Search results for "Filament"

showing 10 items of 405 documents

Complexity of expression of intermediate filament proteins, including glial filament protein, in endometrial and ovarian adenocarcinomas

1991

The expression patterns of intermediate filament proteins of primary and metastatic endometrial (n = 18) and ovarian (n = 24) adenocarcinomas were analyzed by immunocytochemistry using a panel of specific antibodies and by gel electrophoresis of cytoskeletal preparations, followed by immunoblotting. All cells of all endometrial adenocarcinomas studied contained the "simple epithelial"-type cytokeratins (CKs) 8, 18, and (mostly) 19, with variable numbers of cells also positive for CK 7 and vimentin. In addition, most of these tumors contained individual cells or groups of cells that were positive for the stratification-related CKs 4, 5, 6, 13, 14, and 17. The latter CKs were often associated…

AdultPathologymedicine.medical_specialtyCellular differentiationImmunocytochemistryVimentinAdenocarcinomaPathology and Forensic MedicineImmunoenzyme TechniquesIntermediate Filament ProteinsOvarian carcinomaGlial Fibrillary Acidic ProteinmedicineHumansIntermediate filamentAgedOvarian NeoplasmsbiologyCarcinomaMiddle Agedmedicine.diseaseEndometrial NeoplasmsSerous fluidMicroscopy Fluorescencebiology.proteinAdenocarcinomaElectrophoresis Polyacrylamide GelFemaleClear cellHuman Pathology
researchProduct

Primary desminopathies.

2007

•  Introduction •  Desmin is an essential component of the extrasarcomeric cytoskeleton in striated muscle cells •  Distal myopathy,cardiac arrhythmias,cardiomyopathy:classical criteria of primary desminopathies •  Sub-sarcolemmal and cytoplasmic desmin-positive protein aggregates:the morphological hallmark of primary and secondary desminopathies •  The spectrum of pathogenic desmin gene mutations •  The molecular pathogenesis of primary desminopathies: some answers gained,but even more questions raised •  Diagnostic work-up to distinguish primary from secondary desminopathies •  Treatment and clinical management of primary desminopathy patients Abstract Mutations of the human desmin gene o…

Pathologymedicine.medical_specialtyintermediate filamentsCardiomyopathyReviewsgranulofilamentous materialdesmininclusion bodiesmacromolecular substancesBiologymyofibrillar myopathyprotein aggregationdesmin-related myopathySarcolemmaMuscular DiseasesmedicineMyocyteAnimalsHumansIntermediate filamentMyopathyMuscle SkeletalCytoskeletonGenetic heterogeneityCardiac muscleCell Biologymedicine.diseasemusculoskeletal systemmutationsmedicine.anatomical_structuredesminopathyMutationMolecular MedicineDesminmedicine.symptomMyofibrilJournal of cellular and molecular medicine
researchProduct

Transverse nonlinear optics in heavy-metal-oxide glass

2008

6 pags. ; 9 figs.

PhysicsCondensed matter physicsmodulational instabilityComputer Science::Information RetrievalNonlinear opticsComputer Science::Computation and Language (Computational Linguistics and Natural Language and Speech Processing)InstabilityAtomic and Molecular Physics and Opticsnonlinear glassemultiple filamentationFilamentationPicosecondspatial solitonself-focusingAbsorption (logic)Atomic physicsPhase conjugationSelf-phase modulationBeam (structure)
researchProduct

Formin 2 links neuropsychiatric phenotypes at young age to an increased risk for dementia

2017

Age-associated memory decline is due to variable combinations of genetic and environmental risk factors. How these risk factors interact to drive disease onset is currently unknown. Here we begin to elucidate the mechanisms by which post-traumatic stress disorder (PTSD) at a young age contributes to an increased risk to develop dementia at old age. We show that the actin nucleator Formin 2 (Fmn2) is deregulated in PTSD and in Alzheimer's disease (AD) patients. Young mice lacking the Fmn2 gene exhibit PTSD-like phenotypes and corresponding impairments of synaptic plasticity, while the consolidation of new memories is unaffected. However, Fmn2 mutant mice develop accelerated age-associated me…

0301 basic medicineMalememoriaAginggenetics [Stress Disorders Post-Traumatic]Diseasegenetics [Neuronal Plasticity]BioinformaticsdemenciaStress Disorders Post-TraumaticMice0302 clinical medicineRisk FactorsNews & ViewsAge of OnsetMice KnockoutNeuronal PlasticitybiologyGeneral NeuroscienceMicrofilament ProteinsNuclear Proteinsgenetics [Nuclear Proteins]FearadultoMiddle AgedAlzheimer's diseasephysiology [Aging]Phenotype3. Good healthPhenotypemiedoFormin 2Forminsgenetics [Aging]estres postraumaticoepidemiology [Stress Disorders Post-Traumatic]AdultHDAC inhibidorpsychology [Dementia]alzheimerForminsNerve Tissue Proteinsepidemiology [Dementia]Affect (psychology)General Biochemistry Genetics and Molecular Biology03 medical and health sciencesHDAC inhibitorMemorygenetics [Dementia]ddc:570medicineDementiaAnimalsHumansenvejecimientoMolecular Biologyphysiology [Memory]General Immunology and MicrobiologyPost-traumatic stress disordermedicine.diseaseYoung age030104 developmental biologyformin 2 protein mouseCase-Control StudiesSynaptic plasticitybiology.proteinDementiagenetics [Microfilament Proteins]complications [Stress Disorders Post-Traumatic]030217 neurology & neurosurgeryHomeostasis
researchProduct

Measurement of high order Kerr refractive index of major air components: erratum

2010

A clarification is missing concerning the high order Kerr non-linearities deduced from our experimental data published in [Opt. Express 17, 13429-13434 (2009)]. Here, we rectify this omission by making explicit the distinction between cross-Kerr and Kerr effects, and by extrapolating the value of the nonlinear refractive index for the last effect. Since the occurrence of sign inversion in the Kerr effect is not affected, the overall report in [Opt. Express 17, 13429-13434] remains valid.

Kerr effect[ PHYS.PHYS.PHYS-ATOM-PH ] Physics [physics]/Physics [physics]/Atomic Physics [physics.atom-ph](320.2250) Femtosecond phenomena; (350.5400) Plasmas; (190.7110) Ultrafast nonlinear optics; (260.5950) Self-focusing01 natural sciences010309 opticsOptics0103 physical sciencesZ-scan techniqueHigh order[PHYS.PHYS.PHYS-ATOM-PH] Physics [physics]/Physics [physics]/Atomic Physics [physics.atom-ph]010306 general physicsfemtosecondLaser beamsplasmaPhysics[PHYS.PHYS.PHYS-ATOM-PH]Physics [physics]/Physics [physics]/Atomic Physics [physics.atom-ph]Molecular alignmentbusiness.industrySelf-focusingNonlinear refractive indexPolarization (waves)Atomic and Molecular Physics and Opticslaser filamentationbusinessRefractive index) Ultrafast nonlinear optics
researchProduct

Booster vaccination after neonatal priming with acellular pertussis vaccine.

2010

After a birth dose of acellular pertussis (aP) and diphtheria (DT)aP-hepatitis B virus (HBV)-inactivated polio vaccine (IPV)/ Haemophilus influenza type b (Hib) at 2, 4, and 6 months, a booster dose of DTaP-HBV-IPV/Hib at 12 to 23 months induced strong anti-pertussis booster responses. Thus, neonatal aP priming did not lead to immune tolerance to pertussis antigens. However, it elicited bystander interference on HBV, Hib, and diphtheria responses.

Whooping CoughFilamentous haemagglutinin adhesinImmunization SecondaryBooster dosemedicine.disease_causecomplex mixturesVirusPolio vaccineVaccines AcellularmedicineHumansWhooping coughHepatitis B virusPertussis VaccineDose-Response Relationship Drugbusiness.industryDiphtheriaVaccinationInfant Newbornvirus diseasesInfantmedicine.diseasePrognosisVirologyVaccinationPediatrics Perinatology and Child HealthImmunologybusinessFollow-Up StudiesThe Journal of pediatrics
researchProduct

Effects of acclimation time and epigenetic mechanisms on growth of Neurospora in fluctuating environments

2017

AbstractReaction norms or tolerance curves have often been used to predict how organisms deal with fluctuating environments. A potential drawback is that reaction norms measured in different constant environments may not capture all aspects of organismal responses to fluctuating environments. We examined growth of the filamentous fungusNeurospora crassain fluctuating temperatures and tested if growth in fluctuating temperatures can be explained simply by growth in different constant temperatures or if more complex models are needed. In addition, as previous studies on fluctuating environments have revealed that past temperatures that organisms have experienced can affect their response to c…

0106 biological sciences0301 basic medicineAcclimatizationMutantEnvironmentMethylation010603 evolutionary biology01 natural sciencesAcclimatizationNeurosporaArticleEpigenesis GeneticNeurospora crassaHistones03 medical and health sciencesGeneticsEpigeneticsGenetics (clinical)030304 developmental biology0303 health sciencesbiologyCell CyclefungiTemperatureAcetylationDNA MethylationModels Theoreticalbiology.organism_classificationFilamentous fungusNeurospora030104 developmental biologyRNA Interference PathwayH3k4 methylationDNA methylationBiophysicsGene-Environment InteractionRNA Interference
researchProduct

Chronic ethanol exposure alters the levels, assembly, and cellular organization of the actin cytoskeleton and microtubules in hippocampal neurons in …

2010

The organization and dynamics of microtubules (MTs) and the actin cytoskeleton are critical for the correct development and functions of neurons, including intracellular traffic and signaling. In vitro ethanol exposure impairs endocytosis, exocytosis, and nucleocytoplasmic traffic in astrocytes and alters endocytosis in cultured neurons. In astrocytes, these effects relate to changes in the organization and/or function of MTs and the actin cytoskeleton. To evaluate this possibility in hippocampal cultured neurons, we analyzed if chronic ethanol exposure affects the levels, assembly, and cellular organization of both cytoskeleton elements and the possible underlying mechanisms of these effec…

rho GTP-Binding ProteinsRHOAArp2/3 complexmacromolecular substancesToxicologyFilamentous actinHippocampusMicrotubulesActin cytoskeleton organizationActin remodeling of neuronsAnimalsCytoskeletonCells CulturedCytoskeletonNeuronsbiologyEthanolCentral Nervous System DepressantsActin cytoskeletonActinsCell biologyRatsSomatodendritic compartmentbiology.proteinFemaleSignal TransductionToxicological sciences : an official journal of the Society of Toxicology
researchProduct

Silintaphin-1 - interaction with silicatein during structure-guiding bio-silica formation

2011

Silicateins are unique enzymes of sponges (phylum Porifera) that template and catalyze the polymerization of nanoscale silicate to siliceous skeletal elements. These multifunctional spicules are often elaborately shaped, with complex symmetries. They carry an axial proteinaceous filament, consisting of silicatein and the scaffold protein silintaphin-1, which guides silica deposition and subsequent spicular morphogenesis. In vivo, the synthesis of the axial filament very likely proceeds in three steps: (a) assembly of silicatein monomers to form one pentamer; (b) assembly of pentamers to form fractal-like structures; and finally (c) assembly of fractal-like structures to form filaments. The …

chemistry.chemical_classification0303 health sciencesbiologyStereochemistryPentamerSubstrate (chemistry)02 engineering and technologyCell BiologyPolymer021001 nanoscience & nanotechnologybiology.organism_classificationBiochemistryTetraethyl orthosilicateSuberites domunculaProtein filament03 medical and health scienceschemistry.chemical_compoundMonomerchemistryPolymerizationBiophysics0210 nano-technologyMolecular Biology030304 developmental biologyFEBS Journal
researchProduct

Deletion of GLX3 in Candida albicans affects temperature tolerance, biofilm formation and virulence.

2018

Candida albicans is a predominant cause of fungal infections in mucosal tissues as well as life-threatening bloodstream infections in immunocompromised patients. Within the human body, C. albicans is mostly embedded in biofilms, which provides increased resistance to antifungal drugs. The glyoxalase Glx3 is an abundant proteomic component of the biofilm extracellular matrix. Here, we document phenotypic studies of a glx3Δ null mutant concerning its role in biofilm formation, filamentation, antifungal drug resistance, cell wall integrity and virulence. First, consistent with its function as glyoxalase, the glx3 null mutant showed impaired growth on media containing glycerol as the carbon sou…

0106 biological sciencesHot TemperatureMutantAntifungal drugHyphaeVirulence01 natural sciencesApplied Microbiology and BiotechnologyMicrobiologyMicrobiology03 medical and health sciencesFilamentationCell Wall010608 biotechnologyCandida albicansAnimalsCandida albicans030304 developmental biology0303 health sciencesMice Inbred BALB CbiologyVirulenceBiofilmWild typeCandidiasisGeneral Medicinebiology.organism_classificationAldehyde OxidoreductasesSurvival AnalysisCorpus albicansDisease Models AnimalBiofilmsGene DeletionHeat-Shock ResponseFEMS yeast research
researchProduct