Search results for "Genetics and Genomics"

showing 10 items of 46 documents

Evolutionary convergence and nitrogen metabolism in Blattabacterium strain Bge, primary endosymbiont of the cockroach Blattella germanica.

2009

Bacterial endosymbionts of insects play a central role in upgrading the diet of their hosts. In certain cases, such as aphids and tsetse flies, endosymbionts complement the metabolic capacity of hosts living on nutrient-deficient diets, while the bacteria harbored by omnivorous carpenter ants are involved in nitrogen recycling. In this study, we describe the genome sequence and inferred metabolism of Blattabacterium strain Bge, the primary Flavobacteria endosymbiont of the omnivorous German cockroach Blattella germanica. Through comparative genomics with other insect endosymbionts and free-living Flavobacteria we reveal that Blattabacterium strain Bge shares the same distribution of functio…

Cancer Researchfood.ingredientlcsh:QH426-470NitrogenBlochmanniaZoologyCockroachesEvolution Molecular03 medical and health sciencesBlattabacteriumfoodSymbiosisEnterobacteriaceaePhylogeneticsAmmoniabiology.animalBotanyGeneticsAnimalsAmino AcidsSymbiosisMolecular BiologyGenetics (clinical)Ecology Evolution Behavior and SystematicsPhylogeny030304 developmental biology0303 health sciencesGerman cockroachCockroachbiologyPhylogenetic treeEvolutionary Biology/Evolutionary and Comparative Genetics030306 microbiologyAntsBacteroidetesfungiGenomicsbiochemical phenomena metabolism and nutritionbiology.organism_classificationGenetics and Genomics/Microbial Evolution and Genomicslcsh:GeneticsGenetics and Genomics/Genome ProjectsEvolutionary Biology/Microbial Evolution and GenomicsHost-Pathogen InteractionsBacteriaGenome BacterialMetabolic Networks and PathwaysResearch ArticlePLoS genetics
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The Fitness Effects of Random Mutations in Single-Stranded DNA and RNA Bacteriophages

2009

Mutational fitness effects can be measured with relatively high accuracy in viruses due to their small genome size, which facilitates full-length sequencing and genetic manipulation. Previous work has shown that animal and plant RNA viruses are very sensitive to mutation. Here, we characterize mutational fitness effects in single-stranded (ss) DNA and ssRNA bacterial viruses. First, we performed a mutation-accumulation experiment in which we subjected three ssDNA (ΦX174, G4, F1) and three ssRNA phages (Qβ, MS2, and SP) to plaque-to-plaque transfers and chemical mutagenesis. Genome sequencing and growth assays indicated that the average fitness effect of the accumulated mutations was similar…

Cancer Researchlcsh:QH426-470virusesDNA Single-StrandedRNA PhagesBiologymedicine.disease_causeGenomeDNA sequencingGenetics and Genomics/Population GeneticsGeneticsmedicinePoint MutationSelection GeneticMolecular BiologyGenome sizeGenetics (clinical)Ecology Evolution Behavior and SystematicsGeneticsMutationMicrobiology/Microbial Evolution and GenomicsModels GeneticPoint mutationRNARNA PhagesGenetics and Genomics/Microbial Evolution and Genomicslcsh:GeneticsEvolutionary Biology/Microbial Evolution and GenomicsMutagenesisMutationMutagenesis Site-DirectedBacterial virusResearch ArticlePLoS Genetics
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Myc Promoter-Binding Protein-1 (MBP-1) Is a Novel Potential Prognostic Marker in Invasive Ductal Breast Carcinoma

2010

BackgroundAlpha-enolase is a glycolytic enzyme that catalyses the formation of phosphoenolpyruvate in the cell cytoplasm. α-Enolase and the predominantly nuclear Myc promoter-binding protein-1 (MBP-1) originate from a single gene through the alternative use of translational starting sites. MBP-1 binds to the P2 c-myc promoter and competes with TATA-box binding protein (TBP) to suppress gene transcription. Although several studies have shown an antiproliferative effect of MBP-1 overexpression on several human cancer cells, to date detailed observations of α-enolase and MBP-1 relative expression in primary tumors versus normal tissues and their correlation with clinicopathological features ha…

CytoplasmAlpha-enolasePROGRESSIONAged 80 and overRegulation of gene expressionMultidisciplinaryQRGenetics and Genomics/Gene ExpressionMiddle AgedPrognosisPathology/Molecular PathologyNUDE-MICETransport proteinCarcinoma DuctalDNA-Binding ProteinsGene Expression Regulation NeoplasticProtein Transportmedicine.anatomical_structureGLYCOLYTIC ENZYMEOncology/Breast CancerMedicineCELL LUNG-CANCER; ALPHA-ENOLASE; PROTEOMIC ANALYSIS; GLYCOLYTIC ENZYME; NUDE-MICE; GENE; IDENTIFICATION; PROGRESSION; EXPRESSION; METASTASESFemalePROTEOMIC ANALYSISEnolase MBP-1 Breast cancer ImmunohistochemistryResearch ArticleAdultEXPRESSIONScienceCELL LUNG-CANCERBreast NeoplasmsBiologyDNA-binding proteinBiomarkers TumormedicineHumansNeoplasm InvasivenessGeneAgedCell NucleusIDENTIFICATIONBinding proteinALPHA-ENOLASEGENEMolecular biologySettore BIO/18 - GeneticaCell nucleusMETASTASESCytoplasmPhosphopyruvate Hydratasebiology.proteinPLoS ONE
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Lrp4, a Novel Receptor for Dickkopf 1 and Sclerostin, Is Expressed by Osteoblasts and Regulates Bone Growth and Turnover In Vivo

2009

Lrp4 is a multifunctional member of the low density lipoprotein-receptor gene family and a modulator of extracellular cell signaling pathways in development. For example, Lrp4 binds Wise, a secreted Wnt modulator and BMP antagonist. Lrp4 shares structural elements within the extracellular ligand binding domain with Lrp5 and Lrp6, two established Wnt co-receptors with important roles in osteogenesis. Sclerostin is a potent osteocyte secreted inhibitor of bone formation that directly binds Lrp5 and Lrp6 and modulates both BMP and Wnt signaling. The anti-osteogenic effect of sclerostin is thought to be mediated mainly by inhibition of Wnt signaling through Lrp5/6 within osteoblasts. Dickkopf1 …

Genetic Markersmusculoskeletal diseasesmedicine.medical_specialtylcsh:MedicineBiologyBone morphogenetic proteinBone and BonesCell LineMicechemistry.chemical_compoundInternal medicineBiochemistry/Cell Signaling and Trafficking StructuresmedicineAnimalsHumanslcsh:ScienceLDL-Receptor Related ProteinsAdaptor Proteins Signal TransducingGlycoproteinsBone growthBone DevelopmentOsteoblastsMultidisciplinarylcsh:RWnt signaling pathwayLRP6Rheumatology/Bone and Mineral MetabolismLRP5OsteoblastPhenotypemedicine.anatomical_structureEndocrinologyGene Expression RegulationReceptors LDLGenetics and Genomics/Disease ModelschemistryOsteocyteBone Morphogenetic ProteinsIntercellular Signaling Peptides and ProteinsSclerostinlcsh:QSignal TransductionResearch ArticlePLoS ONE
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Genetic Variability of Hepatitis C Virus before and after Combined Therapy of Interferon plus Ribavirin

2008

We present an analysis of the selective forces acting on two hepatitis C virus genome regions previously postulated to be involved in the viral response to combined antiviral therapy. One includes the three hypervariable regions in the envelope E2 glycoprotein, and the other encompasses the PKR binding domain and the V3 domain in the NS5A region. We used a cohort of 22 non-responder patients to combined therapy (interferon alpha-2a plus ribavirin) for which samples were obtained before initiation of therapy and after 6 or/and 12 months of treatment. A range of 25-100 clones per patient, genome region and time sample were sequenced. These were used to detect general patterns of adaptation, t…

Genome evolutionHepatitis C virusEvolutionary Biology/Bioinformaticslcsh:MedicineAlpha interferonGenome ViralHepacivirusBiologyVirology/Immune EvasionInterferon alpha-2Viral Nonstructural Proteinsmedicine.disease_causeGenomeAntiviral AgentsEvolution Molecularchemistry.chemical_compoundGenetics and Genomics/Population GeneticsRibavirinmedicineHumanslcsh:ScienceNS5APhylogenyGenetics:CIENCIAS DE LA VIDA::Genética ::Otras [UNESCO]Virology/Antivirals including Modes of Action and ResistanceMultidisciplinaryEvolutionary Biology/Evolutionary and Comparative GeneticsHepatitis C virusRibavirinlcsh:RGenetic VariationInterferon-alphaVirologyComplementarity Determining RegionsHepatitis CVirology/Virus Evolution and SymbiosisRecombinant ProteinsUNESCO::CIENCIAS DE LA VIDA::Genética ::OtrasHypervariable regionchemistryViral evolutionInterferonlcsh:QGenetic variabilityHepatitis C virus; Genetic variability; Interferon; Ribavirin; Combined therapyCombined therapyResearch ArticlePLoS ONE
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Evidence of Recombination in Intrapatient Populations of Hepatitis C Virus.

2008

Hepatitis C virus (HCV) is a major cause of liver disease worldwide and a potential cause of substantial morbidity and mortality in the future. HCV is characterized by a high level of genetic heterogeneity. Although homologous recombination has been demonstrated in many members of the family Flaviviridae, to which HCV belongs, there are only a few studies reporting recombination on natural populations of HCV, suggesting that these events are rare in vivo. Furthermore, these few studies have focused on recombination between different HCV genotypes/subtypes but there are no reports on the extent of intra-genotype or intra-subtype recombination between viral strains infecting the same patient.…

GenotypeHepatitis C virusHepacivirusPublic Health and Epidemiology/Infectious Diseaseslcsh:MedicineHepacivirusVirology/Immune Evasionmedicine.disease_causeAntiviral AgentsGenetics and Genomics/Population GeneticsGenotypemedicineNS5Alcsh:SciencePhylogenyRecombination GeneticGeneticsLikelihood FunctionsGenomeMultidisciplinaryModels GeneticbiologyGenetic heterogeneitylcsh:RGenetic Variationvirus diseasesRNA virusbiology.organism_classificationGenetics and Genomics/Microbial Evolution and GenomicsVirologyVirology/Virus Evolution and Symbiosislcsh:QComputational Biology/Population GeneticsHomologous recombinationAlgorithmsSoftwareRecombinationResearch Article
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ISWI Regulates Higher-Order Chromatin Structure and Histone H1 Assembly In Vivo

2007

Imitation SWI (ISWI) and other ATP-dependent chromatin-remodeling factors play key roles in transcription and other processes by altering the structure and positioning of nucleosomes. Recent studies have also implicated ISWI in the regulation of higher-order chromatin structure, but its role in this process remains poorly understood. To clarify the role of ISWI in vivo, we examined defects in chromosome structure and gene expression resulting from the loss of Iswi function in Drosophila. Consistent with a broad role in transcriptional regulation, the expression of a large number of genes is altered in Iswi mutant larvae. The expression of a dominant-negative form of ISWI leads to dramatic a…

Imitation SWINucleosome assemblyTranscription GeneticQH301-705.5RNA-POLYMERASE-IIPROTEINCHROMOSOME ARCHITECTUREGeneral Biochemistry Genetics and Molecular BiologyHistones03 medical and health sciencesNUCLEOSOME REMODELING FACTORHigher Order Chromatin StructureHistone H1NucleosomeAnimalsTRANSCRIPTIONBiology (General)LIVING CELLSMolecular Biology030304 developmental biologyGENE-EXPRESSIONRegulation of gene expressionGeneticsAdenosine Triphosphatases0303 health sciencesGeneral Immunology and MicrobiologybiologyGeneral Neuroscience030302 biochemistry & molecular biologyGenetics and GenomicsCell BiologyChromatin Assembly and DisassemblyChromatinChromatinCell biologyDROSOPHILAHistoneGene Expression RegulationLarvaMutationbiology.proteinLINKER HISTONEGeneral Agricultural and Biological SciencesResearch ArticleDevelopmental BiologyTranscription FactorsDOSAGE COMPENSATION
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Phylogeography and genetic variation of Triatoma dimidiata, the main Chagas disease vector in Central America, and its position within the genus Tria…

2008

Background Among Chagas disease triatomine vectors, the largest genus, Triatoma, includes species of high public health interest. Triatoma dimidiata, the main vector throughout Central America and up to Ecuador, presents extensive phenotypic, genotypic, and behavioral diversity in sylvatic, peridomestic and domestic habitats, and non-domiciliated populations acting as reinfestation sources. DNA sequence analyses, phylogenetic reconstruction methods, and genetic variation approaches are combined to investigate the haplotype profiling, genetic polymorphism, phylogeography, and evolutionary trends of T. dimidiata and its closest relatives within Triatoma. This is the largest interpopulational …

Infectious Diseases/Epidemiology and Control of Infectious Diseaseslcsh:Arctic medicine. Tropical medicinelcsh:RC955-962Molecular Sequence DataZoologyPopulation geneticsMolecular Biology/Molecular EvolutionSubspeciesBiologyDNA RibosomalEvolutionary Biology/Animal GeneticsAdaptive radiationGenetics and Genomics/Population Geneticsparasitic diseasesAnimalsChagas DiseaseTriatoma dimidiataTriatomaCladePhylogenyEvolutionary Biology/Evolutionary and Comparative GeneticsEcologylcsh:Public aspects of medicinePublic Health Environmental and Occupational HealthInfectious Diseases/Protozoal InfectionsGenetic VariationCentral Americalcsh:RA1-1270biology.organism_classificationInsect VectorsPhylogeographyInfectious DiseasesInfectious Diseases/Neglected Tropical DiseasesHaplotypesVector (epidemiology)TriatomaResearch Article
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Global Functional Analyses of Cellular Responses to Pore-Forming Toxins

2011

Here we present the first global functional analysis of cellular responses to pore-forming toxins (PFTs). PFTs are uniquely important bacterial virulence factors, comprising the single largest class of bacterial protein toxins and being important for the pathogenesis in humans of many Gram positive and Gram negative bacteria. Their mode of action is deceptively simple, poking holes in the plasma membrane of cells. The scattered studies to date of PFT-host cell interactions indicate a handful of genes are involved in cellular defenses to PFTs. How many genes are involved in cellular defenses against PFTs and how cellular defenses are coordinated are unknown. To address these questions, we pe…

MAPK/ERK pathwayTranscription GeneticImmunology/Innate ImmunityMessengerInteractomeInfectious Diseases/Bacterial InfectionsRNA interference2.1 Biological and endogenous factorsAetiologyBiology (General)Genes HelminthCaenorhabditis elegansOligonucleotide Array Sequence AnalysisGenetics0303 health sciencesGenomebiologyReverse Transcriptase Polymerase Chain ReactionGenetics and Genomics/Functional Genomics030302 biochemistry & molecular biologyrespiratory systemCell biologyInfectious DiseasesMedical MicrobiologyRNA InterferenceSignal transductionDNA microarrayTranscriptionBiotechnologyResearch ArticleSignal TransductionPore Forming Cytotoxic ProteinsQH301-705.5Virulence FactorsMAP Kinase Signaling System1.1 Normal biological development and functioningBacterial ToxinsImmunologyMicrobiologyDNA-binding proteinCell Line03 medical and health sciencesBacterial ProteinsGeneticUnderpinning researchVirologyEscherichia coliHelminthGeneticsAnimalsHumansRNA MessengerCaenorhabditis elegansCaenorhabditis elegans ProteinsMolecular BiologyGene030304 developmental biologyGenome HelminthCell MembraneGenetics and GenomicsRC581-607biology.organism_classificationrespiratory tract diseasesTranscription Factor AP-1Emerging Infectious DiseasesGenesRNAParasitologyGeneric health relevanceRNA HelminthImmunologic diseases. AllergyPLoS Pathogens
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A high incidence of meiotic silencing of unsynapsed chromatin is not associated with substantial pachytene loss in heterozygous male mice carrying mu…

2009

Meiosis is a complex type of cell division that involves homologous chromosome pairing, synapsis, recombination, and segregation. When any of these processes is altered, cellular checkpoints arrest meiosis progression and induce cell elimination. Meiotic impairment is particularly frequent in organisms bearing chromosomal translocations. When chromosomal translocations appear in heterozygosis, the chromosomes involved may not correctly complete synapsis, recombination, and/or segregation, thus promoting the activation of checkpoints that lead to the death of the meiocytes. In mammals and other organisms, the unsynapsed chromosomal regions are subject to a process called meiotic silencing of…

MaleHeterozygoteCancer ResearchDevelopmental Biology/Germ Cellslcsh:QH426-470BiologíaCell Biology/Cell Growth and DivisionChromosomal translocationMeiocyteBiologyTranslocation GeneticMiceMeiosisSpermatocytesGeneticsHomologous chromosomeAnimalsGene SilencingMolecular BiologyMetaphaseGenetics (clinical)Ecology Evolution Behavior and SystematicsGeneticsSex ChromosomesAutosomeSynapsisChromosomeSynapsisChromatinGenetics and Genomics/Chromosome BiologyChromosome PairingMeiosislcsh:GeneticsEvolutionary Biology/Nuclear Structure and FunctionFemalePachytene StageResearch ArticlePLoS Genetics
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