Search results for "Gene Expression"

showing 10 items of 4085 documents

The transcription factor Interferon Regulatory Factor 4 is required for the generation of protective effector CD8 + T cells

2013

Robust cytotoxic CD8 + T-cell response is important for immunity to intracellular pathogens. Here, we show that the transcription factor IFN Regulatory Factor 4 (IRF4) is crucial for the protective CD8 + T-cell response to the intracellular bacterium Listeria monocytogenes . IRF4-deficient ( Irf4 −/− ) mice could not clear L. monocytogenes infection and generated decreased numbers of L. monocytogenes -specific CD8 + T cells with impaired effector phenotype and function. Transfer of wild-type CD8 + T cells into Irf4 −/− mice improved bacterial clearance, suggesting an intrinsic defect of CD8 + T cells in Irf4 −/− mice. Following transfer into wild-type recipients, Irf4 −/− CD8 + T cells bec…

Cellular differentiationGene ExpressionEomesoderminBiologyMiceInterleukin 21AnimalsCytotoxic T cellListeriosisIL-2 receptorAntigen-presenting cellSTAT4Cell ProliferationMice KnockoutMultidisciplinaryCell DifferentiationBiological SciencesListeria monocytogenesMolecular biologyMice Inbred C57BLHost-Pathogen InteractionsInterferon Regulatory FactorsImmunologyPositive Regulatory Domain I-Binding Factor 1CD8T-Lymphocytes CytotoxicTranscription FactorsProceedings of the National Academy of Sciences
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Isolation and characterization of a murine resident liver stem cell.

2008

Increasing evidence provides support that mammalian liver contains stem/progenitor cells, but their molecular phenotype, embryological derivation, biology and their role in liver cell turnover and regeneration remain to be further clarified. In this study, we report the isolation, characterization and reproducible establishment in line of a resident liver stem cell (RLSC) with immunophenotype and differentiative potentiality distinct from other previously described liver precursor/stem cells. RLSCs, derived from fetal and neonatal murine livers as well as from immortalized hepatocytic MMH lines and established in lines, are Sca+, CD34-, CD45-, alpha-fetoprotein+ and albumin-. This molecular…

Cellular differentiationLiver Stem CellCell SeparationBiologyImmunophenotypingLiver progenitor cellsMiceChondrocyteshepatocyteAnimalsCell LineageProgenitor cellLiver progenitor cells; hepatocyte; differentiationMolecular BiologyCells CulturedMultipotent Stem CellOligonucleotide Array Sequence AnalysisNeuronsOsteoblastsAnimalOligonucleotide Array Sequence AnalysiLiver cellOsteoblastGene Expression ProfilingMultipotent Stem CellsMesenchymal stem cellCell DifferentiationCell BiologydifferentiationNeuronChondrocyteMolecular biologyLiver regenerationCell biologyPhenotypeAnimals NewbornLiverMultipotent Stem CellHepatocytesStem cellAnimals; Animals Newborn; Cell Differentiation; Cell Lineage; Cell Separation; Cells Cultured; Chondrocytes; Gene Expression Profiling; Hepatocytes; Immunophenotyping; Liver; Mice; Multipotent Stem Cells; Neurons; Oligonucleotide Array Sequence Analysis; Osteoblasts; Phenotype; Molecular Biology; Cell BiologyCell death and differentiation
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Role of cAMP in mediating AHR signaling.

2009

Regulation of the nuclear import of many transcription factors represents a step in gene regulation which is crucial for a number of cellular processes. The aryl hydrocarbon receptor (AHR), a basic helix-loop-helix protein of the PAS (PER-ARNT-SIM) family of transcriptional regulators is a cytosol-associated and ligand-activated receptor. The environmental toxin dioxin binds with high affinity to AHR rendering it nuclear and leading to the activation of AHR sensitive genes. However, the fact, that the AHR mediates a large variety of physiological events without the involvement of any known exogenous ligand, including liver and vascular system development, maturation of the immune system, re…

Cellular differentiationNuclear translocationSignal transductionDioxinsLigandsBiochemistryCell LineProtein kinase ACyclic AMPCytochrome P-450 CYP1A1AnimalsHumansPhosphorylationReceptorProtein kinase ATranscription factorAryl hydrocarbon receptorPharmacologyRegulation of gene expressionbiologyAryl hydrocarbon receptorCyclic AMP-Dependent Protein KinasesProtein TransportBiochemistryReceptors Aryl HydrocarbonSecond messenger systembiology.proteinEnvironmental PollutantsSignal transductionDioxin toxicitySignal TransductionBiochemical pharmacology
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An essential switch in subunit composition of a chromatin remodeling complex during neural development.

2007

Summary Mammalian neural stem cells (NSCs) have the capacity to both self-renew and to generate all the neuronal and glial cell-types of the adult nervous system. Global chromatin changes accompany the transition from proliferating NSCs to committed neuronal lineages, but the mechanisms involved have been unclear. Using a proteomics approach, we show that a switch in subunit composition of neural, ATP-dependent SWI/SNF-like chromatin remodeling complexes accompanies this developmental transition. Proliferating neural stem and progenitor cells express complexes in which BAF45a, a Kruppel/PHD domain protein and the actin-related protein BAF53a are quantitatively associated with the SWI2/SNF2-…

Cellular differentiationProtein subunitNeuroscience(all)Molecular Sequence DataNeuroepithelial CellsDEVBIONerve Tissue ProteinsBiologyChromatin remodelingMOLNEUROEpigenesis Genetic03 medical and health sciencesMice0302 clinical medicineMultienzyme ComplexesAnimalsAmino Acid SequenceProgenitor cell030304 developmental biologyNeurons0303 health sciencesGeneral NeuroscienceMultipotent Stem CellsGene Expression Regulation DevelopmentalCell DifferentiationChromatin Assembly and DisassemblySTEMCELLNeural stem cellChromatinCell biologyNeuroepithelial cellProtein SubunitsNeural developmentNeuroglia030217 neurology & neurosurgeryTranscription FactorsNeuron
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Murine muscle engineered from dermal precursors: an in vitro model for skeletal muscle generation, degeneration and fatty infiltration.

2013

Skeletal muscle can be engineered by converting dermal precursors into muscle progenitors and differentiated myocytes. However, the efficiency of muscle development remains relatively low and it is currently unclear if this is due to poor characterization of the myogenic precursors, the protocols used for cell differentiation, or a combination of both. In this study, we characterized myogenic precursors present in murine dermospheres, and evaluated mature myotubes grown in a novel three-dimensional culture system. After 5-7 days of differentiation, we observed isolated, twitching myotubes followed by spontaneous contractions of the entire tissue-engineered muscle construct on an extracellul…

Cellular differentiationSarcoplasmMuscle Fibers SkeletalBiomedical EngineeringMedicine (miscellaneous)BioengineeringBiologyMuscle DevelopmentModels BiologicalArticleExtracellular matrixMiceTissue engineeringSpheroids CellularmedicineMyocyteAnimalsCell ProliferationTissue EngineeringMyogenesisCell growthMusclesSkeletal muscleCell DifferentiationDermisLipidsAcetylcholineBiologia experimentalCell biologyExtracellular Matrixmedicine.anatomical_structureBiochemistryGene Expression RegulationFemaleEnginyeria biomèdicaIon Channel GatingBiomarkers
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Symmetric expansion of neural stem cells from the adult olfactory bulb is driven by astrocytes via WNT7A.

2012

Adult neural stem cells (NSCs) located in the subventricular zone (SVZ) persistently produce new neurons destined to the olfactory bulb (OB). Recent research suggests that the OB is also a source of NSCs that remains largely unexplored. Using single/dual-labeling procedures, we address the existence of NSCs in the innermost layers of the OB. In vivo, these cells are more quiescent that their SVZ counterparts, but after in vitro expansion, they behave similarly. Self-renewal and proliferation assays in co-culture with niche astrocytes indicate that OB-glia restricts NSC activity whereas SVZ-glia has the opposite effect. Gene expression profiling identifies WNT7A as a key SVZ-glial factor lac…

Cellular differentiationSubventricular zoneCell Growth ProcessesBiologyMiceNeural Stem CellsIn vivomedicineAnimalsHumansreproductive and urinary physiologyWnt signaling pathwayCell DifferentiationCell BiologyAnatomyOlfactory BulbNeural stem cellnervous system diseasesOlfactory bulbCell biologyGene expression profilingWnt ProteinsWNT7Amedicine.anatomical_structurenervous systemAstrocytesMolecular Medicinebiological phenomena cell phenomena and immunityDevelopmental BiologyStem cells (Dayton, Ohio)
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Monocyte-derived dendritic cells of patients with coronary artery disease show an increased expression of costimulatory molecules CD40, CD80 and CD86…

2007

Background Atherosclerosis is a disease triggered by diverse exogenous stimuli and sustained by chronic inflammatory processes. Dendritic cells (DCs) are key regulatory antigen-presenting cells and play a crucial role in regulating the adaptive and innate immune system in any chronic inflammatory process. DCs are present in atherosclerotic lesions in the areas of the highest T-cell density. So far, their role in atherosclerosis has not been fully elucidated. We investigated the phenotypic properties of DCs in patients with coronary artery disease (CAD) in comparison to healthy individuals. Methods Peripheral blood monocytes were isolated from 50 patients with CAD and 19 healthy individuals …

Cellular differentiationchemical and pharmacologic phenomenaCoronary Artery DiseaseMonocytesFlow cytometryDownregulation and upregulationRisk FactorsMedicineHumansCD40 AntigensAgedRegulation of gene expressionCD86Innate immune systemCD40biologymedicine.diagnostic_testbusiness.industryhemic and immune systemsCell DifferentiationGeneral MedicineDendritic CellsMiddle AgedAtherosclerosisFlow CytometryC-Reactive ProteinGene Expression RegulationImmunologybiology.proteinB7-1 AntigenLeukocytes MononuclearB7-2 AntigenCardiology and Cardiovascular MedicinebusinessCD80Coronary artery disease
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Intrinsic TNFR2 signaling in T regulatory cells provides protection in CNS autoimmunity

2018

Significance In spite of TNF involvement in the pathogenesis of multiple sclerosis (MS), systemic TNF neutralization in MS patients was not successful. One of the possible reasons is that TNF possesses both pathogenic and protective features that may be related to TNFR1 versus TNFR2 receptor engagement. This study uncovers one of such protective functions of TNF mediated by intrinsic TNFR2 signaling in Treg cells. In mice bearing humanized TNF and TNFR2 genetic loci, TNFR2 ablation restricted to Treg cells led to reduced capacity to control Th17 cell responses, exacerbated experimental autoimmune encephalomyelitis (EAE) development, and affected the maintenance of Treg cells. These findings…

Central Nervous System0301 basic medicineEncephalomyelitis Autoimmune ExperimentalT regulatory cellsmedicine.medical_treatmentAutoimmunitychemical and pharmacologic phenomenaBiologymedicine.disease_causeT-Lymphocytes RegulatoryneuroinflammationAutoimmunityMice03 medical and health sciencesImmunology and Inflammation0302 clinical medicineImmune systemmedicineAnimalsHumansReceptors Tumor Necrosis Factor Type IIIL-2 receptorCells CulturedNeuroinflammationMice KnockoutAutoimmune diseaseMultidisciplinaryEAETumor Necrosis Factor-alphaExperimental autoimmune encephalomyelitisFOXP3hemic and immune systemsBiological Sciencesmedicine.diseaseTNF/TNFR2Mice Inbred C57BLDisease Models Animalhumanized mice030104 developmental biologyCytokineGene Expression RegulationImmunology030215 immunologyProceedings of the National Academy of Sciences
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NF-κB inducing kinase (NIK) is an essential post-transcriptional regulator of T-cell activation affecting F-actin dynamics and TCR signaling

2018

NF-κB inducing kinase (NIK) is the key protein of the non-canonical NF-κB pathway and is important for the development of lymph nodes and other secondary immune organs. We elucidated the specific role of NIK in T cells using T-cell specific NIK-deficient (NIKΔT) mice. Despite showing normal development of lymphoid organs, NIKΔT mice were resistant to induction of CNS autoimmunity. T cells from NIKΔT mice were deficient in late priming, failed to up-regulate T-bet and to transmigrate into the CNS. Proteomic analysis of activated NIK-/- T cells showed de-regulated expression of proteins involved in the formation of the immunological synapse: in particular, proteins involved in cytoskeleton dy…

Central Nervous System0301 basic medicineEncephalomyelitis Autoimmune ExperimentalT-LymphocytesT cellPrimary Cell CultureImmunologyReceptors Antigen T-CellPriming (immunology)Protein Serine-Threonine KinasesBiologyLymphocyte ActivationImmunological synapseMice03 medical and health sciences0302 clinical medicineImmune systemmedicineAnimalsImmunology and AllergyProtein kinase BAdaptor Proteins Signal TransducingMice KnockoutMitogen-Activated Protein Kinase 1Mitogen-Activated Protein Kinase 3ZAP-70 Protein-Tyrosine KinasePhospholipase C gammaGene Expression ProfilingZAP70T-cell receptorMembrane ProteinsPhosphoproteinsActinsPeptide FragmentsCell biologyMice Inbred C57BL030104 developmental biologymedicine.anatomical_structureGene Expression Regulation030220 oncology & carcinogenesisMyelin-Oligodendrocyte GlycoproteinLymph NodesSignal transductionT-Box Domain ProteinsProto-Oncogene Proteins c-aktSpleenSignal TransductionJournal of Autoimmunity
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Molecular evidence for the inverse comorbidity between central nervous system disorders and cancers detected by transcriptomic meta-analyses.

2014

There is epidemiological evidence that patients with certain Central Nervous System (CNS) disorders have a lower than expected probability of developing some types of Cancer. We tested here the hypothesis that this inverse comorbidity is driven by molecular processes common to CNS disorders and Cancers, and that are deregulated in opposite directions. We conducted transcriptomic meta-analyses of three CNS disorders (Alzheimer's disease, Parkinson's disease and Schizophrenia) and three Cancer types (Lung, Prostate, Colorectal) previously described with inverse comorbidities. A significant overlap was observed between the genes upregulated in CNS disorders and downregulated in Cancers, as wel…

Central Nervous SystemCancer ResearchGene ExpressionDiseaseComorbidityBioinformaticsProstate cancer0302 clinical medicineNeoplasmsGenetics (clinical)0303 health sciencesWnt signaling pathwayParkinson DiseaseAlzheimer's diseasePeptidylprolyl Isomerase[SDV.BIBS]Life Sciences [q-bio]/Quantitative Methods [q-bio.QM]3. Good health[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Alzheimer's diseaseResearch ArticleSignal Transductionlcsh:QH426-470[SDV.CAN]Life Sciences [q-bio]/CancerProtein degradationBiology03 medical and health sciencesAlzheimer Disease[SDV.BBM.GTP]Life Sciences [q-bio]/Biochemistry Molecular Biology/Genomics [q-bio.GN]medicineGeneticsCancer GeneticsHumansGene NetworksMolecular BiologyBiologyEcology Evolution Behavior and Systematics030304 developmental biologyPeptidylprolyl isomeraseGene Expression ProfilingCancerComputational Biologymedicine.diseaseColorectal cancerComorbidityMalariaNIMA-Interacting Peptidylprolyl IsomeraseMeta-analysislcsh:GeneticsGene Expression RegulationImmunologySchizophrenia[SDV.SPEE]Life Sciences [q-bio]/Santé publique et épidémiologie030217 neurology & neurosurgery
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