Search results for "Estrogen"

showing 10 items of 530 documents

Body Weight, Physical Activity, and Risk of Cancer in Lynch Syndrome

2021

Simple Summary Lifestyle modifies cancer risk in the general public. How lifestyle modifies cancer risk in individuals carrying the inherited pathogenic gene variants in DNA mismatch repair genes (Lynch syndrome) remains understudied. We conducted a retrospective study with cancer register data to investigate associations between body weight, physical activity, and cancer risk among Finnish Lynch syndrome carriers (n = 465, 54% women). The results of our study indicated that longitudinal weight gain increases cancer risk, whereas being highly physically active during adulthood could decrease cancer risk in men. Further, women were observed to be less prone to lifestyle-related risk factors …

elintavatperinnölliset tauditMENOPAUSElifestyleMUTATIONS3122 Cancershereditary non-polyposis colorectal cancerylipainoEXERCISEsuolistosyövätriskitekijätlcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogenslcsh:RC254-282ArticleCOLORECTAL-CANCERESTROGENADIPOSE-TISSUEMASS INDEXAGEDEFINED FAMILIAL RISKepidemiologyepidemiologiaLynchin oireyhtymäfyysinen aktiivisuusASSOCIATIONSCancers
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RASSF1A inhibits estrogen receptor alpha expression and estrogen-independent signalling: implications for breast cancer development

2012

The Ras association domain family 1 isoform A (RASSF1A) is a tumor suppressor whose inactivation is implicated in the development of many human cancers, including breast carcinomas. Little is known about the tumor-suppressive function of RASSF1A in breast tissue and whether its inactivation is mechanistically involved in the initiation and progression of breast tumors. Here, we show that RASSF1A inhibits breast cancer growth in vivo, and suppresses estrogen receptor (ERα) expression and function. Reconstitution of RASSF1A in MCF7 cells led to decreased ERα levels and reduced sensitivity to estrogen (E2). Concomitantly, we observed decreased expression of Id1 as well as the E2-responsive gen…

endocrine systemCancer Researchmedicine.medical_specialtyCell SurvivalGene ExpressionEstrogen receptorApoptosisBreast NeoplasmsCell Cycle ProteinsMice SCIDBiologyMiceBreast cancerDownregulation and upregulationMice Inbred NODInternal medicineGeneticsmedicineAnimalsHumansFulvestrantMolecular BiologyCellular SenescenceCell ProliferationRegulation of gene expressionEstradiolFulvestrantTumor Suppressor ProteinsEstrogen AntagonistsEstrogen Receptor alphaCancerEstrogensCell Cycle Checkpointsmedicine.diseaseGene Expression Regulation NeoplasticEndocrinologyProteolysisMCF-7 CellsCancer researchFemaleEctopic expressionEstrogen receptor alphaNeoplasm TransplantationSignal Transductionmedicine.drugOncogene
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A mutation in the second intracellular loop of the pituitary adenylate cyclase activating polypeptide type I receptor confers constitutive receptor a…

2000

AbstractThe pituitary adenylate cyclase activating polypeptide (PACAP) type I receptor belongs to the glucagon/secretin/vasoactive intestinal polypeptide (VIP) receptor family. We mutated and deleted an amino acid residue (E261) which is located within the second intracellular loop of the rat PACAP type I receptor and which is highly conserved among the receptor family. The wild-type receptor and the mutant receptors were efficiently expressed at the surface of COS-7 cells at nearly the same level and revealed the same high affinity for the agonist PACAP-27. The cAMP contents of COS cells transfected with the E261A, E261Q, and the deletion mutant receptor were 4.6-, 5.7-, and 6.7-fold highe…

endocrine systemGrowth-hormone-releasing hormone receptorMolecular Sequence DataReceptors Pituitary Adenylate Cyclase-Activating PolypeptideBiophysicsGlutamic AcidSignal transductionTransfectionBiochemistryBeta-1 adrenergic receptorConstitutive activityStructural BiologycAMPCyclic AMPGeneticsEnzyme-linked receptorAnimals5-HT5A receptorAmino Acid SequenceReceptors Pituitary HormoneMolecular BiologySequence DeletionPeptide hormone receptorSite-directed mutagenesisPituitary adenylate cyclase activating polypeptideChemistryLiver receptor homolog-1Cell BiologyMolecular biologyRatsInterleukin-21 receptorCOS CellsMutagenesis Site-DirectedEstrogen-related receptor gammaSequence AlignmentGlucagon receptor familyhormones hormone substitutes and hormone antagonistsAdenylyl CyclasesReceptors Pituitary Adenylate Cyclase-Activating Polypeptide Type IFEBS Letters
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Daidzein has neuroprotective effects through ligand-binding-independent PPARγ activation.

2011

Phytoestrogens are a group of plant-derived compounds that include mainly isoflavones like daidzein. Phytoestrogens prevent neuronal damage and improve outcome in experimental stroke; however, the mechanisms of this neuroprotective action have not been fully elucidated. In this context, it has been postulated that phytoestrogens might activate the peroxisome proliferator-activated receptor-γ (PPARγ), which exerts neuroprotective effects in several settings. The aim of this study was to determine whether the phytoestrogen daidzein elicits beneficial actions in neuronal cells by mechanisms involving activation of PPARγ. Our results show that daidzein (0.05-5 μM) decreases cell death induced b…

endocrine systemmedicine.drug_classPyridinesPeroxisome proliferator-activated receptorPharmacologyLigandsNeuroprotectionCellular and Molecular Neurosciencechemistry.chemical_compoundmedicineSynaptic vesicle recyclingAnimalsReceptorCells Culturedchemistry.chemical_classificationNeuronsDaidzeinfood and beveragesCell BiologyIsoflavonesReceptor antagonistIsoflavonesRatsOxygenPPAR gammaGlucoseNeuroprotective AgentschemistryBenzamidesPhytoestrogensNeurochemistry international
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Targeting the vascular endothelial growth factor system to prevent ovarian hyperstimulation syndrome

2008

BACKGROUND: Ovarian hyperstimulation syndrome (OHSS) typically occurs when ovaries are primed with FSH/ LH and subsequently exposed to hCG. The ultimate pathophysiological step underlying this clinical picture is increased vascular permeability (VP). METHODS: A search of the literature was carried out using PubMed and the authors’ files. RESULTS: In rodents and humans, the expression of vascular endothelial growth factor (VEGF) and VEGF receptor 2 (VEGFR-2) mRNA increases during ovarian stimulation. With the administration of hCG, the expression of each rises to a maximum. Expression of VEGF/VEGFR-2 mRNAs correlates with enhanced VP, with both peaking 48 h following an injection of hCG. Imm…

endocrine systemmedicine.medical_specialtyCabergolineIndolesOvarian hyperstimulation syndromeVascular permeabilityBiologyChorionic GonadotropinDopamine agonistCapillary PermeabilityOvarian Hyperstimulation Syndromechemistry.chemical_compoundInternal medicineCabergolinemedicineAnimalsHumansPyrrolesErgolinesPhosphorylationCells CulturedClinical Trials as TopicEstradiolVascular Endothelial Growth FactorsObstetrics and GynecologyEstrogensKinase insert domain receptormedicine.diseaseRatsVascular endothelial growth factorActin CytoskeletonVascular endothelial growth factor Amedicine.anatomical_structureEndocrinologyReproductive MedicinechemistryDopamine AgonistsReproductive Control AgentsFemaleCorpus luteumSignal Transductionmedicine.drugHuman Reproduction Update
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The non-aromatizable androgen dihydrotestosterone (DHT) facilitates sexual behavior in ovariectomized female rats primed with estradiol

2020

Abstract It is still unclear whether Testosterone (T) increases sexual desire through a stimulation of the androgen receptor in relevant brain regions or through its conversion to estrogens. The aim of this study was to clarify the mechanisms of T facilitation of female sexual desire by assessing the effect of a non-aromatizable androgen (Dihydrotestosterone, DHT) in a validated animal model. Ovariectomized (OVX) Long-Evans rats were treated with oil (O) + O, 10 mcg Estradiol Benzoate (EB) + O, 10 mcg EB + 500 mcg Progesterone (P), O + 500 mcg DHT or 10 mcg EB + 500 mcg DHT (n = 12 per group). EB was administered 48 h, while P and DHT 4 h, prior to 4 sexual behavioral testing sessions in bi…

endocrine systemmedicine.medical_specialtyLordosismedicine.drug_classEndocrinology Diabetes and MetabolismOvariectomyReceptivitySolicitationSettore BIO/09 - Fisiologia03 medical and health scienceschemistry.chemical_compoundSexual Behavior Animal0302 clinical medicineEndocrinologyInternal medicineSexual desiremedicineAnimalsRats Long-EvansBiological PsychiatryTestosteroneProgesteroneEstradiolEndocrine and Autonomic SystemsChemistryEstrogensDihydrotestosteroneAndrogenmedicine.diseasePreclinical030227 psychiatryRatsAndrogen receptorPsychiatry and Mental healthEndocrinologyDihydrotestosteroneOvariectomized ratEstradiol benzoateAndrogenssexual behavior female ratRatFemaleProgestinshormones hormone substitutes and hormone antagonists030217 neurology & neurosurgerymedicine.drug
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Ovarian size and blood flow in women with polycystic ovary syndrome (PCOS) and their correlations with some endocrine parameters

2005

Objective: To determine how common polycystic ovarian morphology may be in women given the clinical diagnosis of polycystic ovary syndrome (PCOS) based on chronic anovulation and hyperandrogenism and whether certain hormonal factors correlate with ovarian morphology and blood flow. Design: Prospective study. Setting: Two academic endocrinology centers in Italy. Patient(s): Three hundred twenty-six women with PCOS and 50 age-matched and weight-matched ovulatory women. Intervention(s): Ultrasound assessment of ovarian morphology in patients and controls and ovarian blood flow and fasting hormone levels in a subset of 50 patients and matched controls. Main Outcome Measure(s): Ovarian morpholog…

endocrine systemmedicine.medical_specialtyendocrine system diseasesmedicine.drug_classPolycystic ovary syndrome ovarian size blood flow insulin testosterone inhibin Bmedicine.medical_treatmentHemodynamicsOvaryBiologyStatistics NonparametricAnovulationInsulin resistanceEndocrine GlandsInternal medicinemedicineHumansProspective StudiesGonadal Steroid Hormonesendocrine parametersAnalysis of VarianceInsulinOvaryHyperandrogenismObstetrics and GynecologyOrgan Sizemedicine.diseasePolycystic ovaryfemale genital diseases and pregnancy complicationsmedicine.anatomical_structureEndocrinologyReproductive MedicineRegional Blood FlowEstrogenpolycystic ovary syndromeFemaleHyperandrogenismpolycystic ovary syndrome; endocrine parametersAnovulation
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Análisis del perfil de expresión de microRNA en células endoteliales humanas expuestas a estradiol. Papel del miR-30b-5p en la regulación de la expre…

2017

El endotelio es un tejido dinámico que juega un papel fundamental en el mantenimiento de la función vascular. Está localizado en la capa interna de los vasos sanguíneos, linfáticos y corazón, y resulta clave para las diversas funciones del sistema cardiovascular, tales como la hemostasia, la regulación del tono vascular, la respuesta inflamatoria, la regulación de la permeabilidad vascular y la angiogénesis. El endotelio actúa como una diana de los estrógenos y expresa ambos tipos de receptores de estrógenos (ER): los nucleares clásicos ERα and ERβ, que actúan como factores de transcripción, y el receptor acoplado a proteína G (GPER). Mientras que la unión de estrógenos a ERα y ERβ modula l…

endotheliumendothelinmiRNAestrogens
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Estrogen Receptor and Vascular Aging.

2021

Cardiovascular diseases remain an age-related pathology in both men and women. These pathologies are 3-fold more frequent in men than in women before menopause, although this difference progressively decreases after menopause. The vasculoprotective role of estrogens are well established before menopause, but the consequences of their abrupt decline on the cardiovascular risk at menopause remain debated. In this review, we will attempt to summarize the main clinical and experimental studies reporting the protective effects of estrogens against cardiovascular diseases, with a particular focus on atherosclerosis, and the impact of aging and estrogen deprivation on their endothelial actions. Th…

endotheliumvascular agingGeriatricsestradiolRC952-954.6menopauseGeneral Medicineatherosclerosisestrogen receptorFrontiers in aging
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Effects of combined hormone replacement therapy or its effective agents on the IGF-1 pathway in skeletal muscle.

2010

Objectives To investigate the effects of combined hormone replacement therapy (HRT) and its effective agents on the IGF-1 signaling pathway. Design and methods To examine the effects of HRT on skeletal muscle in vivo, we utilized pre- and post-intervention samples from a randomized double blinded trial with 50–57-year-old women. The intervention included the year-long use of either HRT preparation (2 mg 17β-estradiol, E2; 1 mg norethisterone acetate, NETA, n = 10) or placebo (CO, n = 9). Microarray technology and quantitative PCR (qPCR) were used to study the expression of insulin-like growth factor I (IGF-1) and its splice variants as well as IGF-1 receptor, Akt1, mTOR, FOXO1, FOXO3, atrog…

estradioliTranscription GeneticEndocrinology Diabetes and MetabolismMuscle Fibers SkeletalEstrogen receptorpostmenopausal womenMuscle ProteinsFOXO1Receptor IGF Type 10302 clinical medicineEndocrinologyProtein IsoformsTestosteroneInsulin-Like Growth Factor IReceptorRandomized Controlled Trials as Topic0303 health sciencesEstradiolMyogenesisForkhead Box Protein O1TOR Serine-Threonine KinasesEstrogen Replacement TherapyForkhead Box Protein O3Forkhead Transcription FactorsMiddle Agedmedicine.anatomical_structureReceptors EstrogenReceptors AndrogenFemalemedicine.medical_specialtynorethisterone acetate030209 endocrinology & metabolismBiologypostmenopausaalinen nainen03 medical and health sciencesInternal medicinemedicineHumansnoretisteroniasetaattiluurankolihasskeletal muscleMuscle SkeletalProtein kinase BPI3K/AKT/mTOR pathway030304 developmental biologyhormonikorvaushoitoSKP Cullin F-Box Protein LigasesSkeletal muscleAndrogen receptorNorethindrone AcetateEndocrinologyHormone replacement therapyIGF-1 signalointiNorethindroneIGF-1 signalingProto-Oncogene Proteins c-aktGrowth hormoneIGF research : official journal of the Growth Hormone Research Society and the International IGF Research Society
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