Search results for "FIBROBLASTS"

showing 10 items of 445 documents

Myofibroblasts and increased angiogenesis contribute to periapical cystic injury containment and repair

2019

Background Myofibroblasts (MF) and angiogenesis are important factors in the development and expansion of cystic lesions, where these cells secrete growth factors and proteases, stimulating angiogenesis, matrix deposition and cell migration, affecting the growth of these periapicopathies. The present study aimed to evaluate the immunohistochemical expression of CD34 and α-SMA in radicular cysts (RC) and residual radicular cysts (RRC), with the purpose of contributing to a better understanding of the expansion and progression of these periapical lesions. Material and Methods The present study os a descriptive, quantitative and comparative analysis of positive CD34 and α-SMA immunohistochemic…

Pathologymedicine.medical_specialtyAngiogenesisCD34Neovascularization03 medical and health sciences0302 clinical medicineCell MovementmedicineHumansMyofibroblastsGeneral DentistryMicrovesselRadicular CystOral Medicine and PathologyNeovascularization Pathologicbusiness.industryResearchCell migration030206 dentistry:CIENCIAS MÉDICAS [UNESCO]OtorhinolaryngologyMicrovesselsUNESCO::CIENCIAS MÉDICASImmunohistochemistrySurgerymedicine.symptombusinessMyofibroblastImmunostaining
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SPARC oppositely regulates inflammation and fibrosis in bleomycin-induced lung damage.

2011

Fibrosis results from inflammatory tissue damage and impaired regeneration. In the context of bleomycin-induced pulmonary fibrosis, we demonstrated that the matricellular protein termed secreted protein acidic and rich in cysteine (SPARC) distinctly regulates inflammation and collagen deposition, depending on its cellular origin. Reciprocal Sparc(-/-) and wild-type (WT) bone marrow chimeras revealed that SPARC expression in host fibroblasts is required and sufficient to induce collagen fibrosis in a proper inflammatory environment. Accordingly, Sparc(-/-) >WT chimeras showed exacerbated inflammation and fibrosis due to the inability of Sparc(-/-) macrophages to down-regulate tumor necrosis …

Pathologymedicine.medical_specialtyAnimals; Bleomycin; Bone Marrow Cells; Chimera; Collagen; Down-Regulation; Fibroblasts; Leukocytes; Macrophages; Mice; Mice Inbred BALB C; Osteonectin; Pneumonia; Pulmonary Fibrosis; Transforming Growth Factor beta; Tumor Necrosis Factor-alphaPulmonary FibrosisDown-RegulationInflammationBone Marrow CellsBiologyPathology and Forensic MedicineMiceFibrosisTumor necrosis factor productionTransforming Growth Factor betaPulmonary fibrosismedicineLeukocytesAnimalsOsteonectinInbred BALB CChimeraTumor Necrosis Factor-alphaMacrophagesMatricellular proteinRegular ArticleSPARCTransforming growth factor betaPneumoniaFibroblastsBLEOMYCINmedicine.diseaseSPARC; BLEOMYCIN; LUNG DAMAGELUNG DAMAGECancer researchbiology.proteinTumor necrosis factor alphaCollagenmedicine.symptomOsteonectin
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Expression of T-cadherin in tumor cells influences invasive potential of human hepatocellular carcinoma

2006

Overexpression of T-cadherin (T-cad) transcripts occurs in approximately 50% of human hepatocellular carcinomas (HCCs). To elucidate T-cad functions in HCC, we examined T-cad protein expression in normal and tumoral human livers and hepatoma cell lines and investigated its influence on invasive potential of HCC using RNA interference silencing of T-cad expression in Mahlavu cells. Whereas T-cad expression was restricted to endothelial cells (EC) from large blood vessels in normal livers, it was up-regulated in sinusoidal EC from 8/15 invasive HCCs. Importantly, in three of them (38%) T-cad was detected in tumor cells within regions in which E-cadherin expression was absent. Among six hepato…

Pathologymedicine.medical_specialtyCarcinoma HepatocellularTranscription GeneticLiver cytologyCell Culture TechniquesMotilityBiologyTransfectionBiochemistryRNA interferenceCell MovementCell Line TumorGeneticsmedicineGene silencingAnimalsHumansNeoplasm Invasivenesscardiovascular diseasesRNA Small InterferingMolecular BiologyDNA PrimersWound Healingprimary tumors cadherin switch cell invasion hepatoma cell lines RNA interferenceLiver NeoplasmsEndothelial CellsTransfectionHCCSFibroblastsCadherinsdigestive system diseasesT-cadherinLiverCell cultureCancer researchHepatocytesRabbitsCell DivisionBiotechnology
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Collagenase-3 (MMP-13) Enhances Remodeling of Three-Dimensional Collagen and Promotes Survival of Human Skin Fibroblasts

2006

Collagenase-3 (MMP-13) is a matrix metalloproteinase capable of cleaving a multitude of extracellular matrix proteins in addition to fibrillar collagens. Human MMP-13 is expressed by fibroblasts in chronic cutaneous ulcers, but not in normally healing adult skin wounds. However, MMP-13 is produced by fibroblasts in adult gingival and in fetal skin wounds characterized by rapid collagen remodeling and scarless healing. Here, we have examined the role of human MMP-13 in remodeling of three-dimensional (3D) collagenous matrix by primary adult human skin fibroblasts. The high level of human MMP-13 expression by fibroblasts achieved by adenoviral gene delivery resulted in potent enhancement of r…

Pathologymedicine.medical_specialtyCell SurvivalHuman skinDermatologyMatrix Metalloproteinase InhibitorsMatrix metalloproteinaseBiologyBiochemistryFilamentous actinAdenoviridaeDermal fibroblastExtracellular matrixMatrix Metalloproteinase 13medicineHumansFibroblastMolecular BiologyCells CulturedCell ProliferationWound HealingTissue Inhibitor of Metalloproteinase-1integumentary systemCell BiologyFibroblastsActinsCell biologyEnzyme ActivationCollagen type I alpha 1medicine.anatomical_structureCollagenaseCollagenmedicine.drugJournal of Investigative Dermatology
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Muscle degeneration in neuramindase 1 deficient mice results from infiltration of the muscle fibers by expanded connective tissue

2010

AbstractNeuraminidase 1 (NEU1) regulates the catabolism of sialoglycoconjugates in lysosomes. Congenital NEU1 deficiency in children is the basis of sialidosis, a severe neurosomatic disorder in which patients experience a broad spectrum of clinical manifestations varying in the age of onset and severity. Osteoskeletal deformities and muscle hypotonia have been described in patients with sialidosis. Here we present the first comprehensive analysis of the skeletal muscle pathology associated with loss of Neu1 function in mice. In this animal model, skeletal muscles showed an expansion of the epimysial and perimysial spaces, associated with proliferation of fibroblast-like cells and abnormal …

Pathologymedicine.medical_specialtyMuscle HypotoniaMuscle Fibers SkeletalNeuraminidaseConnective tissueApoptosisNEU1BiologyArticleMiceNecrosisNEU1SarcolemmaCell MovementSettore BIO/10 - BiochimicamedicineAnimalsSialidosisMuscular dystrophyMyopathyMolecular BiologySialidosiMetalloproteinaseCell ProliferationMice KnockoutMuscle biopsySialidosisECMmedicine.diagnostic_testSkeletal muscleFibroblastsMuscular Dystrophy Animalmedicine.diseaseLysosomeExtracellular MatrixMuscular Atrophymedicine.anatomical_structureConnective TissueImmunologyMolecular MedicineMuscle biopsymedicine.symptom
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Fibroblast activation protein inhibitor (FAPi) positive tumour fraction on PET/CT correlates with Ki-67 in liver metastases of neuroendocrine tumours

2021

Abstract Aim Gallium-68-labelled inhibitors of the fibroblast activation protein (FAPi) enable positron emission tomography/computed tomography (PET/CT) imaging of fibroblast activation. We evaluated if [68Ga]Ga-DATA5m.SA.FAPi PET/CT is related to Ki-67 as a marker of tumour aggressiveness in patients with liver metastases of NET. Methods Thirteen patients with liver metastases of a histologically confirmed NET who underwent PET/CT with [68Ga]Ga-DATA5m.SA.FAPi, [18F]FDG and [68Ga]Ga-DOTA-TOC were retrospectively analyzed. PET-positive liver tumour volumes were segmented for calculation of volume, SUVmax and PET-positive tumour fraction (TF). PET parameters were correlated with Ki-67. Result…

Pathologymedicine.medical_specialtyProliferation index030218 nuclear medicine & medical imaging03 medical and health sciences0302 clinical medicineFibroblast activation protein alphaPositron Emission Tomography Computed TomographymedicineHumansRadiology Nuclear Medicine and imagingIn patientFibroblastGrading (tumors)Retrospective StudiesPET-CTbiologymedicine.diagnostic_testbusiness.industryLiver NeoplasmsGeneral MedicineFibroblastsNeuroendocrine TumorsKi-67 Antigenmedicine.anatomical_structurePositron emission tomography030220 oncology & carcinogenesisKi-67biology.proteinbusinessNuklearmedizin - NuclearMedicine
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Transcriptome Analysis of Ullrich Congenital Muscular Dystrophy Fibroblasts Reveals a Disease Extracellular Matrix Signature and Key Molecular Regula…

2015

Background Collagen VI related myopathies encompass a range of phenotypes with involvement of skeletal muscle, skin and other connective tissues. They represent a severe and relatively common form of congenital disease for which there is no treatment. Collagen VI in skeletal muscle and skin is produced by fibroblasts. Aims & Methods In order to gain insight into the consequences of collagen VI mutations and identify key disease pathways we performed global gene expression analysis of dermal fibroblasts from patients with Ullrich Congenital Muscular Dystrophy with and without vitamin C treatment. The expression data were integrated using a range of systems biology tools. Results were validat…

Pathologymedicine.medical_specialtyUllrich congenital muscular dystrophyIntegrin alpha3Integrinlcsh:MedicineDown-RegulationAscorbic AcidBiologyMuscular DystrophiesExtracellular matrixLamininCollagen VImedicineCell AdhesionHumansGene Regulatory NetworksMuscular dystrophylcsh:ScienceWound HealingMultidisciplinarySclerosisGene Expression Profilinglcsh:RFibroblastsmedicine.diseaseMolecular biologyExtracellular MatrixUp-RegulationGene expression profilingMicroRNAsbiology.proteinlcsh:QWound healingResearch ArticleSignal Transduction
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Clinical heterogeneity in infantile galactosialidosis

1987

A new case of infantile galactosialidosis is presented. The condition was diagnosed when the patient was 4 months of age and she died at 20 months. She exhibited some of the symptoms of classical infantile galactosialidosis but no corneal clouding, cherry-red macular spot or limitation of joint mobility. Sonographic examination showed large kidneys and thickened cardiac septa, two symptoms as yet undescribed in this disorder. Urinary oligosaccharide analysis gave grossly pathological results and subsequent fibroblast enzyme analysis showed a deficiency of alpha-neuraminidase and beta-galactosidase. The patient's clinical features are compared with the few cases so far described in the liter…

Pathologymedicine.medical_specialtyUrinary systemNeuraminidaseOligosaccharidesLarge kidneysKidneyLactose IntoleranceJoint mobilityCorneal cloudingClinical heterogeneityHumansMedicinePathologicalUltrasonographybusiness.industryMyocardiumInfantFibroblastsbeta-Galactosidasemedicine.diseaseGalactosidasesPediatrics Perinatology and Child HealthFemalebusinessGalactosialidosisEuropean Journal of Pediatrics
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Low level 809-nm diode laser-induced in vitro stimulation of the proliferation of human gingival fibroblasts

2002

Background and Objective The authors investigated the effects of low level laser irradiation on the proliferation rate of human gingival fibroblasts (HGF) in vitro. Study Design/Materials and Methods HGF were obtained from gingival connective tissue explants and cultured under standard conditions. 110 cell cultures in their logarithmic growth phase were spread on 96-well tissue culture plates and were irradiated at energy fluences of 1.96–7.84 J/cm2. Another 110 cultures served as control. An 809-nm semiconductor laser operated at a power output of 10 mW in the cw-mode was used. The time of exposure varied between 75 and 300 seconds. Laser treatment was performed alternatively once, twice, …

Pathologymedicine.medical_specialtybusiness.industryLasersmedicine.medical_treatmentGingivaConnective tissueDermatologyFibroblastsIn Vitro TechniquesStatistics NonparametricAndrologyTissue culturemedicine.anatomical_structureRelative fluorescence unitsCell culturemedicineHumansSurgeryIrradiationFibroblastbusinessCells CulturedLow level laser therapyExplant cultureLasers in Surgery and Medicine
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Regulation of Anti-Apoptotic SOD2 and BIRC3 in Periodontal Cells and Tissues.

2021

Made available in DSpace on 2021-06-25T10:49:07Z (GMT). No. of bitstreams: 0 Previous issue date: 2021-01-02 Fundação de Amparo à Pesquisa do Estado de São Paulo (FAPESP) Deutscher Akademischer Austauschdienst Coordenação de Aperfeiçoamento de Pessoal de Nível Superior (CAPES) Deutsche Forschungsgemeinschaft The aim of the study was to clarify whether orthodontic forces and periodontitis interact with respect to the anti-apoptotic molecules superoxide dismutase 2 (SOD2) and baculoviral IAP repeat-containing protein 3 (BIRC3). SOD2, BIRC3, and the apoptotic markers caspases 3 (CASP3) and 9 (CASP9) were analyzed in gingiva from periodontally healthy and periodontitis subjects by real-time PCR…

Periodontium0301 basic medicineGingivaApoptosislcsh:ChemistryGingivitis0302 clinical medicineskin and connective tissue diseases610 Medicine &amp; healthperiodontitislcsh:QH301-705.5Cells CulturedSpectroscopybiologyCaspase 3General MedicineGingivitisBaculoviral IAP Repeat-Containing 3 ProteinCaspase 93. Good healthComputer Science Applicationsorthodontic tooth movementHost-Pathogen Interactionscardiovascular systemmedicine.symptomgingivitismedicine.medical_specialtySOD2610 Medicine & healthArticleCatalysisInorganic ChemistrySuperoxide dismutase03 medical and health sciencesstomatognathic systemOrthodontic tooth movementInternal medicinemedicineAnimalsHumansPeriodontal fiberddc:610Physical and Theoretical ChemistryPeriodontitisMolecular BiologyPeriodontitisFusobacterium nucleatumSuperoxide Dismutasebusiness.industry<i>Fusobacterium nucleatum</i>periodontal ligamentOrganic Chemistry030206 dentistryPeriodontiumFibroblastsmedicine.diseasebiology.organism_classificationRatsstomatognathic diseases030104 developmental biologyEndocrinologyGene Expression Regulationlcsh:Biology (General)lcsh:QD1-999Apoptosisbiology.proteinFusobacterium nucleatumbusinessperiodontiumPeriodontal ligament
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