Search results for "Galactosylceramides"

showing 5 items of 5 documents

Prophylaxis of lipopolysaccharide-induced shock by α-galactosylceramide

2008

AbstractThe NKT cell ligand α-galactosylceramide and its synthetic homologue KRN7000 stimulate rapid and copious secretion of IFN-γ and TNF-α release, both of which are key mediators of LPS-induced shock. We showed that KRN7000, injected before or within 2 h after LPS challenge, was able to prevent endotoxic shock. KRN7000 induced survival when the mice were injected 6, 9, or 12 days before the first injection of LPS, and this protective effect was associated with reduction upon subsequent challenge in the levels of IFN-γ, TNF-α, MCP-1, and an increase of IL-10. Further analysis showed that the animals treated with KRN7000 prior to LPS challenge had lower numbers of F4/80+, NKT, and NK cell…

LipopolysaccharidesTime FactorsLipopolysaccharideLps challengeImmunologyCellGalactosylceramidesPharmacologyrodent endotoxic MCP-1 NKTMicechemistry.chemical_compoundAdjuvants Immunologicα galactosylceramidemedicineAnimalsImmunology and AllergySecretionSettore MED/04 - Patologia Generalebusiness.industryCell BiologyNatural killer T cellLigand (biochemistry)Shock SepticMice Inbred C57BLDisease Models Animalmedicine.anatomical_structurechemistryShock (circulatory)ImmunologyCytokinesmedicine.symptombusinessJournal of Leukocyte Biology
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Immune Modulating Effects of NKT Cells in a Physiologically Low Dose Leishmania major Infection Model after αGalCer Analog PBS57 Stimulation

2014

Leishmaniasis is a parasitic infection affecting ∼12 million people worldwide, mostly in developing countries. Treatment options are limited and no effective vaccines exist to date. Natural Killer T (NKT) cells are a conserved innate-like lymphocyte population with immunomodulating effects in various settings. A number of reports state a role of NKT cells in different models of Leishmania infection. Here, we investigated the effect of NKT cells in a physiologically relevant, intradermal low dose infection model. After inoculation of 103 infectious-stage L. major, comparable numbers of skin-immigrating NKT cells in both susceptible BALB/c mice and resistant C57BL/6 mice were noted. Compared …

medicine.medical_treatmentLymphocyteMedizinPathogenesisNK cellsProtozoologyPathology and Laboratory MedicineCellular typesMedicine and Health SciencesLymphoid OrgansLeishmania majorImmune ResponseLeishmania majorSkinProtozoansMice Inbred BALB Ceducation.field_of_studybiologylcsh:Public aspects of medicineNatural killer T cellInfectious Diseasesmedicine.anatomical_structureCytokineMedical MicrobiologyHost-Pathogen InteractionsWhite blood cellsCytokinesAnatomyResearch ArticleCell biologyBlood cellslcsh:Arctic medicine. Tropical medicinelcsh:RC955-962Immune CellsImmunologyPopulationT cellsLeishmaniasis CutaneousGalactosylceramidesSpleenImmunopathologyMicrobiologyLymphatic SystemImmunomodulationImmune ActivationImmune systemImmunityMicrobial ControlmedicineAnimalsImmunologic FactorseducationImmunity to InfectionsMicrobial PathogensBiology and life sciencesImmunityOrganismsPublic Health Environmental and Occupational HealthImmunoregulationlcsh:RA1-1270Molecular Developmentbiology.organism_classificationAcquired Immune SystemParasitic ProtozoansMice Inbred C57BLDisease Models AnimalAnimal cellsImmune SystemImmunologyNatural Killer T-CellsClinical ImmunologyParasitologyDevelopmental Biology
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Pivotal advance: alpha-galactosylceramide induces protection against lipopolysaccharide-induced shock.

2007

Abstract α−galactosylceramide, a natural killer T cell ligand, and its synthetic homolog, KRN7000, consistently influence IFN-γ and TNF-α release, both mediators of LPS-induced shock. To modify the course of endotoxin shock, we injected KRN7000 at different time points of experimental systemic Shwartzman reaction. Mice treated with KRN7000 survived when it was injected within 2 h before and after LPS challenge. Mice survival was associated with low levels of T helper 1 (Th1) cytokines, such as IFN-γ and TNF-α. By contrast, protection from endotoxin shock was associated with an increase of T helper 2 (Th2) cytokines, like IL-4 and IL-10. A role of Th2 cytokines in counteracting LPS-induced s…

LipopolysaccharidesMaleLipopolysaccharideImmunologyStimulationGalactosylceramidesBiologychemistry.chemical_compoundMiceStructure-Activity RelationshipTh2 CellsIn vivomedicineImmunology and AllergyAnimalsrodent cytokines endotoxinT-cell receptorCell BiologyTh1 CellsLigand (biochemistry)Natural killer T cellShock SepticKiller Cells NaturalMice Inbred C57BLDisease Models AnimalchemistryShock (circulatory)Immunologybiology.proteinDisease ProgressionCytokinesmedicine.symptomAntibodyShwartzman Phenomenon
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Clinical and neuroradiological findings in classic infantile and late-onset globoid-cell leukodystrophy (Krabbe disease)

1996

In the present study the clinical course and imaging of early and late-onset forms of Krabbe disease are analyzed. We report on 11 patients with a biochemical diagnosis of galactosyl ceramide β-galactoside deficiency. Two presented as the classic infantile form and died within the second year of life. In 9 children the first clinical signs, such as gait difficulties and visual failure, started after age 2 years. All these patients developed slow regression of motor and mental capacities, and most of them died within their first decade. In patients of both groups computed tomography (CT) and magnetic resonance imaging (MRI) were performed. In the late-onset form, hypodensities of the central…

MalePediatricsmedicine.medical_specialtyGalactosylceramidesDiseaseWhite matterCentral nervous system diseasemedicineHumansAge of OnsetGenetics (clinical)Pyramidal tractsmedicine.diagnostic_testbusiness.industryBrainInfantMagnetic resonance imagingmedicine.diseaseMagnetic Resonance ImagingLeukodystrophy Globoid Cellmedicine.anatomical_structureEl NiñoSpinal CordChild PreschoolKrabbe diseaseFemaleAge of onsetbusinessTomography X-Ray Computed
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Oligomerization of Vibrio cholerae cytolysin yields a pentameric pore and has a dual specificity for cholesterol and sphingolipids in the target memb…

1999

Vibrio cholerae cytolysin permeabilizes animal cell membranes. Upon binding to the target lipid bilayer, the protein assembles into homo-oligomeric pores of an as yet unknown stoichiometry. Pore formation has been observed with model liposomes consisting of phosphatidylcholine and cholesterol, but the latter were much less susceptible to the cytolysin than were erythrocytes or intestinal epithelial cells. We here show that liposome permeabilization is strongly promoted if cholesterol is combined with sphingolipids, whereby the most pronounced effects are observed with monohexosylceramides and free ceramide. These two lipid species are prevalent in mammalian intestinal brush border membranes…

CeramideCell Membrane PermeabilityPentamerProtein ConformationGalactosylceramidesBiologymedicine.disease_causeBiochemistrychemistry.chemical_compoundPhosphatidylcholinemedicineHumansLipid bilayerMolecular BiologyVibrio choleraeCells CulturedLiposomeSphingolipidsCytotoxinsBrainCell BiologyFluoresceinsLipid MetabolismMembraneCholesterolBiochemistrychemistryVibrio choleraeLiposomesElectrophoresis Polyacrylamide GelCytolysinIsoelectric FocusingThe Journal of biological chemistry
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