Search results for "G-Protein-Coupled"

showing 10 items of 110 documents

Host microbiota constantly control maturation and function of microglia in the CNS.

2015

As the tissue macrophages of the CNS, microglia are critically involved in diseases of the CNS. However, it remains unknown what controls their maturation and activation under homeostatic conditions. We observed substantial contributions of the host microbiota to microglia homeostasis, as germ-free (GF) mice displayed global defects in microglia with altered cell proportions and an immature phenotype, leading to impaired innate immune responses. Temporal eradication of host microbiota severely changed microglia properties. Limited microbiota complexity also resulted in defective microglia. In contrast, recolonization with a complex microbiota partially restored microglia features. We determ…

Central Nervous SystemMaleCellGut–brain axis610 Medicine & healthBiologydigestive systemReceptors G-Protein-CoupledMiceImmunitymedicineAnimalsHomeostasis10239 Institute of Laboratory Animal ScienceReceptorInnate immune systemMicrogliaGeneral NeuroscienceMicrobiota2800 General NeuroscienceFatty Acids VolatilePhenotypeImmunity InnateMice Inbred C57BLmedicine.anatomical_structurenervous systemImmunology570 Life sciences; biology590 Animals (Zoology)FemaleMicrogliaNeuroscienceHomeostasisNature neuroscience
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The evolutionary history and tissue mapping of GPR123: specific CNS expression pattern predominantly in thalamic nuclei and regions containing large …

2007

The Adhesion family of G protein-coupled receptors (GPCRs) includes 33 receptors and is the second largest GPCR family. Most of these proteins are still orphans and fairly little is known of their tissue distribution and evolutionary context. We report the evolutionary history of the Adhesion family protein GPR123 as well as mapping of GPR123 mRNA expression in mouse and rat using in situ hybridization and real-time PCR, respectively. GPR123 was found to be well conserved within the vertebrate lineage, especially within the transmembrane regions and in the distal part of the cytoplasmic tail, containing a potential PDZ binding domain. The real-time PCR data indicates that GPR123 is predomin…

Central Nervous SystemMaleModels MolecularNeuronal signal transductionPDZ domainGene ExpressionContext (language use)In situ hybridizationBiologyBiochemistryReceptors G-Protein-CoupledMiceCellular and Molecular NeuroscienceAnimalsHumansTissue DistributionRNA MessengerNeural Cell Adhesion MoleculesIn Situ HybridizationPhylogenyG protein-coupled receptorReverse Transcriptase Polymerase Chain ReactionPyramidal CellsSubiculumRatsCell biologySignal transductionSequence AlignmentNeuroscienceBinding domainJournal of Neurochemistry
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Primary cilia are required for cerebellar development and Shh-dependent expansion of progenitor pool

2008

Cerebellar granule cell precursors (GCPs), which give rise to the most abundant neuronal type in the mammalian brain, arise from a restricted pool of primary progenitors in the rhombic lip (RL). Sonic hedgehog (Shh) secreted by developing Purkinje cells is essential for the expansion of GCPs and for cerebellar morphogenesis. Recent studies have shown that the primary cilium concentrates components of Shh signaling and that this structure is required for Shh signaling. GCPs have a primary cilium on their surface [Del Cerro, M.P., Snider, R.S. (1972). Studies on the developing cerebellum. II. The ultrastructure of the external granular layer. J Comp Neurol 144, 131-64.]. Here, we show that 1)…

CerebellumKinesinsReceptors G-Protein-CoupledMicePurkinje Cells0302 clinical medicinePrimary ciliaCerebellumSonic hedgehogPromoter Regions GeneticRhombic lipGenetics0303 health scienceseducation.field_of_studyCiliumStem CellsSonic hedgehogjoubert syndromeCerebellar developmentSmoothened ReceptorCell biologyneurogenesismedicine.anatomical_structurecerebellar developmentembryonic structuresanimal structuresNeurogenesisPopulationMice TransgenicBiologyKif3aArticle03 medical and health sciencessonic hedgehogprimary ciliaJoubert syndromeGlial Fibrillary Acidic ProteinmedicineAnimalsHumansKIF3AHedgehog ProteinsCiliaeducationMolecular Biology030304 developmental biologyCell BiologyGranule cellMice Inbred C57BLbiology.proteinSmoothened030217 neurology & neurosurgeryDevelopmental Biology
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The Bile Acid Receptor GPBAR-1 (TGR5) Modulates Integrity of Intestinal Barrier and Immune Response to Experimental Colitis

2011

Background GP-BAR1, a member G protein coupled receptor superfamily, is a cell surface bile acid-activated receptor highly expressed in the ileum and colon. In monocytes, ligation of GP-BAR1 by secondary bile acids results in a cAMP-dependent attenuation of cytokine generation. Aims To investigate the role GP-BAR1 in regulating intestinal homeostasis and inflammation-driven immune dysfunction in rodent models of colitis. Methods Colitis was induced in wild type and GP-BAR1−/− mice by DSS and TNBS administration. Potential GP-BAR1 agonists were identified by in silico screening and computational docking studies. Results GP-BAR1−/− mice develop an abnormal morphology of colonic mucous cells a…

Cholera ToxinCD14Biophysicslcsh:MedicineInflammationGastroenterology and HepatologyBiologyLigandsBiochemistryPermeabilityReceptors G-Protein-CoupledTight JunctionsMiceCrohn DiseaseCiprofloxacinMolecular Cell BiologymedicineAnimalsUlcerative ColitisIntestinal MucosaProtein PrecursorsBiomacromolecule-Ligand InteractionsColitislcsh:ScienceReceptorBiologyMice KnockoutMultidisciplinaryIntestinal permeabilityHaptoglobinsPhysicsInflammatory Bowel Diseaselcsh:RImmunityZonulinColitisFlow Cytometrymedicine.diseaseMolecular biologyG protein-coupled bile acid receptorImmunologyTLR4Medicinelcsh:Qmedicine.symptomCytometryResearch ArticlePLoS ONE
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A variant of Smurf2 protects mice against colitis-associated colon cancer by inducing transforming growth factor β signaling.

2011

Background & Aims Transforming growth factor (TGF)-β signaling, which is down-regulated by the E3 ubiquitin ligase Smad ubiquitin regulating factor 2 (Smurf2), promotes development of cancer. We identified a splice variant of Smurf2 (ΔE2Smurf2) and investigated its role in colon carcinogenesis in mice. Methods Colitis-associated colon cancer was induced in mice by administration of azoxymethane, followed by 3 cycles of oral administration of dextran sodium sulfate. Messenger RNA levels of Smurf2 in colon tumors and control tissue were measured by quantitative polymerase chain reaction; lymphocyte and cytokine levels were measured in tumor and tissue samples. Results Tumor-infiltrating CD4 +…

Colorectal cancermedicine.medical_treatmentLymphocyteUbiquitin-Protein LigasesSMADBiologyProinflammatory cytokineReceptors G-Protein-Coupledchemistry.chemical_compoundMiceTransforming Growth Factor betamedicineAnimalsCells CulturedHepatologyAzoxymethaneGene Expression ProfilingGastroenterologymedicine.diseaseColitisMolecular biologyUbiquitin ligaseMice Inbred C57BLProto-Oncogene Proteins c-kitCytokinemedicine.anatomical_structureHyaluronan ReceptorschemistryColonic Neoplasmsbiology.proteinCancer researchTransforming growth factorSignal TransductionGastroenterology
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Molecular cloning of rat G-protein-coupled receptor kinase 6 (GRK6) from brain tissue, and its mRNA expression in different brain regions and periphe…

1997

The rat G-protein-coupled receptor kinase 6 (GRK6) cDNA was cloned from rat brain tissue by a combination of reverse-transcription polymerase chain reactions (RT-PCR), based on homology to the cloned human GRK6, and rapid amplification of cDNA ends (RACE-PCR). We obtained a clone of 2817 bp with an open reading frame of 1731 bp encoding for a protein of 576 amino acids that is 96.7% identical and 97.9% similar to its human counterpart. mRNA was detectable in all brain areas examined. In addition, GRK6 was expressed in skeletal muscle, small intestine, aorta, liver, heart, lung, thymus, stomach, uterus and kidney.

DNA ComplementaryTranscription GeneticMolecular Sequence DataProtein Serine-Threonine KinasesMolecular cloningBiologyPolymerase Chain ReactionOpen Reading FramesCellular and Molecular NeuroscienceRapid amplification of cDNA endsGTP-Binding ProteinsComplementary DNAGene expressionAnimalsHumansAmino Acid SequenceRNA MessengerCloning MolecularProtein kinase AMolecular BiologyG protein-coupled receptor kinaseMessenger RNABase SequenceSequence Homology Amino AcidBrainReceptor Protein-Tyrosine KinasesG-Protein-Coupled Receptor KinasesMolecular biologyRatsOpen reading frameOrgan SpecificityFemaleSequence AlignmentMolecular Brain Research
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Leucine-rich repeat-containing G-protein-coupled receptor 5 (Lgr5) as a putative human endometrial stem cell marker†

2013

The endometrium is recognized for its remarkable regenerative and remodeling capacity. Every month this hormonally regu- lated organ undergoes cycles of growth (from 0.5-2 to 7 mm), regression and shedding of two-third of the tissue, leading to its monthly renewal that occurs � 400 times in a woman's reproductive lifetime. Several groups have suggested the existence of a human endometrial somatic stem cell (SSC) population located around the spiral arterioles of the basalis. Different groups have isolated, identified and charac- terized putative endometrial SSC populations in human endometrium based on the general features of undifferentiated cells, such as slow cycling detected using the 5…

Embryologymedicine.medical_specialtyPopulationBiologyEndometriumStem cell markerLgr5 human endometrium regenerative tissues somatic stem cells stem cell markerReceptors G-Protein-CoupledEndometriumMiceSide populationInternal medicineGeneticsmedicineAnimalsHumanseducationMolecular Biologyeducation.field_of_studyWnt signaling pathwayLGR5Obstetrics and GynecologyCell BiologyCell biologyAdult Stem Cellsmedicine.anatomical_structureEndocrinologyReproductive MedicineFemaleStem cellDevelopmental BiologyAdult stem cellMHR: Basic science of reproductive medicine
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Regulation of cuticular hydrocarbon profile maturation by Drosophila tanning hormone, bursicon, and its interaction with desaturase activity

2016

International audience; Shortly after emergence the exoskeleton (cuticle) of adult insects is rapidly expanded, hardened (sclerotized), and pigmented (melanized). In parallel with this process, the oenocytes, which are large polyploid cells located below the abdominal epidermis, secrete onto the cuticle a cocktail of cuticular hydrocarbons (CHs) and waxes. These improve the waterproofing of the cuticle, and also provide important chemosensory and pheromonal cues linked with gender, age, and species differentiation. The hardening and pigmentation of the new cuticle are controlled by the neurohormone, bursicon, and its receptor, encoded by the DLGR2 receptor, rickets (rk); by contrast, little…

Fatty Acid DesaturasesMale0106 biological sciences0301 basic medicinemelanogasterInvertebrate HormonesreceptorMutantInsectMolting[ SDV.BA ] Life Sciences [q-bio]/Animal biology01 natural sciencesBiochemistryReceptors G-Protein-Coupledchemistry.chemical_compoundMelanogasterNeurohormoneDrosophila ProteinsReceptormedia_commonBursiconbiologyPigmentationcis-Vaccenyl acetate[SDV.BA]Life Sciences [q-bio]/Animal biologysex-pheromonesCell biologyExoskeletonDrosophila melanogasterontogenySex pheromonecourtshipFemaleMoultingmedia_common.quotation_subjectmales010603 evolutionary biology03 medical and health sciencesBiosynthesisBotanyevolutionexpressionAnimalsMolecular Biologybehaviorfungibiology.organism_classificationHydrocarbons030104 developmental biologychemistryInsect ScienceEclosionbiosynthesisInsect
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G-protein-coupled receptor kinase 5 polymorphism and Takotsubo cardiomyopathy.

2015

BACKGROUND: Takotsubo cardiomyopathy (TTC) is an increasingly reported clinical syndrome that mimics acute myocardial infarction without obstructive coronary artery disease and is characterized by transient systolic dysfunction of the apical and/or mid-segments of the left ventricle. The syndrome mainly occurs in postmenopausal women with high adrenergic state conditions. Nowadays, the pathophysiology of TTC is not yet known and the possibility of a genetic predisposition is controversial. AIMS: The purpose of this study was to assess the genetic susceptibility to TTC through analysis of the L41Q polymorphism of the G-protein-coupled receptor kinase 5 (GRK5). METHODS AND RESULTS: In a cohor…

G-Protein-Coupled Receptor Kinase 5Malemedicine.medical_specialtyCardiomyopathyInfarctionCohort StudiesGene FrequencyG-Protein-Coupled Receptor Kinase 5Takotsubo CardiomyopathyInternal medicineGenotypemedicineGenetic predispositionHumansSettore MED/05 - Patologia ClinicaGenetic Predisposition to Diseasecardiovascular diseasesgenotype G-protein-coupled receptor kinase 5 gene polymorphism Takotsubo cardiomyopathyAgedPolymorphism Geneticbusiness.industryCase-control studyGeneral MedicineMiddle Agedmedicine.diseaseSettore MED/11 - Malattie Dell'Apparato Cardiovascolaremedicine.anatomical_structureVentricleCase-Control StudiesCardiologyFemaleCardiology and Cardiovascular MedicinebusinessCohort study
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Myocardial G Protein Receptor–Coupled Kinase Expression Correlates With Functional Parameters and Clinical Severity in Advanced Heart Failure

2010

In heart failure (HF), sympathetic hyperactivation induces deleterious effects in myocardial β-adrenergic signaling, with receptor down-regulation and desensitization mediated by G protein receptor-coupled kinases (GRKs). We hypothesised that changes in GRK isoforms may be associated with clinical status in advanced HF, using the Interagency Registry for Mechanically Assisted Circulatory Support (INTERMACS) scale.We included 31 patients with advanced HF undergoing transplantation. According to INTERMACS profiles, mRNA and protein levels of GRK isoforms in left ventricular (LV) myocardium were analyzed and compared with nonfailing LV samples.In failing LV myocardium, GRK2 and GRK5 (but not G…

G-Protein-Coupled Receptor Kinase 5Malemedicine.medical_specialtyG-Protein-Coupled Receptor Kinase 2Down-RegulationPolymerase Chain ReactionSeverity of Illness IndexGene Expression Regulation EnzymologicInternal medicinemedicineHumansRNA MessengerRegistriesReceptorG protein-coupled receptorHeart FailureG protein-coupled receptor kinasebiologybusiness.industryMyocardiumBeta adrenergic receptor kinaseStroke volumeMiddle AgedG-Protein-Coupled Receptor Kinasesmedicine.diseaseTransplantationEndocrinologySpainHeart failureCirculatory systembiology.proteinFemaleHeart-Assist DevicesCardiology and Cardiovascular MedicinebusinessJournal of Cardiac Failure
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