Search results for "Generation"

showing 10 items of 3050 documents

Mucolipidosis I — A sialidosis

1977

Mucolipidosis I is characterized by Hurler-like features and skeletal dysplasia with a cherry-red macular spot and signs of neurodegeneration involving neuronal cells and myelin. Excessive amounts of sialic acid-containing compounds were found in cultured fibroblasts, leukocytes, and urine of a patient with a clinical phenotype of mucolipidosis I. In cultured fibroblasts, profoundly diminished activity of an alpha-N-acetylneuraminidase (sialidase) was found. Mucolipidosis I thus appears to be a distinct disorder of complex carbohydrate catabolism caused by the genetic deficiency of a neuraminidase.

Malemedicine.medical_specialtyHydrolasesNeuraminidaseSialidaseMyelinMucolipidosesInternal medicinemedicineHumansSialidosisChildCells CulturedGenetics (clinical)SkinbiologyMucolipidosisCatabolismNeurodegenerationmedicine.diseasePhenotypeEndocrinologymedicine.anatomical_structureDysplasiaChild PreschoolImmunologySialic Acidsbiology.proteinLysosomesNeuraminidaseFollow-Up StudiesAmerican Journal of Medical Genetics
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Chronic Fluoxetine Treatment Increases the Expression of PSA-NCAM in the Medial Prefrontal Cortex

2006

Recent hypotheses suggest that changes in neuronal structure and connectivity may underlie the etiology of depression. The medial prefrontal cortex (mPFC) is affected by depression and shows neuronal remodeling during adulthood. This plasticity may be mediated by the polysialylated form of the neural cell adhesion molecule (PSA-NCAM), which is intensely expressed in the adult mPFC. As the expression of PSA-NCAM is increased by serotonin in other cerebral regions, antidepressants acting on serotonin reuptake may influence PSA-NCAM expression and thus counteract the effects of depression by modulating neuronal structural plasticity. Using immunohistochemistry, we have studied the relationship…

Malemedicine.medical_specialtyInterneuronFluorescent Antibody TechniquePrefrontal CortexCell CountNeural Cell Adhesion Molecule L1urologic and male genital diseasesSerotonergicRats Sprague-Dawleychemistry.chemical_compoundFluoxetineInternal medicinemedicineNeuropilAnimalsPrefrontal cortexNeurotransmitter5-HT receptorNeuronsPharmacologyAnalysis of VarianceRatsPsychiatry and Mental healthmedicine.anatomical_structureEndocrinologyGene Expression Regulationnervous systemchemistryReceptors SerotoninSialic AcidsAntidepressive Agents Second-GenerationNeural cell adhesion moleculeSerotoninPsychologyNeuroscienceNeuropsychopharmacology
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Early Onset of Guillain–Barré Syndrome Following Lumbar Disc Herniation Surgery: An Unexpected Clinical Evolution

2021

Letter: Guillain–Barré syndrome (GBS), is a neurologic complication rarely reported following a spinal surgery procedure.1, 2, 3, 4, 5, 6 GBS is a potentially fatal, immune-mediated disease of the peripheral nerves and nerve roots that is usually triggered by infections. It is the most common cause of acute flaccid paralysis, with an annual global incidence of approximately 1–2 per 100,000 person-years.7 Although the clinical presentation of the disease is heterogeneous, patients typically present with weakness and sensory signs in the legs that progress to the arms and cranial muscles. Disease progression can be rapid in approximately 20% of patients with respiratory failure requiring mech…

Malemedicine.medical_specialtyLumbar VertebraeGuillain-Barre syndromebusiness.industryGuillaineBarré Syndrome Lumbar Disc Herniation surgeryIntervertebral Disc DegenerationAged Guillain-Barre Syndrome Diagnosis Differential Humans Intervertebral Disc Degeneration Intervertebral Disc Displacement Postoperative Complications Lumbar Vertebrae MaleGuillain-Barre Syndromemedicine.diseaseSurgeryDiagnosis DifferentialPostoperative ComplicationsmedicineHumansSurgeryNeurology (clinical)Lumbar disc herniationbusinessIntervertebral Disc DisplacementAgedEarly onset
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Prednisolone decreases exercise-induced acid hydrolase response in mouse skeletal muscle.

1984

Male NMRI-mice were subjected to exhaustive treadmill exercise. 3 and 6 days after the exertion, quadriceps femoris muscles were examined histologically and analyzed for acid hydrolases in order to follow the degree and progress of injuries. Prednisolone (PRED), an anti-inflammatory corticosteroid, was given to some of the animals in order to modify the exercise response. The PRED administration began 14 h before exercise and continued until the end of the experiment (6 days). The doses were 25 and 50 mg . kg-1 i.p. twice a day. The activities of both arylsulphatase and beta-glucuronidase increased significantly in the exercise control group after 3 and 6 days. The increase in activity corr…

Malemedicine.medical_specialtyNecrosisPhysiologymedicine.drug_classPrednisolonePhysical ExertionPhysical exerciseInflammationMice Inbred StrainsBiologyMiceMuscular DiseasesPhysiology (medical)Internal medicinemedicineAnimalsRegenerationOrthopedics and Sports MedicineExertionArylsulfatasesGlucuronidaseMyositisMusclesPublic Health Environmental and Occupational HealthSkeletal muscleGeneral Medicinemedicine.anatomical_structureEndocrinologyGlucoseDepression ChemicalPrednisolonebiology.proteinExercise TestCorticosteroidmedicine.symptomSulfatasesAcid hydrolasemedicine.drugEuropean journal of applied physiology and occupational physiology
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Differential sensitivity to the effects of nicotine and bupropion in adolescent and adult male OF1 mice during social interaction tests.

2008

Few studies have compared the action of both nicotine (NIC) and bupropion (BUP), an antidepressant used to treat NIC dependence, on social and aggressive behavior at different ages. This study aims to determine whether these drugs produce differential effects in adolescent (postnatal day: 36–37) and adult (postnatal day: 65–66) mice that have been housed individually for 2 weeks in order to induce aggressive behavior. Mice received BUP (40, 20, or 10 mg/kg), NIC (1, 0.5, and 0.25 mg/kg as base), or vehicle earlier to a social interaction test. BUP (40 mg/kg) decreased social investigation and increased nonsocial exploration in both adolescent and adult mice. The same effects were also obser…

Malemedicine.medical_specialtyNicotineAdult malePhysiologyMice Inbred StrainsNicotineMiceArts and Humanities (miscellaneous)Age groupsDopamine Uptake InhibitorsStatistical significanceDevelopmental and Educational PsychologymedicineAnimalsNicotinic AgonistsPsychiatryPostnatal daySocial BehaviorBupropionGeneral PsychologyBupropionDose-Response Relationship DrugAge FactorsGroomingSocial relationAggressionSocial IsolationAntidepressantAntidepressive Agents Second-GenerationPsychologyAgonistic Behaviormedicine.drugAggressive behavior
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Elevation of striatal urate in experimental models of Parkinson's disease: a compensatory mechanism triggered by dopaminergic nigrostriatal degenerat…

2014

Epidemiological studies have indicated an inverse association between high uricemia and incidence of Parkinson's disease (PD). To investigate the link between endogenous urate and neurotoxic changes involving the dopaminergic nigrostriatal system, this study evaluated the modifications in the striatal urate levels in two models of PD. To this end, a partial dopaminergic degeneration was induced by 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP) in mice, while a severe dopaminergic degeneration was elicited by unilateral medial forebrain bundle infusion of 6-hydroxydopamine (6-OHDA) in rats. Urate levels were measured by in vivo microdialysis at 7 or 14 days from toxin exposure. The resu…

Malemedicine.medical_specialtyParkinson's diseaseDopamineStriatumBiochemistryNeuroprotectionRats Sprague-DawleyCellular and Molecular Neurosciencechemistry.chemical_compoundHydroxydopaminesMiceDopamineInternal medicinemedicineAnimalsParkinson Disease SecondaryMedial forebrain bundleMPTPDopaminergic NeuronsNeurodegenerationDopaminergicMPTP Poisoningmedicine.diseaseRatsUric AcidMice Inbred C57BLNeostriatumSubstantia NigraEndocrinologynervous systemchemistryNeurosciencemedicine.drugJournal of neurochemistry
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Stage-dependent agreement between cerebrospinal fluid proteins and FDG-PET findings in Alzheimer's disease.

2011

Cerebral hypometabolism and abnormal levels of amyloid beta (Aβ), total (t-tau) and phosphorylated tau (ptau) proteins in cerebrospinal fluid (CSF) are established biomarkers of Alzheimer's disease (AD). We examined the agreement between these biomarkers in a single center study of patients with AD of severity extending over a wide range. Forty seven patients (MMSE 21.4 ± 3.6, range 13-28 points) with incipient and probable AD underwent positron emission tomography with [18F]-fluorodeoxyglucose (FDG-PET) and lumbar puncture for CSF assays of Aβ1-42, p-tau181, and t-tau. All findings were classified as either positive or negative for AD. Statistical analyses were performed for the whole samp…

Malemedicine.medical_specialtyPathologyAmyloid betaApolipoprotein E4tau ProteinsNeuropsychological TestsGastroenterologySensitivity and SpecificityCerebrospinal fluidAlzheimer DiseaseFluorodeoxyglucose F18Internal medicinemental disordersmedicineDementiaHumansAgedRetrospective StudiesPsychiatric Status Rating ScalesAmyloid beta-Peptidesmedicine.diagnostic_testbiologyLumbar punctureNeurodegenerationCerebrospinal Fluid ProteinsMiddle Agedmedicine.diseasePeptide FragmentsNeurologyPositron emission tomographyArea Under CurvePositron-Emission Tomographybiology.proteinFemaleNeurology (clinical)Alzheimer's diseasePsychologyCognition DisordersKappaBiomarkersFollow-Up StudiesCurrent Alzheimer research
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Nerve degeneration in inguinal hernia specimens

2011

BACKGROUND: The histological study of the herniated inguinal area is rare in the literature. This report is focused on the detection of structural changes of the nerves within tissues bordering the inguinal hernia of cadavers. Their physiopathological consequences are hypothesized. MATERIALS AND METHODS: Primary inguinal hernia was diagnosed in 30 fresh cadavers. Tissue specimens from the inguinal region close to and around the hernia opening were excised for histological examination. A control of the data was achieved through tissue samples excised from equivalent sites of the inguinal region in 15 cadavers without hernia. RESULTS: The detected nerves in the inguinal area demonstrated path…

Malemedicine.medical_specialtyPathologyInguinal CanalHernia InguinalDegeneration (medical)INGUINAL HERNIAGroinAbdominal wallAtrophyCadavermedicineCadaverHumansHerniaPeripheral NervesInguinal hernia nerve degenerationMuscle SkeletalAgedbusiness.industryAbdominal WallDystrophyAnatomyMiddle Agedmedicine.diseaseFibrosisSurgeryINGUINAL HERNIA.Inguinal herniaSettore MED/18 - Chirurgia Generalemedicine.anatomical_structureNerve DegenerationSurgerybusinessAbdominal surgery
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Increased blood mercury levels in patients with Alzheimer's disease.

1998

Alzheimer's disease (AD) is a common neurodegenerative disorder that leads to dementia and death. In addition to several genetic parameters, various environmental factors may influence the risk of getting AD. In order to test whether blood levels of the heavy metal mercury are increased in AD, we measured blood mercury concentrations in AD patients (n = 33), and compared them to age-matched control patients with major depression (MD) (n = 45), as well as to an additional control group of patients with various non-psychiatric disorders (n = 65). Blood mercury levels were more than two-fold higher in AD patients as compared to both control groups (p = 0.0005, and p = 0.0000, respectively). In…

Malemedicine.medical_specialtyPathologyNeurologychemistry.chemical_elementtau ProteinsCentral nervous system diseaseDegenerative diseaseAlzheimer DiseaseInternal medicineBlood plasmamedicineDementiaHumansBiological PsychiatryAgedAged 80 and overDepressive DisorderAmyloid beta-PeptidesNeurodegenerationMercuryMiddle Agedmedicine.diseaseMercury (element)Psychiatry and Mental healthEndocrinologyNeurologychemistryLinear ModelsFemaleNeurology (clinical)Alzheimer's diseasePsychologyBiomarkersJournal of neural transmission (Vienna, Austria : 1996)
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The Adult Pituitary Shows Stem/Progenitor Cell Activation in Response to Injury and Is Capable of Regeneration

2012

The pituitary gland constitutes, together with the hypothalamus, the regulatory core of the endocrine system. Whether the gland is capable of cell regeneration after injury, in particular when suffered at adult age, is unknown. To investigate the adult pituitary's regenerative capacity and the response of its stem/progenitor cell compartment to damage, we constructed a transgenic mouse model to conditionally destroy pituitary cells. GHCre/iDTR mice express diphtheria toxin (DT) receptor after transcriptional activation by Cre recombinase, which is driven by the GH promoter. Treatment with DT for 3 d leads to gradual GH+ (somatotrope) cell obliteration with a final ablation grade of 80–90% 1…

Malemedicine.medical_specialtyPituitary glandTime FactorsSomatotropic cellMice TransgenicBiologyMiceEndocrinologySOX2Internal medicinemedicineAnimalsRegenerationCell LineageProgenitor cellPromoter Regions GeneticSOXB1 Transcription FactorsStem CellsDeoxyuridineEndothelial stem cellEndocrinologymedicine.anatomical_structureGrowth HormonePituitary GlandIntercellular Signaling Peptides and ProteinsFemaleStem cellHeparin-binding EGF-like Growth FactorAdult stem cellEndocrine glandEndocrinology
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