Search results for "GENOMICS"

showing 10 items of 1255 documents

Translation of genomics-guided RNA-based personalised cancer vaccines: towards the bedside

2014

Cancer is a disease caused by DNA mutations. Cancer therapies targeting defined functional mutations have shown clinical benefit. However, as 95% of the mutations in a tumour are unique to that single patient and only a small number of mutations are shared between patients, the addressed medical need is modest. A rapidly determined patient-specific tumour mutation pattern combined with a flexible mutation-targeting drug platform could generate a mutation-targeting individualised therapy, which would benefit each single patient. Next-generation sequencing enables the rapid identification of somatic mutations in individual tumours (the mutanome). Immunoinformatics enables predictions of mutat…

Cancer ResearchMutationbusiness.industryGenetic enhancementDrug Evaluation PreclinicalCancerGenomicsmedicine.diseasePrecision medicinemedicine.disease_causeBioinformaticsCancer VaccinesTranslational Research BiomedicalBreast cancerOncologyImmunologyMutationMedicineHumansPersonalized medicineCancer vaccineMinireviewRNA NeoplasmPrecision MedicinebusinessBritish Journal of Cancer
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Broad-spectrum Cross-resistance to Anticancer Drugs Mediated by Epidermal Growth Factor Receptor

2019

BACKGROUND The oncogenic role of epidermal growth factor receptor (EGFR) has been intensively studied. However, its emerging role in drug resistance has not been fully addressed. MATERIALS AND METHODS This study systematically investigated the correlation of mRNA and protein expression of EGFR, as well as gene amplification and mutations with the log-transformed half-maximal inhibitory concentration (log10IC50) values obtained from the NCI panel of 60 human tumor cell lines against 83 standard anticancer agents and the top 10 natural cytotoxic products previously screened by us. RESULTS EGFR protein expression, rather than other measurements, was most frequently associated with drug respons…

Cancer ResearchOncogenebiologyChemistryTopoisomeraseAntineoplastic AgentsGeneral MedicineDrug resistanceErbB Receptors03 medical and health sciences0302 clinical medicineOncologyDrug Resistance NeoplasmCell cultureCell Line TumorNeoplasms030220 oncology & carcinogenesisPharmacogenomicsbiology.proteinCancer researchHumansCytotoxic T cellRNA MessengerEpidermal growth factor receptorCross-resistanceAnticancer Research
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Regulon-Specific Control of Transcription Elongation across the Yeast Genome

2009

Transcription elongation by RNA polymerase II was often considered an invariant non-regulated process. However, genome-wide studies have shown that transcriptional pausing during elongation is a frequent phenomenon in tightly-regulated metazoan genes. Using a combination of ChIP-on-chip and genomic run-on approaches, we found that the proportion of transcriptionally active RNA polymerase II (active versus total) present throughout the yeast genome is characteristic of some functional gene classes, like those related to ribosomes and mitochondria. This proportion also responds to regulatory stimuli mediated by protein kinase A and, in relation to cytosolic ribosomal-protein genes, it is medi…

Cancer ResearchSaccharomyces cerevisiae Proteinslcsh:QH426-470Transcription GeneticComputational Biology/Transcriptional RegulationRNA polymerase IISaccharomyces cerevisiaeRegulonGenètica molecularSaccharomycesTranscripció genèticaTranscription (biology)GeneticsTranscriptional regulationMolecular BiologyRNA polymerase II holoenzymeGeneGenetics (clinical)Ecology Evolution Behavior and SystematicsGeneticsbiologyGenetics and Genomics/Functional GenomicsMolecular Biology/Transcription ElongationHigh Mobility Group ProteinsGenetics and Genomics/Gene ExpressionElongation factorDNA-Binding Proteinslcsh:GeneticsTAF4biology.proteinRNARNA Polymerase IITranscription factor II DGenome FungalTranscriptional Elongation FactorsBiochemistry/Transcription and TranslationResearch Article
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Serratia symbiotica from the aphid Cinara cedri: a missing link from facultative to obligate insect endosymbiont.

2011

The genome sequencing of Buchnera aphidicola BCc from the aphid Cinara cedri, which is the smallest known Buchnera genome, revealed that this bacterium had lost its symbiotic role, as it was not able to synthesize tryptophan and riboflavin. Moreover, the biosynthesis of tryptophan is shared with the endosymbiont Serratia symbiotica SCc, which coexists with B. aphidicola in this aphid. The whole-genome sequencing of S. symbiotica SCc reveals an endosymbiont in a stage of genome reduction that is closer to an obligate endosymbiont, such as B. aphidicola from Acyrthosiphon pisum, than to another S. symbiotica, which is a facultative endosymbiont in this aphid, and presents much less gene decay…

Cancer ResearchSerratialcsh:QH426-470RiboflavinPseudogeneGenomeDNA sequencingBacterial ProteinsBuchneraEnterobacteriaceaePhylogeneticsBotanyGeneticsAnimalsAmino AcidsSymbiosisMolecular BiologyPhylogenyGenetics (clinical)Ecology Evolution Behavior and SystematicsGeneticsComparative genomicsObligatebiologyTryptophanbiology.organism_classificationBiological EvolutionAcyrthosiphon pisumlcsh:GeneticsAphidsBuchneraGenome BacterialMetabolic Networks and PathwaysPseudogenesPLoS Genetics
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Cell volume homeostatically controls the rDNA repeat copy number and rRNA synthesis rate in yeast

2021

[Abstract] The adjustment of transcription and translation rates to the changing needs of cells is of utmost importance for their fitness and survival. We have previously shown that the global transcription rate for RNA polymerase II in budding yeast Saccharomyces cerevisiae is regulated in relation to cell volume. Total mRNA concentration is constant with cell volume since global RNApol II-dependent nascent transcription rate (nTR) also keeps constant but mRNA stability increases with cell size. In this paper, we focus on the case of rRNA and RNA polymerase I. Contrarily to that found for RNA pol II, we detected that RNA polymerase I nTR increases proportionally to genome copies and cell s…

Cancer ResearchTranscription GeneticCellGene ExpressionRNA polymerase IIYeast and Fungal ModelsProtein SynthesisQH426-470HaploidyBiochemistryPolymerasesSirtuin 2Transcription (biology)RNA Polymerase IHomeostasisCell Cycle and Cell DivisionGenetics (clinical)Silent Information Regulator Proteins Saccharomyces cerevisiaebiologyTranscriptional ControlEukaryotaChemical SynthesisGenomicsCell biologyNucleic acidsmedicine.anatomical_structureExperimental Organism SystemsRibosomal RNARNA polymeraseCell ProcessesRNA Polymerase IIResearch ArticleCell biologyCellular structures and organellesSaccharomyces cerevisiae ProteinsBiosynthetic TechniquesSaccharomyces cerevisiaeSaccharomyces cerevisiaeResearch and Analysis MethodsDNA RibosomalSaccharomycesModel OrganismsCyclinsDNA-binding proteinsmedicineRNA polymerase IGeneticsGene RegulationNon-coding RNAMolecular BiologyEcology Evolution Behavior and SystematicsCell SizeMessenger RNACèl·lules eucariotesOrganismsFungiRNABiology and Life SciencesProteinsGenes rRNARibosomal RNAModels Theoreticalbiology.organism_classificationYeastGenòmicabiology.proteinAnimal StudiesRNARibosomes
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Abstract LB-099: Metabolic vulnerabilities of mesenchymal-like EGFR-mutant NSCLC cells with acquired resistance to tyrosine kinase inhibitors

2018

Abstract Despite the availability of the effective targeted therapies in lung cancer, such as EGFR tyrosine kinase inhibitors (TKIs), drug tolerance and acquired resistance are two common problems that negatively impact lung cancer patient survival. Consequently it is important to understand the molecular basis of the drug tolerance and resistance so that we could formulate effective strategies to ameliorate the efficacy of existing drug and to suppress the emergence of drug resistance. A burgeoning body of literature demonstrated that epigenetic changes by the methylation of DNA and histones are critical in acquired drug resistance, especially in those cancer cells with stem cell-like prop…

Cancer Researchbusiness.industryCancerDrug resistanceMethylationmedicine.diseaseOncologyTumor progressionCancer cellDNA methylationCancer researchMedicineEpigeneticsbusinessEpigenomicsCancer Research
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Evolutionary convergence and nitrogen metabolism in Blattabacterium strain Bge, primary endosymbiont of the cockroach Blattella germanica.

2009

Bacterial endosymbionts of insects play a central role in upgrading the diet of their hosts. In certain cases, such as aphids and tsetse flies, endosymbionts complement the metabolic capacity of hosts living on nutrient-deficient diets, while the bacteria harbored by omnivorous carpenter ants are involved in nitrogen recycling. In this study, we describe the genome sequence and inferred metabolism of Blattabacterium strain Bge, the primary Flavobacteria endosymbiont of the omnivorous German cockroach Blattella germanica. Through comparative genomics with other insect endosymbionts and free-living Flavobacteria we reveal that Blattabacterium strain Bge shares the same distribution of functio…

Cancer Researchfood.ingredientlcsh:QH426-470NitrogenBlochmanniaZoologyCockroachesEvolution Molecular03 medical and health sciencesBlattabacteriumfoodSymbiosisEnterobacteriaceaePhylogeneticsAmmoniabiology.animalBotanyGeneticsAnimalsAmino AcidsSymbiosisMolecular BiologyGenetics (clinical)Ecology Evolution Behavior and SystematicsPhylogeny030304 developmental biology0303 health sciencesGerman cockroachCockroachbiologyPhylogenetic treeEvolutionary Biology/Evolutionary and Comparative Genetics030306 microbiologyAntsBacteroidetesfungiGenomicsbiochemical phenomena metabolism and nutritionbiology.organism_classificationGenetics and Genomics/Microbial Evolution and Genomicslcsh:GeneticsGenetics and Genomics/Genome ProjectsEvolutionary Biology/Microbial Evolution and GenomicsHost-Pathogen InteractionsBacteriaGenome BacterialMetabolic Networks and PathwaysResearch ArticlePLoS genetics
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The Fitness Effects of Random Mutations in Single-Stranded DNA and RNA Bacteriophages

2009

Mutational fitness effects can be measured with relatively high accuracy in viruses due to their small genome size, which facilitates full-length sequencing and genetic manipulation. Previous work has shown that animal and plant RNA viruses are very sensitive to mutation. Here, we characterize mutational fitness effects in single-stranded (ss) DNA and ssRNA bacterial viruses. First, we performed a mutation-accumulation experiment in which we subjected three ssDNA (ΦX174, G4, F1) and three ssRNA phages (Qβ, MS2, and SP) to plaque-to-plaque transfers and chemical mutagenesis. Genome sequencing and growth assays indicated that the average fitness effect of the accumulated mutations was similar…

Cancer Researchlcsh:QH426-470virusesDNA Single-StrandedRNA PhagesBiologymedicine.disease_causeGenomeDNA sequencingGenetics and Genomics/Population GeneticsGeneticsmedicinePoint MutationSelection GeneticMolecular BiologyGenome sizeGenetics (clinical)Ecology Evolution Behavior and SystematicsGeneticsMutationMicrobiology/Microbial Evolution and GenomicsModels GeneticPoint mutationRNARNA PhagesGenetics and Genomics/Microbial Evolution and Genomicslcsh:GeneticsEvolutionary Biology/Microbial Evolution and GenomicsMutagenesisMutationMutagenesis Site-DirectedBacterial virusResearch ArticlePLoS Genetics
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Genome-Wide Haplotype Analysis of Cis Expression Quantitative Trait Loci in Monocytes

2013

In order to assess whether gene expression variability could be influenced by several SNPs acting in cis, either through additive or more complex haplotype effects, a systematic genome-wide search for cis haplotype expression quantitative trait loci (eQTL) was conducted in a sample of 758 individuals, part of the Cardiogenics Transcriptomic Study, for which genome-wide monocyte expression and GWAS data were available. 19,805 RNA probes were assessed for cis haplotypic regulation through investigation of ∼2,1×109 haplotypic combinations. 2,650 probes demonstrated haplotypic p-values >104-fold smaller than the best single SNP p-value. Replication of significant haplotype effects were tested f…

Cancer Researchmedicine.medical_specialtyHereditylcsh:QH426-470Immune Cells[SDV]Life Sciences [q-bio]Quantitative Trait LociImmunologyGene ExpressionGenome-wide association studySingle-nucleotide polymorphismQuantitative trait locusBiologyRegulatory Sequences Nucleic AcidPolymorphism Single NucleotideMonocytes03 medical and health sciences0302 clinical medicineMolecular geneticsmedicineGeneticsGenome-Wide Association StudiesSNPHumansGenetic Predisposition to DiseaseMolecular BiologyBiologyGenetics (clinical)Ecology Evolution Behavior and Systematics030304 developmental biologyGenetics0303 health sciencesQuantitative TraitsComplex TraitsHaplotypeGenomicslcsh:GeneticsGene Expression RegulationHaplotypesExpression quantitative trait lociGenome Expression Analysis030217 neurology & neurosurgeryImputation (genetics)Population GeneticsGenome-Wide Association StudyResearch Article
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EPCT-15. RAPID EPIGENOMIC CLASSIFICATION OF BRAIN TUMORS ENABLES INTRAOPERATIVE NEUROSURGICAL RISK MODULATION

2021

Abstract Background Clear identification of tumor subtype is the main predictor of patient outcome and ultimately what is considered an adequate level of surgical risk. At brain tumor resection, imaging modalities and intraoperative histology often give an ambigious diagnosis, complicating intraoperative surgical decision-making. Here, we report a nanopore DNA methylation analysis (NDMA) sequencing approach combined with machine learning for classification of tumor entities that could be used intraoperatively. Methods We analyzed 50 biopsies obtained from biobanked tissue (43, prospective) or sampled at surgery (7, intraoperative) from 20 female and 30 male patients with a median age of 8 y…

Cancer Researchmedicine.medical_specialtyIntra operativebusiness.industryTranslational/Early Phase Clinical TrialsTumor excisionOncologymedicineAcademicSubjects/MED00300AcademicSubjects/MED00310Neurology (clinical)RadiologyOperative riskbusinessEpigenomicsNeuro-Oncology
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