Search results for "Genetic therapy"

showing 10 items of 121 documents

Altered intracellular sorting signals do not influence the efficacy of genetic melanoma vaccines incorporating helper determinants in mice.

2004

Background A genetic melanoma vaccine consisting of cDNA encoding the model self-antigen tyrosinase-related protein 2 (TRP2) fused in-frame to the immunogenic enhanced green fluorescent protein (EGFP) was able to break immune tolerance and stimulate CD8+ T cells in vivo. In the present study we investigated whether alteration of the intracellular antigen localization as a result of the linkage with immune-enhancing helper proteins affects the resulting immune response. Methods Expression plasmids and recombinant adenoviruses were constructed encoding various fusion proteins with different intracellular sorting signals which direct the antigen to the cytosol, the endoplasmic reticulum or the…

Skin Neoplasmsmedicine.medical_treatmentRecombinant Fusion ProteinsGenetic VectorsGreen Fluorescent ProteinsMelanoma ExperimentalAutoimmunityBiologyCancer VaccinesMelanoma VaccineImmune toleranceMiceImmune systemAntigenDrug DiscoveryGeneticsmedicineAnimalsMolecular BiologyGenetics (clinical)MelanomaELISPOTImmunotherapyGenetic TherapyT-Lymphocytes Helper-Inducermedicine.diseaseMolecular biologyFusion proteinCell biologyIntramolecular OxidoreductasesMice Inbred C57BLProtein TransportCD4 AntigensMolecular MedicineImmunizationThe journal of gene medicine
researchProduct

Antisense gene therapy using anti-k-ras and antitelomerase oligonucleotides in colorectal cancer

2005

Aim: to test the efficacy of anti-k-ras and antitelomerase oligonucleotides for disabling colorectal cancer cell growth. Material and methods: an established human colorectal cancer cell line (SW 480, ATTC ® ) was used. Oligodeoxiribonucleotides (ODNs) have a phosphorotioate modification to ensure intracellular intake. We used an antitelomerase ODN (Telp5) and two anti-k-ras ODNs (AS-KRAS and ISIS). AS-KRAS is designed to join the k-ras oncogene’s exon 1. ISIS links to the terminal transcription unit 5’ of k-ras. Telp5 joins the template region of the hTR telomerase subunit. ODNs have been tested in different concentrations (1, 5, 10, 20 micromolar). Cell viability has been tested at 48 and…

TelomeraseColorectal cancerAntisense therapyK-ras oncogenemedicine.disease_causeOligodeoxyribonucleotides AntisenseCell Line TumormedicineHumansViability assayTelomeraseOligoribonucleotidesOncogeneOligonucleotideCell growthbusiness.industryGastroenterologyGenetic TherapyGeneral Medicinemedicine.diseaseColorectal cancerGenes rasImmunologyCancer researchKRASColorectal NeoplasmsbusinessSoftwareIntracellularRevista Española de Enfermedades Digestivas
researchProduct

Expression of the genetic suppressor element 24.2 (GSE24.2) decreases DNA damage and oxidative stress in X-linked dyskeratosis congenita cells.

2014

This is an open-access article distributed under the terms of the Creative Commons Attribution License.-- et al.

TelomeraseDNA repairDNA damagelcsh:MedicineCell Cycle ProteinsComputingMilieux_LEGALASPECTSOFCOMPUTINGBiologyTransfectionBioinformaticsmedicine.disease_causeBiochemistryDyskeratosis CongenitaDyskerinCell LineMiceHeterochromatinMolecular Cell BiologyMedicine and Health SciencesmedicineAnimalsHumanslcsh:ScienceMutationMultidisciplinarylcsh:RBiology and Life SciencesNuclear ProteinsCell BiologyHematologyGenetic TherapyTransfectionTelomeremedicine.diseaseTelomereCell biologyOxidative StressGene Expression Regulationlcsh:QPeptidesDyskeratosis congenitaResearch ArticleDNA DamagePLoS ONE
researchProduct

Persistent episomal transgene expression in liver following delivery of a scaffold/matrix attachment region containing non-viral vector

2008

An ideal gene therapy vector should enable persistent transgene expression without limitations of safety and reproducibility. Here we report the development of a non-viral episomal plasmid DNA (pDNA) vector that appears to fulfil these criteria. This pDNA vector combines a scaffold/matrix attachment region (S/MAR) with a human liver-specific promoter (alpha1-antitrypsin (AAT)) in such a way that long-term expression is enabled in murine liver following hydrodynamic injection. Long-term expression is demonstrated by monitoring the longitudinal luciferase expression profile for up to 6 months by means of in situ bioluminescent imaging. All relevant control pDNA constructs expressing luciferas…

Time FactorsTransgeneGenetic VectorsGene ExpressionMice Inbred StrainsGene deliveryBiologyTransfectionViral vectorInjectionsMiceSettore MED/38 - Pediatria Generale E SpecialisticaGene expressionGeneticsGene silencingAnimalsHepatectomyHumansLuciferaseTransgenesScaffold/matrix attachment regionLuciferasesPromoter Regions GeneticMolecular BiologyGenetic Therapynon-viral episomal plasmid DNA (pDNA) vector S/MAR element hydrodynamic injection.DNA MethylationMatrix Attachment RegionsMolecular biologyImmunohistochemistryLiveralpha 1-AntitrypsinDNA methylationMolecular MedicinePlasmids
researchProduct

Transcriptional targeting of dendritic cells for gene therapy using the promoter of the cytoskeletal protein fascin.

2003

Strong cell-type-specific promoters are basic tools in gene therapy allowing for novel applications and focused strategies by transcriptionally targeting gene expression to selected cells. In immunotherapy, dendritic cells (DC) are of central importance, since they represent the principal inducers of immune responses. Here we describe isolation and use of the promoter of the murine actin-bundling protein fascin to target transcriptionally gene expression to cutaneous DC. Using the reporter gene enhanced green fluorescent protein (EGFP), we demonstrate that the fascin promoter mediates a strong antigen expression that is restricted to mature DC. DNA vaccination with antigen-encoding expressi…

Transcription GeneticBiologyCD8-Positive T-LymphocytesDNA vaccinationMiceGenes ReporterGene expressionGeneticsVaccines DNAAnimalsPromoter Regions GeneticMolecular BiologyFascinReporter geneMice Inbred BALB CExpression vectorMicrofilament ProteinsPromoterDendritic cellTransfectionDendritic CellsGenetic TherapyBiolisticsMolecular biologyMice Inbred C57BLbiology.proteinMolecular MedicineCarrier ProteinsGene therapy
researchProduct

Generation and characterization of tTS-H4: a novel transcriptional repressor that is compatible with the reverse tetracycline-controlled TET-ON system

2007

Background Conditional gene regulatory systems ensuring tight and adjustable expression of therapeutic genes are central for developing future gene therapy strategies. Among various regulatory systems, tetracycline-controlled gene expression has emerged as a safe and reliable option. Moreover, the tightness of tetracycline-regulated gene switches can be substantially improved by complementing transcriptional activators with antagonizing repressors. Methods To develop novel tetracycline-responsive transcriptional repressors, we fused various transcriptional silencing domains to the TetR (B/E) DNA-binding and dimerization domain of the Tn10-encoded tetracycline resistance operon (TetR (B/E)).…

Transcription GeneticOperonRepressorBiologyHistone DeacetylasesHistonesMicechemistry.chemical_compoundGenes ReporterDrug DiscoveryGeneticsAnimalsHumansGene silencingTetRPromoter Regions GeneticMolecular BiologyGenetics (clinical)Regulation of gene expressionYY1Genetic TherapyTetracyclineMolecular biologyHDAC4Repressor ProteinsGene Expression RegulationchemistryGATAD2BNIH 3T3 CellsMolecular Medicine
researchProduct

Improving mRNA-Based Therapeutic Gene Delivery by Expression-Augmenting 3' UTRs Identified by Cellular Library Screening.

2019

Synthetic mRNA has emerged as a powerful tool for the transfer of genetic information, and it is being explored for a variety of therapeutic applications. Many of these applications require prolonged intracellular persistence of mRNA to improve bioavailability of the encoded protein. mRNA molecules are intrinsically unstable and their intracellular kinetics depend on the UTRs embracing the coding sequence, in particular the 3′ UTR elements. We describe here a novel and generally applicable cell-based selection process for the identification of 3′ UTRs that augment the expression of proteins encoded by synthetic mRNA. Moreover, we show, for two applications of mRNA therapeutics, namely, (1) …

Untranslated regionCellular differentiationRNA StabilityInduced Pluripotent Stem CellsBlood DonorsComputational biologyGene deliveryBiologyCancer Vaccines03 medical and health sciencesMice0302 clinical medicineDrug DiscoveryGeneticsCoding regionAnimalsHumansRNA MessengerInduced pluripotent stem cellMolecular BiologyGene3' Untranslated RegionsCells Cultured030304 developmental biologyGene LibraryPharmacology0303 health sciencesMessenger RNAMice Inbred BALB CVaccinationGene Transfer TechniquesGenetic TherapyFibroblastsCellular Reprogramming030220 oncology & carcinogenesisMolecular MedicineFemaleOriginal ArticleReprogrammingHalf-LifeMolecular therapy : the journal of the American Society of Gene Therapy
researchProduct

Direct and long-term detection of gene doping in conventional blood samples

2010

The misuse of somatic gene therapy for the purpose of enhancing athletic performance is perceived as a coming threat to the world of sports and categorized as 'gene doping'. This article describes a direct detection approach for gene doping that gives a clear yes-or-no answer based on the presence or absence of transgenic DNA in peripheral blood samples. By exploiting a priming strategy to specifically amplify intronless DNA sequences, we developed PCR protocols allowing the detection of very small amounts of transgenic DNA in genomic DNA samples to screen for six prime candidate genes. Our detection strategy was verified in a mouse model, giving positive signals from minute amounts (20 μl)…

Vascular Endothelial Growth Factor ACandidate geneAthletic PerformanceBiologyPolymerase Chain ReactionDNA sequencinglaw.inventionMicelawGene dopingGeneticsAnimalsHumansTransgenesMolecular BiologyGenePolymerase chain reactionDoping in SportsGeneticsGenetic transferGenetic TherapyNucleic acid amplification techniqueDependovirusgenomic DNAGene ComponentsMolecular MedicineNucleic Acid Amplification TechniquesGene Therapy
researchProduct

Capillary enlargement, not sprouting angiogenesis, determines beneficial therapeutic effects and side effects of angiogenic gene therapy.

2010

Aims Currently, it is still unclear which mechanisms drive metabolic benefits after angiogenic gene therapy. The side-effect profile of efficient angiogenic gene therapy is also currently incompletely understood. In this study, the effects of increasing doses of adenoviral (Ad) vascular endothelial growth factor-A (VEGF-A) were evaluated on vascular growth, metabolic benefits, and systemic side effects. Methods and results Adenoviral vascular endothelial growth factor-A or AdLacZ control was injected intramuscularly (109–1011 vp/mL) or intra-arterially (5 × 1011 vp/mL) into rabbit ( n = 102) hindlimb muscles and examined 6 or 14 days later. Blood flow, tissue oedema, metabolic benefits, and…

Vascular Endothelial Growth Factor Amedicine.medical_specialtyAngiogenesisGenetic VectorsNeovascularization PhysiologicEnzyme-Linked Immunosorbent Assayta3111Injections IntramuscularAdenoviridaeNeovascularizationchemistry.chemical_compoundInternal medicinemedicineAnimalsMuscle SkeletalUltrasonography InterventionalSprouting angiogenesisDose-Response Relationship Drugbusiness.industryGene Transfer TechniquesMetabolic acidosisGenetic Therapymedicine.diseaseCapillariesHindlimbVascular endothelial growth factorVascular endothelial growth factor AEndocrinologychemistryLac OperonCirculatory systemRabbitsmedicine.symptomCardiology and Cardiovascular MedicinebusinessPerfusionEuropean heart journal
researchProduct

Treatment of Anderson-Fabry Disease

2020

Fabry disease is an X-linked disorder of glycosphingolipid metabolism that results in progressive accumulation of neutral glycosphingolipids, predominantly globotriaosylsphingosine (Gb3) in lysosomes, as well as other cellular compartments of several tissues, causing multi-organ manifestations (acroparesthesias, hypohidrosis, angiokeratomas, signs and symptoms of cardiac, renal, cerebrovascular involvement). Pathogenic mutations lead to a deficiency of the lysosomal enzyme alpha-galactosidase A (GLA). In the presence of high clinical suspicion, a careful physical examination and specific laboratory tests are required. Finally, the diagnosis of Fabry’s disease is confirmed by the demonstrat…

Viral vectorsMaleGenetic enhancementChaperone therapyPhysical examinationDiseaseKidneyViral vector03 medical and health sciencesGene therapy0302 clinical medicineDrug DiscoverymedicineHumansEnzyme Replacement Therapy030304 developmental biologyPharmacology0303 health sciencesmedicine.diagnostic_testbusiness.industryPharmacologicalGenetic TherapyEnzyme replacement therapymedicine.diseaseFabry diseasePharmacological chaperonealpha-GalactosidaseImmunologyFabry DiseaseFemaleStem cellbusiness030217 neurology & neurosurgerymedicine.drugCurrent Pharmaceutical Design
researchProduct