Search results for "EuP"

showing 10 items of 423 documents

Are the effects of the antidepressants amitriptyline, maprotiline, and fluoxetine on inhibitory avoidance state-dependent?

2005

Abstract State-dependent learning (SDL) is a phenomenon in which the retrieval of newly acquired information is possible if the subject is in the same physiological state as during the encoding phase. SDL makes it possible to separate the effects of drugs per se on learning from the effects due to changes in drug state during the task. The present work was designed to investigate whether the antidepressants amitriptyline (30 mg/kg), maprotiline (25 mg/kg), and fluoxetine (15 mg/kg) produce SDL of the inhibitory avoidance conditioning in male and female CD1 mice. In three separate experiments, independent groups were used for each pharmacological treatment and for each sex using a 2 × 2 expe…

Malemedicine.medical_specialtyAmitriptylinePharmacologyMiceBehavioral Neurosciencechemistry.chemical_compoundSex FactorsFluoxetineAvoidance LearningmedicineAnimalsAmitriptylineNeurotransmitterPsychiatryMaprotilineFluoxetineBehavior AnimalAntidepressive AgentsInhibition PsychologicalMaprotilinechemistryFacilitationConditioningFemaleSerotoninReuptake inhibitorPsychologymedicine.drugBehavioural Brain Research
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Non-competitive inhibition of clomipramineN-demethylation by fluvoxamine

1995

The selective serotonin reuptake inhibitor fluvoxamine interferes with the metabolism of tricyclic antidepressants. The present investigation was set out to characterize these interactions in vitro using rat liver microsomes and in vivo by analysing levels of clomipramine and metabolites in sera of depressed patients treated concomitantly with fluvoxamine and clomipramine. Clomipramine was N-demethylated and hydroxylated in vitro by microsomes to N-desmethyl-clomipramine, 8-hydroxyclomipramine, and 10-hydroxyclomipramine. Kinetic analyses revealed Km values of 6.2 microM for N-demethylation and 1.2 microM for 8-hydroxylation. Fluvoxamine was a non-competitive inhibitor for N-demethylation w…

Malemedicine.medical_specialtyClomipramineSerotonin reuptake inhibitorFluvoxamineIn Vitro TechniquesPharmacologyHydroxylationRats Sprague-DawleyPharmacokineticsInternal medicinemedicineAnimalsHumansPharmacologychemistry.chemical_classificationDrug interactionRatsEndocrinologychemistryDealkylationFluvoxamineDepression ChemicalClomipramineMicrosomes LiverSerotoninReuptake inhibitormedicine.drugTricyclicPsychopharmacology
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Nicotine receptors do not modulate the 3H-noradrenaline release from the isolated rat heart evoked by sympathetic nerve stimulation.

1982

Isolated rat hearts with the right sympathetic nerves attached were perfused at a constant flow rate of 7 ml/min with Tyrode's solution. (-)-3H-Noradrenaline (final concentration 10–13.9 nM) was infused for 10 min to label the noradrenaline stores. After wash-out the sympathetic nerves were stimulated electrically (3 Hz, 180 impulses, 1 ms, 20–30 mA) three times (S1–S3) at intervals of 15 min. 3H-Noradrenaline and its metabolites were determined by liquid scintillation counting according to Graefe et al. (1973). Both, nicotine 50 μM and p-aminophenethyltrimethylammonium (PAPETA) 30 μM, enhanced the 3H-noradrenaline overflow in the absence of nerve stimulation. The effect of PAPETA was bipha…

Malemedicine.medical_specialtyNicotineSympathetic Nervous SystemSympathetic nerveStimulationIn Vitro TechniquesReceptors NicotinicTritiumReuptakeMethoxyhydroxyphenylglycol3h noradrenalineNicotinechemistry.chemical_compoundNorepinephrineInternal medicinemedicineAnimalsReceptors CholinergicReceptorPharmacologyNeuronsMyocardiumHeartRats Inbred StrainsGeneral MedicineRat heartElectric StimulationRatsQuaternary Ammonium CompoundsEndocrinologychemistryMandelic AcidsHexamethoniumFemalemedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
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Autoinhibition of noradrenaline release from the rat heart as a function of the biophase concentration. Effects of exogenous alpha-adrenoceptor agoni…

1984

1. Rat isolated perfused hearts with the right sympathetic nerves intact were loaded with 3H-(-)-noradrenaline. The nerves were stimulated with trains of 180 pulses at 3 Hz and at 10 min intervals. The overflow of 3H-noradrenaline and 3H-metabolites was determined by liquid scintillation spectrometry. 2. Clonidine (IC50 17 nM), oxymetazoline (IC50 63 nM), and α-methylnoradrenaline (apparent IC50 35 nM, determined in the presence of cocaine and propranolol) decreased the stimulation-evoked overflow of 3H-noradrenaline by 26, 49, and 78%, respectively, but not methoxamine up to 100 μM (propranolol present). Oxymetazoline and α-methyl-noradrenaline did not cause desensitization of the presynap…

Malemedicine.medical_specialtyOxymetazolinePropranololIn Vitro TechniquesMethoxamineReuptakeFeedbackchemistry.chemical_compoundNorepinephrinePhentolamineCocaineInternal medicinemedicineAnimalsNeurotransmitterPhentolaminePharmacologyMyocardiumYohimbineRats Inbred StrainsGeneral MedicineElectric StimulationYohimbineRatsPerfusionEndocrinologychemistryPerfusionAdrenergic alpha-Agonistsmedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
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Effects of oxotremorine and physostigmine on the inhibitory avoidance impairment produced by amitriptyline in male and female mice.

2009

We have previously observed that amitriptyline and other antidepressants produce impairing effects on inhibitory avoidance (also called passive avoidance) in mice of both sexes. In the present study we investigated the involvement of the cholinergic system in the inhibitory avoidance impairment produced by acute amitriptyline in male and female CD1 mice. For this purpose, the effects on said task of acute i.p. administration of several doses of amitriptyline, either alone or in combination with the cholinergic agonists oxotremorine and physostigmine, were evaluated. Pre-training administration of 5, 7.5, 10 or 15 mg/kg of amitriptyline produced a significant impairment of inhibitory avoidan…

Malemedicine.medical_specialtyPhysostigmineTime FactorsAmitriptylinePhysostigmineMice Inbred StrainsPharmacologyAntidepressive Agents TricyclicCholinergic AgonistsBehavioral Neurosciencechemistry.chemical_compoundMiceRandom AllocationInternal medicineOxotremorineAvoidance LearningMedicineAnimalsAmitriptylineNeurotransmitterCholinesteraseSex Characteristicsbiologybusiness.industryLearning DisabilitiesOxotremorineEndocrinologychemistrybiology.proteinAntidepressantCholinergicFemalebusinessReuptake inhibitormedicine.drugBehavioural brain research
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Effects of co-administration of amitriptyline and fluoxetine on inhibitory avoidance in mice

2010

We have previously observed that, while the impairing effects of amitriptyline on inhibitory avoidance in mice are consistently observed, those of acute fluoxetine are negligible. Two experiments were designed to investigate whether a regular dose of fluoxetine potentiates the effect of a low dose of amitriptyline that is ineffective when administered alone. Male and female CD1 mice were administered i.p. 30 min before training, as follows. In the first experiment, they were injected with saline, one of three doses of amitriptyline (2.5, 5, 10 mg/kg), one dose of fluoxetine (15 mg/kg), or a combination of amitriptyline (2.5 mg/kg) and fluoxetine (15 mg/kg). In the second experiment, the mic…

Malemedicine.medical_specialtyRatónAmitriptylinemedicine.medical_treatmentMice Inbred StrainsPharmacologyMiceBehavioral Neurosciencechemistry.chemical_compoundSex FactorsFluoxetineInternal medicineAvoidance LearningmedicineAnimalsAmitriptylineNeurotransmitterSalineFluoxetineDose-Response Relationship Drugbusiness.industryDrug SynergismAntidepressive AgentsEndocrinologychemistryCatecholamineFemaleSerotoninReuptake inhibitorbusinessmedicine.drugBehavioural Brain Research
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Association Between Citalopram Serum Levels and Clinical Improvement of Patients With Major Depression

2011

Imaging studies have shown that serum concentrations of the selective serotonin reuptake inhibitor citalopram correlate with serotonin transporter (5-HTT) occupancy in vivo. In patients with major depressive disorders treated with citalopram, 80% 5-HTT occupancy was considered to be necessary for maximal therapeutic effects, which requires citalopram serum concentrations of at least 50 ng/mL. The aim of this study was to compare treatment outcome in patients with citalopram serum concentrations greater than and less than 50 ng/mL after 7 days of treatment. This study included inpatients with acute major depressive disorder according to International Classification of Disease, 10th Revision …

Malemedicine.medical_specialtySerotonin reuptake inhibitorCitalopramCitalopramSeverity of Illness Indexbehavioral disciplines and activitiesGastroenterologyInternal medicinemental disordersSeverity of illnessmedicineHumansPharmacology (medical)Adverse effectDepressive Disorder Majormedicine.diagnostic_testTherapeutic effectLength of StayMiddle Agedmedicine.diseasePsychiatry and Mental healthTherapeutic drug monitoringAnesthesiaMajor depressive disorderFemaleDrug MonitoringReuptake inhibitorPsychologySelective Serotonin Reuptake Inhibitorsmedicine.drugJournal of Clinical Psychopharmacology
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Astroglial plasticity in the hippocampus is affected by chronic psychosocial stress and concomitant fluoxetine treatment.

2006

Analysis of post-mortem tissue from patients with affective disorders has revealed a decreased number of glial cells in several brain areas. Here, we examined whether long-term psychosocial stress influences the number and morphology of hippocampal astrocytes in an animal model with high validity for research on the pathophysiology of major depression. Adult male tree shrews were submitted to 5 weeks of psychosocial stress, after which immunocytochemical and quantitative stereological techniques were used to estimate the total number and somal volume of glial fibrillary acidic protein-positive astrocytes in the hippocampal formation. Stress significantly decreased both the number (-25%) and…

Malemedicine.medical_specialtySerotonin reuptake inhibitorHippocampusHippocampal formationHippocampus03 medical and health scienceschemistry.chemical_compound0302 clinical medicineInternal medicineFluoxetinemedicineAnimalsNeurotransmitter030304 developmental biologyPharmacologyTupaia0303 health sciencesFluoxetineNeuronal PlasticityPsychiatry and Mental healthEndocrinologymedicine.anatomical_structurechemistryAstrocytesChronic DiseaseAntidepressantNeurogliaPsychologyNeuroscienceNeuroglia030217 neurology & neurosurgeryStress PsychologicalAstrocytemedicine.drugNeuropsychopharmacology : official publication of the American College of Neuropsychopharmacology
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Serum concentrations of hydroxybupropion for dose optimization of depressed patients treated with bupropion.

2014

Background Bupropion is a dopamine and norepinephrine reuptake inhibitor approved for the treatment of depression and smoking cessation. According to the recently published reviews, it is a candidate for therapeutic drug monitoring (TDM) to improve therapeutic outcomes and reduce risks of intolerability or intoxication. In practice, however, the use of TDM is limited due to the chemical instability of bupropion. This investigation sought to determine if the major, active, and chemically stable metabolite 4-hydroxybupropion is a suitable measure to guide antidepressant drug therapy with bupropion. Methods 4-Hydroxybupropion serum levels were measured using a newly developed and validated hig…

Malemedicine.medical_specialtyUrologyNorepinephrine reuptake inhibitorPharmacokineticsmedicineHumansPharmacology (medical)BupropionChromatography High Pressure LiquidRetrospective StudiesPharmacologyBupropionDepressive Disordermedicine.diagnostic_testbusiness.industryTherapeutic effectHydroxybupropionMiddle AgedTherapeutic drug monitoringArea Under CurveClinical Global ImpressionAntidepressantAntidepressive Agents Second-GenerationFemalebusinessmedicine.drugHalf-LifeTherapeutic drug monitoring
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Prolactin secretion before, during, and after chronic gonadotropin-releasing hormone agonist treatments in children.

2005

Objective To examine the effect of long-term administration of GnRH agonists (GnRHa) on PRL secretion in children affected by central precocious puberty (CPP) and growth hormone deficiency (GHD). Design Prospective analysis of blood sampling before, during, and after GnRHa treatments. Setting Pediatric endocrine center. Patient(s) One hundred nineteen and 93 children with a diagnosis of CPP and GHD, respectively. Intervention(s) Monthly depot injections of GnRHa drugs (leuprorelin acetate 3.75 mg [LA] and triptorelin 3.75 mg [TR]) administered to CPP and GHD patients for 40 and 24 months, respectively. Main Outcome Measure(s) Serum PRL levels at baseline and after 6, 12, 18, 24, 30, 36, and…

Malemedicine.medical_specialtymedicine.drug_classPuberty PrecociousGonadotropin-releasing hormoneGrowth hormone deficiencyGonadotropin-Releasing HormoneLeuprorelinInternal medicineGonadotropin-releasing hormone agonistmedicinePrecocious pubertyHumansProspective StudiesChildbusiness.industryHuman Growth HormoneObstetrics and Gynecologymedicine.diseaseTriptorelinProlactinProlactinEndocrinologyReproductive MedicineChild PreschoolFemalebusinessBlood samplingmedicine.drugFertility and sterility
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