Search results for "GeV"

showing 10 items of 709 documents

Aging, longevity, inflammation, and cancer.

2005

Cancer rates increase sharply with age in both sexes, and the majority of cases of cancer occur in patients over the age of 65 years. However, the incidence and mortality for cancer level off around 85-90 years of age, followed by a plateau, or even a decline in the last decades of life. Therefore, it seems reasonable to conclude that centenarians are endowed with a peculiar resistance to cancer. Tumor progression is a complex process that depends on interactions between tumor and host cells. One aspect of the host response, the inflammatory response, is of particular interest because it includes the release of proinflammatory cytokines, some of which may promote tumor growth and hence infl…

AdultAgingTime FactorsGenotypemedia_common.quotation_subjectInflammationDiseaseBiologyGeneral Biochemistry Genetics and Molecular BiologyProinflammatory cytokineImmune systemHistory and Philosophy of ScienceNeoplasmsmedicineHumansRNA MessengerAntigensInterleukin 6media_commonAgedAged 80 and overInflammationInterleukin-6General NeuroscienceLongevityCancerMiddle Agedmedicine.diseaseInterleukin-10Protein Structure TertiaryTumor progressionImmunologybiology.proteinCytokinesmedicine.symptomAnnals of the New York Academy of Sciences
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Association of Klotho Polymorphisms with Healthy Aging: A Systematic Review and Meta-Analysis

2013

Today it is clearly evident that genetic background constitutes an integral part of aging and longevity. Many studies on long-lived people have been conducted emphasizing the role of certain genes in long life. Classic case-control studies, genome-wide association studies, and high-throughput sequencing have permitted identification of a variety of genetic variants seemingly associated with longevity. Over the years, aging research has focused on the insulin/insulin-like growth factor-1 (IGF-1) signaling pathway because of its evolutionarily conserved correlation with life-span extension in model animals. Indeed, many single-nucleotide polymorphisms (SNPs) associated with longevity were ide…

AdultAgingmedia_common.quotation_subjectGenome-wide association studySingle-nucleotide polymorphismBiologyPolymorphism Single NucleotideYoung AdultHumansGenetic Predisposition to DiseaseKlotho ProteinsKlothoGeneAgedGlucuronidasemedia_commonGenetic associationLongevity Ageing Klotho Meta-AnalysisSettore MED/04 - Patologia GeneraleAged 80 and overGeneticsInfant NewbornLongevityInfantMiddle AgedMembrane proteinHealthCase-Control StudiesGeriatrics and GerontologySignal transductionGenome-Wide Association StudyRejuvenation Research
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LPS-mediated production of pro/anti-inflammatory cytokines and eicosanoids in whole blood samples: Biological effects of +896A/G TLR4 polymorphism in…

2011

Toll-like receptors (TLRs) are the principal mediators of rapid microbial recognition: the lipopolysaccharide (LPS) receptor TLR4 seems to have a paradigmatic role. Single nucleotide polymorphisms (SNPs) in the TLR4 gene, such as +896A/G, known to attenuate receptor signaling, have been described. The +896A/G SNP is significantly less frequent in patients with myocardial infarction, Alzheimer's disease or prostate cancer, whereas it is overrepresented in centenarians. To clarify and confirm the biological effects of +896A/G SNP and its role in the pathophysiology of age-related diseases and longevity, we assessed the levels of IL-6, TNF-α, IL-10 and eicosanoids (LTB4 and PGE2) in LPS-stimul…

AdultLipopolysaccharidesMaleAgingAgeing Cytokines Eicosanoids Genetics Inflammation Longevity TLR4PopulationInflammationSingle-nucleotide polymorphismBiologyLeukotriene B4Polymorphism Single NucleotideDinoprostonemedicineHumansSNPeducationReceptorSettore MED/04 - Patologia Generaleeducation.field_of_studyMiddle AgedToll-Like Receptor 4ItalyEicosanoidImmunologyTLR4CytokinesFemalelipids (amino acids peptides and proteins)medicine.symptomDevelopmental BiologyEicosanoid ProductionMechanisms of Ageing and Development
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Role of TLR4 polymorphisms in inflammatory responses: implications for unsuccessful aging.

2007

The total burden of infection at various sites may affect the progression of atherosclerosis and Alzheimer's disease (AD), the risk being modulated by host genotype. The role of lipopolysaccharide (LPS) receptor TLR4 is paradigmatic. It initiates the innate immune response against gram-negative bacteria, and TLR4 single nucleotide polymorphisms (SNPs), such as +896A/G, known to attenuate receptor signaling, have been described. This SNP shows a significantly lower frequency in patients affected by myocardial infarction or AD. Thus, people genetically predisposed to developing lower inflammatory activity seem to have less chance of developing cardiovascular disease (CVD) or AD. In the presen…

AdultLipopolysaccharidesMaleAgingTime FactorsLipopolysaccharideGenotypeLeukotriene B4Myocardial InfarctionInflammationSingle-nucleotide polymorphismBiologyLeukotriene B4Polymorphism Single NucleotideGeneral Biochemistry Genetics and Molecular BiologyDinoprostoneProinflammatory cytokinechemistry.chemical_compoundHistory and Philosophy of ScienceAlzheimer DiseaseGenotypemedicineTLR4 SNPAgeing related disease longevityEscherichia coliHumansCells CulturedEscherichia coli InfectionsSettore MED/04 - Patologia GeneraleInflammationInnate immune systemBlood CellsGeneral NeuroscienceMiddle AgedImmunity InnateToll-Like Receptor 4chemistryImmunologyTLR4lipids (amino acids peptides and proteins)Femalemedicine.symptomAnnals of the New York Academy of Sciences
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Inflammation, Longevity, and Cardiovascular Diseases: Role of Polymorphisms of TLR4

2006

The total burden of infection at various sites may affect the progression of atherosclerosis, the risk being modulated by host genotype. The role of lipopolysaccaride receptor TLR4 is paradigmatic. It initiates the innate immune response against gram-negative bacteria; and TLR4 polymorphisms, as ASP299GLY, suggested to attenuate receptor signaling, have been described. We demonstrated that TLR4 ASP299GLY polymorphism shows a significantly lower frequency in patients affected by myocardial infarction compared to controls, whereas centenarians show a higher frequency. Thus, people genetically predisposed to developing weak inflammatory activity, seem to have fewer chances of developing cardio…

AdultLipopolysaccharidesMaleHeterozygoteTime Factorsmedia_common.quotation_subjectmedicine.medical_treatmentLongevityMyocardial InfarctionEnzyme-Linked Immunosorbent AssayInflammationBiologyGeneral Biochemistry Genetics and Molecular BiologyAMIHistory and Philosophy of SciencemedicineHumansGenetic Predisposition to DiseaseTLR4Interleukin 6media_commonInflammationPolymorphism GeneticInnate immune systemInterleukin-6General NeuroscienceLongevityInterleukinHeterozygote advantageMiddle AgedToll-Like Receptor 4CytokineAcute DiseaseMutationImmunologyTLR4biology.proteinFemalemedicine.symptomAnnals of the New York Academy of Sciences
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Analysis of Candidate Genes in Celiac Disease: A Tool to Identify Life-Threatening Associated Genes?

2006

The authors have recently reported that celiac patients show a proinflammatory cytokine genetic profile characterized by the contemporaneous presence of both the tumour necrosis factor-alpha-308A and the interferon-gamma +874T allele-positive genotypes. The same alleles are considered risk factors for aging associated disease, whereas an anti-inflammatory cytokine genotype profile might be associated with an extended life expectancy. This paper reports data on the 1249-1250InsACAA/Non-Ins transforming growth factor (TGF)-beta2, a multifunctional anti-inflammatory cytokine, polymorphism distribution in 88 celiac disease (CD) patients, 99 age- and sex-matched controls, and 2895-year-old healt…

AdultMaleAgeing genes.AgingCandidate geneGenotypemedicine.medical_treatmentLongevityDiseaseBiologyProinflammatory cytokineInterferon-gammaTransforming Growth Factor beta2Gene FrequencyTransforming Growth Factor betaPolymorphism (computer science)GenotypemedicineHumansAlleleAgedAged 80 and overPolymorphism GeneticTumor Necrosis Factor-alphaMiddle AgedGenotype frequencyCeliac DiseaseCytokineImmunologyFemaleGeriatrics and GerontologyRejuvenation Research
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The role of IL-1 gene cluster in longevity: a study in Italian population.

2003

In this study, we analysed the polymorphic variants of IL-1alpha (C-T transition at position -889), IL-1beta (C-T transition at position -511) and IL-1 receptor antagonist (Ra) (86-bp repeated sequence in intron 2) in 1131 subjects (453 females and 678 males) from Northern and Central Italy, including 134 centenarians, to evaluate whether IL-1 cluster alleles might be differently represented in people selected for longevity. In addition, IL-1Ra and IL-1beta plasma levels were quantified by ELISA in 130 randomly selected subjects. No significant differences in the genotype and allele frequency distributions were observed between young, elderly and centenarian subjects. IL-1Ra plasma levels s…

AdultMaleAgingGenotypemedia_common.quotation_subjectLongevityBiologyGenetic determinismRandom AllocationGene FrequencyPolymorphism (computer science)Gene clusterGenotypeHumansAlleleAllele frequencymedia_commonAgedGeneticsAged 80 and overPolymorphism GeneticLongevityMiddle AgedItalyMultigene FamilyFemaleCentenarianDevelopmental BiologyInterleukin-1Mechanisms of ageing and development
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Evidence for Less Marked Potential Signs of T-Cell Immunosenescence in Centenarian Offspring Than in the General Age-Matched Population

2014

People may reach the upper limits of the human life span at least partly because they have maintained more appropriate immune function, avoiding changes to immunity termed "immunosenescence." Exceptionally long-lived people may be enriched for genes that contribute to their longevity, some of which may bear on immune function. Centenarian offspring would be expected to inherit some of these, which might be reflected in their resistance to immunosenescence, and contribute to their potential longevity. We have tested this hypothesis by comparing centenarian offspring with age-matched controls. We report differences in the numbers and proportions of both CD4(+) and CD8(+) early- and late-diffe…

AdultMaleAgingImmunosenescenceOffspringHealth StatusT-LymphocytesT cellmedia_common.quotation_subjectLongevityPopulationCD4-CD8 RatioT cellsBiologyLymphocyte Activation03 medical and health sciences0302 clinical medicineImmune systemAntigenmedicineHumanseducationAged030304 developmental biologymedia_commonAged 80 and overSettore MED/04 - Patologia Generale0303 health scienceseducation.field_of_studyAge FactorsLongevityImmunosenescencemedicine.anatomical_structureCase-Control StudiesCentenarian offspring.ImmunologyAdult ChildrenFemaleGeriatrics and GerontologyCentenarian030215 immunologyThe Journals of Gerontology Series A: Biological Sciences and Medical Sciences
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Comparison of telomere length between population-specific mitochondrial haplogroups among different age groups in a Latvian population

2014

Population studies have demonstrated that telomere length (TL) displays great diversity among different populations. Previously described controversial findings associated longevity with specific mitochondrial DNA haplogroups (hgs) (e.g., J and U). These observations may be influenced by population diversity, geographic location, and/or specific historic background. The aims of this study were to identify a specific hg which correlates with aging in a Latvian populating and to evaluate the possible association of TL variability with specific mitochondrial hgs. The results show no significant correlation between TL, mitochondrial DNA hgs and longevity. A slight increase in frequency was obse…

AdultMaleAgingMitochondrial DNAmedia_common.quotation_subjectLongevityPopulationBiologyDNA MitochondrialHaplogroupTelomere HomeostasisHumanseducationAgedmedia_commonAged 80 and overGeneticseducation.field_of_studyHaplotypeLongevityTelomere HomeostasisMiddle AgedTelomereLatviaTelomereHaplotypesFemaleDevelopmental BiologyHuman mitochondrial DNA haplogroupMechanisms of Ageing and Development
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Allele frequencies of +874T→A single nucleotide polymorphism at the first intron of interferon-γ gene in a group of Italian centenarians

2002

Ageing is characterized by a pro-inflammatory status which could contribute to the onset of major age-related diseases such as cardiovascular diseases, neurodegeneration, osteoarthritis and osteoporosis, and diabetes. Thus, it can be hypothesized that genetic variations in pro- or anti-inflammatory cytokines might influence successful ageing and longevity. We have studied the distribution of +874T--A interferon-gamma (IFN-gamma) polymorphisms in a large number of Italian centenarians to evaluate if the two alleles might be differently represented in people selected for longevity. DNA samples were obtained from 174 Italian centenarians (99 years old, 142 women and 32 men) and from 24860-year…

AdultMaleAgingmedia_common.quotation_subjectSingle-nucleotide polymorphismImmunogeneticsBiologyPolymorphism Single NucleotideBiochemistryInterferon-gammaEndocrinologyGene FrequencyGenetic variationGeneticsHumansAlleleMolecular BiologyAllele frequencyGeneAllelesAgedmedia_commonAged 80 and overGeneticsLongevityCell BiologyMiddle AgedIntronsItalyAgeingImmunologyFemaleExperimental Gerontology
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