Search results for "Genomic Hybridization"

showing 10 items of 95 documents

Interstitial deletions of chromosome 1p: novel 1p31.3p22.2 microdeletion in a newborn with craniosynostosis, coloboma and cleft palate, and review of…

2022

Abstract Background Rearrangements of unstable DNA sequences may alter the structural integrity or the copy number of dose-sensitive genes, resulting in copy number variations. They may lead more frequently to deletions, in addition to duplications and/or inversions, which are the underlying pathogenic mechanism of a group of conditions known as genomic disorders (or also contiguous gene syndromes). Interstitial deletions of the short arm of chromosome 1 are rare, and only about 30 patients have been reported. Their clinical features are variable, in respect of the extent of the deleted region. They include global developmental delay, central nervous system (CNS) malformations, craniosynost…

Cleft PalateColobomaComparative Genomic HybridizationCraniosynostosesPhenotypeDNA Copy Number VariationsChromosomes Human Pair 1HumansFemaleGenomicsChromosome Deletion1p31.1 deletion syndrome Array-CGH Case report Chromosome 1 Contiguous gene syndrome Chromosome Deletion Chromosomes Human Pair 1 Comparative Genomic Hybridization DNA Copy Number Variations Female Genomics Humans Phenotype Cleft Palate Coloboma Craniosynostoses
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Copy number variation and missense mutations of the agouti signaling protein (ASIP) gene in goat breeds with different coat colors.

2009

In goats, classical genetic studies reported a large number of alleles at the Agouti locus with effects on coat color and pattern distribution. From these early studies, the dominant A(Wt) (white/tan) allele was suggested to cause the white color of the Saanen breed. Here, we sequenced the coding region of the goat ASIP gene in 6 goat breeds (Girgentana, Maltese, Derivata di Siria, Murciano-Granadina, Camosciata delle Alpi, and Saanen), with different coat colors and patterns. Five single nucleotide polymorphisms (SNPs) were identified, 3 of which caused missense mutations in conserved positions of the cysteine-rich carboxy-terminal domain of the protein (p.Ala96Gly, p.Cys126Gly, and p.Val1…

Comparative Genomic HybridizationBase SequenceDNA Copy Number VariationsGoatsCapra hircuCNVMolecular Sequence DataMutation MissenseSNPColorBreedingPolymorphism Single NucleotideCOAT COLORSettore AGR/17 - Zootecnica Generale E Miglioramento GeneticoBREEDSaCGHAgouti locuASIP GENEGOATAgouti Signaling ProteinAnimalsHumansAmino Acid SequenceSequence AlignmentCytogenetic and genome research
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Comparative genomics and proteomics of Helicobacter mustelae, an ulcerogenic and carcinogenic gastric pathogen

2010

Abstract Background Helicobacter mustelae causes gastritis, ulcers and gastric cancer in ferrets and other mustelids. H. mustelae remains the only helicobacter other than H. pylori that causes gastric ulceration and cancer in its natural host. To improve understanding of H. mustelae pathogenesis, and the ulcerogenic and carcinogenic potential of helicobacters in general, we sequenced the H. mustelae genome, and identified 425 expressed proteins in the envelope and cytosolic proteome. Results The H. mustelae genome lacks orthologs of major H. pylori virulence factors including CagA, VacA, BabA, SabA and OipA. However, it encodes ten autotransporter surface proteins, seven of which were detec…

DNA BacterialProteomicslcsh:QH426-470Proteomelcsh:BiotechnologyMolecular Sequence DataVirulenceCarcinogenicHelicobacter mustelaeProteomicsFN555004MicrobiologyUlcerogenic03 medical and health sciencesBacterial ProteinsHelicobacterlcsh:TP248.13-248.65medicineGeneticsCagAHelicobacterAmino Acid SequencePhylogeny030304 developmental biology0303 health sciencesComparative Genomic HybridizationbiologyHelicobacter pyloriVirulence030306 microbiologyCancerGene Expression Regulation BacterialGenomicsSequence Analysis DNAHelicobacter pylorimedicine.diseasebiology.organism_classificationdigestive system diseases3. Good healthlcsh:GeneticsProteomeGastritismedicine.symptomSequence AlignmentH. pyloriGenome BacterialBiotechnologyResearch ArticleBMC Genomics
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Transformation of follicular lymphoma to diffuse large cell lymphoma is associated with a heterogeneous set of DNA copy number and gene expression al…

2002

AbstractGenomic aberrations in a series of paired biopsy samples from patients who presented initially with follicle center lymphoma (FCL) and subsequently transformed to diffuse large B-cell lymphoma (DLBCL) were measured by array comparative genomic hybridization (CGH). The consequences of these aberrations on gene expression were determined by comparison with expression analysis on these specimens using cDNA microarrays. A heterogeneous pattern of acquired genomic abnormalities was observed upon transformation, some of which were recurrent in small subsets of patients. Some of the genomic aberration acquired upon transformation, such as gain/amplification of 1q21-q24, 2p16 (REL/BCL11A ge…

DNA ComplementaryImmunologyFollicular lymphomaLocus (genetics)BiologyAllelic ImbalanceBiochemistryGene duplicationmedicineChromosomes HumanHumansGeneLymphoma FollicularOligonucleotide Array Sequence AnalysisGeneticsChromosome AberrationsGene Expression ProfilingGene AmplificationCell BiologyHematologyDNA Neoplasmmedicine.diseaseBCL6Gene Expression Regulation NeoplasticCell Transformation NeoplasticDisease ProgressionLymphoma Large B-Cell DiffuseDNA microarrayChromosome DeletionDiffuse large B-cell lymphomaComparative genomic hybridizationBlood
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Complete karyotype characterization of the K562 cell line by combined application of G-banding, multiplex-fluorescence in situ hybridization, fluores…

2001

This study combines conventional cytogenetics, fluorescence in situ hybridization (FISH), multiplex-FISH and comparative genomic hybridization (CGH). In applying this multimodal approach on the human leukemia cell line K562, the chromosome composition was refined in detail and compared with data from the literature. A hypotriploid karyotype with a modal chromosome number of 67, and 21 unique marker chromosomes were identified. The classification of six markers was identical to published data and the composition of five further markers from the literature could be fully clarified for the first time. The composition of another five markers, which have been interpreted in divergent ways in dif…

Genetic MarkersCancer Researchmedicine.medical_specialtyG bandingIn situ hybridizationComputational biologyBiologyChromosome PaintingCytogeneticsmedicineHumansIn Situ Hybridization FluorescenceGeneticsmedicine.diagnostic_testCytogeneticsChromosome MappingNucleic Acid HybridizationKaryotypeHematologyModal Chromosome NumberOncologyKaryotypingK562 CellsVirtual karyotypeComparative genomic hybridizationFluorescence in situ hybridizationLeukemia research
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Molecular and clinical characterization of a small duplication Xp in a human female with psychiatric disorders

2011

CGH techniques allow us to detect small duplications thatoccur in humans with phenotypic manifestations and demon-strate the importance of these duplications in the etiologyof neurodevelopmental impairment. As in the case of otherX-linked disorders, X-inactivation plays a major role in theclinical expression of such X chromosomal imbalances withusually milder symptoms in females than in males. Mostmale patients carrying Xp duplication have mental retarda-tion (X-linked mental retardation) and variable facial dys-morphic features (Gimelli

GeneticsChromosomes Human XComparative Genomic HybridizationMental Disordershuman geneticsBiologyPhenotypeHuman geneticspsychiatric disorderfunctional Xp disomySettore MED/38 - Pediatria Generale E SpecialisticaSettore MED/03 - Genetica MedicaX Chromosome InactivationChild PreschoolGene duplicationChromosome DuplicationGeneticsMental Retardation X-LinkedHumansarray CGHFemaleChildfunctional Xp disomy; array CGH; psychiatric disorders; human geneticsGenetic Association StudiesSex Chromosome Aberrations
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Molecular biology approaches utilized in preimplantation genetics: real-time PCR, microarrays, next-generation sequencing, karyomapping, and others

2020

Abstract Over the past few decades the development of new molecular technologies has revolutionized diagnosis in the reproductive medicine field, with the evaluation of the two main factors involved in a successful pregnancy: the embryo and the endometrium. The detection of genetically abnormal embryos, as well as the identification of an optimum endometrium using transcriptomics have become a priority in assisted reproductive treatments to increase pregnancy rates. This chapter provides an overview of the molecular techniques currently employed in assisted reproduction for embryo evaluation such as preimplantation genetic testing karyotyping, fluorescence in situ hybridization, polymerase …

Geneticsmedicine.diagnostic_testBiologyDNA sequencinglaw.inventionTranscriptomeReal-time polymerase chain reactionlawmedicineDNA microarrayPolymerase chain reactionComparative genomic hybridizationFluorescence in situ hybridizationGenetic testing
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Search forReCQL4mutations in 39 patients genotyped for suspected Rothmund-Thomson/Baller-Gerold syndromes

2014

Three overlapping conditions, namely Rothmund-Thomson (RTS), Baller-Gerold (BGS) and RAPADILINO syndromes, have been attributed to RECQL4 mutations. Differential diagnoses depend on the clinical presentation, but the numbers of known genes remain low, leading to the widespread prescription of RECQL4 sequencing. The aim of our study was therefore to determine the best clinical indicators for the presence of RECQL4 mutations in a series of 39 patients referred for RECQL4 molecular analysis and belonging to the RTS (27 cases) and BGS (12 cases) spectrum. One or two deleterious RECQL4 mutations were found in 10/27 patients referred for RTS diagnosis. Clinical and molecular reevaluation led to a…

Geneticsmedicine.medical_specialtybusiness.industryPoikilodermaConsanguinityBaller–Gerold syndromemedicine.diseaseDermatology3. Good healthHereditary sclerosing poikilodermaGenotypeGeneticsmedicinebusinessRothmund–Thomson syndromeGenetics (clinical)Comparative genomic hybridizationPorokeratosisClinical Genetics
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Cardio-facio-cutaneous syndrome and gastrointestinal defects: report on a newborn with 19p13.3 deletion including the MAP 2 K2 gene

2022

Abstract Background Cardio-facio-cutaneous syndrome (CFCS) belongs to RASopathies, a group of conditions caused by mutations in genes encoding proteins of the rat sarcoma/mitogen-activated protein kinase (RAS/MAPK) pathway. It is a rare syndrome, with about 300 patients reported. Main clinical manifestations include facial dysmorphisms, growth failure, heart defects, developmental delay, and ectodermal abnormalities. Mutations (mainly missense) of four genes (BRAF, MAP 2 K1, MAP 2 K2, and KRAS) have been associated to CFCS. However, whole gene deletions/duplications and chromosomal microdeletions have been also reported. Specifically, 19p13.3 deletion including MAP 2 K2 gene are responsible…

Heart Defects CongenitalComparative Genomic HybridizationEctodermal DysplasiaPregnancyCFCS RASopathies Contiguous gene syndrome Array-CGH Genotype-phenotype correlations HPS Case reportFaciesHumansFemaleSyndromeHernia UmbilicalFailure to ThriveItalian Journal of Pediatrics
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Array-CGH and clinical characterization in a patient with subtelomeric 6p deletion without ocular dysgenesis

2011

Subtelomeric terminal 6p deletion has been recognized as a clinically identifiable syndrome including facial dysmorphism, malformation of the anterior eye chamber, hearing loss, heart defect and developmental delay. Genotype –phenotype correlations of previously published patients have been strongly suggested anterior eye segment anomalies as one of major malformation of the syndrome if the critical 6p25 region containing the FOXC 1 gene. In addition it has been hypothesized the presence in this region of one or more genes involved in hearing loss. We report on a case of terminal 6p deletion in a 47, XYY karyotype. Further characterization of the deletion with array comparative genome hybri…

Heart Defects CongenitalMaleHearing lossDevelopmental DisabilitiesKaryotypeBiologyEyeDysgenesisSettore MED/38 - Pediatria Generale E SpecialisticaChromosome 19GeneticsmedicineHumansarray-CGH.Eye AbnormalitiesGeneGenetics (clinical)Genetic Association StudiesIn Situ Hybridization FluorescenceGeneticsComparative Genomic Hybridizationeye abnormalitieInfantKaryotypeForkhead Transcription Factorshearing loSubtelomereAnterior Eye SegmentSettore MED/03 - Genetica MedicaChromosomes Human Pair 6FOXC1medicine.symptomChromosome Deletionchromosome 6p deletionComparative genomic hybridization
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