Search results for "Extracellular"

showing 10 items of 1220 documents

Cigarette smoke extract activates human bronchial epithelial cells affecting non-neuronal cholinergic system signalling in vitro.

2010

Abstract Aims Acetylcholine (ACh) is synthesized by Choline Acetyl-Transferase (ChAT) that exerts its physiological effects in airway epithelial cells via muscarinic receptor (MR) activation. We evaluate the effect of ACh stimulation on human bronchial epithelial cells (16-HBE) and test whether cigarette smoke extract (CSE) can modify the basal cellular response to ACh affecting the non-neuronal cholinergic system signalling. Main methods ACh stimulated 16-HBE were tested for ACh-binding, Leukotriene B 4 (LTB 4 ) release and ERK1/2 and NFkB pathway activation. Additionally, we investigated all the aforementioned parameters as well as ChAT and MR proteins and mRNA expression and endogenous A…

medicine.medical_specialtyLeukotriene B4Blotting WesternEndogenyStimulationBronchiPharmacologyBiologyComplex MixturesIn Vitro TechniquesLeukotriene B4General Biochemistry Genetics and Molecular BiologyCell LineCholine O-Acetyltransferasechemistry.chemical_compoundInternal medicineSmokeparasitic diseasesMuscarinic acetylcholine receptorTobaccomedicineHumansGeneral Pharmacology Toxicology and PharmaceuticsReceptorExtracellular Signal-Regulated MAP KinasesAnalysis of VarianceReverse Transcriptase Polymerase Chain ReactionNF-kappa BCholine Acetyl-TransferaseAcetylcholine muscarinic receptorhuman bronchial epithelial cellsGeneral MedicineFlow CytometryCholine acetyltransferaseReceptors MuscarinicAcetylcholineEndocrinologychemistryGene Expression RegulationTelenzepineAcetylcholinemedicine.drugSignal Transduction
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Sequential release of TNFα and phospholipase A2 in a rat model of LPS-induced pleurisy

1997

The levels of extracellular phospholipase A2(sPLA2) and TNFα, and cell accumulation were measured in the pleural washings obtained at different times following the induction ofEscherichia colilipopolysaccharide (LPS, 100 μg/cavity) pleurisy in rats. TNFα peaked at 2 hours (3036 ± 160.3 units/ml) and decreased thereafter. Conversely, levels of sPLA2peaked at 48 hours (1.97 ± 0.64 ng/ml) and were increased further (14.02 ± 4.16 ng/ml) by pretreatment with anti-TNFα antibody. Cell accumulation was not affected by antibody pretreatment. These data indicate that the sPLA2enzyme is involved in LPS-induced pleurisy. The enzyme seems not to be stimulated by TNFα which may be involved in the downreg…

medicine.medical_specialtyLipopolysaccharideImmunologypleurisyInflammationchemistry.chemical_compoundPhospholipase A2Downregulation and upregulationInternal medicinemedicineExtracellularlcsh:Pathologyratchemistry.chemical_classificationbiologybusiness.industrylipopolysaccharideCell Biologymedicine.diseaseEndocrinologyEnzymechemistryPleurisyImmunologybiology.proteinTumor necrosis factor alphaphospholipase A2medicine.symptombusinessResearch Articlelcsh:RB1-214Mediators of Inflammation
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Effects of Leptin on Lipopolysaccharide-Induced Remodeling in an In Vitro Model of Human Myometrial Inflammation1

2013

Reorganization of myometrial extracellular matrix (ECM) is essential for the uterus to achieve powerful synchronous contractions during labor. Remodeling of the ECM has been implicated in membrane rupture and cervical ripening. Because maternal obesity is associated with both delivery disorders and elevated circulating leptin levels, this study aimed to assess the ability of leptin to interfere with lipopolysaccharide (LPS)-induced myometrial ECM remodeling. Myometrial biopsy samples were obtained from women undergoing cesarean delivery before labor onset. Myometrial explants were incubated for 48 h with LPS and leptin. LPS challenge was associated with a marked decrease in collagen content…

medicine.medical_specialtyMMP2LeptinMyometriumInflammationCell BiologyGeneral MedicineBiologyMatrix metalloproteinaseMMP9Extracellular matrixEndocrinologyReproductive MedicineInternal medicineHeat shock proteinmedicinemedicine.symptomBiology of Reproduction
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Intestinal ischaemia during cardiac arrest and resuscitation: comparative analysis of extracellular metabolites by microdialysis.

2003

Intestinal ischaemia is a major complication of shock syndromes causing translocation of bacteria and endotoxins and multiple organ failure in intensive care patients. The present study was designed to use microdialysis as a tool to monitor intestinal ischaemia after cardiac arrest and resuscitation in pigs. For this purpose, microdialysis probes were implanted in pig jejunal wall, peritoneum, skeletal muscle and brain, and interstitial fluid was obtained during circulatory arrest (induced by ventricular fibrillation) and after return of spontaneous circulation (ROSC). Cardiac arrest for 4 min caused a prolonged (60 min) reduction of blood flow in jejunal wall, muscle and brain as determine…

medicine.medical_specialtyMicrodialysisResuscitationTime FactorsSwinemedicine.medical_treatmentMicrodialysisIschemiaGlutamic AcidEmergency NursingReturn of spontaneous circulationCholineIschemiaInternal medicineIntensive caremedicineAnimalsCardiopulmonary resuscitationLactic AcidIntestinal MucosaMuscle Skeletalbusiness.industrySkeletal muscleBrainmedicine.diseaseCardiopulmonary ResuscitationHeart ArrestIntestinesEndocrinologymedicine.anatomical_structureGlucoseAnesthesiaVentricular fibrillationEmergency MedicinePeritoneumCardiology and Cardiovascular MedicinebusinessExtracellular SpaceResuscitation
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Serotonergic modulation of hippocampal acetylcholine release after long-term neuronal grafting

2000

Adult female rats sustained aspirative fimbria-fornix lesions and, 2 weeks later, received intrahippocampal grafts of fetal septal or mixed septal-raphe cell suspensions. Twenty-four months later, the extracellular concentration of hippocampal acetylcholine (ACh) was determined by microdialysis. Basal ACh levels (5-65 fmol/5 microl sham-operated rats) were strongly reduced after lesioning (3-7 fmol/5 microl). In septally transplanted and septal-raphe co-transplanted rats, hippocampal ACh concentrations were restored to near-normal levels (15-25 fmol/5 microl), indicating long-term functional survival of hippocampal transplants. After administration of citalopram (100 microM by infusion) and…

medicine.medical_specialtyMicrodialysisSerotoninTime FactorsFenfluramine[SDV]Life Sciences [q-bio][SDV.NEU.NB]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/NeurobiologyFornix BrainHippocampal formationBiologySerotonergicHippocampus03 medical and health sciences0302 clinical medicineFetusInternal medicineExtracellularmedicineAnimalsBrain Tissue TransplantationRats Long-EvansComputingMilieux_MISCELLANEOUS030304 developmental biology0303 health sciencesFetusNeuronal PlasticityRapheGeneral NeuroscienceGraft SurvivalAcetylcholineRatsEndocrinologyCholinergic FibersRaphe NucleiSeptal Nuclei[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]030217 neurology & neurosurgeryAcetylcholinemedicine.drug
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The neuronal efflux of noradrenaline: Dependency on sodium and facilitation by ouabain

1974

Rabbit hearts were isolated after pretreatment with the MAO inhibitor pargyline and with reserpine and were perfused with 200 ng/ml noradrenaline for 1 h. During the subsequent wash-out with an amine-free solution for 2 h, the neuronal efflux of noradrenaline declined mono-exponentially with a mean halftime of 42 min. Both Na+-free solution and ouabain caused facilitation of the efflux which thereafter declined in a multi-exponential fashion. The maximum facilitation was reached after 3 min of Na+-free perfusion and 25 min after introduction of ouabain. The amount of exogenous noradrenaline accumulated in the heart was only partially released when the extracellular Na+-concentration was nor…

medicine.medical_specialtyMonoamine Oxidase InhibitorsReserpineTime FactorsSodiumchemistry.chemical_elementAdrenergicOuabainNorepinephrineHeart RateInternal medicinemedicineExtracellularAnimalsOuabainNeuronsPharmacologyMyocardiumSodiumGeneral MedicineReserpinePargylineEndocrinologyPargylinechemistryRabbitsEffluxPerfusionmedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
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The pathological hallmarks of Alzheimer’s disease derive from compensatory responses to NMDA receptor insufficiency

2018

AbstractAlzheimer’s disease is characterized by intracellular aggregates of hyperphosphorylated tau protein and extracellular plaques of amyloid β peptide, a product of APP processing. The origin of these pathological hallmarks has remained elusive. Here, we have tested the idea that both alterations, at the onset of the disease, may constitute compensatory responses to the same causative and initial trigger, namely NMDA receptor insufficiency. Treatment of rat cortical neurons with the specific NMDA receptor antagonist AP5 within 4 h caused a significant increase in tau phosphorylation at the AT8 and S404 epitopes as well as an increase in APP expression and Aβ 40 secretion. Single intrape…

medicine.medical_specialtyMutationbiologybusiness.industryTau proteinNeurotransmissionmedicine.disease_causeEndocrinologyInternal medicineExtracellularbiology.proteinExcitatory postsynaptic potentialNMDA receptorMedicineSecretionbusinessIntracellular
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Inhibitory effects of okadaic acid on rat uterine contractile responses to different spasmogens

1997

In the present study, we examined the effects of okadaic acid, a selective inhibitor of type I and 2A protein phosphatases, on the mechanical responses evoked by oxytocin, K + - and Na + -modified solutions and ouabain in estrogen-primed rat myometrium. Oxytocin elicited a rapid, phasic contraction followed by rhythmic oscillations. The phasic response was partially resistant to the absence of external Ca 2+ . Okadaic acid (1 μM) and the L-type calcium channel blocker nifedipine (1 μM) abolished the oscillatory component and reduced the initial, phasic response to about 80% of the control response. High K + (60 mM) solution, ouabain (1 mM), K + -free medium and low Na + (25 mM) solution ind…

medicine.medical_specialtyNifedipinePhosphatasechemistry.chemical_elementCalciumOxytocinOuabainUterine Contractionchemistry.chemical_compoundNifedipineInternal medicineOkadaic AcidmedicineExtracellularAnimalsPharmacology (medical)Rats WistarOuabainPharmacologyDose-Response Relationship DrugSodiumMyometriumOkadaic acidCalcium Channel BlockersPhosphoric Monoester HydrolasesRatsAmilorideEndocrinologychemistryPotassiumCalciumFemalemedicine.drugFundamental & Clinical Pharmacology
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Oxygen and glucose deprivation induces major dysfunction in the somatosensory cortex of the newborn rat

2005

The mechanisms and functional consequences of ischemia-induced injury during perinatal development are poorly understood. Subplate neurons (SPn) play a central role in early cortical development and a pathophysiological impairment of these neurons may have long-term detrimental effects on cortical function. The acute and long-term consequences of combined oxygen and glucose deprivation (OGD) were investigated in SPn and compared with OGD-induced dysfunction of immature layer V pyramidal cortical neurons (PCn) in somatosensory cortical slices from postnatal day (P)0-4 rats. OGD for 50 min followed by a 10-24-h period of normal oxygenation and glucose supply in vitro or in culture led to pron…

medicine.medical_specialtyPatch-Clamp TechniquesTolbutamideIn Vitro TechniquesBiologySomatosensory systemMembrane PotentialsInternal medicineSubplatemedicineExtracellularAnimalsHypoglycemic AgentsMagnesiumEnzyme InhibitorsHypoxiaOuabainNeuronsMembrane potentialCaspase 3General NeuroscienceDose-Response Relationship RadiationDepolarizationSomatosensory CortexHyperpolarization (biology)ImmunohistochemistryElectric StimulationRatsGlucoseNeuroprotective AgentsEndocrinologymedicine.anatomical_structureAnimals Newbornnervous systemApoptosisCaspasesNMDA receptorDizocilpine MaleateEuropean Journal of Neuroscience
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Effect of coronary perfusion rate on the hydrolysis of exogenous and endogenous acetylcholine in the isolated heart

1977

1. The effect of perfusion rate on the hydrolysis of acetylcholine in isolated chicken hearts was studied by measuring both the spontaneous and the evoked output of endogenous acetylcholine into the perfusate in response to vagal stimulation and the arterio-venous difference of exogenous acetylcholine. 2. A decrease in the perfusion rate from 30 to 20 and 10 ml/min caused a graded and significant decline of both the spontaneous overflow of acetylcholine and the overflow evoked by stimulation of both vagus nerves (20 Hz, 1 ms, 40V) for 20 min. The spontaneous and evoked overflow at 30 ml/min were 2 and 3 times, respectively, the overflow at 10 ml/min. 3. Physostigmine (10−6M) raised both the…

medicine.medical_specialtyPhysostigminePhysostigmineEndogenyStimulationIn Vitro TechniquesHydrolysisCoronary CirculationInternal medicinemedicineExtracellularAnimalsCholinesterasePharmacologybiologyChemistryHydrolysisMyocardiumVagus NerveGeneral MedicineAcetylcholineElectric StimulationPerfusionEndocrinologybiology.proteinChickensPerfusionAcetylcholinemedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
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