Search results for "Forensic"

showing 10 items of 1701 documents

Comprehensive three‐dimensional morphology of neoangiogenesis in pulmonary veno‐occlusive disease and pulmonary capillary hemangiomatosis

2019

Abstract Pulmonary veno‐occlusive disease (PVOD) is a rare lung disease characterized by fibrotic narrowing of pulmonary veins leading to pulmonary hypertension (PH) and finally to death by right heart failure. PVOD is often accompanied by pulmonary capillary hemangiomatosis (PCH), a marked abnormal proliferation of pulmonary capillaries. Both morphological patterns often occur together and are thought to be distinct manifestations of the same disease process and accordingly are classified together in group 1′ of the Nice classification of PH. The underlying mechanisms of these aberrant remodeling processes remain poorly understood. In this study, we investigated the three‐dimensional struc…

Pathologymedicine.medical_specialtyLung NeoplasmsHypertension Pulmonarypulmonary veno‐occlusive diseasePulmonary capillary hemangiomatosis030204 cardiovascular system & hematologypulmonary capillary hemangiomatosisPathology and Forensic Medicine03 medical and health sciencesThree dimensional morphology0302 clinical medicineRight heart failurepulmonary hypertensionmedicinelcsh:PathologyHumansHemangioma CapillaryLungNeovascularization Pathologicbusiness.industryBrief Definitive Reportintussusceptive neoangiogenesismedicine.diseasePulmonary hypertension3. Good healthmedicine.anatomical_structurePulmonary VeinsLung disease030220 oncology & carcinogenesisPulmonary Veno-Occlusive DiseaseImmunohistochemistryPulmonary Veno-Occlusive Diseasebusinesspulmonary vascular remodelinglcsh:RB1-214The Journal of Pathology: Clinical Research
researchProduct

Increased numbers of cytokeratin-positive interstitial reticulum cells (CIRC) in reactive, inflammatory and neoplastic lymphadenopathies: hyperplasia…

1995

A total of 291 enlarged lymph nodes showing a range of reactive-inflammatory processes, primary and metastatic neoplasms were studied to determine the distribution and immunoprofile of their cytokeratin-positive interstitial reticulum cells (CIRC) in comparison with normal nodes. In 258/291 nodes (89%), CIRC numbers were distinctly increased in the subcapsular, paracortical and, occasionally, in the medullary zones; often, these increased CIRC formed networks around follicles, sinuses and vessels. CIRC had comparatively small, irregularly shaped bodies and dendritic processes; occasionally, giant forms were noted. CIRC contained cytokeratins (CK) 8 and 18 but not 19, as shown by immunohisto…

Pathologymedicine.medical_specialtyLymphomaLymphangitisVimentinLymphoid hyperplasiaInterstitial cellPathology and Forensic MedicineMetastasisCytokeratinNeoplasmsmedicineHumansMolecular BiologyLymph nodeLymphatic DiseasesHyperplasiabiologyCell BiologyGeneral MedicineDendritic CellsHyperplasiamedicine.diseaseImmunohistochemistryCytoskeletal Proteinsmedicine.anatomical_structureMicroscopy Fluorescencebiology.proteinKeratinsLymphLymph Nodesmedicine.symptomVirchows Archiv : an international journal of pathology
researchProduct

Biological and prognostic significance of stratified epithelial cytokeratins in infiltrating ductal breast carcinomas

1998

The biological significance of the differential expression of cytokeratin (CK) polypeptides in breast carcinomas is unclear. We examined the CK profiles of 101 primary infiltrating ductal breast carcinomas using monoclonal antibodies directed against 11 different CKs and against vimentin. Two major CK phenotypes were distinguished: first, a phenotype expressing only the simple-epithelial CKs 7 (variably), 8, 18 and 19, and secondly, a bimodal phenotype co-expressing significant amounts of one or more of the stratified-epithelial CKs 4, 14 and 17. The vast majority of G1 and G2 carcinomas had the simple-epithelium phenotype, as did a subgroup of G3 carcinomas. Interestingly, the majority (62…

Pathologymedicine.medical_specialtyMammary glandBreast NeoplasmsVimentinDisease-Free SurvivalEpitheliumPathology and Forensic MedicineCytokeratinBreast cancerCarcinomamedicineFrozen SectionsHumansVimentinMolecular BiologybiologyCarcinoma Ductal BreastAntibodies MonoclonalCell BiologyGeneral MedicineDuctal carcinomaPrognosismedicine.diseasePhenotypemedicine.anatomical_structureReceptors EstrogenLymphatic Metastasisbiology.proteinKeratinsImmunohistochemistryFemaleLymph NodesVirchows Archiv
researchProduct

Caspase 9 and Caspase 3 Immunohistochemical Pattern in Skeletal and Cardiac Muscles at Different Times after Death: An Experimental Study on PMI Esti…

2021

(1) Background: The estimation of the post mortem interval (PMI) is a challenge for forensic pathologists because data emerging from methods commonly applied are not always conclusive, since several conditions exist that may affect the reliability of these parameters. Thus, new approaches have been proposed to overcome such a limit. In recent years, several studies have been performed on proteins analyzing their expression/degradation patterns in relation to the progressing of the post mortem interval. (2) Methods: The immunoreactivity patterns of two apoptosis mediators—Caspase 9 and Caspase 3—have been tested in order to evaluate their potential role as markers of the post mortem interval…

Pathologymedicine.medical_specialtyMedicine (General)cardiac muscleforensic sciencesClinical BiochemistryCaspase 3Article03 medical and health sciences0302 clinical medicineR5-920caspase 3Medicine030216 legal & forensic medicineskeletal musclecaspase 9Caspase030304 developmental biologyPost-mortem intervalCaspase-90303 health sciencespost mortem intervalbiologybusiness.industryCardiac muscleSkeletal musclemedicine.anatomical_structureApoptosisbiology.proteinImmunohistochemistrybusinesstime after deathDiagnostics
researchProduct

HMB-45 Immunostaining and Ultrastructure of Melanocytic Hyperplasia in Pigmented Basal Cell Carcinomas

1999

The pigmented variant of basal cell carcinoma (PBCC) may be clinically misinterpreted as malignant melanoma. Histologically this variant is characterized by the presence of functional melanocytes distributed among epithelial tumor cells, particularly in superficial areas. Our finding of a case of PBCC with numerous atypical HMB-45-positive melanocytes prompted us to analyze the morphologic and immunohistochemical features of 20 consecutive cases of PBCC. Four additional cases with numerous large HMB-45-positive melanocytes, also present in the most invasive tumor nests, were found. Ultrastructural examination was performed in two selected cases. Large melanocytes with immature (stages I an…

Pathologymedicine.medical_specialtyMelanomaMelanocytic hyperplasiaBiologymedicine.diseasePathology and Forensic MedicineHMB-45medicineUltrastructureImmunohistochemistrySurgeryBasal cell carcinomaAnatomyImmunostainingMelanosomeInternational Journal of Surgical Pathology
researchProduct

Biomaterial-Induced Sarcoma

2000

In the study of carcinogenesis most interest has focused on carcinomas, as they represent the majority of human cancers. The recognition of the adenoma-carcinoma sequence both in humans and in animal experimental models has given the field of basic oncology the opportunity to elucidate individual mechanisms in the multistep development of carcinoma. The relative scarcity of human sarcomas coupled with the lack of adequate animal models has hampered understanding of the molecular genetic steps involved. We present an experimental model in the rat in which a high incidence of malignant mesenchymal tumors arise around a subcutaneously implanted biomaterial. Nine commercially available biomater…

Pathologymedicine.medical_specialtyMesenchymal stem cellBiologyMalignancymedicine.diseasemedicine.disease_causePathology and Forensic MedicineTumor progressionmedicineCarcinomaImmunohistochemistrySarcomaCarcinogenesisPreneoplastic ChangeThe American Journal of Pathology
researchProduct

Postischemic Brain Infiltration of Leukocyte Subpopulations Differs among Murine Permanent and Transient Focal Cerebral Ischemia Models

2012

Cellular and humoral inflammations play important roles in ischemic brain injury. The effectiveness of immunomodulatory therapies may critically depend on the chosen experimental model. Our purpose was to compare the post-ischemic neuroinflammation among murine permanent and transient middle cerebral artery occlusion (MCAO) models. Permanent MCAO was induced by transtemporal electrocoagulation and 30 minutes or 90 minutes transient MCAO was induced by intraluminal filament in C57BL/6 mice. Infiltration of leukocyte subpopulations was quantified by immunohistochemistry and fluorescence-activated cell sorting. Cerebral cytokine and adhesion molecule expression was measured by real-time polyme…

Pathologymedicine.medical_specialtyMicrogliabusiness.industryCell adhesion moleculeanimal diseasesGeneral Neurosciencemedicine.medical_treatmentIschemiaInflammationmedicine.diseasenervous system diseasesPathology and Forensic Medicinemedicine.anatomical_structureCytokinenervous systemCerebral cortexcardiovascular systemMedicinecardiovascular diseasesNeurology (clinical)medicine.symptombusinessInfiltration (medical)NeuroinflammationBrain Pathology
researchProduct

Mitochondrial and chromosomal DNA alterations in human chromophobe renal cell carcinomas

1992

Renal cell tumours are characterized by the loss of chromosome 3p and trisomy of 5q segments (common, non-papillary renal cell carcinoma), or by trisomy of chromosomes 7 and 17 and loss of the Y chromosome (papillary renal cell carcinoma), or by random karyotype changes and mitochondrial DNA alterations (renal oncocytoma). We have studied by means of RFLP analysis the genomic and mitochondrial DNA in 11 chromophobe renal cell carcinomas, which have a unique morphology among kidney cancers. We found a loss of the constitutional heterozygosity at chromosomal regions 3p, 5q, 17p, and 17q, a combination of allelic losses that has not been found in other types of renal cell tumours. Three of the…

Pathologymedicine.medical_specialtyMitochondrial DNAChromophobe Renal Cell CarcinomaChromophobe cellBiologyurologic and male genital diseasesDNA MitochondrialChromosomesPathology and Forensic MedicineLoss of heterozygosityRenal cell carcinomamedicineHumansRenal oncocytomaCarcinoma Renal CellChromosome AberrationsKidneyStaining and LabelingPapillary renal cell carcinomasDNA Neoplasmmedicine.diseaseKidney NeoplasmsBlotting Southernmedicine.anatomical_structurePolymorphism Restriction Fragment LengthThe Journal of Pathology
researchProduct

Assessment of proliferative activity in breast cancer: MIB-1 immunohistochemistry versus mitotic figure count.

1999

Abstract The proliferative activity is one of the most important single prognostic parameters in breast cancer diagnosis and the time-honored measure of proliferative activity, the mitotic figure count, is an integral component of most combined prognostic scores. The detection of the cell cycle-specific antigens Ki-67, and the development of anti-Ki-67 antibodies, including the paraffin-reactive antibody MIB-1, have established immunohistochemical detection of cell cycle-specific antigens as a measure of proliferative activity in breast cancer diagnosis. The current study was performed to correlate mitotic figure counts with the proliferative activity as assessed by MIB-1 immunohistochemist…

Pathologymedicine.medical_specialtyMitotic indexMammary glandBreast NeoplasmsPathology and Forensic MedicineBreast cancermedicineCarcinomaMitotic IndexHumansHigh-power fieldObserver VariationbiologyCarcinoma Ductal BreastNuclear ProteinsAntigens Nuclearmedicine.diseaseImmunohistochemistrymedicine.anatomical_structureKi-67 AntigenKi-67biology.proteinMitotic FigureImmunohistochemistryCell DivisionHuman pathology
researchProduct

Identification of a Novel BRCA1 Alteration in Recurrent Melanocytoma Resulting in Increased Proliferation

2020

Abstract Primary meningeal melanocytomas are rare tumors of the central nervous system. Although they are considered benign neoplasms, some reports describe recurrent rates up to 45%. Little is known about their genetic and epigenetic landscape because of their infrequency. Even less has been described about markers with prognostic value. Here we describe a patient who developed a primary meningeal melanocytoma, suffered 3 recurrences in a period of 6 years and died of the tumor. The genetic and epigenetic changes explored confirmed GNAQ mutation as an initiating event. We found an epigenetic alteration of GSTP1, a feature that has recently been described in meningiomas, from the beginning …

Pathologymedicine.medical_specialtyMitotic indexProliferation indexDiseasePathology and Forensic MedicineMeningiomaLoss of heterozygosity03 medical and health sciencesCellular and Molecular NeuroscienceFatal Outcome0302 clinical medicineMeningeal NeoplasmsmedicineHumansEpigeneticsMelanomaCell ProliferationBRCA1 Proteinbusiness.industryGeneral MedicineMiddle Agedmedicine.diseaseGlutathione S-Transferase piNeurology030220 oncology & carcinogenesisMutationGTP-Binding Protein alpha Subunits Gq-G11FemaleNeurology (clinical)Neoplasm Recurrence LocalMelanocytomabusiness030217 neurology & neurosurgeryGNAQJournal of Neuropathology & Experimental Neurology
researchProduct