Search results for "Fibril"

showing 10 items of 836 documents

Fibril formation and toxicity of the non-amyloidogenic rat amylin peptide.

2012

Full-length native rat amylin 1-37 has previously been widely shown to be unable to form fibrils and to lack the toxicity of the human amylin form leading to its use as a non-amyloidogenic control peptide. A recent study has suggested that rat amylin 1-37 forms amyloidogenic β-sheet structures in the presence of the human amylin form and suggested that this property could promote toxicity. Using TEM analysis we show here fibril formation by synthetic rat amylin 1-37 and 8-37 peptides when the lyophilized HPLC purified peptides are initially dissolved in 20 mM Tris-HCl. Dissolution of synthetic rat amylin 1-37 and 8-37 peptides in H(2)O or phosphate buffered saline failed to produce fibrils.…

endocrine systemAmyloidendocrine system diseasesAmyloidmedicine.drug_classGeneral Physics and AstronomyAmylinPeptideAmyloidogenic Proteinsmacromolecular substancesFibrilAmino Acid Chloromethyl Ketoneschemistry.chemical_compoundIslets of LangerhansMicroscopy Electron TransmissionStructural BiologymedicineAnimalsHumansGeneral Materials ScienceCells Culturedchemistry.chemical_classificationgeographygeography.geographical_feature_categoryCell BiologyIsletReceptor antagonistCatalasePeptide FragmentsCongo redIslet Amyloid PolypeptideRatsNeuroprotective AgentsBiochemistrychemistryCell cultureMicron (Oxford, England : 1993)
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Cardiac changes in subclinical and overt hyperthyroid women: retrospective study

2003

This retrospective and descriptive 4-year study was undertaken to describe cardiac changes in subclinical and overt hyperthyroidism.We revised the charts of 386 consecutive cardiopathic women whose blood samples were referred to the Radioimmunoassay Laboratory to evaluate thyroid function from 1 January 1997 through 31 December 2000.After excluding women because euthyroid or hypothyroid, or taking amiodarone and women with hypertension, rheumatic disease, myocardial infarction, a total of 31 hyperthyroid women were thus selected for the study: 19 with subclinical hyperthyroidism and 12 with overt hyperthyroidism. The prevalence of atrial fibrillation did not differ between subclinical (48%)…

endocrine systemmedicine.medical_specialtyHeart Diseasesendocrine system diseasesHeart diseaseConcentric hypertrophyThyroid Function TestsAmiodaroneHyperthyroidismStatistics NonparametricInternal medicinemedicineHumansEuthyroidMyocardial infarctionAgedRetrospective StudiesSubclinical infectionAnalysis of Variancebusiness.industryAtrial fibrillationMiddle Agedmedicine.diseaseCase-Control StudiesLinear ModelsCardiologyFemaleThyroid functionCardiology and Cardiovascular Medicinebusinesshormones hormone substitutes and hormone antagonistsmedicine.drugInternational Journal of Cardiology
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Transgenic overexpression of corticotropin releasing hormone provides partial protection against neurodegeneration in an in vivo model of acute excit…

2008

Abstract Corticotropin releasing hormone (CRH) is the central modulator of the mammalian hypothalamic–pituitary–adrenal (HPA) axis. In addition, CRH affects other processes in the brain including learning, memory, and synaptic plasticity. Moreover, CRH has been shown to play a role in nerve cell survival under apoptotic conditions and to serve as an endogenous neuroprotectant in vitro . Employing mice overexpressing murine CRH in the CNS, we observed a differential response of CRH-overexpressing mice (CRH-COE hom -Nes) to acute excitotoxic stress induced by kainate compared with controls (CRH-COE con -Nes). Interestingly, CRH-overexpression reduced the duration of epileptic seizures and pre…

endocrine systemmedicine.medical_specialtyIndolesRNA UntranslatedCorticotropin-Releasing HormoneExcitotoxicityMice TransgenicNerve Tissue ProteinsBiologymedicine.disease_causeNeuroprotectionHippocampusNestinCorticotropin-releasing hormoneMiceIntermediate Filament ProteinsNeurotrophic factorsNeurofilament ProteinsSeizuresInternal medicineGlial Fibrillary Acidic Proteinpolycyclic compoundsmedicineExcitatory Amino Acid AgonistsReaction TimeAnimalsNeuroinflammationBrain-derived neurotrophic factorAnalysis of VarianceKainic AcidCell DeathGeneral NeuroscienceBrain-Derived Neurotrophic FactorNeurodegenerationProteinsLong-term potentiationmedicine.diseaseDisease Models AnimalEndocrinologynervous systemGene Expression RegulationNerve DegenerationNeurotoxicity SyndromesPlant Lectinshormones hormone substitutes and hormone antagonistsNeuroscience
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Influence of Oral Contraceptive Use on Adaptations to Resistance Training

2019

Introduction: The majority of young women use oral contraceptives (OCs). Use of OCs has been associated with lower myofibrillar protein and tendon collagen synthesis rates, but it is unknown whether OCs will limit the adaptive response of myotendinous tissue to resistance training. Design and Methods: Fourteen healthy untrained young regular OC users (24 +/- 1 years, fat% 32 +/- 1, 35 +/- 2 ml.min(-1).kg(-1)) and 14 NOC users (non-OC, controls) (24 +/- 1 years, fat% 32 +/- 2, 34 +/- 2 ml.min(-1).kg(-1)) performed a 10-week supervised lower extremity progressive resistance training program. Before and after the intervention biopsies from the vastus lateralis muscle and the patellar tendon we…

estrogeenitmedicine.medical_specialtyestradioliTESTOSTERONE LEVELStendonPhysiologyVastus lateralis muscleUrologyIsometric exerciselcsh:PhysiologyMuscle hypertrophy03 medical and health sciences0302 clinical medicinePhysiology (medical)estradiolmedicineestrogenmuscle hypertrophyIN-VIVOHUMAN ACHILLES-TENDONOriginal Researchlcsh:QP1-981ehkäisypilleritexerciseMUSCLE-FIBER SIZETENDON COLLAGEN-SYNTHESISbusiness.industryResistance training030229 sport sciencesMECHANICAL-PROPERTIESTendonmedicine.anatomical_structureContraceptive uselihasmassaBIOMECHANICAL PROPERTIESMuscle strengthmuscle strengthPROTEIN-SYNTHESISSKELETAL-MUSCLEvoimaharjoitteluwomenMENSTRUAL-CYCLEbusinessMyofibril030217 neurology & neurosurgerylihasvoima
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FIBRILLAZIONE ATRIALE E DETERIORAMENTO COGNITIVO CEREBRALE

2010

fibrillazione atriale
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RUOLO DELLA SINDROME METABOLICA NELLA FIBRILLAZIONE ATRIALE

2010

fibrillazione atriale sindrome metabolica
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Entrapment of Aβ(1-40) peptide in unstructured aggregates

2012

Recognizing the complexity of the fibrillogenesis process provides a solid ground for the development of therapeutic strategies aimed at preventing or inhibiting protein-protein aggregation. Under this perspective, it is meaningful to identify the possible aggregation pathways and their relative products. We found that Aβ-peptide dissolved in a pH 7.4 solution at small peptide concentration and low ionic strength forms globular aggregates without typical amyloid β-conformation. ThT binding kinetics was used to monitor aggregate formation. Circular dichroism spectroscopy, AFM imaging, static and dynamic light scattering were used for structural and morphological characterization of the aggre…

fibrillogenesis
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Fibrillogenesis of Hen Egg-White Lysozyme at acid pH

2009

fibrillogenesis lysozyme electrostatic interactions
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Role of repulsive interaction on the fibrillogenesis of hen egg-white lysozyme

2010

fibrillogenesis lysozyme oligomer electrostatic interaction
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Fibrillogenesis of hen egg-white lysozyme at acidic pH

2009

fibrillogenesis lysozyme oligomers electrostatic interactionSettore FIS/07 - Fisica Applicata(Beni Culturali Ambientali Biol.e Medicin)
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