Search results for "FRAGMENTS"

showing 10 items of 422 documents

Interactions of the hormones leptin, ghrelin, adiponectin, resistin, and PYY3-36 with the reproductive system.

2006

Objective To summarize the effects of novel hormones (leptin, ghrelin, adiponectin, resistin, and PYY3-36) secreted from adipose tissue and the gastrointestinal tract that have been discovered to exert different effects on several reproductive functions, such as the hypothalamic–pituitary–gonadal axis, embryo development, implantation physiology, and clinically relevant conditions. Design A MEDLINE computer search was performed to identify relevant articles. Result(s) Leptin and ghrelin exert important roles on body weight regulation, eating behavior, and reproduction, acting on the central nervous system and target reproductive organs. As a marker of adequate nutritional stores, these horm…

LeptinMalemedicine.medical_specialtyMEDLINEPeptide HormonesAdipose tissueHypothalamic–pituitary–gonadal axisBiologyGenitalia MaleInternal medicineProtein Interaction MappingmedicineAnimalsHumansPeptide YYResistinReproductive systemGonadal Steroid HormonesReproductive functionEvidence-Based MedicineAdiponectinLeptinReproductiondigestive oral and skin physiologyObstetrics and GynecologyGenitalia FemaleGhrelinPeptide FragmentsGastrointestinal TractEndocrinologyReproductive MedicineAdipose TissueGhrelinFemaleAdiponectinhormones hormone substitutes and hormone antagonistsHormoneSignal TransductionFertility and sterility
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Monoclonal anti-fosB antibody specific for predetermined, nonstructural region of the fosB protein.

1997

Comparison of the primary structures and theoretical prediction of the potential antigenic determinant of the deduced Fos proteins reveals the presence of a nonstructural and hydrophilic region juxtaposed to the leucine zipper and nonconserved among the Fos protein family. To develop monoclonal anti-peptide antibodies capable of distinguishing all Fos-proteins, synthetic peptides specific for the mentioned predicted region were synthesized manually by the "tea-bag" method. Immunization of Balb/c mice with fosB-related synthetic peptide BSA gave rise to mouse hybridoma cell line K21 (IgG1, kappa) secreting highly specific antibodies against corresponding human fosB protein. Fine mapping of t…

Leucine zippermedicine.drug_classImmunologyMolecular Sequence DataEnzyme-Linked Immunosorbent AssayMonoclonal antibodyEpitopeMiceAntibody SpecificityGeneticsmedicineAnimalsHumansAmino Acid SequencePeptide sequencebiologyProtein primary structureDrug Resistance MicrobialMolecular biologyPeptide FragmentsEpitope mappingbiology.proteinAntibodyProto-Oncogene Proteins c-fosEpitope MappingFOSBHybridoma
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Anti-inflammatory effects of annexin-1: stimulation of IL-10 release and inhibition of nitric oxide synthesis.

2003

Annexin-1 (ANX-1) is an anti-inflammatory protein induced by glucocorticoids. Like glucocorticoids, ANX-1 and derived peptides inhibit eicosanoid synthesis, block leukocyte migration and induce apoptosis of inflammatory cells. Cytokines may possess either pro-inflammatory, i.e. interleukin(IL)-1beta, tumor necrosis factor (TNF)-alpha, IL-12 or anti-inflammatory properties, i.e. IL-4, IL-10. The experiments described in the present study have been performed to answer the question whether the anti-inflammatory action of ANX-1 may be mediated, at least in part, by the release of IL-10. In macrophage (J774) cell line cultures primed with lipolysaccharide (LPS), recombinant ANX-1 stimulated IL-1…

Leukocyte migrationCell SurvivalImmunologyAnti-Inflammatory AgentsNitric Oxide Synthase Type IINitric OxideNitric oxideCell Linechemistry.chemical_compoundMiceAnnexinImmunology and AllergyAnimalsRNA MessengerEnzyme InhibitorsAnnexin A1PharmacologybiologyReverse Transcriptase Polymerase Chain ReactionMacrophagesInterleukinPeptide FragmentsRecombinant ProteinsCell biologyInterleukin-10Nitric oxide synthasechemistryBiochemistryApoptosisbiology.proteinTumor necrosis factor alphaNitric Oxide SynthaseAnnexin A1International immunopharmacology
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NT pro BNP plasma level and atrial volume are linked to the severity of liver cirrhosis.

2013

BACKGROUND AND AIMS: Plasma levels of NT-pro-BNP, a natriuretic peptide precursor, are raised in the presence of fluid retention of cardiac origin and can be used as markers of cardiac dysfunction. Recent studies showed high levels of NT pro BNP in patients with cirrhosis. We assessed NT pro-BNP and other parameters of cardiac dysfunction in patients with cirrhosis, with or without ascites, in order to determine whether the behaviour of NT pro BNP is linked to the stage of liver disease or to secondary cardiac dysfunction. METHODS: Fifty eight consecutive hospitalized patients mostly with viral or NAFLD-related cirrhosis were studied. All underwent abdominal ultrasound and upper GI endoscop…

Liver CirrhosisMaleCirrhosisAnatomy and Physiologylcsh:MedicineCardiovascularCardiovascular SystemLiver diseaseAscitesNatriuretic Peptide BrainNatriuretic peptidePathologylcsh:ScienceMultidisciplinaryLiver DiseasesMiddle AgedHypertensioncardiovascular systemCardiologyMedicineFemalemedicine.symptomhormones hormone substitutes and hormone antagonistscirculatory and respiratory physiologyResearch Articlemedicine.medical_specialtymedicine.drug_classGastroenterology and HepatologyCardiac dysfunctionDiagnostic MedicineInternal medicinemedicineHumanscardiovascular diseasesHeart Atriabusiness.industryAcute Cardiovascular Problemscirrhosislcsh:RCase-control studyPlasma levelsmedicine.diseasePeptide FragmentsEndocrinologyCase-Control Studieslcsh:QN terminal pro b type natriuretic peptidebusinesshuman activitiesBiomarkersGeneral Pathology
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p42 MAPK phosphorylates 80 kDa MARCKS at Ser-113.

1996

Abstract It is demonstrated here that p42 MAPKinase (p42 MAPK) phosphorylates the M yristoylated A lanine- R ich C - K inase S ubstrate (MARCKS) at Ser-113. In permeabilised Swiss 3T3 cells activation of protein kinase C (PKC) leads to p42 MAPK activation, but only the protein kinase C sites in MARCKS become phosphorylated and not Ser-113. The mitogen platelet-derived growth factor (PDGF) elicits the same response. These results demonstrate that while Ser-113 is a substrate for p42 MAPK in vitro and can be phosphorylated in vivo as shown by Taniguchi et al. [(1994) J. Biol. Chem. 269, 18299–18302], its phosphorylation is not subject to acute regulation by p42 MAPK in Swiss 3T3 cells.

MAPK/ERK pathwayMARCKSmedicine.medical_treatmentMitogen-activated protein kinase kinaseBiochemistryenvironment and public healthSubstrate SpecificityMiceStructural BiologySerinep42MAPKinasePhosphorylationMyristoylated Alanine-Rich C Kinase SubstrateCells CulturedProtein Kinase CMitogen-Activated Protein Kinase 1Platelet-Derived Growth FactorbiologyChemistryIntracellular Signaling Peptides and Proteins3T3 CellsProtein-Tyrosine KinasesCell biologyBiochemistryMitogen-activated protein kinasePhosphorylationTetradecanoylphorbol Acetatebiological phenomena cell phenomena and immunityPlatelet-derived growth factor receptorhormones hormone substitutes and hormone antagonistsendocrine systemRecombinant Fusion ProteinsMolecular Sequence DataBiophysicsGeneticsmedicineAnimalsAmino Acid SequenceMARCKSMolecular BiologyProtein kinase CGrowth factorMembrane ProteinsProteinsCell BiologyPeptide FragmentsEnzyme ActivationMolecular Weightenzymes and coenzymes (carbohydrates)Calcium-Calmodulin-Dependent Protein Kinasesbiology.proteinMutagenesis Site-DirectedMitogensFEBS letters
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Onco-fetal/laminin-binding collagen from colon carcinoma: detection of new sequences.

1995

We have recently identified an oncofetal-laminin binding collagen (OF/LB) composed of three alpha chains, with the apparent molecular mass of about 100 kDa each, but bearing different pI. One of the chains appears markedly acidic in a bidimensional electrophoretic system, where the NEPHGE is used as first dimension separating gel, while the two more basic chains have similar migration as alpha 1(III) and alpha 1(I) collagen chains, respectively. Sequence analyses have been performed on CNBr-peptides, derived from pepsinized triple helical molecules and on tryptic fragments obtained after in gel digestion of the acidic band. The research of sequence homology with computerized databases indic…

Macromolecular SubstancesBiopsyMolecular Sequence DataBiophysicsSequence (biology)In-gel digestionBiochemistryMoleculeHumansElectrophoresis Gel Two-DimensionalTrypsinAmino Acid SequenceCyanogen BromideLaminin bindingMolecular BiologyGeneChromatography High Pressure LiquidFetusMolecular massSequence Homology Amino AcidChemistryCell BiologyMolecular biologyPeptide FragmentsElectrophoresisBiochemistryColonic NeoplasmsCollagenBiochemical and biophysical research communications
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An NMR Study of the Interaction of 15N-Labelled Bradykinin with an Antibody Mimic of the Bradykinin B2 Receptor

1997

An isotope-edited NMR study of the peptide hormone bradykinin (RPPGFSPFR) bound to the Fab fragment of a monoclonal antibody against bradykinin (MBK3) is reported. MBK3 was previously shown to provide a binding site model of the B2 bradykinin receptor [Haasemann, M., Buschko, J., Faussner, A., Roscher, A. A., Hoebeke, J., Burch, R. M. & Muller-Esterl, W. (1991) Anti-idiotypic antibodies bearing the internal image of a bradykinin epitope, J. Immunol. 147, 3882-3892]. Bradykinin was obtained in a uniformly 15N-labelled form using recombinant expression of a fusion protein consisting of the glutathione-binding domain of glutathione S-transferase fused to residues 354-375 of the high-molecular-…

Magnetic Resonance SpectroscopyReceptor Bradykinin B2Protein ConformationStereochemistryRecombinant Fusion ProteinsBradykininIn Vitro TechniquesBradykininBiochemistryImmunoglobulin Fab FragmentsMicechemistry.chemical_compoundAnimalsHumansAmino Acid SequenceBradykinin receptorDNA PrimersKininogenBinding SitesBase SequenceNitrogen IsotopesChemistryReceptors BradykininImmunoglobulin Fab FragmentsProteolytic enzymesAntibodies MonoclonalNuclear magnetic resonance spectroscopyB2 Bradykinin ReceptorTwo-dimensional nuclear magnetic resonance spectroscopyProtein BindingEuropean Journal of Biochemistry
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Intake of Vitamin K Antagonists and Worsening of Cardiac and Vascular Disease: Results From the Population‐Based Gutenberg Health Study

2018

Background Preclinical data have indicated a link between use of vitamin K antagonists ( VKA ) and detrimental effects on vascular structure and function. The objective of the present study was to determine the relationship between VKA intake and different phenotypes of subclinical cardiovascular disease in the population. Methods and Results Clinical and laboratory data, as well as medical–technical examinations were assessed from 15 010 individuals aged 35 to 74 years during a highly standardized 5‐hour visit at the study center of the population‐based Gutenberg Health Study. In total, the study sample comprised 287 VKA users and 14 564 VKA nonusers. Multivariable analysis revealed an in…

Male0301 basic medicineEpidemiologyPROGRESSION030204 cardiovascular system & hematologyVitamin kCarotid Intima-Media ThicknessTHERAPYGastroenterologyAdrenomedullin0302 clinical medicineRisk Factorscardiovascular diseaseGermanyAtrial FibrillationNatriuretic Peptide BrainMatrix gla proteinOriginal ResearchVenous Thrombosisoral anticoagulationRISKbiologyMiddle AgedStrokevitamin K antagonistsC-Reactive ProteinCardiovascular DiseasesFemaleCardiology and Cardiovascular MedicineAtrial Natriuretic Factormedicine.drugAdultmedicine.medical_specialtyPopulation basedMATRIX GLA-PROTEINWARFARIN03 medical and health sciencesVascular StiffnessInternal medicineORAL ANTICOAGULANTmedicineHumansAnkle Brachial IndexVascular structureProtein PrecursorsAgedInflammationVascular diseasebusiness.industryWarfarinAnticoagulantsFibrinogenStroke Volumepharmacogenomic variantsARTERIALmedicine.diseasePreclinical dataPeptide FragmentsCALCIFICATION030104 developmental biologyAsymptomatic DiseasesPhenprocoumonbiology.proteinPulmonary EmbolismbusinessCalcificationJournal of the American Heart Association
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Impact of Resilience on the Association Between Amyloid-β and Longitudinal Cognitive Decline in Cognitively Healthy Older Adults

2019

The present study aims at investigating if the association between amyloid-β and longitudinal cognitive decline in cognitively healthy elderly is modulated by resilience capacity. Resilience capacity was quantified by education, which is a common proxy of resilience and has been shown to be related to a wide range of behaviors promoting resilience. Analyses were conducted with longitudinal cognitive data from the Alzheimer's Disease Neuroimaging Initiative (ADNI). 276 cognitively healthy older individuals (≥56 years) were included in the study. Baseline amyloid pathology was quantified using CSF amyloid-β 1-42 measurements. Longitudinal cognitive decline was assessed using ADAS13, Clinical …

Male0301 basic medicineGerontologyAmyloid pathologyAmyloid βClinical Dementia RatingDiseaseNeuropsychological Tests03 medical and health sciences0302 clinical medicineNeuroimagingAlzheimer Diseasemental disordersHumansMedicineCognitive DysfunctionLongitudinal StudiesCognitive declineAgedAged 80 and overAmyloid beta-Peptidesbusiness.industryGeneral NeuroscienceCognitionGeneral MedicineMiddle AgedResilience PsychologicalPeptide FragmentsPsychiatry and Mental healthClinical Psychology030104 developmental biologyMixed effectsFemaleGeriatrics and GerontologybusinessBiomarkers030217 neurology & neurosurgeryFollow-Up StudiesJournal of Alzheimer's Disease
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Outcome of urgent and elective percutaneous coronary interventions after pharmacologic reperfusion with tenecteplase combined with unfractionated hep…

2003

Item does not contain fulltext OBJECTIVES: The aim of this study was to evaluate percutaneous coronary intervention (PCI) in the Assessment of the Safety and Efficacy of New Thrombolytic Regimens (ASSENT-3) trial. BACKGROUND: In the ASSENT-3 trial, co-therapy with abciximab (ABC) or enoxaparin (ENOX) reduced ischemic complications after ST-elevation acute myocardial infarction treated with tenecteplase when compared with unfractionated heparin (UFH). The effect of these new co-therapies on the results of PCI is unknown. METHODS: Clinical outcomes in patients who received co-therapy with ABC, ENOX, or UFH and subsequently underwent an elective (n = 1,064) or urgent (n = 716) PCI in the ASSEN…

MaleAbciximabmedicine.medical_treatmentMyocardial InfarctionAlbertaBelgiumRecurrenceGermanyAbciximabAngioplasty Balloon CoronaryHeart lung and circulation [UMCN 2.1]Netherlandseducation.field_of_studyAntibodies MonoclonalMiddle AgedTreatment OutcomeItalyElective Surgical ProceduresTissue Plasminogen ActivatorDrug Therapy CombinationFemaleCardiology and Cardiovascular MedicineEnoxaparin sodiummedicine.drugmedicine.medical_specialtymedicine.drug_classPopulationLow molecular weight heparinTenecteplasePlatelet Glycoprotein GPIIb-IIIa ComplexDrug Administration ScheduleImmunoglobulin Fab FragmentsFibrinolytic AgentsInternal medicineNorth CarolinamedicineHumansEnoxaparineducationEmergency TreatmentSwedenHeparinbusiness.industryAnticoagulantsPercutaneous coronary interventionSurvival AnalysisSurgerySpainConventional PCITenecteplasebusinessFibrinolytic agentJournal of the American College of Cardiology
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