysis of CD15-marked immaturity in the vessel walls between normal and pathological term placentas of clinically and structurally heterogenous groups with normal, low and high weight. Study design 165 clinically normal and pathological placentas of gestational age 39–42 with normal …

0301 basic medicineCD31medicine.medical_specialtyTerm BirthPlacentaCD34Lewis X AntigenGestational AgePlacental insufficiency03 medical and health sciences0302 clinical medicineVasculogenesisImmunophenotypingPregnancyInternal medicinemedicineHumansPlacental villous immaturityFetus030219 obstetrics & reproductive medicinebusiness.industryObstetrics and GynecologyGestational ageOrgan SizePlacental Insufficiencymedicine.disease030104 developmental biologyEndocrinologyReproductive MedicineMicrovesselsImmunologyFemaleEndothelium VascularbusinessEuropean Journal of Obstetrics & Gynecology and Reproductive Biology
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BIOMARKERS OF CORONARY MICROVASCULAR DYSFUNCTION IN PATIENTS WITH MICROVASCULAR ANGINA: A NARRATIVE REVIEW

2021

The current gold standard for diagnosis of coronary microvascular dysfunction (CMD) in the absence of myocardial diseases, whose clinical manifestation is microvascular angina (MVA), is reactivity testing using adenosine or acetylcholine during coronary angiography. This invasive test can be difficult to perform, expensive, and harmful. The identification of easily obtainable blood biomarkers which reflect the pathophysiology of CMD, characterized by high reliability, precision, accuracy, and accessibility may reduce risks and costs related to invasive procedures and even facilitate the screening and diagnosis of CMD. In this review, we summarized the results of several studies that have i…

0301 basic medicineCoronary angiographymedicine.medical_specialtyClinical manifestationCoronary Artery Disease030204 cardiovascular system & hematology03 medical and health sciences0302 clinical medicinemicrovascular dysfunction (CMD) oxidative stress inflammation endothelial dysfunction microvascular angina (MVA)Internal medicineCoronary CirculationMedicineHumansIn patientEndothelial dysfunctionMicrovascular Anginabusiness.industryMicrocirculationMicrovascular anginaReproducibility of ResultsGold standard (test)medicine.disease030104 developmental biologyCardiologyNarrative reviewCardiology and Cardiovascular MedicinebusinessBiomarkersReactivity Testing
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Mothering under the influence: How perinatal drugs of abuse alter the mother-infant interaction

2018

AbstractAlthough drug-abusing women try to moderate their drug and alcohol use during pregnancy, they often relapse at a time when childcare needs are high and maternal bonding is critical to an infant’s development. In the clinical setting, the search for the neural basis of drug-induced caregiving deficits is complex due to several intervening variables. Rather, the preclinical studies that control for drug dose and regimen, as well as for gestational and postpartum environment, allow a precise determination of the effects of drugs on maternal behaviour. Given the relevance of the issue, this review will gather reports on the phenotypic correlates of maternal behaviour in preclinical stud…

0301 basic medicineDrugMalecannabiSubstance-Related Disordersmedia_common.quotation_subjectMother infantcocainePrenatal Exposure Delayed EffectMaternal behaviourNicotine03 medical and health sciences0302 clinical medicinePregnancymedicineHumansMaternal Behaviormedia_commonPregnancymaternal behaviourbiologybusiness.industryalcoholGeneral Neurosciencestimulantperinatal substance usemedicine.diseasebiology.organism_classificationSubstance-Related DisorderMother-Child RelationsopiateRegimen030104 developmental biologyPrenatal Exposure Delayed EffectsSettore BIO/14 - FarmacologiaGestationFemaleCannabisbusiness030217 neurology & neurosurgeryClinical psychologymedicine.drugnicotineHuman
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Does Embryo Culture Medium Influence the Health and Development of Children Born after In Vitro Fertilization?

2016

International audience; In animal studies, extensive data revealed the influence of culture medium on embryonic development, foetal growth and the behaviour of offspring. However, this impact has never been investigated in humans. For the first time, we investigated in depth the effects of embryo culture media on health, growth and development of infants conceived by In Vitro Fertilization until the age of 5 years old. This single-centre cohort study was based on an earlier randomized study. During six months, in vitro fertilization attempts (No. 371) were randomized according to two media (Single Step Medium—SSM group) or Global medium (Global group). This randomized study was stopped prem…

0301 basic medicineEmbryologyPediatricsMaternal HealthHealth Statusmedicine.medical_treatmentlcsh:MedicineSocial SciencesGrowthPediatricslaw.inventionCohort StudiesFamiliesLabor and Delivery[SCCO]Cognitive science0302 clinical medicineRandomized controlled triallawMedicine and Health SciencesPsychologyTermination of Pregnancylcsh:ScienceChildren[ SDV.MHEP.GEO ] Life Sciences [q-bio]/Human health and pathology/Gynecology and obstetricsLanguage030219 obstetrics & reproductive medicineMultidisciplinaryObstetricsObstetrics and GynecologyLaboratory EquipmentChild PreschoolEngineering and TechnologyGestationEpigeneticsBiological Cultures[ SCCO ] Cognitive scienceResearch ArticleCohort studymedicine.medical_specialtyOffspringGross motor skillEquipmentFertilization in Vitro[SDV.MHEP.GEO]Life Sciences [q-bio]/Human health and pathology/Gynecology and obstetricsResearch and Analysis Methods03 medical and health sciencesGeneticsmedicineHumansPregnancyIn vitro fertilisationbusiness.industrylcsh:REmbryosCognitive PsychologyInfant NewbornBiology and Life SciencesInfantEmbryo culturemedicine.diseaseCulture Media030104 developmental biologyAge GroupsPeople and PlacesBirthWomen's HealthCognitive Sciencelcsh:QPopulation GroupingsbusinessDevelopmental BiologyNeuroscience
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Parental risk factors of anorectal malformations: Analysis with a regional population-based control group

2015

BACKGROUND Adequate evidence on environmental risk factors for anorectal malformations (ARMs) is very limited. We assessed maternal body weight and several prenatal exposures of the parents to tobacco, pregestational diabetes, chronic cardiovascular and respiratory diseases, periconceptional folic acid and multivitamin intake. METHODS Data from the German Network for Congenital Uro-REctal malformations (CURE-Net) were compared with data from the Malformation Monitoring Centre Saxony-Anhalt of the Otto-von-Guericke University in Magdeburg, Germany. Controls were matched to cases by gender and birth year of the child. Crude and adjusted odds ratios (95% confidence intervals) were calculated f…

0301 basic medicineEmbryologyPediatricsmedicine.medical_specialtyPregnancybusiness.industryOffspringCase-control studyGestational ageGeneral MedicineOdds ratio030105 genetics & heredityLogistic regressionmedicine.diseaseConfidence interval03 medical and health sciences0302 clinical medicine030225 pediatricsPediatrics Perinatology and Child HealthMedicinebusinessBirth YearDevelopmental BiologyBirth Defects Research Part A: Clinical and Molecular Teratology
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Autism is a prenatal disorder: Evidence from late gestation brain overgrowth

2018

This retrospective study aimed to specify the critical period for atypical brain development in individuals with autism spectrum disorder (ASD) using prenatal and postnatal head growth parameters. The sample consisted of 80 Caucasian, unrelated, idiopathic patients with ASD born after 1995. Fetal ultrasound parameters (head circumference [HC], abdominal circumference, and femur length) were obtained during the second and third trimesters of gestation. HC at birth and postnatal parameters at 12 and 24 months of age were also collected. Head overgrowth, assessed by HC, was highlighted during the second (20-26 weeks of amenorrhea) and third (28-36 weeks of amenorrhea) trimesters. Normal growth…

0301 basic medicineFetusPediatricsmedicine.medical_specialtybusiness.industryGeneral Neurosciencemedicine.disease03 medical and health sciences030104 developmental biology0302 clinical medicineNeurodevelopmental disorderAutism spectrum disorderIn uteromedicineGestationAutismAmenorrheaNeurology (clinical)medicine.symptombusinessPathological030217 neurology & neurosurgeryGenetics (clinical)Autism Research
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Neonatal hyperinsulinemic hypoglycemia: case report of kabuki syndrome due to a novel KMT2D splicing-site mutation

2020

Abstract Background Persistent neonatal hypoglycemia, owing to the possibility of severe neurodevelopmental consequences, is a leading cause of neonatal care admission. Hyperinsulinemic hypoglycemia is often resistant to dextrose infusion and needs rapid diagnosis and treatment. Several congenital conditions, from single gene defects to genetic syndromes should be considered in the diagnostic approach. Kabuki syndrome type 1 (MIM# 147920) and Kabuki syndrome type 2 (MIM# 300867), can be associated with neonatal hyperinsulinemic hypoglycemia. Patient presentation We report a female Italian (Sicilian) child, born preterm at 35 weeks gestation, with persistent hypoglycemia. Peculiar facial dys…

0301 basic medicineHeterozygotePediatricsmedicine.medical_specialtyFacial dysmorphismNeonatal hypotoniaCase ReportHypoglycemiamedicine.disease_causeDiagnosis DifferentialNervous system malformation03 medical and health sciences0302 clinical medicineHyperinsulinismmedicineHumansAbnormalities MultipleHyperinsulinemic hypoglycemiaPathologicalbusiness.industryNeonatal hypoglycemiaInfant Newbornlcsh:RJ1-570lcsh:Pediatricsmedicine.diseaseHematologic DiseasesNeoplasm ProteinsDNA-Binding ProteinsPhenotype030104 developmental biologyNeonatal hypotoniaItalyVestibular DiseasesFaceMutationGestationFemalebusinessHyperinsulinismKabuki syndromeInfant PrematureNeonatal hypoglycemia030217 neurology & neurosurgeryItalian Journal of Pediatrics
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