Search results for "Extracellular"

showing 10 items of 1220 documents

MHCII-independent CD4+ T cells protect injured CNS neurons via IL-4

2015

A body of experimental evidence suggests that T cells mediate neuroprotection following CNS injury; however, the antigen specificity of these T cells and how they mediate neuroprotection are unknown. Here, we have provided evidence that T cell-mediated neuroprotection after CNS injury can occur independently of major histocompatibility class II (MHCII) signaling to T cell receptors (TCRs). Using two murine models of CNS injury, we determined that damage-associated molecular mediators that originate from injured CNS tissue induce a population of neuroprotective, IL-4-producing T cells in an antigen-independent fashion. Compared with wild-type mice, IL-4-deficient animals had decreased functi…

CD4-Positive T-LymphocytesCancer ResearchMAP Kinase Signaling SystemPopulationReceptors Antigen T-CellInflammationBiologyNeuroprotectionMiceAntigenClinical investigationAnimalsMedicineExtracellular Signal-Regulated MAP KinaseseducationReceptorInterleukin 4Mice Knockouteducation.field_of_studybusiness.industryT-cell receptorHistocompatibility Antigens Class IINeurodegenerative DiseasesGeneral MedicineAxonsCell biologyBrain InjuriesMyeloid Differentiation Factor 88Immunologybiology.proteinInterleukin-4medicine.symptomFunction and Dysfunction of the Nervous SystemCorrigendumbusinessProto-Oncogene Proteins c-aktResearch ArticleNeurotrophinJournal of Clinical Investigation
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In vitro model for the activation of CD4 and CD8 T cell receptors.

2008

Previously, most models that sought to explain the misregulation of immune cell function assumed molecular similarities between the disease-causing pathogens and the host's proteins. In recent time several different models have been proposed and in this study, these concepts are compared to a new hypothesis proposing another explanation for this immune dysregulation: the possibility that the mislocalization of proteins may be responsible for autoimmune activity. Based on this hypothesis, proteins are recognized as self or non-self depending on where they appear in sufficiently high concentrations. To examine this new idea, the intracellular human proteins beta-actin, GAPDH, and hemoglobin a…

CD4-Positive T-LymphocytesCytoplasmImmunologyReceptors Antigen T-CellAutoimmunityCell SeparationCD8-Positive T-Lymphocytesmedicine.disease_causeLymphocyte ActivationHemoglobinsAlbuminsmedicineExtracellularImmunology and AllergyCytotoxic T cellHumansInsulinReceptorGlyceraldehyde 3-phosphate dehydrogenaseCells CulturedbiologyAlbuminModels ImmunologicalGlyceraldehyde-3-Phosphate DehydrogenasesGeneral MedicineImmune dysregulationFlow CytometryActinsCell biologyProtein Transportbiology.proteinCell activationExtracellular SpaceIntracellularHuman immunology
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Modulation of proliferation and lymphokine secretion of murine CD4+ T cells and cloned Th1 cells by proteins of the extracellular matrix.

1997

In this study we investigated the co-stimulatory signaling capacity of diverse proteins of the extracellular matrix (ECM) for murine resting CD4+ T cells and Th1 clone cells, activated by immobilized anti-CD3 mAb. ECM proteins used in various concentrations had no effect on IL-2 production or proliferation of highly purified CD4+ T cell populations. When the preparation of CD4+ T cells contained contaminating accessory cells, IL-2 secretion and proliferation was enhanced in the presence of co-immobilized collagens or fibronectin. However, the level of proliferation attainable by added irradiated splenocytes was not reached. Using Th1 cell clone M4, enhanced production of IL-2 in the presenc…

CD4-Positive T-LymphocytesT cellImmunologyLymphocyte ActivationExtracellular matrixInterleukin 21MicemedicineImmunology and AllergyCytotoxic T cellAnimalsSecretionAntigen-presenting cellExtracellular Matrix ProteinsLymphokinesMice Inbred BALB CMice Inbred C3HbiologyChemistryIntegrin beta1LymphokineReceptors Interleukin-2General MedicineTh1 CellsMolecular biologyCell biologyClone CellsFibronectinMice Inbred C57BLmedicine.anatomical_structurebiology.proteinInternational immunology
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HSP90 AND ENOS PARTIALLY CO-LOCALIZE AND CHANGE CELLULAR LOCALIZATION IN RELATION TO DIFFERENT ECM COMPONENTS IN 2D AND 3D CULTURES OF ADULT RAT CARD…

2007

BACKGROUND INFORMATION: Cultivation techniques promoting three-dimensional organization of mammalian cells are of increasing interest, since they confer key functionalities of the native ECM (extracellular matrix) with a power for regenerative medicine applications. Since ECM compliance influences a number of cell functions, Matrigel-based gels have become attractive tools, because of the ease with which their mechanical properties can be controlled. In the present study, we took advantage of the chemical and mechanical tunability of commonly used cell culture substrates, and co-cultures to evaluate, on both two- and three-dimensional cultivated adult rat cardiomyocytes, the impact of ECM c…

CO-COLTURESARTIFICIAL EXTRACELLULAR MATRIXELECTRON MICROSCOPYCONFOCAL MICROSCOPYPROTEIN CO-LOCALIZATION.
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Versican and collagen-III expression in bronchial and pulmonary muscular arteries in COPD patients

2015

Background: We demonstrated that COPD patients have thicker bronchial and pulmonary muscular arteries (MA) compared with never and non-obstructed smokers. The composition of the extracellular matrix of these altered MA has been not fully described. Objectives: To investigate the expression of versican and collagen-III in bronchial and pulmonary MA in the lungs of mild/moderate COPD patients. Methods: Eighteen mild/moderate COPD subjects (67±8y, FEV 1 : 67±15%pred), 14 non-obstructed smokers (64±8y, FEV 1 : 95±10%pred) and 11 never-smoking controls (CTRL) (51±12y, FEV 1 : 108±15%pred) were studied. Lung tissue obtained during surgery (ethical approval obtained) was used to analyze the expres…

COPDmedicine.medical_specialtybiologyCollagen iiibusiness.industryrespiratory systemmedicine.diseaseTunica intimaGastroenterologyrespiratory tract diseasesExtracellular matrixFEV1/FVC ratiomedicine.anatomical_structureInternal medicineAdventitiamedicinebiology.proteinImmunohistochemistryVersicanbusiness4.3 Pulmonary Circulation and Pulmonary Vascular Disease
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Effects of cadmium exposure on the ultrastructure of hepatopancreatic cells of Thais haemastoma (gastropoda, prosobranchia)

1993

Abstract Specific histopathological parameters have been proposed as good indicators of heavy-metal pollution. Hence the study of ultrastructural alterations in target organs from sentinel animals after heavy-metal exposure may provide a useful tool in understanding the cellular role in heavy-metal metabolism. The main purpose of this study was the determination of the ultrastructural changes in the different hepatopancreatic-cell types of Thais haemastoma after experimental exposure to cadmium. Specimens of this species from the Gulf of Valencia were exposed to sublethal doses of cadmium during a period of 96 h. The hepatopancreas was excised and prepared in a routine manner for electron m…

CadmiumbiologyEndoplasmic reticulumExtracellular digestionchemistry.chemical_elementGeneral MedicineMetabolismAnatomyAquatic ScienceOceanographybiology.organism_classificationPollutionExcretionBasophilicBiochemistrychemistryUltrastructureHepatopancreasMarine Environmental Research
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Dibutyryl cyclic AMP and adrenaline increase contractile force and 45Ca uptake in mammalian cardiac muscle

1973

The effects of dibutyryl cyclic AMP (DB-AMP; 10−3M) and adrenaline (2.2×10−6 M) on contractile force, 45Ca uptake, and total myocardial Ca concentration were investigated in electrically driven left auricles isolated from rat hearts. The experiments were performed at an extracellular Ca concentration of 0.45 mM and at low frequency of stimulation (15 beats/min). 45Ca exposure was 5 min. Under the conditions used, both drugs increased contractile force and enhanced 45Ca uptake (expressed as relative specific activity) by about 30% (DB-AMP) and 40% (adrenaline), respectively. Thus, the results provide evidence that the effects of adrenaline on 45Ca uptake in mammalian cardiac muscle can be mi…

Calcium Isotopesmedicine.medical_specialtyContraction (grammar)Epinephrinechemistry.chemical_elementStimulationIn Vitro TechniquesCalciumInternal medicineCyclic AMPmedicineExtracellularAnimalsCa uptakeCardiac OutputPharmacologyMyocardiumCardiac muscleHeartGeneral MedicineDibutyryl Cyclic AMPRatsEndocrinologymedicine.anatomical_structurechemistryCalciumFemaleSpecific activityNaunyn-Schmiedeberg's Archives of Pharmacology
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Rebalancing β-Amyloid-Induced Decrease of ATP Level by Amorphous Nano/Micro Polyphosphate: Suppression of the Neurotoxic Effect of Amyloid β-Protein …

2017

Morbus Alzheimer neuropathology is characterized by an impaired energy homeostasis of brain tissue. We present an approach towards a potential therapy of Alzheimer disease based on the high-energy polymer inorganic polyphosphate (polyP), which physiologically occurs both in the extracellular and in the intracellular space. Rat pheochromocytoma (PC) 12 cells, as well as rat primary cortical neurons were exposed to the Alzheimer peptide Aβ25-35. They were incubated in vitro with polyphosphate (polyP); ortho-phosphate was used as a control. The polymer remained as Na+ salt; or complexed in a stoichiometric ratio to Ca2+ (Na-polyP[Ca2+]); or was processed as amorphous Ca-polyP microparticles (C…

Calcium Phosphates0301 basic medicineIntracellular SpacePeptidelcsh:Chemistrychemistry.chemical_compoundAdenosine TriphosphateX-Ray DiffractionPolyphosphatesSpectroscopy Fourier Transform Infraredprimary rat cortex neuronslcsh:QH301-705.5SpectroscopyCerebral CortexNeuronschemistry.chemical_classificationmicroparticlesChemistryβ-amyloidGeneral Medicinepathological conditions signs and symptomsComputer Science Applicationsneurotoxic effectsurgical procedures operativeBiochemistryAlzheimer's diseaseIntracellularCell Survivalβ-amyloid; calcium polyphosphate; microparticles; neurotoxic effect; adenosine triphosphate level; PC12 cells; primary rat cortex neuronsArticleCatalysisInorganic Chemistry03 medical and health sciencesmedicineExtracellularotorhinolaryngologic diseasesAnimalsPhysical and Theoretical ChemistryMolecular BiologyneoplasmsAmyloid beta-PeptidesPolyphosphateOrganic ChemistryNeurotoxicityPC12 cellsmedicine.diseaseIn vitrodigestive system diseasesRats030104 developmental biologylcsh:Biology (General)lcsh:QD1-999BiophysicsNanoparticlesAdenosine triphosphatecalcium polyphosphateadenosine triphosphate levelInternational Journal of Molecular Sciences
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Characterisation of tumour-associated antigens in colon cancer.

2002

In order to search for clinically relevant cancer-associated genes and to define further the spectrum of immunogenic proteins, we applied SEREX (serological identification of antigens by recombinant expression cloning) to analyse genes expressed in colon adenocarcinoma. Eight different serum-reactive cDNA clones were isolated by immunoscreening from a colon cancer-derived cDNA expression library. mRNA expression studies showed that 2 of them, RHAMM and AD034, have a differential tissue distribution, and that 3 genes, NAP1L1, RHAMM and AD034, are overexpressed in tumours in comparison with the adjacent non-cancerous tissues. 5' RLM-RACE analysis of AD034, a sequence with a tyrosine kinase mo…

Cancer ResearchAntibodies NeoplasmImmunologyMolecular Sequence DataBiologyAdenocarcinomaProtein Serine-Threonine KinasesAntigenAntigens NeoplasmIsoantibodiesComplementary DNAImmunoscreeningGene expressionmedicineImmunology and AllergyHumansspliceTissue DistributionRNA MessengerCloning MolecularGeneGene LibraryGeneticsExtracellular Matrix ProteinsCancermedicine.diseaseMolecular biologyAlternative SplicingHyaluronan ReceptorsOncologyColonic NeoplasmsAdenocarcinomaCancer immunology, immunotherapy : CII
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Glioblastoma cells induce differential glutamatergic gene expressions in human tumor-associated microglia/macrophages and monocyte-derived macrophages

2015

Glioblastoma cells produce and release high amounts of glutamate into the extracellular milieu and subsequently can trigger seizure in patients. Tumor-associated microglia/macrophages (TAMs), consisting of both parenchymal microglia and monocytes-derived macrophages (MDMs) recruited from the blood, are known to populate up to 1/3 of the glioblastoma tumor environment and exhibit an alternative, tumor-promoting and supporting phenotype. However, it is unknown how TAMs respond to the excess extracellular glutamate in the glioblastoma microenvironment. We investigated the expressions of genes related to glutamate transport and metabolism in human TAMs freshly isolated from glioblastoma resecti…

Cancer ResearchAntigens Differentiation MyelomonocyticGlutamic AcidglutamateAMPA receptorSLC7A11Antigens CDTumor Cells CulturedExtracellularmedicineHumansReceptors AMPAGRIA2PharmacologyCD11b AntigenbiologyMicrogliaBrain NeoplasmsMacrophagesmonocyte-derived macrophagesCalcium-Binding ProteinsMicrofilament Proteinsglioblastomatumor-associated microglia/macrophagesGlutamate receptorSLC1A2Coculture TechniquesDNA-Binding ProteinsGlutaminemedicine.anatomical_structureGene Expression RegulationOncologyAstrocytesImmunologybiology.proteinCancer researchLeukocyte Common AntigensMolecular MedicineMicrogliaResearch PaperCancer Biology & Therapy
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