Search results for "Forming"

showing 10 items of 1719 documents

The T-box transcription factor eomesodermin controls CD8 T cell activity and lymph node metastasis in human colorectal cancer.

2007

An efficient cytolytic T cell function is essential for immune mediated rejection of colorectal cancer. However, the molecular mechanisms driving T cell mediated cancer rejection are still poorly understood. Here, we assessed the relevance of the T-box transcription factor eomesodermin in colorectal cancer. METHODS/ RESULTS: By analysing tissue probes from 88 different colorectal tumours, a significant (p0.02) inverse correlation between eomesodermin expression in colorectal cancers and the presence of lymph node metastases could be shown, whereas no such correlation was noted for the master transcription factor of regulatory T cells, FoxP3 and CD8 alpha expression. To evaluate whether this…

MaleT cellEomesoderminEnzyme-Linked Immunosorbent AssayCD8-Positive T-LymphocytesTransforming Growth Factor betamedicineCytotoxic T cellHumansTranscription factorColorectal Cancerbiologybusiness.industryReverse Transcriptase Polymerase Chain ReactionGastroenterologyCancerT lymphocytemedicine.diseasemedicine.anatomical_structurePerforinLymphatic MetastasisImmunologybiology.proteinFemaleInterleukin-4businessColorectal NeoplasmsT-Box Domain ProteinsCD8Gut
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Partial Sleep Restriction Activates Immune Response-Related Gene Expression Pathways: Experimental and Epidemiological Studies in Humans

2013

Epidemiological studies have shown that short or insufficient sleep is associated with increased risk for metabolic diseases and mortality. To elucidate mechanisms behind this connection, we aimed to identify genes and pathways affected by experimentally induced, partial sleep restriction and to verify their connection to insufficient sleep at population level. The experimental design simulated sleep restriction during a working week: sleep of healthy men (N = 9) was restricted to 4 h/night for five nights. The control subjects (N = 4) spent 8 h/night in bed. Leukocyte RNA expression was analyzed at baseline, after sleep restriction, and after recovery using whole genome microarrays complem…

MaleTBX21NF-KAPPA-Blcsh:MedicineNK cellsBioinformaticskokeellinen tutkimusReceptors G-Protein-Coupled0302 clinical medicineCARDIOMETABOLIC RISKLeukocytesta319geeniekspressiolcsh:Scienceta515Sleep restrictionRegulation of gene expression0303 health sciencesMultidisciplinaryNATURAL-KILLERNF-kappa Bta3142Sleep in non-human animalsC-REACTIVE PROTEIN3. Good healthMACROPHAGE APOPTOSISINSUFFICIENT SLEEPSTAT1 Transcription FactorCARDIOVASCULAR-DISEASEimmuunivasteProteoglycansmedicine.symptomResearch ArticleAdulteducationENDOPLASMIC-RETICULUMMETABOLIC CONSEQUENCESSyntaxin 16Biologyepidemiologinen tutkimusuni (lepotila)03 medical and health sciencesImmune systemmedicineHumans030304 developmental biologyTOLL-LIKE RECEPTORSB cellsuniMicroarray analysis techniquesGene Expression Profilingsytokiinitlcsh:RMicroarray AnalysisGene expression profilingSleep deprivationGene Expression RegulationImmunologyRNASleep Deprivationlcsh:Q3111 BiomedicineT-Box Domain ProteinsReceptors Transforming Growth Factor beta030217 neurology & neurosurgery
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Time-restricted eating effects on performance, immune function, and body composition in elite cyclists: a randomized controlled trial.

2020

Abstract Background Although there is substantial interest in intermittent fasting as a dietary approach in active individuals, information regarding its effects in elite endurance athletes is currently unavailable. The present parallel randomized trial investigated the effects of a particular intermittent fasting approach, called time-restricted eating (TRE), during 4 weeks of high-level endurance training. Methods Sixteen elite under-23 cyclists were randomly assigned either to a TRE group or a control group (ND). The TRE group consumed 100% of its estimated daily energy needs in an 8-h time window (from 10:00 a.m. to 6:00 p.m.) whilst energy intake in the ND group was distributed in 3 me…

MaleTime FactorsElite cyclists Endurance Immune system Inflammation Intermittent fastingPhysiologyIntermittent fastinglaw.inventionEnduranceLeukocyte Count0302 clinical medicineRandomized controlled trialWeight losslawIntermittent fastingElectric ImpedanceTestosteroneInsulin-Like Growth Factor INutrition and DieteticsFastingCreatinineBody Compositionmedicine.symptomlcsh:RC1200-1245lcsh:Nutrition. Foods and food supplySettore M-EDF/01 - Metodi E Didattiche Delle Attivita' MotorieBioelectrical impedance analysisResearch ArticleElite cyclistslcsh:TX341-641030209 endocrinology & metabolismClinical nutritionAthletic Performance03 medical and health sciencesYoung AdultEndurance trainingWeight LossmedicineHumansLymphocyte Countlcsh:Sports medicineInflammationbusiness.industryInterleukin-6Elite cyclists; Endurance; Immune system; Inflammation; Intermittent fasting030229 sport sciencesTransforming Growth Factor alphaGas analyzerBicyclingDietSports Nutritional Physiological PhenomenaImmune systemAthletesBasal metabolic rateBasal MetabolismbusinessEnergy IntakeFood ScienceJournal of the International Society of Sports Nutrition
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Complement Activation in Peritoneal Dialysis–Induced Arteriolopathy

2017

Cardiovascular disease (CVD) is the leading cause of increased mortality in patients with CKD and is further aggravated by peritoneal dialysis (PD). Children are devoid of preexisting CVD and provide unique insight into specific uremia- and PD-induced pathomechanisms of CVD. We obtained peritoneal specimens from children with stage 5 CKD at time of PD catheter insertion (CKD5 group), children with established PD (PD group), and age-matched nonuremic controls (n=6/group). We microdissected omental arterioles from tissue layers not directly exposed to PD fluid and used adjacent sections of four arterioles per patient for transcriptomic and proteomic analyses. Findings were validated in omenta…

MaleVascular Endothelial Growth Factor A0301 basic medicinePathologyProteomemedicine.medical_treatmentComplement Membrane Attack ComplexSmad2 ProteinSeverity of Illness IndexTransforming Growth Factor betaMedicinePhosphorylationChildComplement ActivationCatheter insertionGeneral MedicineArteriosclerosisArteriolesComplement C3dNephrologyChild PreschoolFemaleOmentumPeritoneal DialysisSignal Transductionmedicine.medical_specialtyAdolescentPeritoneal dialysis03 medical and health sciencesDownregulation and upregulationClinical ResearchTGF beta signaling pathwayHumansSmad3 ProteinVascular DiseasesUremiabusiness.industryVascular diseaseComplement C1qInfant NewbornInfantComplement System Proteinsmedicine.diseaseUremiaComplement systemGene Ontology030104 developmental biologyCase-Control StudiesKidney Failure ChronicTranscriptomebusinessJournal of the American Society of Nephrology
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Liver-specific p38α deficiency causes reduced cell growth and cytokinesis failure during chronic biliary cirrhosis in mice

2012

p38α mitogen-activated protein kinases (MAPK) may be essential in the up-regulation of proinflammatory cytokines and can be activated by transforming growth factor β, tumor necrosis factor-α, interleukin-1β, and oxidative stress. p38 MAPK activation results in hepatocyte growth arrest, whereas increased proliferation has been considered a hallmark of p38α-deficient cells. Our aim was to assess the role of p38α in the progression of biliary cirrhosis induced by chronic cholestasis as an experimental model of chronic inflammation associated with hepatocyte proliferation, apoptosis, oxidative stress, and fibrogenesis. Cholestasis was induced in wildtype and liver-specific p38α knockout mice by…

Malemedicine.medical_specialtyBiliary cirrhosisMAP Kinase Kinase 2ApoptosisBiologymedicine.disease_causeProinflammatory cytokineMitogen-Activated Protein Kinase 14MiceCholestasisInternal medicinemedicineAnimalsCyclin D1Cyclin B1Cell ProliferationCytokinesisMice KnockoutHepatologyLiver Cirrhosis BiliaryHepatologymedicine.diseaseMice Inbred C57BLSurvival RateDisease Models AnimalOxidative Stressmedicine.anatomical_structureEndocrinologyLiverHepatocyteChronic DiseaseDisease ProgressionHepatocytesTumor necrosis factor alphaOxidative stressSignal TransductionTransforming growth factorHepatology
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Relationship between transforming growth factor beta1 and progression of hypertensive renal disease.

2002

In this study the role of circulating transforming growth factor beta1 (TGFbeta1) on progression of renal hypertensive disease has been investigated. Fifty consecutive outpatients with essential hypertension were enrolled and divided into three groups, according to their urinary albumin excretion (UAE). Group A comprised 10 hypertensives with UAEor=20 mg/24 h (normoalbuminuric group); Group B included 21 hypertensives with UAE20300 mg/24 h (microalbuminuric group); Group C encompassed 19 hypertensives with UAEor= 300 mg/24 h (proteinuric group). In all patients UAE by immunonephelometric assay, circulating TGFbeta1 by a solid phase specific sandwich ELISA technique, BUN and creatinine by ro…

Malemedicine.medical_specialtyDiastoleLeft ventricular hypertrophyEssential hypertensionStatistics NonparametricMuscle hypertrophyExcretionTransforming Growth Factor beta1chemistry.chemical_compoundTransforming Growth Factor betaInternal medicineInternal MedicinemedicinePrevalenceAlbuminuriaHumansInterventricular septumCreatinineEjection fractionbusiness.industryMiddle Agedmedicine.diseasemedicine.anatomical_structureEndocrinologyHypertension RenovascularchemistryEchocardiographyCardiologyDisease ProgressionRegression AnalysisFemaleHypertrophy Left VentricularbusinessBiomarkersJournal of human hypertension
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Effects of cerivastatin on adrenergic pathways, hypertrophic growth and TGFbeta expression in adult ventricular cardiomyocytes.

2010

Abstract The effects of statin treatment in the setting of heart failure have already been shown. Nevertheless, there is little knowledge about its influence on adrenergic pathways in cardiomyocytes. Therefore, this study investigated the impact of cerivastatin on adrenoceptor-mediated signalling pathways in isolated adult ventricular cardiomyocytes. It focused on two endpoints: hypertrophic growth and TGFbeta expression. Cultured cardiomyocytes were used to study rac activation (analysed by its translocation into the membrane fraction), ROS formation (H 2 DCF fluorescence) and hypertrophic growth ( 14 C-phenylalanine incorporation). Alpha- and beta-adrenoceptor stimulation showed significa…

Malemedicine.medical_specialtyHistologyAdrenergic receptorMAP Kinase Signaling SystemPyridinesp38 mitogen-activated protein kinasesHeart VentriclesAdrenergicAlpha (ethology)StimulationPharmacologyp38 Mitogen-Activated Protein KinasesPathology and Forensic MedicineTransforming Growth Factor betaInternal medicineReceptors Adrenergic betamedicineAnimalsMyocytes CardiacRats WistarCells CulturedHeart FailurebiologyCerivastatinCell BiologyGeneral MedicineReceptors Adrenergic alphaRatsEnzyme ActivationEndocrinologyGene Expression RegulationNAD(P)H oxidaseMitogen-activated protein kinasebiology.proteinHydroxymethylglutaryl-CoA Reductase InhibitorsReactive Oxygen SpeciesProto-Oncogene Proteins c-aktmedicine.drugEuropean journal of cell biology
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Administration of transforming growth factor-α reduces infarct volume after transient focal cerebral ischemia in the rat

2001

Growth factors promote cell growth and survival and protect the brain from developing injury after ischemia. In this article, the authors examined whether transforming growth factor-α (TGF-α was protective in transient focal ischemia and whether alteration of cerebral circulation was involved. Rats received intraventricular TGF-α (50 ng, either split into 2 doses given 30 minutes before and 30 minutes after middle cerebral artery occlusion (MCAO), or 1 dose given 30 minutes after MCAO) or vehicle. Rats were subjected to 1-hour intraluminal MCAO and cerebral blood flow was recorded continuously by laser-Doppler flowmetry. Infarct volume was measured 1 and 4 days later. The effects of TGF-α o…

Malemedicine.medical_specialtyMicrovascular perfusionCerebral arteriesIschemiaRabbitIn Vitro Techniques030218 nuclear medicine & medical imagingMicrocirculationBrain IschemiaRats Sprague-Dawley03 medical and health sciencesCerebral circulation0302 clinical medicineInternal medicinemedicineAnimalscardiovascular diseasesMiddle cerebral artery occlusionCerebral infarctionbusiness.industryMicrocirculationInfarction Middle Cerebral ArteryTransforming Growth Factor alphamedicine.diseaseRatsCarotid ArteriesNeuroprotective AgentsNeurologyCerebral blood flowVasoconstrictionAnesthesiaBasilar ArteryCerebrovascular CirculationCardiologyRatNeurology (clinical)Rabbitsmedicine.symptomCardiology and Cardiovascular MedicinebusinessGrowth factors030217 neurology & neurosurgeryVasoconstrictionTransforming growth factor
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Post-weaning xenohormone intake affects adult rat submandibular gland in a sex-dependent manner

2018

International audience; OBJECTIVES: We previously reported that maternal exposure to genistein, vinclozolin, ingested alone or in combination, affects submandibular salivary glands of rats' offspring's. Here, we investigated the responsiveness of submandibular gland when such xenohormone exposure occurs later in life. MATERIALS AND METHODS: Chemicals were given orally to male and female Wistar rats (1mg/kg body weight per day), from weaning to adulthood. Submandibular glands and plasma were collected at postnatal day 100 for histological and molecular analysis. RESULTS: Whereas no effect was observed in females, increases in granular convoluted tubules area coupled with a modification of sa…

Malemedicine.medical_specialtyOffspringSubmandibular GlandPhytoestrogenssalivary glandWeaning03 medical and health scienceschemistry.chemical_compound0302 clinical medicineSex Factorsstomatognathic systemInternal medicineProgesterone receptormedicineWeaningAnimalsVinclozolinRNA MessengerCystatin CGeneral DentistryOxazolesbiologySalivary glandbusiness.industryMucinsex-dimorphismAndrogen Antagonists030206 dentistryTransforming Growth Factor alphaSubmandibular glandGenisteinRatsEndocrinologymedicine.anatomical_structureendocrine disruptorsepidermal growth factorOtorhinolaryngologyCystatin CchemistryReceptors Androgen030220 oncology & carcinogenesisbiology.proteinFemalebusiness[SDV.AEN]Life Sciences [q-bio]/Food and Nutrition[SDV.MHEP]Life Sciences [q-bio]/Human health and pathology
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Postexercise myostatin and activin IIb mRNA levels: effects of strength training.

2007

ABSTRACTHULMI, J. J., J. P. AHTIAINEN, T. KAASALAINEN, E. PO¨LLA¨NEN, K. HA¨KKINEN, M. ALEN, H. SELA¨NNE, V. KOVANEN,and A. A. MERO. Postexercise Myostatin and Activin IIb mRNA Levels: Effects of Strength Training. Med. Sci. Sports Exerc., Vol.39, No. 2, pp. 289–297, 2007. Purpose: Muscle hypertrophy is likely to result from the cumulative effects of repeated bouts ofresistance exercise (RE) on postexercise molecular responses. Therefore, we determined muscle growth- and regeneration-relatedmRNA expression in response to a single RE bout both before and after a strength-training (ST) period. By means of this novellongitudinal setting, we examined whether postexercise gene expression at the …

Malemedicine.medical_specialtyTime FactorsWeight LiftingVastus lateralis muscleGene ExpressionPhysical Therapy Sports Therapy and RehabilitationPhysical exerciseMyostatinMyoDMuscle hypertrophyQuadriceps MuscleTransforming Growth Factor betaInternal medicinemedicineHumansOrthopedics and Sports MedicineRNA MessengerLeg pressMuscle SkeletalExerciseMyogeninAgedbiologybusiness.industryAge FactorsMiddle AgedMyostatinmusculoskeletal systemActivinsEndocrinologyCase-Control Studiesbiology.proteinmedicine.symptombusinessMuscle contractionMuscle ContractionMedicine and science in sports and exercise
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