Search results for "Fluoxetin"

showing 10 items of 38 documents

Are the effects of the antidepressants amitriptyline, maprotiline, and fluoxetine on inhibitory avoidance state-dependent?

2005

Abstract State-dependent learning (SDL) is a phenomenon in which the retrieval of newly acquired information is possible if the subject is in the same physiological state as during the encoding phase. SDL makes it possible to separate the effects of drugs per se on learning from the effects due to changes in drug state during the task. The present work was designed to investigate whether the antidepressants amitriptyline (30 mg/kg), maprotiline (25 mg/kg), and fluoxetine (15 mg/kg) produce SDL of the inhibitory avoidance conditioning in male and female CD1 mice. In three separate experiments, independent groups were used for each pharmacological treatment and for each sex using a 2 × 2 expe…

Malemedicine.medical_specialtyAmitriptylinePharmacologyMiceBehavioral Neurosciencechemistry.chemical_compoundSex FactorsFluoxetineAvoidance LearningmedicineAnimalsAmitriptylineNeurotransmitterPsychiatryMaprotilineFluoxetineBehavior AnimalAntidepressive AgentsInhibition PsychologicalMaprotilinechemistryFacilitationConditioningFemaleSerotoninReuptake inhibitorPsychologymedicine.drugBehavioural Brain Research
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Effects of fluoxetine on behavioral deficits evoked by chronic social stress in rats.

2006

Recently, we described an advanced model of chronic social stress in male rats based on the resident intruder paradigm. In this model, rats subjected to daily social stress for 5 weeks showed behavioral changes resembling anhedonia and motivational deficits in humans. In the present study, male Wistar rats were subjected to 5 weeks of daily social defeat by an aggressive conspecific and concomitant treatment with the antidepressant drug fluoxetine (10 mg/kg) after the first week of stress. Compared with controls, rats exposed to chronic stress had significantly reduced locomotor and exploratory activity (rearing and sniffing) and diminished preference for sucrose solution. These effects wer…

Malemedicine.medical_specialtyBehavioral SymptomsMotor ActivitySocial defeat03 medical and health sciencesBehavioral NeuroscienceFood Preferences0302 clinical medicineSniffingInternal medicineFluoxetineAdrenal GlandsmedicineAnimalsChronic stressTestosteroneRats WistarTestosterone030304 developmental biologySocial stress0303 health sciencesFluoxetineAnalysis of VarianceBehavior AnimalBody WeightAnhedoniaRatsEndocrinologyExploratory BehaviorAntidepressantAntidepressive Agents Second-Generationmedicine.symptomPsychology030217 neurology & neurosurgeryStress Psychologicalmedicine.drugBehavioural brain research
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Chronic Fluoxetine Treatment Increases the Expression of PSA-NCAM in the Medial Prefrontal Cortex

2006

Recent hypotheses suggest that changes in neuronal structure and connectivity may underlie the etiology of depression. The medial prefrontal cortex (mPFC) is affected by depression and shows neuronal remodeling during adulthood. This plasticity may be mediated by the polysialylated form of the neural cell adhesion molecule (PSA-NCAM), which is intensely expressed in the adult mPFC. As the expression of PSA-NCAM is increased by serotonin in other cerebral regions, antidepressants acting on serotonin reuptake may influence PSA-NCAM expression and thus counteract the effects of depression by modulating neuronal structural plasticity. Using immunohistochemistry, we have studied the relationship…

Malemedicine.medical_specialtyInterneuronFluorescent Antibody TechniquePrefrontal CortexCell CountNeural Cell Adhesion Molecule L1urologic and male genital diseasesSerotonergicRats Sprague-Dawleychemistry.chemical_compoundFluoxetineInternal medicinemedicineNeuropilAnimalsPrefrontal cortexNeurotransmitter5-HT receptorNeuronsPharmacologyAnalysis of VarianceRatsPsychiatry and Mental healthmedicine.anatomical_structureEndocrinologyGene Expression Regulationnervous systemchemistryReceptors SerotoninSialic AcidsAntidepressive Agents Second-GenerationNeural cell adhesion moleculeSerotoninPsychologyNeuroscienceNeuropsychopharmacology
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Effects of antidepressants in adrenergic neurotransmission of human vas deferens

2000

Objectives. To evaluate the effects of sertraline, fluoxetine, and amitriptyline on the contractile responses of the human vas deferens muscle elicited by norepinephrine, electrical field stimulation, and KCl, because the therapeutic action of antidepressants may be accompanied by sexual dysfunction related to the contractility of the vas deferens smooth muscle. Methods. Ring segments of the epididymal part of the vas deferens were taken from 32 elective vasectomies and mounted in organ baths for isometric recording of tension. We then studied the effects of sertraline, fluoxetine, and amitriptyline on the neurogenic and agonist-induced contractile responses. Results. Amitriptyline caused c…

Malemedicine.medical_specialtyNifedipineAdrenergic receptorAmitriptylineUrologyAdrenergicSynaptic TransmissionNorepinephrine (medication)Vas DeferensNifedipineCulture TechniquesFluoxetineSertralineInternal medicinemedicineHumansAmitriptylineSertralineFluoxetineDose-Response Relationship Drugbusiness.industryVas deferensCalcium Channel BlockersAntidepressive AgentsReceptors AdrenergicEndocrinologymedicine.anatomical_structureCalciumbusinessMuscle Contractionmedicine.drugUrology
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Antidepressant-like activity of hyperforin and changes in BDNF and zinc levels in mice exposed to chronic unpredictable mild stress.

2019

Abstract Chronic unpredictable mild stress (CUMS) - a rodent model of depression mimics a variety of neurochemical and behavioral alterations similar to those seen in human depression. This study evaluated the antidepressant activity of hyperforin in the CUMS model using fluoxetine (FLX) as a reference drug. The antidepressant-like effects of hyperforin and FLX were evaluated in the tail suspension test (TST), forced swim test (FST), and splash test (SPT). CUMS induced an increase in immobility time in mice (pro-depressive effects) in the FST and TST. CUMS-induced changes were reversed by chronic treatment with hyperforin (2.5 and 5 mg/kg), as well as FLX (10 mg/kg). SPT results revealed a …

Malemedicine.medical_specialtyPhloroglucinolCREBHippocampus03 medical and health sciencesBehavioral Neurosciencechemistry.chemical_compoundMice0302 clinical medicineNeurochemicalInternal medicineFluoxetinemedicineHippocampus (mythology)Animals030304 developmental biology0303 health sciencesFluoxetineDepressive DisorderbiologyDepressionTerpenesBrain-Derived Neurotrophic FactorTail suspension testAntidepressive AgentsFrontal LobeMice Inbred C57BLHyperforinZincEndocrinologychemistrybiology.proteinAntidepressant030217 neurology & neurosurgeryStress Psychologicalmedicine.drugBehavioural despair testBehavioural brain research
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Effects of co-administration of amitriptyline and fluoxetine on inhibitory avoidance in mice

2010

We have previously observed that, while the impairing effects of amitriptyline on inhibitory avoidance in mice are consistently observed, those of acute fluoxetine are negligible. Two experiments were designed to investigate whether a regular dose of fluoxetine potentiates the effect of a low dose of amitriptyline that is ineffective when administered alone. Male and female CD1 mice were administered i.p. 30 min before training, as follows. In the first experiment, they were injected with saline, one of three doses of amitriptyline (2.5, 5, 10 mg/kg), one dose of fluoxetine (15 mg/kg), or a combination of amitriptyline (2.5 mg/kg) and fluoxetine (15 mg/kg). In the second experiment, the mic…

Malemedicine.medical_specialtyRatónAmitriptylinemedicine.medical_treatmentMice Inbred StrainsPharmacologyMiceBehavioral Neurosciencechemistry.chemical_compoundSex FactorsFluoxetineInternal medicineAvoidance LearningmedicineAnimalsAmitriptylineNeurotransmitterSalineFluoxetineDose-Response Relationship Drugbusiness.industryDrug SynergismAntidepressive AgentsEndocrinologychemistryCatecholamineFemaleSerotoninReuptake inhibitorbusinessmedicine.drugBehavioural Brain Research
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Astroglial plasticity in the hippocampus is affected by chronic psychosocial stress and concomitant fluoxetine treatment.

2006

Analysis of post-mortem tissue from patients with affective disorders has revealed a decreased number of glial cells in several brain areas. Here, we examined whether long-term psychosocial stress influences the number and morphology of hippocampal astrocytes in an animal model with high validity for research on the pathophysiology of major depression. Adult male tree shrews were submitted to 5 weeks of psychosocial stress, after which immunocytochemical and quantitative stereological techniques were used to estimate the total number and somal volume of glial fibrillary acidic protein-positive astrocytes in the hippocampal formation. Stress significantly decreased both the number (-25%) and…

Malemedicine.medical_specialtySerotonin reuptake inhibitorHippocampusHippocampal formationHippocampus03 medical and health scienceschemistry.chemical_compound0302 clinical medicineInternal medicineFluoxetinemedicineAnimalsNeurotransmitter030304 developmental biologyPharmacologyTupaia0303 health sciencesFluoxetineNeuronal PlasticityPsychiatry and Mental healthEndocrinologymedicine.anatomical_structurechemistryAstrocytesChronic DiseaseAntidepressantNeurogliaPsychologyNeuroscienceNeuroglia030217 neurology & neurosurgeryStress PsychologicalAstrocytemedicine.drugNeuropsychopharmacology : official publication of the American College of Neuropsychopharmacology
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Animal Models of Stress - Current Knowledge and Potential Directions

2021

Finding new therapies and new antidepressant agents is of high clinical priority given that many cases of depressive disorder do not respond to conventional monoaminergic antidepressants such as selective serotonin reuptake inhibitors, tricyclic antidepressants, and monoamine oxidase inhibitors The authors demonstrated that electroacupuncture and fluoxetine, a second-generation antidepressant categorized as a selective serotonin reuptake inhibitor (Perez-Caballero et al , 2014), regulate the expression of key proteins in the calmodulin kinase (CAMK) signaling pathway, which are related to depression in the hippocampi of rats (Takemoto-Kimura et al , 2017;Xie et al , 2019) In a paper on “Sho…

Monoamine oxidaseCognitive NeuroscienceSerotonin reuptake inhibitorContext (language use)Pharmacologyalternative therapylcsh:RC321-571stress03 medical and health sciencesBehavioral Neurosciencechemistry.chemical_compound0302 clinical medicineorganoselenium compoundsMonoaminergicmedicineNeurotransmitterlcsh:Neurosciences. Biological psychiatry. Neuropsychiatry030304 developmental biologychemistry.chemical_classification0303 health sciencesFluoxetinebusiness.industryanimal modelsEditorialNeuropsychology and Physiological PsychologychemistryAntidepressantmajor depressionbusiness030217 neurology & neurosurgeryTricyclicmedicine.drugFrontiers in Behavioral Neuroscience
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Lamotrigine compared to placebo and other agents with antidepressant activity in patients with unipolar and bipolar depression: a comprehensive meta-…

2016

ObjectivesTo meta-analytically summarize lamotrigine’s effectiveness and safety in unipolar and bipolar depression.MethodsWe conducted systematic PubMed and SCOPUS reviews (last search =10/01/2015) of randomized controlled trials comparing lamotrigine to placebo or other agents with antidepressant activity in unipolar or bipolar depression. We performed a random-effects meta-analysis of depression ratings, response, remission, and adverse effects calculating standardized mean difference (SMD) and risk ratio (RR) ±95% confidence intervals (CIs).ResultsEighteen studies (n=2152, duration=9.83 weeks) in patients with unipolar depression (studies=4, n=187; monotherapy vs lithium=1, augmentation …

Olanzapinemedicine.medical_specialtyBipolar DisorderLithium (medication)Bipolar depressionBipolar depression bipolar disorder lamotrigine major depressive disorder trials unipolar depressionCitalopramLithiumCitalopramLamotrigineLamotriginePlaceboBipolar depression; bipolar disorder; lamotrigine; major depressive disorder; trials; unipolar depressionBenzodiazepines03 medical and health sciences0302 clinical medicineAntimanic AgentsFluoxetineInternal medicinemedicineHumansBipolar disorderunipolar depressionPsychiatryDepressive Disorder MajorFluoxetinemajor depressive disorderDepressionTriazinesbusiness.industrytrialsmedicine.diseaseAntidepressive Agents030227 psychiatryDrug CombinationsPsychiatry and Mental healthVitamin B ComplexMajor depressive disorderAnticonvulsantsNeurology (clinical)businessInositol030217 neurology & neurosurgeryAntipsychotic Agentsmedicine.drug
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Determination of fluoxetine enantiomers in pharmaceutical formulations by electrokinetic chromatography-counter current technique

2012

In this work, an electrokinetic chromatography–counter current procedure for the separation of fluoxetine enantiomers using highly sulfated β-cyclodextrin was optimized and applied to the determination of the enantiomers in three pharmaceutical formulations according to the matrix features. Quality criteria were applied to facilitate its transferability to testing laboratories. Fluoxetine was used therapeutically as the racemate, although a stereospecificity associated with its interactions with the neuronal serotonin-uptake carrier was demonstrated. In this context, the development of enantioselective methods for the chiral analysis of pharmaceuticals allowing stereoisomer ratio estimation…

PharmacologyFluoxetineChromatographyChemistryClinical BiochemistryTransferabilityEnantioselective synthesisCounter currentContext (language use)General MedicineBiochemistryAnalytical ChemistryMatrix (chemical analysis)Electrokinetic phenomenaDrug DiscoverymedicineEnantiomerMolecular Biologymedicine.drugBiomedical Chromatography
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