Search results for "Free Radical"

showing 10 items of 263 documents

Oxidative stress triggers cytokinesis failure in hepatocytes upon isolation

2015

Primary hepatocytes are highly differentiated cells and proliferatively quiescent. However, the stress produced during liver digestion seems to activate cell cycle entry by proliferative/dedifferentiation programs that still remain unclear. The aim of this work was to assess whether the oxidative stress associated with hepatocyte isolation affects cell cycle and particularly cytokinesis, the final step of mitosis. Hepatocytes were isolated from C57BL/6 mice by collagenase perfusion in the absence and presence of N-acetyl cysteine (NAC). Polyploidy, cell cycle, and reactive oxygen species (ROS) were studied by flow cytometry (DNA, phospho-histone 3, and CellROX(®) Deep Red) and Western blott…

MaleCellular differentiationGene ExpressionCell Cycle ProteinsCell SeparationBiologymedicine.disease_causeBiochemistrychemistry.chemical_compoundmedicineAnimalsMitosisCells CulturedCytokinesisCyclinFree Radical ScavengersGeneral MedicineGlutathioneCell cycleFlow CytometryMolecular biologyAcetylcysteineCell biologyMice Inbred C57BLOxidative Stressmedicine.anatomical_structurechemistryHepatocyteHepatocytesReactive Oxygen SpeciesCytokinesisOxidative stressFree Radical Research
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Mandarin Juice Improves the Antioxidant Status of Hypercholesterolemic Children

2008

Background: Oxidative stress has been linked to such degenerative diseases as atherosclerosis, and it has been suggested that increased dietary intake of antioxidants may reduce its progression. Objective: To determine the effect of mandarin juice consumption on biomarkers related to oxidative stress in hypercholesterolemic children. Materials and Methods: The diet of 48 children with plasma cholesterol >200 mg/dL and low-density lipoprotein cholesterol >130 mg/dL was supplemented for 28 days with 500 mL/day of pure (100%) mandarin juice (Citrus clementina Hort. ex Tan.). The composition of the mandarin juice was analyzed, and its antioxidant antiradical activity was evaluated in vitro. Mal…

MaleCitrusmedicine.medical_specialtyAntioxidantmedicine.medical_treatmentHypercholesterolemiaNutritional StatusAscorbic AcidProtein oxidationmedicine.disease_causeAntioxidantsBeverageschemistry.chemical_compoundMalondialdehydeInternal medicinemedicineHumansVitamin EChildbusiness.industryCholesterolVitamin EGastroenterologyFree Radical ScavengersGlutathioneMalondialdehydeLipidsOxidative StressCholesterolEndocrinologychemistryDietary SupplementsPediatrics Perinatology and Child HealthFemaleLipid PeroxidationbusinessOxidation-ReductionBiomarkersOxidative stressLipoproteinJournal of Pediatric Gastroenterology & Nutrition
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Allopurinol has Immunomodulating Activity following Topical and Systemic Application in Experimental Autoimmune Uveitis

2001

PurposeAllopurinol has beneficial effects in the systemic treatment of lens-induced uveitis and experimental autoimmune uveitis (EAU). This is believed to be due to a reduction of oxidative tissue damage through its dose-dependent free radical scavenging ability, and to an immunomodulating effect. The purpose of this study was to demonstrate the immunological effects on the IgG-antibody repertoire in EAU after topical and systemic allopurinol and steroids.MethodsWe assigned 43 male Lewis rats to 6 different groups: healthy rats (BASE, n=3), EAU without therapy (CTRL, n=9); systemic treatment with allopurinol (ALSYS, n=9, 50 mg/kg body wt.i.v., given every three days for two weeks), topical …

MaleDrugmedicine.medical_specialtyAdministration TopicalAllopurinolmedicine.medical_treatmentmedia_common.quotation_subjectBlotting WesternAllopurinol hypersensitivity syndromeAllopurinol030204 cardiovascular system & hematologyPharmacologyAutoantigens030226 pharmacology & pharmacyAutoimmune DiseasesUveitis03 medical and health sciences0302 clinical medicineAdjuvants ImmunologicInternal medicinemedicineAnimalsGlucocorticoidsAutoantibodiesmedia_commonAutoimmune diseaseChemotherapybusiness.industryAutoantibodyFree Radical ScavengersGeneral Medicinemedicine.diseaseRatsDisease Models AnimalOphthalmologyEndocrinologyMethylprednisoloneRats Inbred LewImmunoglobulin GInjections IntravenousElectrophoresis Polyacrylamide GelbusinessUveitismedicine.drugEuropean Journal of Ophthalmology
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Heme oxygenase-1: a novel key player in the development of tolerance in response to organic nitrates.

2007

Objective— Nitrate tolerance is likely attributable to an increased production of reactive oxygen species (ROS) leading to an inhibition of the mitochondrial aldehyde dehydrogenase (ALDH-2), representing the nitroglycerin (GTN) and pentaerythrityl tetranitrate (PETN) bioactivating enzyme, and to impaired nitric oxide bioactivity and signaling. We tested whether differences in their capacity to induce heme oxygenase-1 (HO-1) might explain why PETN and not GTN therapy is devoid of nitrate and cross-tolerance. Methods and Results— Wistar rats were treated with PETN or GTN (10.5 or 6.6 μg/kg/min for 4 days). In contrast to GTN, PETN did not induce nitrate tolerance or cross-tolerance as assess…

MaleEndotheliumPharmacologySensitivity and SpecificityNitric oxidechemistry.chemical_compoundNitroglycerinRandom AllocationDrug toleranceReference ValuesmedicineAnimalsPentaerythritol TetranitrateRats WistarHemeCyclic GMPChromatography High Pressure LiquidProbabilitychemistry.chemical_classificationReactive oxygen speciesbiologyDrug ToleranceFree Radical ScavengersAldehyde DehydrogenaseRatsHeme oxygenaseFerritinDisease Models Animalmedicine.anatomical_structurechemistryBiochemistrycardiovascular systembiology.proteinEndothelium VascularCardiology and Cardiovascular MedicineReactive Oxygen SpeciesHeme Oxygenase-1HeminArteriosclerosis, thrombosis, and vascular biology
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In vivo and in vitro antioxidant properties of furosemide

2003

The aim of this study was to investigate in vivo and in vitro antioxidant properties of furosemide. In vitro, human red blood cells were submitted to oxidative stress (AAPH), in absence or in presence of different concentrations of furosemide. Potassium efflux was measured in order to quantify the oxidative stress after the action of AAPH on red blood cells. Allophycocyanin assay was also used to investigate antioxidant capacities of furosemide. For the in vivo experiment, male Wistar rats were used. A control group (n = 5) was treated by a daily intraperitoneal injection of saline solution (0.2 ml); 2 other groups (J0 and J+) were treated for 7 days by one daily intraperitoneal injection o…

MaleErythrocytesAntioxidantmedicine.medical_treatmentIntraperitoneal injectionAmidinesOxidative phosphorylationIn Vitro TechniquesPharmacologymedicine.disease_causeAntioxidantsGeneral Biochemistry Genetics and Molecular BiologyFurosemideIn vivomedicineAnimalsHumansGeneral Pharmacology Toxicology and PharmaceuticsDiureticsSalineChemistryPhycocyaninFurosemideFree Radical ScavengersGeneral MedicineIn vitroRatsOxidative StressAnesthesiaPotassiumOxidative stressmedicine.drugLife Sciences
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Calcium overload increases oxidative stress in old rat gastrocnemius muscle

2005

International audience; In order to challenge in vivo muscle Ca2+ homeostasis and analyze consequences on mitochondrial H2O2 release (MHR) and sarcopenia, we injected Ca2+ ionophore A23187 (200 µg/kg, ip) in adult and old rats and measured gastrocnemius mass and mitochondrial Ca2+ content (MCC) using radioactive Ca2+ 48 h after injection. In a second experiment performed in old rats, we measured isocitrate dehydrogenase (ICDH) activity as an index of MCC, MHR, mitochondrial respiration, citrate synthase, COX and antioxydant enzyme activities 24 h after a 150 µg/kg injection. In adult rats, muscle mass and MCC were unchanged by A23187. In old rats, MCC increased 24 h after injection as refle…

MaleGPXAGINGOxygen ConsumptionMITOCHONDRIAAnimalsIONOPHORE A 23187Rats WistarFREE RADICALSODMuscle SkeletalCalcimycinIonophoresfood and beveragesHydrogen PeroxideOrgan Size[SDV.SP]Life Sciences [q-bio]/Pharmaceutical sciencesA23187Mitochondria MuscleRatsENZYME GLUTATHION PEROXYDASE[SDV.SP] Life Sciences [q-bio]/Pharmaceutical sciencesOxidative StressENZYME SUPEROXYDE DISMUTASECalciumSKELETAL MUSCLE
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Lymphocytic Mitochondrial Aconitase Activity is Reduced in Alzheimer's Disease and Mild Cognitive Impairment

2015

Background: Specific mechanisms behind the role of oxidative/nitrosative stress and mitochondrial dysfunction in Alzheimer's disease (AD) pathogenesis remain elusive. Mitochondrial aconitase (ACO2) is a Krebs cycle enzyme sensitive to free radicalmediated damage. Objective: We assessed activity and expression of ACO2 extracted from blood lymphocytes of subjects with AD, mild cognitive impairment (MCI), older adults with normal cognition (OCN, age >= 65 years), and younger adults with normal cognition (YCN, age < 65 years). Plasma levels and activities of antioxidants were also measured. Methods: Blood samples were collected from 28 subjects with AD, 22 with MCI, 21 OCN, and 19 YCN. ACO2 act…

MalePathologyantioxidantAntioxidantmedicine.medical_treatmentLymphocyteMitochondrionmedicine.disease_causePolymerase Chain ReactionPathogenesisVitamin Eoxidative stressLymphocytesaconitase (aconitate hydratase)Aconitate Hydratasereactive oxygen speciesGeneral NeuroscienceACO2General MedicineAlzheimer's diseasemitochondriaPsychiatry and Mental healthClinical Psychologyantioxidantsmedicine.anatomical_structureDisease ProgressionSettore MED/26 - NeurologiaFemaleAlzheimer diseaseAlzheimer's diseaseAzheimer diseasereactive nitrogen speciemedicine.medical_specialtyaconitase (aconitate hydratase); Alzheimer disease; antioxidants; free radicals; lymphocyte; mild cognitive impairment; mitochondria; oxidative stress; reactive nitrogen species; reactive oxygen speciesBlotting Westernfree radicalslymphocytemild cognitive impairmentInternal medicinemedicineHumansCognitive DysfunctionRNA MessengerAgedfree radicaloxidative strebusiness.industryVitamin EAconitasimedicine.diseasereactive nitrogen speciesEndocrinologyGeriatrics and GerontologyAlzheimer's disease; Aconitasi; oxidative stress; Aconitase (aconitate hydratase) Azheimer disease antioxidants free radicals lymphocyte mild cognitive impairment mitochondria oxidative stress reactive nitrogen species reactive oxygen speciesMental Status SchedulebusinessBiomarkersOxidative stressJournal of Alzheimer's Disease
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Increased Hypoxic Tolerance by Chemical Inhibition of Oxidative Phosphorylation: “Chemical Preconditioning”

1997

A short ischemic episode preceding sustained ischemia is known to increase tolerance against ischemic cell death. We report early-onset long-lasting neuroprotection against in vitro hypoxia by preceding selective chemical inhibition of oxidative phosphorylation: “chemical preconditioning.” The amplitude of CA1population spikes (psap) in hippocampal slices prepared from control animals (control slices) was 31 ± 27% (mean ± SD) upon 45-min recovery from 15-min in vitro hypoxia. In slices prepared from animals treated in vivo with 20 mg/kg 3-nitropropionate (3-np) 1–24 h prior to slice preparation (preconditioned slices), psap improved to 90 ± 15% (p &lt; 0.01). Posthypoxic oxygen free radical…

MalePotassium ChannelsFree RadicalsPopulationIschemiaNerve Tissue ProteinsBiologyPharmacologyHippocampusNeuroprotectionOxidative PhosphorylationBrain Ischemia030218 nuclear medicine & medical imagingGlibenclamide03 medical and health sciencesAdenosine Triphosphate0302 clinical medicineSlice preparationIn vivoGlyburidemedicineAnimalsEnzyme InhibitorsRats WistarHypoxia BraineducationNeuronseducation.field_of_studyAntagonistHypoxia (medical)NADNitro Compoundsmedicine.diseaseCell HypoxiaRatsSuccinate DehydrogenaseNeuroprotective AgentsNeurologyAnesthesiaNeurology (clinical)Propionatesmedicine.symptomReactive Oxygen SpeciesCardiology and Cardiovascular Medicine030217 neurology & neurosurgerymedicine.drugJournal of Cerebral Blood Flow &amp; Metabolism
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Glutamine potentiates TNF-α-induced tumor cytotoxicity

2001

L-glutamine (Gln) sensitizes tumor cells to tumor necrosis factor (TNF)-alpha-induced cytotoxicity. The type and mechanism of cell death induced by TNF-alpha was studied in Ehrlich ascites tumor (EAT)-bearing mice fed a Gln-enriched diet (GED; where 30% of the total dietary nitrogen was from Gln). A high rate of Gln oxidation promotes a selective depletion of mitochondrial glutathione (mtGSH) content to approximately 58% of the level found in tumor mitochondria of mice fed a nutritionally complete elemental diet (standard diet, SD). The mechanism of mtGSH depletion involves a glutamate-induced inhibition of GSH transport from the cytosol into mitochondria. The increase in reactive oxygen in…

MaleProgrammed cell deathFree RadicalsCell SurvivalGlutamineApoptosisCytochrome c GroupMitochondrionBiologyBiochemistryMembrane PotentialsMiceNecrosischemistry.chemical_compoundAdenosine TriphosphateSuperoxidesPhysiology (medical)Tumor Cells CulturedAnimalsButhionine sulfoximineCaspase 3Tumor Necrosis Factor-alphaDrug SynergismHydrogen PeroxideGlutathioneGlutathioneMolecular biologyDietMitochondriaCell biologyOxygenGlutamineOxidative StressCytosolProto-Oncogene Proteins c-bcl-2chemistryApoptosisCaspasesReactive Oxygen SpeciesOxidation-ReductionCell DivisionIntracellularFree Radical Biology and Medicine
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Oral N-acetylcysteine reduces bleomycin-induced lung damage and mucin Muc5ac expression in rats

2003

Oxidative stress is involved in the pathogenesis of pulmonary fibrosis, therefore antioxidants may be of therapeutic value. Clinical work indicates that N ‐acetylcysteine (NAC) may be beneficial in this disease. The activity of this antioxidant was examined on bleomycin-induced lung damage, mucus secretory cells hyperplasia and mucin Muc5ac gene expression in rats. NAC (3 mmol·kg−1·day−1) or saline was given orally to Sprague-Dawley rats for 1 week prior to a single intratracheal instillation of bleomycin (2.5 U·kg−1) and for 14 days postinstillation. NAC decreased collagen deposition in bleomycin-exposed rats (hydroxyproline content was 4,257±323 and 3,200±192 µg·lung−1 in vehicle- and NAC…

MalePulmonary and Respiratory MedicinePathologymedicine.medical_specialtyPulmonary FibrosisAdministration OralGene ExpressionMucin 5ACPharmacologyBleomycinmedicine.disease_causeRats Sprague-DawleyAcetylcysteineBleomycinHydroxyprolinechemistry.chemical_compoundAdministration InhalationPulmonary fibrosismedicineAnimalsLungAntibiotics AntineoplasticHyperplasiabusiness.industryMucinMucinsFree Radical Scavengersrespiratory systemmedicine.diseaseMucusAcetylcysteineRatsrespiratory tract diseasesOxidative StresschemistryModels AnimalRespiratory epitheliumbusinessOxidative stressmedicine.drugEuropean Respiratory Journal
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