Search results for "Forming"

showing 10 items of 1719 documents

Central obesity and hypertensive renal disease: association between higher levels of BMI, circulating transforming growth factor beta1 and urinary al…

2004

OBJECTIVE: In this study, the relationship between circulating transforming growth factor beta1 (TGFbeta1) and urinary albumin excretion (UAE) has been investigated in non-obese and central obese hypertensive patients. DESIGN AND PATIENTS: Fifty-eight consecutive hypertensive outpatients both lean and with central obesity were enrolled and divided in three groups, according to their body mass index (BMI) values. Group A: 16 lean hypertensives (men with BMI or = 25 kg/m2 and 24.7 kg/m2 and or = 30 kg/m2 and women with BMI > or = 27.3 kg/m2). MEASURES: In all patients, UAE, by immunonephelometric assay, circulating TGFbeta1 by a solid-phase specific sandwich enzyme-linked immunosorbent assay …

AdultMalemedicine.medical_specialtyHypertension RenalSettore MED/09 - Medicina Internacentral obesity hypertension transforming growth factor beta1 urinary albumin excretionOverweightBody Mass IndexExcretionTransforming Growth Factor beta1chemistry.chemical_compoundElectrocardiographyTransforming Growth Factor betaInternal medicineInternal MedicineMedicineAlbuminuriaHumansObesityBlood urea nitrogenAgedSettore MED/14 - NefrologiaCreatininebusiness.industryBody Weightnutritional and metabolic diseasesGeneral MedicineMiddle Agedmedicine.diseaseObesitySettore MED/45 - Scienze Infermieristiche Generali Cliniche E PediatricheEndocrinologychemistryHeart Function TestsAlbuminuriaRegression AnalysisFemaleKidney Diseasesmedicine.symptomCardiology and Cardiovascular MedicinebusinessBody mass indexKidney disease
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Effect of dual blockade of renin–angiotensin system on TGFβ1 and left ventricular structure and function in hypertensive patients

2007

The effects of 24 weeks losartan and ramipril treatment, both alone and in combination, on left ventricular mass (LVM), circulating transforming growth factor beta1 (TGFbeta1), procollagen type I (PIP) and III (PIIIP), have been evaluated in hypertensive (HT) patients. A total of 57 HT with stage 1 and 2 essential hypertension were included. After 4 weeks run in, a randomized double-blind, three arms, double dummy, independent trial was used. All HT patients were randomly allocated to three treatment arms consisting of losartan (50 mg/daily), ramipril (5 mg/ daily) and combined (losartan 50 mg/daily + ramipril 5 mg/daily) for 24 weeks. TGFbeta1, PIP and PIIIP, LVM, LVM/h(2.7) and other echo…

AdultMalemedicine.medical_specialtyLeft ventricular structureHeart VentriclesBlood PressureEnzyme-Linked Immunosorbent AssayAngiotensin II Receptor BlockersPeptide hormoneSeverity of Illness IndexDual blockadeCollagen Type ILosartanVentricular Function LeftRenin-Angiotensin SystemTransforming Growth Factor beta1Double-Blind MethodRamiprilInternal medicineRenin–angiotensin systemPrevalenceInternal MedicineHumansMedicineAntihypertensive AgentsUltrasonographyAnalysis of Variancebusiness.industryMiddle AgedAngiotensin IICollagen Type IIITreatment OutcomeEndocrinologyItalyHypertensionACE inhibitorDrug Therapy CombinationFemaleHypertrophy Left VentricularbusinessBiomarkersmedicine.drugJournal of Human Hypertension
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Transforming growth factor beta1 T29C gene polymorphism and hypertension: relationship with cardiovascular and renal damage.

2008

Distribution of T29C TGFβ1 gene polymorphism was analysed in 260 hypertensive and 134 normotensive subjects. Circulating TGFβ1 and procollagen type III levels, microalbuminuria, left ventricular geometry and function were evaluated in all the hypertensives subgrouped according to T29C TGFβ1 gene polymorphism. Circulating TGFβ1 by ELISA technique, procollagen type III by a specific radioimmunoassay, microalbuminuria by radioimmunoassay, left ventricular geometry and function by echocardiography were determined. All groups were comparable for gender, age and sex. Regarding T29C TGFβ1 gene polymorphism, prevalence of TC or CC genotypes was significantly (p < 0.05) higher in hypertensives th…

AdultMalemedicine.medical_specialtyMetabolic Clearance RateRadioimmunoassayEnzyme-Linked Immunosorbent AssayLeft ventricular hypertrophyTransforming Growth Factor beta1Procollagen type IIIElectrocardiographyInternal medicineGenotypeInternal MedicinemedicineAlbuminuriaHumansCirculating TGFβ1 Hypertension Left ventricular hypertrophy Microalbuminuria Procollagen type III TGFβ1 gene polymorphismPolymorphism GeneticRenal damagebusiness.industryRadioimmunoassayGeneral MedicineMiddle Agedmedicine.diseaseEndocrinologyCollagen Type IIICase-Control StudiesCreatinineHypertensionMicroalbuminuriaFemaleHypertrophy Left VentricularGene polymorphismCardiology and Cardiovascular MedicinebusinessProcollagenTransforming growth factorBlood pressure
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Supramaximal exercise mobilizes hematopoietic progenitors and reticulocytes in athletes

2005

Am J Physiol Regul Integr Comp Physiol. 2005 Nov;289(5):R1496-503. Epub 2005 Jul 14. Supramaximal exercise mobilizes hematopoietic progenitors and reticulocytes in athletes. Morici G, Zangla D, Santoro A, Pelosi E, Petrucci E, Gioia M, Bonanno A, Profita M, Bellia V, Testa U, Bonsignore MR. SourceDepartment of Experimental Medicine, University of Palermo, Italy. Abstract Marathon runners show increased circulating CD34+ cell counts and postexercise release of interleukin-6 (IL-6), granulocyte-colony stimulating factor (G-CSF) and flt3-ligand (Bonsignore MR, Morici G, Santoro A, Pegano M, Cascio L, Bonnano A, Abate P, Mirabella F, Profita M, Insalaco G, Gioia M, Vignola AM, Majolino I, Testa…

AdultMalemedicine.medical_specialtyReticulocytesAdolescentHydrocortisonePhysiologyCD34Physical exerciseSettore MED/10 - Malattie Dell'Apparato RespiratorioBiologySettore BIO/09 - FisiologiaMonocytesColony-Forming Units AssayBlood cellPhysiology (medical)Internal medicineGranulocyte Colony-Stimulating Factorgrowth factorscytokinemedicineHumansProgenitor cellExercise physiologyGrowth SubstancesErythropoietinExerciseangiogenetic precursorhypoxiaHypoxia (medical)Hematopoietic Stem CellsGranulocyte colony-stimulating factormedicine.anatomical_structureEndocrinologyPhysical EnduranceCytokinesFemalemedicine.symptomLeukocyte ElastaseGlucocorticoidGranulocytesmedicine.drugAmerican Journal of Physiology-Regulatory, Integrative and Comparative Physiology
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Left ventricular filling abnormalities and obesity-associated hypertension: relationship with overproduction of circulating transforming growth facto…

2005

This study has been designed to evaluate the relationship among transforming growth factor beta1 (TGFbeta1) and some measurements of diastolic function in a population of hypertensive subjects with normal left ventricular ejection fraction. We studied 67 hypertensive outpatients who according to their BMI levels were subdivided into three groups: lean (L), overweight (OW) and obese (OB) hypertensives (HT). Circulating TGFbeta1 and M- and B-mode echocardiography was determined. All hypertensives were further subgrouped, according to European Society of Cardiology Guidelines, into two subsets of patients with normal diastolic function or with diastolic dysfunction. Prevalence of left ventricu…

AdultMalemedicine.medical_specialtySettore MED/09 - Medicina InternaHeart VentriclesEnzyme-Linked Immunosorbent Assayobesity-associated hypertension; TGFb1; left ventricular hypertrophy; left ventricular diastolic functionLeft ventricular hypertrophyTransforming Growth Factor beta1Ventricular Dysfunction LeftDiastoleRisk FactorsTransforming Growth Factor betaVentricule gaucheInternal medicineInternal MedicinemedicineHumansObesityOverproductionAgedbusiness.industryStroke VolumeNutritional statusMiddle Agedmedicine.diseaseMyocardial ContractionSettore MED/11 - Malattie Dell'Apparato CardiovascolareObesityEndocrinologyEchocardiographyHypertensionAdult Aged Biological Markers/blood Diastole Echocardiography Enzyme-Linked Immunosorbent Assay Female Heart Ventricles/physiopathology Heart Ventricles/ultrasonography Humans Hypertension/blood Hypertension/complications* Hypertension/physiopathology Hypertrophy Left Ventricular/blood Hypertrophy Left Ventricular/complications* Hypertrophy Left Ventricular/physiopathology Male Middle Aged Myocardial Contraction/physiology* Obesity/blood Obesity/complications* Obesity/physiopathology Regression Analysis Risk FactorsCardiologyRegression AnalysisFemaleHypertrophy Left VentricularbusinessVentricular fillingBiomarkersTransforming growth factorJournal of Human Hypertension
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Lattice Corneal Dystrophy Type 1

2014

PURPOSE To evaluate the question whether lattice corneal dystrophy type 1 (LCD1) is of epithelial or stromal origin. METHODS The landmark of advanced LCD1 shows central superficial haze and paracentral stromal lattice lines. In 16 eyes of 8 affected individuals of 2 families, a penetrating keratoplasty was performed. The follow-up was 8 to 16 years after penetrating keratoplasty. Slit-lamp documentation of the patients was evaluated in direct and indirect illumination with dilated pupil to assess the horizontal and vertical pattern of new deposits on the corneal graft. Three affected patients of 2 families are demonstrated in detail. A DNA analysis was performed. RESULTS Gene identification…

AdultMalemedicine.medical_specialtyStromal cellCorneal StromaDNA Mutational AnalysisCorneal graftCorneal KeratocytesCorneal erosionDirect illuminationTransforming Growth Factor betaOphthalmologyTGFBI genemedicineHumansCorneal Dystrophies HereditaryExtracellular Matrix Proteinsbusiness.industryEpithelium Cornealmedicine.diseaseeye diseasesOphthalmologyMutationLattice corneal dystrophyFemalebusinessKeratoplasty PenetratingTGFBICornea
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Relationship of transforming growth factor-beta(1) with tumour necrosis factor-alpha and endothelial activation in patients with stable renal transpl…

2008

SUMMARY: Aim:  To evaluate whether or not transforming growth factor-beta1 is related to inflammation markers and to intercellular and vascular cell adhesion molecules in patients with stable renal transplantation. Methods:  Serum concentrations of transforming growth factor-beta1, tumour necrosis factor-alpha, C-reactive protein and adhesion molecules were analysed in 33 renal transplanted patients, 33 patients with chronic renal insufficiency (matched to the transplanted group for level of renal function), and 33 hypertensives with normal renal function. anova, Student's t-test and simple regression analysis were used to analyse the data. Results:  Transplanted patients showed higher valu…

AdultMalemedicine.medical_specialtyTime Factorsmedicine.medical_treatmentIntercellular Adhesion Molecule-1Renal functionVascular Cell Adhesion Molecule-1Transforming Growth Factor beta1Internal medicinemedicineHumansRenal Insufficiency ChronicKidney transplantationInflammationbiologyCell adhesion moleculebusiness.industryTumor Necrosis Factor-alphaC-reactive proteinGraft SurvivalGeneral MedicineTransforming growth factor betaMiddle Agedmedicine.diseaseIntercellular Adhesion Molecule-1Kidney TransplantationTransplantationEndocrinologyCytokineC-Reactive ProteinCross-Sectional StudiesNephrologyCase-Control StudiesHypertensionbiology.proteinEndothelium VascularbusinessBiomarkersGlomerular Filtration RateNephrology (Carlton, Vic.)
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De novo 15q21.1q21.2 deletion identified through FBN1 MLPA and refined by 244K array-CGH in a female teenager with incomplete Marfan syndrome

2010

International audience; Interstitial deletions involving the 15q21.1 band are very rare. Only 4 of these cases have been studied using molecular cytogenetic techniques in order to confirm the deletion of the whole FBN1 gene. The presence of clinical features of the Marfan syndrome (MFS) spectrum associated with mental retardation has been described in only 2/4 patients. Here we report on a 16-year-old female referred for suspicion of MFS (positive thumb and wrist sign, scoliosis, joint hyperlaxity, high-arched palate with dental crowding, dysmorphism, mitral insufficiency with dystrophic valve, striae). She had therefore 3 minor criteria according to the Ghent nosology. She also had speech …

AdultMalemusculoskeletal diseasesProbandMarfan syndromecongenital hereditary and neonatal diseases and abnormalitiesAdolescent[SDV]Life Sciences [q-bio]Fibrillin-1BiologyFibrillinsBioinformaticsPolymerase Chain ReactionMarfan SyndromeLoss of heterozygosity03 medical and health sciencesTransforming Growth Factor betaIntellectual DisabilityGeneticsmedicineHumansMultiplex ligation-dependent probe amplificationAlleleChildGeneIn Situ Hybridization FluorescenceGenetics (clinical)Oligonucleotide Array Sequence AnalysisSequence Deletion030304 developmental biologyGeneticsChromosomes Human Pair 15Comparative Genomic Hybridization0303 health sciencesMicrofilament Proteins030305 genetics & heredityGeneral Medicinemedicine.diseasePedigree3. Good healthPhenotypeMutationMicrosatelliteFemaleDNA ProbesHaploinsufficiencyMicrosatellite RepeatsEuropean Journal of Medical Genetics
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Transforming Growth Factor Beta 1 Serum Levels in Patients with Preinvasive and Invasive Lesions of the Breast

2004

Transforming growth factor beta (TGF-beta)1 is thought to be involved in breast carcinogenesis. TGF-beta1 acts in an antiproliferative manner in the early stages of breast carcinogenesis, but promotes tumor progression and metastases in the advanced stages of the disease. No data have been published on serum TGF-beta1 in breast cancer. We investigated TGF-beta1 serum levels in patients with breast cancer (n=135), ductal carcinoma in situ (DCIS) I to III (n=67) or fibroadenoma (n=35), and in healthy women (n=40) to determine its value as a differentiation marker between malignant, pre-invasive and benign diseases and as a predictive marker for metastatic spread. Median (range) TGF-beta1 seru…

AdultOncology0301 basic medicinemedicine.medical_specialtyPathologyCancer ResearchMammary glandClinical BiochemistryCA 15-3Breast NeoplasmsPathology and Forensic MedicineTransforming Growth Factor beta103 medical and health sciencesBreast cancer0302 clinical medicineTransforming Growth Factor betaInternal medicinemedicineHumansNeoplasm Invasivenessskin and connective tissue diseasesEstrogen Receptor StatusAgedNeoplasm StagingAged 80 and overPredictive markerbusiness.industryMiddle AgedDuctal carcinomamedicine.diseaseFibroadenomamedicine.anatomical_structure030104 developmental biologyOncologyTumor progression030220 oncology & carcinogenesisbusinessThe International Journal of Biological Markers
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Induction of bone morphogenetic protein-6 in skin wounds. Delayed reepitheliazation and scar formation in BMP-6 overexpressing transgenic mice.

1998

Growth factors of the transforming growth factor-beta superfamily are involved in cutaneous wound healing. In this study we analyze the expression of the bone morphogenetic protein-6 (BMP-6) gene, a transforming growth factor-beta related gene, in skin wounds. In normal mouse skin high levels of BMP-6 mRNA and protein are expressed by postmitotic keratinocytes of stratified epidermis until day 6 after birth. BMP-6 expression is strongly reduced in adult epidermis with diminished mitotic activity. After skin injury we found large induction of BMP-6-specific RNA and protein in keratinocytes at the wound edge and keratinocytes of the newly formed epithelium as well as in fibroblast shaped cell…

AdultPathologymedicine.medical_specialtyBone Morphogenetic Protein 6Gene ExpressionMice TransgenicDermatologyBiologyBiochemistryMiceTransforming Growth Factor betaGene expressionmedicineAnimalsHumansFibroblastMolecular BiologySkinMessenger RNAWound Healingintegumentary systemEpidermis (botany)RNACell DifferentiationCell BiologyCell biologyUp-RegulationBone morphogenetic protein 6medicine.anatomical_structureBone Morphogenetic ProteinsRNAWounds and InjuriesWound healingCell DivisionTransforming growth factorThe Journal of investigative dermatology
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