Search results for "FRAGMENTS"

showing 10 items of 422 documents

Tumor necrosis factor-alpha converting enzyme is processed by proprotein-convertases to its mature form which is degraded upon phorbol ester stimulat…

2003

Tumor necrosis factor-alpha converting enzyme (TACE or ADAM17) is a member of the ADAM (a disintegrin and metalloproteinase) family of type I membrane proteins and mediates the ectodomain shedding of various membrane-anchored signaling and adhesion proteins. TACE is synthesized as an inactive zymogen, which is subsequently proteolytically processed to the catalytically active form. We have identified the proprotein-convertases PC7 and furin to be involved in maturation of TACE. This maturation is negatively influenced by the phorbol ester phorbol-12-myristate-13-acetate (PMA), which decreases the cellular amount of the mature form of TACE in PMA-treated HEK293 and SH-SY5Y cells. Furthermore…

DNA ComplementaryTime FactorsADAM10Blotting WesternGenetic VectorsADAM17 ProteinTransfectionBiochemistryCell LineAmyloid beta-Protein PrecursorAlzheimer DiseaseZymogenEndopeptidasesPhorbol EstersCell AdhesionTumor Cells CulturedAnimalsAspartic Acid EndopeptidasesHumansSubtilisinsProtein kinase A signalingFurinProtein Kinase CProtein kinase CFurinMetalloproteinasebiologyChemistryMetalloendopeptidasesCyclic AMP-Dependent Protein KinasesPeptide FragmentsRatsCell biologyADAM ProteinsEctodomainBiochemistrybiology.proteinTetradecanoylphorbol AcetateCattleTumor necrosis factor alphaProprotein ConvertasesAmyloid Precursor Protein SecretasesSignal TransductionEuropean Journal of Biochemistry
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Design, construction and cooling system performance of a prototype cryogenic stopping cell for the Super-FRS at FAIR

2015

A cryogenic stopping cell for stopping energetic radioactive ions and extracting them as a low energy beam was developed. This first ever cryogenically operated stopping cell serves as prototype device for the Low-Energy Branch of the Super-FRS at FAIR. The cell has a stopping volume that is 1 m long and 25 cm in diameter. Ions are guided by a DC field along the length of the stopping cell and by a combined RF and DC fields provided by an RE carpet at the exit-hole side. The ultra-high purity of the stopping gas required for optimum ion survival is reached by cryogenic operation. The design considerations and construction of the cryogenic stopping cell, as well as some performance character…

Dc fieldNuclear and High Energy PhysicsSPACE-CHARGEPhysics::Instrumentation and DetectorsNuclear engineering7. Clean energy01 natural sciencesIonNuclear physicsSuper-FRSENERGYCryogenic stopping cell0103 physical sciencesWater coolingddc:530FACILITYradioactive ion beams010306 general physicsInstrumentationRADIOACTIVE IONSFinal versionPhysicsCATCHERSPECTROSCOPYta114010308 nuclear & particles physicsCYCLOTRON GAS STOPPERCryocoolerSpace chargeVolume (thermodynamics)13. Climate actionIon catcherRadioactive on beamsFLIGHT MASS-SPECTROMETRYPROJECTILE FRAGMENTSBeam (structure)ION GUIDE
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Role of NK1 and NK2 receptors in mouse gastric mechanical activity

2006

1. The aim of the present study was to examine the role of NK1 and NK2 receptors in the control of mechanical activity of mouse stomach. In this view, the motor effects induced by NK1 and NK2 receptor agonists and antagonists were analyzed, measuring motility as intraluminal pressure changes in mouse-isolated stomach preparations. In parallel, immunohistochemical studies were performed to identify the location of NK1 and NK2 receptors on myenteric neurons and smooth muscle cells. 2. Substance P (SP) induced biphasic effects: a contraction followed by relaxation; neurokinin A (NKA) and [beta-Ala8]-NKA(4-10), selective agonist of NK2 receptors, evoked concentration-dependent contractions, whe…

Dose-Response Relationship Drugsubstance PMice Inbred StrainsReceptors Neurokinin-2Receptors Neurokinin-1ImmunohistochemistrySettore BIO/09 - FisiologiaPeptide FragmentsMice Inbred C57BLMiceneurokinin ANeurokinin-1 Receptor Antagonistsgastric relaxationnitric oxidePapersAnimalsTachykininGastrointestinal Motility
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Interferon-alpha 2a increases serum concentration of hyaluronic acid and type III procollagen aminoterminal propeptide in patients with chronic hepat…

1994

Interferon-alpha (IFN-alpha) has become an important drug for the treatment of chronic viral liver diseases. However, the action of IFN-alpha remains unclear. We investigated whether human recombinant IFN-alpha modulates serum concentrations of hyaluronic acid (HA) and type III procollagen aminoterminal propeptide (P-III-NP) in 56 patients with chronic hepatitis-B under IFN-alpha therapy. IFN-alpha increased the HA serum level in 44 of 46 patients and, after cessation of treatment, HA serum levels returned to the pretherapy levels. The increase of HA serum level was higher in patients with active cirrhosis (aC) than in patients with chronic persistent hepatitis (CPH) and in patients with se…

Drugmedicine.medical_specialtyCirrhosisPhysiologymedia_common.quotation_subjectBiopsyInterferon alpha-2Viruslaw.invention03 medical and health scienceschemistry.chemical_compound0302 clinical medicinelawInternal medicineHyaluronic acidMedicineHumansHyaluronic AcidProtein precursor030304 developmental biologymedia_commonHepatitis Chronic0303 health sciencesbusiness.industryGastroenterologyInterferon-alphaHepatologymedicine.diseaseHepatitis BPeptide FragmentsRecombinant Proteins3. Good healthProcollagen peptidaseEndocrinologychemistry030220 oncology & carcinogenesisImmunologyChronic DiseaseRecombinant DNAbusinessProcollagenFollow-Up StudiesDigestive diseases and sciences
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Gene within gene configuration and expression of the Drosophila melanogaster genes lethal(2) neighbour of tid [l(2)not] and lethal(2) relative of tid…

1997

In this paper, we describe the structure and temporal expression pattern of the Drosophila melanogaster genes l(2)not and l(2)rot located at locus 59F5 vis a vis the tumor suppressor gene l(2)tid described previously and exhibiting a gene within gene configuration. The l(2)not protein coding region, 1530 nt, is divided into two exons by an intron, 2645 nt, harboring the genes l(2)rot, co-transcribed from the same DNA strand, and l(2)tid, co-transcribed from the opposite DNA strand, located vis a vis. To determine proteins encoded by the genes described in this study polyclonal rabbit antibodies (Ab), anti-Not and anti-Rot, were generated. Immunostaining of developmental Western blots with t…

Embryo NonmammalianTranscription GeneticMolecular Sequence DataRestriction MappingGenes Insectmacromolecular substancesBiologyMannosyltransferasesAntibodiesExonTranscription (biology)GeneticsAnimalsDrosophila ProteinsNorthern blotAmino Acid SequenceMicroscopy ImmunoelectronGeneBody PatterningRegulation of gene expressionBase SequenceSequence Homology Amino Acidtechnology industry and agricultureIntronRNAGene Expression Regulation DevelopmentalMembrane ProteinsGeneral MedicineExonsMolecular biologyIntronsPeptide FragmentsAntisense RNADrosophila melanogasterGene Expression RegulationInsect ProteinsRabbitsSequence AlignmentGene
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MAPK3 deficiency drives autoimmunity via DC arming.

2010

DC are professional APC that instruct T cells during the inflammatory course of EAE. We have previously shown that MAPK3 (Erk1) is important for the induction of T-cell anergy. Our goal was to determine the influence of MAPK3 on the capacity of DC to arm T-cell responses in autoimmunity. We report that DC from Mapk3(-/-) mice have a significantly higher membrane expression of CD86 and MHC-II and--when loaded with the myelin oligodendrocyte glycoprotein--show a superior capacity to prime naive T cells towards an inflammatory phenotype than Mapk3(+/+) DC. Nonetheless and as previously described, Mapk3(-/-) mice were only slightly but not significantly more susceptible to myelin oligodendrocyt…

Encephalomyelitis Autoimmune ExperimentalMAP Kinase Signaling SystemOvalbuminImmunologyMedizinAutoimmunityMice TransgenicT-Cell Antigen Receptor SpecificityBiologymedicine.disease_causeAutoimmunityMyelinMiceImmune systemT-Lymphocyte SubsetsmedicineImmunology and AllergyAnimalsNeuroinflammationGlycoproteinsCD86Mitogen-Activated Protein Kinase 3KinaseHistocompatibility Antigens Class IIDendritic Cellsmedicine.diseaseOligodendrocytePeptide FragmentsSpecific Pathogen-Free OrganismsMice Inbred C57BLmedicine.anatomical_structureRadiation ChimeraImmunologyCytokinesMyelin-Oligodendrocyte GlycoproteinB7-2 AntigenInfiltration (medical)European journal of immunology
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Reversible neural stem cell niche dysfunction in a model of multiple sclerosis

2011

Objective The subventricular zone (SVZ) of the brain constitutes a niche for neural stem and progenitor cells that can initiate repair after central nervous system (CNS) injury. In a relapsing-remitting model of experimental autoimmune encephalomyelitis (EAE), the neural stem cells (NSCs) become activated and initiate regeneration during acute disease, but lose this ability during the chronic phases of disease. We hypothesized that chronic microglia activation contributes to the failure of the NSC repair potential in the SVZ. Methods Using bromodeoxyuridine injections at different time points during EAE, we quantified the number of proliferating and differentiating progenitors, and evaluate…

Encephalomyelitis Autoimmune ExperimentalMultiple SclerosisTime FactorsSubventricular zoneCell CountMinocyclineBiologyArticleMiceSOX2Microscopy Electron TransmissionNeural Stem CellsCell MovementmedicineSecondary PreventionAnimalsProgenitor cellStem Cell NicheMyelin Proteolipid ProteinCell ProliferationMicrogliaExperimental autoimmune encephalomyelitismedicine.diseaseNeural stem cellOligodendrocytePeptide FragmentsAnti-Bacterial Agentsnervous system diseasesDisease Models AnimalOligodendrogliamedicine.anatomical_structureNeurologyBromodeoxyuridinenervous systemNeurology (clinical)MicrogliaStem cellNeuroscience
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IL-17A and IL-17F do not contribute vitally to autoimmune neuro-inflammation in mice

2009

The clear association of Th17 cells with autoimmune pathogenicity implicates Th17 cytokines as critical mediators of chronic autoimmune diseases such as EAE. To study the impact of IL-17A on CNS inflammation, we generated transgenic mice in which high levels of expression of IL-17A could be initiated after Cre-mediated recombination. Although ubiquitous overexpression of IL-17A led to skin inflammation and granulocytosis, T cell–specific IL-17A overexpression did not have a perceptible impact on the development and health of the mice. In the context of EAE, neither the T cell–driven overexpression of IL-17A nor its complete loss had a major impact on the development of clinical disease. Sin…

Encephalomyelitis Autoimmune Experimentalmedicine.medical_treatmentT cellEncephalomyelitisPopulation610 Medicine & healthMice TransgenicInflammation2700 General Medicine10263 Institute of Experimental ImmunologyMyelin oligodendrocyte glycoproteinMicemedicineAnimalseducationCells CulturedGlycoproteinseducation.field_of_studybiologybusiness.industryInterleukin-17General MedicineTh1 Cellsmedicine.diseasePeptide FragmentsMice Inbred C57BLCytokinemedicine.anatomical_structureImmunologybiology.protein570 Life sciences; biologyExperimental pathologyFemaleMyelin-Oligodendrocyte GlycoproteinInterleukin 17medicine.symptombusinessGranulocytesResearch Article
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Conical nanopores highlight the pro-aggregating effects of pyrimethanil fungicide on Aβ(1-42) peptides and dimeric splitting phenomena.

2022

International audience; The Aβ(1-42) aggregation is a key event in the physiopathology of Alzheimer's disease (AD). Exogenous factors such as environmental pollutants, and more particularly pesticides, can corrupt Aβ(1-42) assembly and could influence the occurrence and pathophysiology of AD. However, pesticide involvement in the early stages of Aβ(1-42) aggregation is still unknown. Here, we employed conical track-etched nanopore in order to analyse the Aβ(1-42) fibril formation in the presence of pyrimethanil, a widely used fungicide belonging to the anilinopyrimidine class. Our results evidenced a pro-aggregating effect of pyrimethanil on Aβ(1-42). Aβ(1-42) assemblies were successfully d…

Environmental EngineeringAmyloidHealth Toxicology and MutagenesisDimerSettore ING-IND/06track-etchedMolecular dynamicschemistry.chemical_compoundNanoporesFibril formationPEG ratio[CHIM] Chemical SciencesfungicideEnvironmental Chemistry[CHIM]Chemical SciencesnanoporeAmyloid beta-PeptidesChemistryPublic Health Environmental and Occupational HealthamyloidGeneral MedicineGeneral ChemistryPollutionresistive pulsePeptide FragmentsAβ(1-42)Fungicides IndustrialNanoporePyrimidinesAβ(1–42)Biophysicslag phasePyrimethanilChemosphere
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Interaction between N-terminal domain of H4 and DNA is regulated by the acetylation degree.

1998

Abstract To study whether the acetylation of one or more of the four acetylatable lysines of histone H4 affects its binding to DNA, we have designed a protection experiment with a model system consisting in phage lambda DNA as substrate, Stu I as restriction endonuclease and histone H4 with different degrees of acetylation as the protective agent. It can be deduced from the experimental data that the protection afforded by the histone is not dependent on the number of positive charges lost by acetylation. Thus, non-acetylated H4 and mono-acetylated H4 cause similar protection, while di-acetylation of the histone seems to be the crucial step in significantly weakening the interaction between…

ErythrocytesBiophysicsAcetylationDNABiologySAP30Chemical FractionationChromatography Ion ExchangeBiochemistryPeptide FragmentsHistone H4HistonesBiochemistryHistone H1Structural BiologyHistone H2AGeneticsHistone codeNucleosomeAnimalsHistone octamerHistone deacetylaseChickens
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