Search results for "Gene expression"

showing 10 items of 4085 documents

Investigation into mechanisms mediating the inhibitory effect of 1,4-benzodiazepines on mast cells by gene expression profiling.

2013

Abstract Aims This study aims to identify by a molecular genetic approach potential targets in mast cells at which 1,4-benzodiazepines may cause their inhibitory effect on mast cell activity. Main methods Gene expression analyses with microarray gene chip and/or quantitative PCR were performed using 1,4-benzodiazepine-treated human mast cell leukemia HMC-1.2 cells, promyelocytic leukemia HL-60 cells and human mast cells from healthy volunteers and patients with mast cell activation disease (MCAD). Pathway analysis was applied to search for enriched biological functions and canonical pathways within differentially regulated genes. Key findings Both neoplastic and normal human mast cells expr…

AdultMalegenetics [Mastocytosis]Gene ExpressionHL-60 CellsFlunitrazepamBiologyPolymerase Chain ReactionGeneral Biochemistry Genetics and Molecular BiologyClonazepamLYNddc:570medicineTranslocator proteinpharmacology [Flunitrazepam]HumansMast CellsGeneral Pharmacology Toxicology and Pharmaceuticsmethods [Polymerase Chain Reaction]Interleukin 5AgedRegulation of gene expressionBenzodiazepinonesGene Expression Profilingdrug effects [Gene Expression]General MedicineMiddle AgedMast cell leukemiamedicine.diseaseMast cellMicroarray Analysis4'-chlorodiazepamCell biologyInterleukin 33Gene expression profilingmedicine.anatomical_structuremethods [Microarray Analysis]biology.proteinpharmacology [Clonazepam]drug effects [Mast Cells]Femalepharmacology [Benzodiazepinones]Mastocytosismethods [Gene Expression Profiling]
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PTCH-1 and MDM2 expression in ameloblastoma from a West African sub-population: implication for chemotherapeutics

2015

INTRODUCTION: ameloblastoma is a slow growing, painless odontogenic swelling which can attain sizes that result in severe deformities of the craniofacial complex. It is the most commonly encountered odontogenic tumor in Nigeria. Surgical intervention is currently the method of treatment; however identification of altered molecular pathways may inform chemotherapeutic potential. The Protein Patched homolog 1 (PTCH-1) is overexpressed in ameloblastoma. Also, mutation in the MDM2 gene can reduce the tumor suppressor function of p53 and promote ameloblastoma growth. No study however has characterized the molecular profile of African cases of ameloblastoma with a view to developing chemotherapeu…

AdultMalemdm2Pathologymedicine.medical_specialtyPopulationNigeriaPathology and Forensic MedicinePTCH-1 MdM2 ameloblastoma chemotherapeuticsameloblastomaptch-1HumansMedicineRadiology Nuclear Medicine and imagingDentistry (miscellaneous)ddc:610educationAmeloblastomaStellate reticulumProtein Patched Homolog 1education.field_of_studylcsh:R5-920biologybusiness.industryResearchlcsh:Public aspects of medicinechemotherapeuticsOdontogenic tumorProto-Oncogene Proteins c-mdm2lcsh:RA1-1270General Medicinemedicine.diseaseJaw NeoplasmsGene Expression Regulation NeoplasticPatched-1 ReceptorWest africanstomatognathic diseasesMutationMonoclonalCancer researchbiology.proteinMdm2FemaleSurgeryOral SurgeryAntibodybusinesslcsh:Medicine (General)The Pan African Medical Journal
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Epigenetic differences arise during the lifetime of monozygotic twins.

2005

Monozygous twins share a common genotype. However, most monozygotic twin pairs are not identical; several types of phenotypic discordance may be observed, such as differences in susceptibilities to disease and a wide range of anthropomorphic features. There are several possible explanations for these observations, but one is the existence of epigenetic differences. To address this issue, we examined the global and locus-specific differences in DNA methylation and histone acetylation of a large cohort of monozygotic twins. We found that, although twins are epigenetically indistinguishable during the early years of life, older monozygous twins exhibited remarkable differences in their overall…

AdultMalemedicine.medical_specialtyADNRestriction MappingTwinsMonozygotic twinX-inactivationEpigenesis GeneticHistonesX Chromosome InactivationSurveys and QuestionnairesGenotypemedicineHumansEpigeneticsOligonucleotide Array Sequence AnalysisGeneticsAnalysis of VarianceMultidisciplinarybiologyReverse Transcriptase Polymerase Chain ReactionElectrophoresis CapillaryGene Expression Regulation DevelopmentalAcetylationNucleic acid amplification techniqueDNASequence Analysis DNATwins MonozygoticDNA MethylationExpressió gènicaFenotipHistonePhenotypeSpainDNA methylationbiology.protein5-MethylcytosineCommentaryMedical geneticsBessonsFemaleGene expressionNucleic Acid Amplification TechniquesProceedings of the National Academy of Sciences of the United States of America
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Sensory profiles and immune-related expression patterns of patients with and without neuropathic pain after peripheral nerve lesion

2019

In this multicenter cross-sectional study, we determined sensory profiles of patients with (NL-1) and without neuropathic pain (NL-0) after nerve lesion and assessed immune-related systemic gene expression. Patients and matched healthy controls filled in questionnaires and underwent neurological examination, neurophysiological studies, quantitative sensory testing, and blood withdrawal. Neuropathic pain was present in 67/95 (71%) patients (NL-1). Tactile hyperalgesia was the most prominent clinical sign in NL-1 patients (P < 0.05). Questionnaires showed an association between neuropathic pain and the presence of depression, anxiety, and catastrophizing (P < 0.05 to P < 0.01). Neuropathic pa…

AdultMalemedicine.medical_specialtyAdolescentGene ExpressionNeurological examinationNerve fiberSensory systemGastroenterologyCohort StudiesYoung Adult03 medical and health sciencesNerve Fibers0302 clinical medicine030202 anesthesiologyInternal medicinemedicineHumansYoung adultDepression (differential diagnoses)AgedPain MeasurementAged 80 and overmedicine.diagnostic_testbusiness.industryCatastrophizationChronic painMiddle Agedmedicine.diseaseCross-Sectional StudiesAnesthesiology and Pain Medicinemedicine.anatomical_structureNeurologyNeuropathic painHyperalgesiaNeuralgiaFemaleNeurology (clinical)Inflammation Mediatorsmedicine.symptombusiness030217 neurology & neurosurgeryPain
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Revised nomenclature and classification of inherited ichthyoses: Results of the First Ichthyosis Consensus Conference in Soreze 2009

2010

Background: Inherited ichthyoses belong to a large, clinically and etiologically heterogeneous group of mendelian disorders of cornification; typically involving the entire integument. Over the recent years, much progress has been made defining their molecular causes. However, there is no internationally accepted classification and terminology. Objective: We sought to establish a consensus for the nomenclature and classification of inherited ichthyoses. Methods: The classification project started at the First World Conference on Ichthyosis in 2007. A large international network of expert clinicians, skin pathologists, and geneticists entertained an interactive dialogue over 2 years, eventua…

AdultMalemedicine.medical_specialtyCongenital ichthyosiform erythrodermaAdolescentautosomal recessive congenital ichthyosisDermatologySeverity of Illness Indexhistologymendelian disorders of cornificationYoung AdultCYP4F22Terminology as TopicCongenital ichthyosisMedicineHumansGenetic Predisposition to DiseasegeneticsABCA12Childbiologybusiness.industryIchthyosisInfant NewbornIchthyosisInfantkeratinopathic ichthyosisLamellar ichthyosisHarlequin IchthyosisCongresses as TopicIchthyosiform Erythroderma Congenitalepidermolytic ichthyosismedicine.diseasePrognosisDermatologyultrastructureGene Expression RegulationPractice Guidelines as Topicbiology.proteinFemaleDermatologic AgentsFrancesuperficial epidermolytic ichthyosisbusinessIchthyosis vulgarisJournal of the American Academy of Dermatology
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Enhanced reduction in oxidative stress and altered glutathione and thioredoxin system response to unsaturated fatty acid load in familial hypercholes…

2014

Abstract Objectives Familial hypercholesterolemia (FH) is characterized by increased oxidative stress (OS) levels. In the postprandial state, lipids and lipoproteins modulate OS status through their impact on pro-oxidant and antioxidant mechanisms. The objective of this study was to evaluate in patients with FH the response to an unsaturated oral fat load test (OFLT) by analyzing the mRNA levels of genes involved in the glutathione and thioredoxin antioxidant systems. Design and Methods We analyzed 14 FH patients and 20 normolipidemic and normoglycemic controls. In both groups, mRNA values of antioxidant enzyme genes (glutathione and thioredoxin systems) were determined at baseline and at 2…

AdultMalemedicine.medical_specialtyGPX1Antioxidantmedicine.medical_treatmentGlutamate-Cysteine LigaseClinical Biochemistrymedicine.disease_causeGPX4Gene Expression Regulation EnzymologicGlutathione SynthaseHyperlipoproteinemia Type IIchemistry.chemical_compoundThioredoxinsDietary Fats UnsaturatedInternal medicinemedicineHumansUnsaturated fatty acidGlutathione PeroxidaseChemistryReverse Transcriptase Polymerase Chain ReactionGeneral MedicineGlutathioneFastingMiddle AgedPhospholipid Hydroperoxide Glutathione PeroxidaseGlutathioneOxidative StressPostprandialEndocrinologyGlutathione ReductaseFemaleThioredoxinOxidation-ReductionOxidative stressClinical biochemistry
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The UCP2 -866 G>A promoter region polymorphism is associated with nonalcoholic steatohepatitis

2015

Background & Aims Uncoupling protein 2 - UCP2 - regulates mitochondrial lipid fluxes and reactive oxygen species production by the respiratory chain. The −866 G>A UCP2 promoter region polymorphism has been linked to insulin resistance and lipid metabolism. The aim of this study was to assess whether the −866 G>A UCP2 polymorphism predisposes to nonalcoholic steatohepatitis in patients at risk, and the relationship with lipid metabolism and hepatic UCP2 expression. Methods We considered 688 Italian patients who underwent liver biopsy for suspected NASH, and 232 healthy controls. The UCP2 −866 G>A polymorphism was determined by allele specific oligonucleotide probes, hepatic UCP2 mRNA levels …

AdultMalemedicine.medical_specialtyGenotypeRespiratory chainGene ExpressionBiologyIon ChannelsMitochondrial Proteinsgenetic polymorphism; lipid metabolism;liver; mitochondria; nonalcoholic steatohepatitis; uncoupling protein-2Insulin resistanceNon-alcoholic Fatty Liver DiseaseRisk FactorsDiabetes mellitusInternal medicineGenotypemedicineHumansUncoupling Protein 2Promoter Regions GeneticUncoupling protein-2AllelesAgedPolymorphism GeneticGenetic polymorphismmedicine.diagnostic_testHepatologyLipid metabolismMiddle Agedmedicine.diseaseImpaired fasting glucoseMitochondriaEndocrinologyLogistic ModelsLipid metabolismLiverLiver biopsyCase-Control StudiesFemaleSteatosisInsulin ResistanceNonalcoholic steatohepatiti
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Adrenomedullin and arterial stiffness: integrative approach combining monocyte ADM expression, plasma MR-Pro-ADM, and genome-wide association study.

2014

Background— Adrenomedullin (ADM) is a circulating vasoactive peptide involved in vascular homeostasis and endothelial function. Single nucleotide polymorphisms of the ADM gene are associated with blood pressure variability, and elevated levels of plasma midregional proadrenomedullin (MR-pro-ADM) are associated with cardiovascular diseases. Methods and Results— We investigated the sources of variability of ADM gene expression and plasma MR-pro-ADM concentrations in the general population, and their relationship with markers of atherosclerosis. MR-pro-ADM levels were assessed in 4155 individuals who underwent evaluation of carotid intima-media thickness and arterial rigidity (reflection inde…

AdultMalemedicine.medical_specialtyGenotypeTranscription Geneticmedicine.drug_classPopulationGenome-wide association studySingle-nucleotide polymorphismBiologyCarotid Intima-Media ThicknessPolymorphism Single NucleotideMonocytesArticleCohort Studieschemistry.chemical_compoundAdrenomedullinVascular StiffnessInternal medicineGene expressionGeneticsmedicineNatriuretic peptideHumansProtein PrecursorseducationGenetics (clinical)AgedCreatinineeducation.field_of_studyGene Expression ProfilingGenetic VariationMiddle Agedmedicine.diseaseAtherosclerosisAdrenomedullinEndocrinologyCarotid ArterieschemistryGene Expression RegulationArterial stiffnessLinear ModelsFemaleCardiology and Cardiovascular MedicineTranscriptomeGenome-Wide Association StudyCirculation. Cardiovascular genetics
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Expression of PSA-NCAM and synaptic proteins in the amygdala of psychiatric disorder patients.

2011

Neuroimaging has revealed structural abnormalities in the amygdala of different psychiatric disorders. The polysialylated neural cell adhesion molecule (PSA-NCAM), a molecule related to neuronal structural plasticity, which expression is altered in schizophrenia, major depression and in animal models of these disorders, may participate in these changes. However, PSA-NCAM has not been studied in the human amygdala. To know whether its expression and that of presynaptic markers, was affected in psychiatric disorders, we have analyzed post-mortem sections from the Stanley Neuropathology Consortium, which includes controls, schizophrenia, bipolar and major depression patients. PSA-NCAM was expr…

AdultMalemedicine.medical_specialtyGlutamate decarboxylaseSynaptophysinNeural Cell Adhesion Molecule L1NeuropathologyAmygdalamental disordersNeuropilmedicineHumansBipolar disorderPsychiatryBiological PsychiatryAgedNeuronsbiologyGlutamate DecarboxylaseMood DisordersMiddle Agedmedicine.diseaseAmygdalaPsychiatry and Mental healthmedicine.anatomical_structurenervous systemGene Expression RegulationSchizophreniaPhosphopyruvate HydratasePostmortem ChangesVesicular Glutamate Transport Protein 1Synaptophysinbiology.proteinAcetylcholinesteraseSchizophreniaSialic AcidsNeural cell adhesion moleculeFemalePsychologyCalcium-Calmodulin-Dependent Protein Kinase Type 2NeuroscienceJournal of psychiatric research
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Expression patterns and prognostic role of transketolase-like 1 in muscle-invasive bladder cancer

2015

PURPOSE: The pentose phosphate pathway (PPP) has been shown to play an important role in the metabolism of cancer cells. The transketolase-like 1 gene (TKTL1) encodes an enzyme representing an essential component of this pathway. Its expression has been demonstrated to correlate with stage and outcome in various tumors. The aim of the present study was to assess expression patterns and the prognostic role of TKTL1 in muscle-invasive bladder cancer (MIBC). PATIENTS AND METHODS: The expression of TKTL1 was assessed in a tissue microarray consisting of histopathologically benign and malign tissue of 112 patients who underwent radical cystectomy due to MIBC. Cytoplasmatic and nuclear expression…

AdultMalemedicine.medical_specialtyGlycolysiUrologymedicine.medical_treatmentBiopsyDiabetePentose phosphateSettore MED/24 - UrologiaCystectomyInternal medicineBiopsymedicineHumansNeoplasm InvasivenessUrotheliumLymph nodeAgedRetrospective StudiesAged 80 and overBladder cancerTissue microarraymedicine.diagnostic_testbusiness.industryBladder cancerTransketolase-like 1DNA NeoplasmBiomarkerMiddle Agedmedicine.diseaseImmunohistochemistryGene Expression Regulation Neoplasticmedicine.anatomical_structureEndocrinologyUrinary Bladder NeoplasmsTissue Array AnalysisCancer cellCancer researchImmunohistochemistryFemaleTransketolaseUrotheliumbusinessFollow-Up Studies
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