Search results for "Exchange"

showing 10 items of 2035 documents

Analysis of the Possible Persistent Genotoxic Damage in Workers Linked to the Ardystil Syndrome

2016

[EN] Background: A combination of several factors including a change in the paint application system; a lack of proper hygiene; and inadequate safety measures caused a severe health impact in the workers of some textile painting factories. This outbreak, mainly characterized by respiratory disorders, caused the death of six people and it has been classified as Ardystil syndrome. Materials and Methods: Fifty-two workers involved in the outbreak and 48 healthy subjects not known to have exposed to the potentially mutagenic agents participated in the study. The program evaluated possible genotoxic damage through the sister chromatid exchange (SCE) cytogenetic biomarker assay. We determined the…

Male0301 basic medicineHealth impactPhysiologySister chromatid exchangeINGENIERIA NUCLEARToxicology03 medical and health sciencesOccupational ExposureHumansMedicineGenetics (clinical)business.industrySignificant differenceHealthy subjectsOutbreakSyndromeGeneral MedicineRespiration Disorders030104 developmental biologyTextile IndustryDistribution patternBiomarker (medicine)FemalebusinessSister Chromatid ExchangeDNA DamageGenetic Testing and Molecular Biomarkers
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A new family with an SLC9A6 mutation expanding the phenotypic spectrum of Christianson syndrome

2016

Using targeted next generation sequencing, we have identified a splicing mutation (c.526-9_526-5del) in the SLC9A6 gene in a 9-year-old boy with mild intellectual disability (ID), microcephaly, and social interaction disabilities. This intronic microdeletion leads to the skipping of exon 3 and to an in-frame deletion of 26 amino acids in the TM4 domain. It segregates with cognitive impairment or learning difficulties in other members of the family. Mutations in SLC9A6 have been reported in X-linked Christianson syndrome associating severe to profound intellectual deficiency and an Angelman-like phenotype with microcephaly, absent speech, ataxia with progressive cerebellar atrophy, ophthalmo…

Male0301 basic medicineProbandMicrocephalyDNA Mutational Analysisx-chromosome inactivationSLC9A6Gene mutationexchangerEpilepsyOcular Motility Disorders0302 clinical medicineangelman-syndromeX Chromosome InactivationIntellectual disabilitymicrocephalyChild10. No inequalityGenetics (clinical)Sequence DeletionGeneticsBrainGenetic Diseases X-LinkedtoolMagnetic Resonance ImagingPedigree3. Good healthPhenotypeFemaleCerebellar atrophyChristianson syndromemedicine.symptomAdultHeterozygoteSodium-Hydrogen ExchangersAtaxiaAdolescentlearning disabilities linked mental-retardation03 medical and health sciencescerebellar atrophyIntellectual Disability[ SDV.MHEP ] Life Sciences [q-bio]/Human health and pathologyAngelman syndromeGeneticsmedicineHumansFamilygeneGenetic Association Studiesbusiness.industryFaciesmedicine.disease030104 developmental biologysplicing signalsMutationepilepsyAtaxiaRNA Splice Sitesbusiness030217 neurology & neurosurgery[SDV.MHEP]Life Sciences [q-bio]/Human health and pathology
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Apremilast, a novel phosphodiesterase 4 (PDE4) inhibitor, regulates inflammation through multiple cAMP downstream effectors

2015

Introduction This work was undertaken to delineate intracellular signaling pathways for the PDE4 inhibitor apremilast and to examine interactions between apremilast, methotrexate and adenosine A2A receptors (A2AR). Methods After apremilast and LPS incubation, intracellular cAMP, TNF-α, IL-10, IL-6 and IL-1α were measured in the Raw264.7 monocytic murine cell line. PKA, Epac1/2 (signaling intermediates for cAMP) and A2AR knockdowns were performed by shRNA transfection and interactions with A2AR and A2BR, as well as with methotrexate were tested in vitro and in the murine air pouch model. Statistical differences were determined using one or two-way ANOVA or Student’s t test. The alpha nominal…

MaleAdenosineReceptor Adenosine A2AImmunologyBlotting WesternAdenosine A2A receptorGene ExpressionPharmacologyBiologyCell LineMiceRheumatologyPhenethylaminesmedicineCyclic AMPImmunology and AllergyAnimalsGuanine Nucleotide Exchange FactorsReceptorIC50ArtritisReverse Transcriptase Polymerase Chain ReactionTumor Necrosis Factor-alphaMacrophagesAdenosineMolecular biologyCyclic AMP-Dependent Protein KinasesThalidomideMethotrexateMechanism of actionAntirheumatic AgentsCytokinesTumor necrosis factor alphaRNA InterferenceApremilastPhosphodiesterase 4 Inhibitorsmedicine.symptomInflammation MediatorsIntracellularMedicamentsmedicine.drugResearch Article
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Conserved role of Ras-GEFs in promoting aging: from yeast to mice

2011

RasGRF1 is a Ras-guanine nucleotide exchange factor implicated in a variety of physiological processes including learning and memory and glucose homeostasis. To determine the role of RASGRF1 in aging, lifespan and metabolic parameters were analyzed in aged RasGrf1(-/-) mice. We observed that mice deficient for RasGrf1(-/-) display an increase in average and most importantly, in maximal lifespan (20% higher than controls). This was not due to the role of Ras in cancer because tumor-free survival was also enhanced in these animals. Aged RasGrf1(-/-) displayed better motor coordination than control mice. Protection against oxidative stress was similarly preserved in old RasGrf1(-/-). IGF-I lev…

MaleAgingpositron emission tomographyProtein familyCellular differentiationLongevityCellSaccharomyces cerevisiaeSaccharomyces cerevisiaeMiceSirtuin 1RNA Ribosomal 16SmedicineAnimalsInsulin-Like Growth Factor IGEFCaloric RestrictionMice KnockoutBase Sequenceaging stress resistance yeast lifespanbiologyras-GRF1SUPERFAMILYCell Biologybiology.organism_classificationMolecular biologyYeastLiver GlycogenCell biologyMice Inbred C57BLOxidative StressGlucosemedicine.anatomical_structureRanCommentaryMetabolomeIGF-1Femaleras Guanine Nucleotide Exchange FactorsRabmetabolismPsychomotor PerformanceResearch PaperRasAging
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Expression and developmental regulation of the cystine/glutamate exchanger (xc-) in the rat.

2007

The cystine/glutamate exchanger (antiporter x c − ) is a membrane transporter involved in the uptake of cystine, the rate-limiting amino acid in the synthesis of glutathione. Recent studies suggest that the antiporter plays a role in the slow oxidative excitotoxity and in the pathological effects of β-N-oxalylamino-l-alanine, the molecule responsible for neurolathyrism, a neurotoxic upper motor neuron disease. The mouse cystine/glutamate exchanger has been cloned and showed to be composed of two distinct proteins, one of which being a novel protein, named xCT, of 502 amino acids and 12 putative trans-membrane domains. We have generated and purified a polyclonal antibody to mouse xCT and stu…

MaleAmino Acid Transport SystemsAntiporterProtein subunitBlotting WesternImmunoblottingCystineGlutamic AcidBiologyBiochemistryRats Sprague-DawleyCellular and Molecular Neurosciencechemistry.chemical_compoundMiceWestern blotChlorocebus aethiopsmedicineAnimalsHumansCystine/glutamate exchanger Protein expression Cell cultures Developmenchemistry.chemical_classificationCerebral CortexNeuronsmedicine.diagnostic_testGlutamate receptorGene Expression Regulation DevelopmentalGeneral MedicineGlutathioneFibroblastsImmunohistochemistryAmino acidRatsBiochemistrychemistryAstrocytesCOS CellsCystineSettore MED/26 - NeurologiaElectrophoresis Polyacrylamide GelCell fractionationSubcellular FractionsNeurochemical research
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Maternal medication use and the risk of brain tumors in the offspring: The SEARCH international case-control study

2006

International audience; N-nitroso compounds (NOC) have been associated with carcinogenesis in a wide range of species, including humans. There is strong experimental data showing that nitrosamides (R(1)NNO.COR(2)), a type of NOC, are potent neuro-carcinogens when administered transplacentally. Some medications are a concentrated source of amides or amines, which in the presence of nitrites under normal acidic conditions of the stomach can form NOC. Therefore, these compounds, when ingested by women during pregnancy, may be important risk factors for tumors of the central nervous system in the offspring. The aim of the present study was to test the association between maternal use of medicat…

MaleCancer ResearchMESH: Maternal-Fetal ExchangeMESH: Pregnancy0302 clinical medicinePregnancyRisk FactorsMESH: Risk FactorsMESH: ChildRecall biasEpidemiologyMedicine030212 general & internal medicineAminesChildMaternal-Fetal Exchangeeducation.field_of_studyBrain NeoplasmsN-nitroso compoundsMESH: AminesMESH: InfantMESH: AmidesMESH: Case-Control StudiesMESH: Mothers3. Good healthOncologyChild Preschool030220 oncology & carcinogenesisMESH: Brain NeoplasmsFemaleDisease SusceptibilityAdultmedicine.medical_specialtyAdolescentOffspringcase-control studyPopulationMESH: Disease SusceptibilityMothers[SDV.CAN]Life Sciences [q-bio]/Cancerchildhood brain tumors03 medical and health sciencesInternal medicineGliomamaternal medicationHumansRisk factoreducationMESH: AdolescentPregnancyMESH: Humansbusiness.industryMESH: Child PreschoolCase-control studyInfantMESH: Adultmedicine.diseaseAmidesMESH: MaleCase-Control StudiesbusinessMESH: FemaleInternational Journal of Cancer
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Criteria for the determination of maximal oxygen uptake in patients newly diagnosed with cancer : Baseline data from the randomized controlled trial …

2020

IntroductionMaximal oxygen uptake ([Formula: see text]) is a measure of cardiorespiratory fitness often used to monitor changes in fitness during and after treatment in cancer patients. There is, however, limited knowledge in how criteria verifying [Formula: see text] work for patients newly diagnosed with cancer. Therefore, the aim of this study was to describe the prevalence of fulfillment of typical criteria verifying [Formula: see text] and to investigate the associations between the criteria and the test leader's evaluation whether a test was performed "to exhaustion". An additional aim was to establish new cut-points within the associated criteria.MethodsFrom the Phys-Can randomized c…

MaleCancer Treatmentlaw.inventionCohort Studies0302 clinical medicineRandomized controlled trialHeart RateReference ValueslawNeoplasmsMedicine and Health SciencesPublic and Occupational HealthTreadmillRespiratory exchange ratioRating of perceived exertioneducation.field_of_studyMultidisciplinaryQRVO2 maxMiddle AgedVDP::Medisinske Fag: 700::Idrettsmedisinske fag: 850Sports ScienceExercise TherapyChemistryTreatment OutcomeOncologyCardiorespiratory FitnessResearch Design030220 oncology & carcinogenesisPhysical SciencesMedicineFemaleResearch ArticleChemical Elementsmedicine.medical_specialtySciencePhysical ExertionPopulationCardiologyResearch and Analysis Methods03 medical and health sciencesOxygen ConsumptionDiagnostic MedicineInternal medicineHeart rateCancer Detection and DiagnosismedicineHumansSports and Exercise MedicineeducationExerciseAgedColorectal Cancerbusiness.industryBiology and Life SciencesCancers and NeoplasmsCardiorespiratory fitnessPhysical Activity030229 sport sciencesOxygenROC CurvePhysical FitnessExercise Testbusinesshuman activities
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Genome-wide association scan of the time to onset of attention deficit hyperactivity disorder.

2008

Contains fulltext : 70149.pdf (Publisher’s version ) (Closed access) A time-to-onset analysis for family-based samples was performed on the genomewide association (GWAS) data for attention deficit hyperactivity disorder (ADHD) to determine if associations exist with the age at onset of ADHD. The initial dataset consisted of 958 parent-offspring trios that were genotyped on the Perlegen 600,000 SNP array. After data cleaning procedures, 429,981 autosomal SNPs and 930 parent-offspring trios were used found suitable for use and a family-based logrank analysis was performed using that age at first ADHD symptoms as the quantitative trait of interest. No SNP achieved genome-wide significance, and…

MaleCandidate geneGenetics and epigenetic pathways of disease [NCMLS 6]2804 Cellular and Molecular NeuroscienceMedizinGenome-wide association studyNeuroinformatics [DCN 3]Linkage Disequilibrium2738 Psychiatry and Mental Health0302 clinical medicinePerception and Action [DCN 1]Genetics(clinical)Age of OnsetChildGenetics (clinical)Genetics0303 health sciences10058 Department of Child and Adolescent PsychiatryPedigreePsychiatry and Mental healthChild PreschoolFemaleFunctional Neurogenomics [DCN 2]SNP arrayGenetic Markers2716 Genetics (clinical)Sodium-Hydrogen ExchangersAdolescentQuantitative Trait Loci610 Medicine & healthSingle-nucleotide polymorphismBiologyQuantitative trait locusMental health [NCEBP 9]Polymorphism Single NucleotideArticleGenomic disorders and inherited multi-system disorders [IGMD 3]03 medical and health sciencesCellular and Molecular NeuroscienceCognitive neurosciences [UMCN 3.2]medicineAttention deficit hyperactivity disorderSNPHumansGenetic Predisposition to Diseaseddc:610Medizinische Fakultät » Universitätsklinikum Essen » LVR-Klinikum Essen » Klinik für Psychiatrie Psychosomatik und Psychotherapie des Kindes- und Jugendalters030304 developmental biologyProbabilityRetrospective StudiesGenome Humanmedicine.diseaseGenetic defects of metabolism [UMCN 5.1]HaplotypesAttention Deficit Disorder with HyperactivityAge of onset030217 neurology & neurosurgeryGenome-Wide Association Study
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The genomic and clinical landscape of fetal akinesia

2020

International audience; Fetal akinesia has multiple clinical subtypes with over 160 gene associations, but the genetic etiology is not yet completely understood.Methods: In this study, 51 patients from 47 unrelated families were analyzed using next-generation sequencing (NGS) techniques aiming to decipher the genomic landscape of fetal akinesia (FA).Results: We have identified likely pathogenic gene variants in 37 cases and report 41 novel variants. Additionally, we report putative pathogenic variants in eight cases including nine novel variants. Our work identified 14 novel disease-gene associations for fetal akinesia: ADSSL1, ASAH1, ASPM, ATP2B3, EARS2, FBLN1, PRG4, PRICKLE1, ROR2, SETBP1…

MaleCandidate geneMyopathyVARIANTSFetal akinesiaMESH: Ryanodine Receptor Calcium Release Channel0302 clinical medicineMESH: ChildGuanine Nucleotide Exchange FactorsMESH: Guanine Nucleotide Exchange FactorsExomeCopy-number variationChildExomeMESH: High-Throughput Nucleotide SequencingGenetics (clinical)GeneticsArthrogryposisArthrogryposis0303 health sciencesMESH: Infant NewbornMESH: Genetic Predisposition to DiseaseHigh-Throughput Nucleotide SequencingRNA-Binding ProteinsMESH: Infant3. Good healthFetal DiseasesCopy-number variationMESH: Fetal DiseasesMESH: Young AdultChild PreschoolASAH1FemaleMESH: DNA Copy Number Variationsmedicine.symptomAdultGENETICSAdolescentDNA Copy Number VariationsMESH: Trans-ActivatorsMESH: ArthrogryposisBiologyASPMYoung Adult03 medical and health sciencesMuscular DiseasesmedicineHumansGenetic Predisposition to DiseaseGene030304 developmental biologyMESH: Adolescent[SDV.MHEP.PED]Life Sciences [q-bio]/Human health and pathology/PediatricsMESH: HumansMUTATIONSMESH: Child PreschoolInfant NewbornMESH: Muscular DiseasesInfantNEMALINE MYOPATHYRyanodine Receptor Calcium Release ChannelMESH: Adultmedicine.diseaseCongenital myopathyMESH: MaleMESH: RNA-Binding Proteins[SDV.GEN.GH]Life Sciences [q-bio]/Genetics/Human geneticsDISTAL ARTHROGRYPOSISTrans-ActivatorsMESH: Female030217 neurology & neurosurgery
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The use of high-pressure liquid cation-exchange chromatography for determination of the 5-methylcytosine content of DNA.

1976

MaleChromatographyTroutHydrophilic interaction chromatographyOrganic ChemistryIon chromatographyFishesGeneral MedicineDNAPlantsChromatography Ion ExchangeBiochemistryHigh-performance liquid chromatographySpermatozoaAnalytical Chemistrychemistry.chemical_compound5-MethylcytosineCytosinechemistrySpecies SpecificityHigh pressureMethodsAnimalsDNAChromatography High Pressure LiquidJournal of chromatography
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