Search results for "Genetic Variation"

showing 10 items of 836 documents

Comparative analysis of variation and selection in the HCV genome

2016

AbstractGenotype 1 of the hepatitis C virus (HCV) is the most prevalent of the variants of this virus. Its two main subtypes, HCV-1a and HCV-1b, are associated to differences in epidemic features and risk groups, despite sharing similar features in most biological properties. We have analyzed the impact of positive selection on the evolution of these variants using complete genome coding regions, and compared the levels of genetic variability and the distribution of positively selected sites. We have also compared the distributions of positively selected and conserved sites considering different factors such as RNA secondary structure, the presence of different epitopes (antibody, CD4 and C…

CD4-Positive T-Lymphocytes0301 basic medicineMicrobiology (medical)GenotypeEpitopes T-LymphocyteGenome ViralHepacivirusCD8-Positive T-LymphocytesBiologyGenoma humàMicrobiologyGenomeEpitopeNucleic acid secondary structureEvolution MolecularViral Proteins03 medical and health sciencesNegative selection0302 clinical medicineGenotypeGeneticsHumansCoding regionAmino Acid SequenceGenetic variabilitySelection GeneticMolecular BiologyGenePhylogenyEcology Evolution Behavior and SystematicsSelection (genetic algorithm)GeneticsGenetic VariationSequence Analysis DNAHepatitis C AntibodiesHepatitis C ChronicVirus030104 developmental biologyInfectious DiseasesRNA Viral030211 gastroenterology & hepatology
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Genetic variation and evolutionary analysis of Cucumber mosaic virus in medicinal, aromatic and ornamental plants from Northern Italy

2011

The genetic variation and evolution of Cucumber mosaic virus (CMV) (1) infecting aromatic, medicinal and ornamental plants in Northern Italy were studied by sequence analysis of the movement protein gene and comparison with equivalent sequences of isolates from other countries. Comparison of nonsynonymous and synonymous substitutions at each codon suggested that 30% of amino acid sites were under negative selection and only one was under positive selection. Phylogenetic inference, nucleotide diversity estimation, neutrality tests and genetic differentiation analysis revealed a high nucleotide diversity of CMV in Northern Italy with respect to areas of other countries [1,2] and suggested tha…

CMV Genetic variationPIANTE OFFICINALICMVSettore AGR/12 - Patologia VegetaleCARATTERIZIONE MOLECOLARE
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Evidence for cryptic glacial refugia from North American mountain sheep mitochondrial DNA

2006

The separation of populations by ice sheets into large refugia can account for much of the genetic diversity found in present day populations. The evolutionary implications of small glacial refugia have not been as thoroughly explored. To examine refugial origins of North American mountain sheep Ovis spp., we analyzed a 604 bp portion of the mitochondrial DNA (mtDNA) control region from 223 O. dalli and O. canadensis. Major refugia were identified in eastern Beringia and southern North America, and we found evidence for two smaller refugia situated between the Laurentide and Cordilleran glaciers. Our results are the first to demonstrate support for survival of any organism in the latter two…

CanadaMitochondrial DNAClimateAnimals WildEnvironmentBiologyDNA MitochondrialBeringiaMountain sheepEvolution Molecularcvg.developerGenetic variationAnimalsIce CoverGlacial periodcvgEcology Evolution Behavior and SystematicsgeographyGenetic diversitySheepgeography.geographical_feature_categoryModels GeneticEcologyAltitudeGenetic VariationGlacierNorth AmericaIce sheetJournal of Evolutionary Biology
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BCL2 gene polymorphisms and splicing variants in chronic myeloid leukemia.

2015

Recent data suggest that constitutional genetic variation in the antiapoptotic BCL2 gene could be associated with the susceptibility to develop chronic myeloid leukemia (CML) and the clinical outcome in several hematological malignancies. The present study examines whether BCL2 single nucleotide polymorphisms (SNPs) predispose to CML or may potentially influence the disease characteristics at diagnosis. Notably, no association was observed between the four candidate BCL2 SNPs and the risk of developing CML. Instead, the 4777C>A (rs2279115) and the 5735A>G (rs1801018) SNPs were significantly associated with the disease risk profile as determined by the Sokal score. We found that such polymor…

Cancer ResearchBCL2business.industryAlternative splicingChronic myeloid leukemiaClinical courseMyeloid leukemiaSingle-nucleotide polymorphismHematologyBioinformaticsSplicingBCL2 Chronic myeloid leukemia Polymorphisms Splicing SusceptibilityOncologyimmune system diseasesSusceptibilityhemic and lymphatic diseasesGenetic variationRNA splicingMedicinebiological phenomena cell phenomena and immunitySokal ScorebusinessPolymorphismsGeneneoplasms
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Presence on a human melanoma of multiple antigens recognized by autologous CTL.

1989

We derived from blood lymphocytes of a melanoma patient a large number of cytolytic T-cell clones directed against a cell line of the autologous tumor. Three distinct groups of antigens were recognized by these CTL on the autologous melanoma cells: group A consisted of stable antigens present on all sublines, whereas antigens B and C appeared unstable and were expressed by distinct sublines. In vitro immunoselections with various anti-A CTL clones were applied to the melanoma cells and variants resistant to 3 different CTL clones were obtained. These variants remained sensitive to other anti-A CTL clones, indicating that group A comprises at least 4 different antigens (D, E, F and A'). From…

Cancer ResearchCellular immunitySkin NeoplasmsLymphocyteGenes MHC Class IHuman leukocyte antigenBiologyCell LineAntigenAntigens NeoplasmHLA AntigensmedicineTumor Cells CulturedHumansPan-T antigensMelanomaMelanomaGenetic Variationmedicine.diseaseClone CellsGene Expression Regulation NeoplasticCytolysisCTL*medicine.anatomical_structureOncologyImmunologyLymphocyte Culture Test MixedT-Lymphocytes CytotoxicInternational journal of cancer
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Novel insulin receptor substrate 1 and 2 variants in breast and colorectal cancer

2013

The insulin/insulin-like growth factor pathway is involved in breast and colorectal cancer (CRC) development. In the present study, we analyzed the coding region and short intron-exon borders of the insulin receptor substrate 1 and 2 (IRS‑1 and IRS‑2) genes in 12 cell lines derived from breast cancer (BC), 14 cell lines derived from CRC and 33 primary CRCs. The nucleotide variants identified in BC were 3 in IRS‑1, 1 of which (p.Arg267Cys) was novel and with a pathogenic potential as predicted by in silico analysis and 6 in IRS‑2. Twenty‑one variants in IRS‑1 and 18 in IRS‑2 were identified in the CRC samples. These included 11 novel IRS‑1 variants detected exclusively in CRCs, which include…

Cancer ResearchInsulin Receptor Substrate ProteinsSettore MED/06 - Oncologia MedicaIn silicoMutation MissenseBreast NeoplasmsColorectal NeoplasmBiologymedicine.disease_causeFrameshift mutationBreast cancerBreast cancerMCF-7 CellCell Line TumormedicineHumansMissense mutationFrameshift MutationInsulin Receptor Substrate ProteinSequence DeletionGeneticsMutationCaco-2 CellPolymorphism GeneticCancerGenetic VariationInsulin receptor substrate 1ArticlesGeneral MedicineInsulin receptor substrate 2HCT116 Cellsmedicine.diseaseColorectal cancerIRS1Mutagenesis InsertionalCell Transformation NeoplasticHT29 CellOncologyHCT116 CellBreast cancer; Colorectal cancer; Insulin receptor substrate 1; Insulin receptor substrate 2; Breast Neoplasms; Caco-2 Cells; Cell Line Tumor; Cell Transformation Neoplastic; Colorectal Neoplasms; Female; Frameshift Mutation; Genetic Variation; HCT116 Cells; HT29 Cells; Humans; Insulin Receptor Substrate Proteins; MCF-7 Cells; Mutagenesis Insertional; Mutation Missense; Polymorphism Genetic; Sequence Deletion; Signal Transduction; Cancer Research; OncologyInsulin Receptor Substrate ProteinsMCF-7 CellsFemaleCaco-2 CellsColorectal NeoplasmsHT29 CellsBreast NeoplasmHumanSignal Transduction
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The effect of genetic complementation on the fitness and diversity of viruses spreading as collective infectious units

2019

Viruses can spread collectively using different types of structures such as extracellular vesicles, virion aggregates, polyploid capsids, occlusion bodies, and even cells that accumulate virions at their surface, such as bacteria and dendritic cells. Despite the mounting evidence for collective spread, its implications for viral fitness and diversity remain poorly understood. It has been postulated that, by increasing the cellular multiplicity of infection, collective spread could enable mutually beneficial interactions among different viral genetic variants. One such interaction is genetic complementation, whereby deleterious mutations carried by different genomes are compensated. Here, we…

Cancer ResearchMutation rateViral diversityEvolutionPopulationViral transmissionGenome ViralBiologyVirus ReplicationGenomeEvolution Molecular03 medical and health sciencesMultiplicity of infectionPolyploidVirologyeducation030304 developmental biologyGenetics0303 health scienceseducation.field_of_study030306 microbiologyVirionDefective VirusesGenetic VariationDendritic cellGenetic complementationMutation AccumulationModels TheoreticalCollective spread3. Good healthComplementationInfectious DiseasesMutationGenetic FitnessVirus Research
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Geographical distribution and oncogenic risk association of human papillomavirus type 58 E6 and E7 sequence variations.

2013

Human papillomavirus (HPV) 58 accounts for a notable proportion of cervical cancers in East Asia and parts of Latin America, but it is uncommon elsewhere. The reason for such ethnogeographical predilection is unknown. In our study, nucleotide sequences of E6 and E7 genes of 401 HPV58 isolates collected from 15 countries/cities across four continents were examined. Phylogenetic relationship, geographical distribution and risk association of nucleotide sequence variations were analyzed. We found that the E6 genes of HPV58 variants were more conserved than E7. Thus, E6 is a more appropriate target for type-specific detection, whereas E7 is more appropriate for strain differentiation. The frequ…

Cancer ResearchSettore MED/07 - Microbiologia E Microbiologia Clinicacervical cancerSettore MED/06 - Oncologia MedicaPapillomavirus E7 ProteinsUterine Cervical NeoplasmsCervix UteriphylogenyPolymerase Chain ReactionViralPapillomaviridaePapillomaviridaePhylogenyCancerOncogene ProteinsCervical cancerGeneticsTumorGeographybiologyNucleic acid sequenceDNA NeoplasmPrognosisInfectious DiseasesOncologyHIV/AIDSFemaleHPVhuman papillomavirus type 58 E6 and E7 sequence variationsOncology and CarcinogenesisCervical intraepithelial neoplasiaRisk AssessmentArticleVaccine Relatedoncogenic riskClinical ResearchPhylogeneticsGenetic variationGeneticsBiomarkers TumormedicineHumansOncology & CarcinogenesisGenePreventionPapillomavirus InfectionsGenetic VariationInternational AgenciesDNAOncogene Proteins ViralOdds ratioUterine Cervical Dysplasiamedicine.diseasebiology.organism_classificationVirologyvariantNeoplasmSexually Transmitted InfectionsCapsid ProteinsBiomarkersFollow-Up Studies
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Single-Cell Analysis of RNA Virus Infection Identifies Multiple Genetically Diverse Viral Genomes within Single Infectious Units

2015

Summary Genetic diversity enables a virus to colonize novel hosts, evade immunity, and evolve drug resistance. However, viral diversity is typically assessed at the population level. Given the existence of cell-to-cell variation, it is critical to understand viral genetic structure at the single-cell level. By combining single-cell isolation with ultra-deep sequencing, we characterized the genetic structure and diversity of a RNA virus shortly after single-cell bottlenecks. Full-length sequences from 881 viral plaques derived from 90 individual cells reveal that sequence variants pre-existing in different viral genomes can be co-transmitted within the same infectious unit to individual cell…

Cancer Research[SDE.MCG]Environmental Sciences/Global ChangesvirusesGenome Viralmedicine.disease_causeMicrobiologyArticleVirus[SDV.EE.ECO]Life Sciences [q-bio]/Ecology environment/EcosystemsSingle-cell analysisViral entry[SDV.MHEP.MI]Life Sciences [q-bio]/Human health and pathology/Infectious diseasesCricetinaeVirologyImmunology and Microbiology(all)Genetic variationmedicineAnimalsMolecular BiologyCells CulturedComputingMilieux_MISCELLANEOUSGenetics[SDV.EE.SANT]Life Sciences [q-bio]/Ecology environment/HealthGenetic diversityMutation[SDV.MHEP.ME]Life Sciences [q-bio]/Human health and pathology/Emerging diseasesbiologyGenetic VariationHigh-Throughput Nucleotide SequencingEpithelial CellsRNA virusVesiculovirusbiology.organism_classification[SDV.MP.BAC]Life Sciences [q-bio]/Microbiology and Parasitology/Bacteriology3. Good healthGenetic structure[SDV.MP.VIR]Life Sciences [q-bio]/Microbiology and Parasitology/VirologyParasitologySingle-Cell Analysis[SDE.BE]Environmental Sciences/Biodiversity and Ecologyhuman activitiesCell Host & Microbe
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Genome-Wide Analyses Identifies Known and New Markers Responsible of Chicken Plumage Color

2020

Simple Summary In order to assess sources of variation related to Polverara breed plumage color (black vs. white), we carried out genome-wide analyses to identify the genomic regions involved in this trait. The present work has revealed new candidate genes involved in the phenotypic variability in local chicken populations. These results also contribute insights into the genetic basis for plumage color in poultry, and confirm the great complexity of the mechanisms that control this trait. Abstract Through the development of the high-throughput genotyping arrays, molecular markers and genes related to phenotypic traits have been identified in livestock species. In poultry, plumage color is a…

Candidate geneCandidate genegenome-wide analysesCandidate genes; Genome-wide analyses; Local chicken populations; Plumage color; SNPSNPSingle-nucleotide polymorphismGenome-wide association studyBiologyArticleFixation indexSettore AGR/17 - Zootecnica Generale E Miglioramento Genetico03 medical and health scienceslcsh:ZoologyGenetic variationlcsh:QL1-991GenotypingLocal chicken population030304 developmental biologyGeneticsGenome-wide analyse0303 health scienceslcsh:Veterinary medicineGeneral Veterinarylocal chicken populations0402 animal and dairy science04 agricultural and veterinary sciencesPhenotypic traitlocal chicken populations; genome-wide analyses; SNP; plumage color; candidate genes040201 dairy & animal sciencePlumage colorPlumagelcsh:SF600-1100Animal Science and Zoologycandidate genesAnimals
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