Search results for "Gliomas"

showing 10 items of 13 documents

10-Year Journey Through Brain Gliomas: Case Report

2020

Abstract We present the case of a 28-year-old doctor who was diagnosed in 2009 with grade II oligodendroglioma. In the next 10 years he underwent 4 surgeries, passing in turn through 3 different types of cerebral gliomas: grade II oligodendroglioma, grade III oligoastrocytoma and IDH Wild-Type glioblastoma. In the spirit of the surgical resection limited by the preservation of the function, he was able to practice his medical profession until the last months of his life. Life expectancy at the time of diagnosis complied with all the statistical data. Although the etiology, pathophysiology and treatment of cerebral gliomas is still full of unknowns, knowing our limits we can provide a good q…

03 medical and health sciencesPathologymedicine.medical_specialty0302 clinical medicinebusiness.industry030220 oncology & carcinogenesisMedicineGeneral Medicinebusinessneoplasms030217 neurology & neurosurgeryBrain gliomasnervous system diseasesActa Medica Transilvanica
researchProduct

Quantitative Imaging of D-2-Hydroxyglutarate in Selected Histological Tissue Areas by a Novel Bioluminescence Technique

2016

Abstract Patients with malignant gliomas have a poor prognosis with average survival of less than one year. Whereas in other tumor entities the characteristics of tumor metabolism are successfully used for therapeutic approaches, such developments are very rare in brain tumors, notably in gliomas. One metabolic feature characteristic of gliomas, in particular diffuse astrocytomas and oligodendroglial tumors, is the variable content of D-2-hydroxyglutarate (D2HG), a metabolite, which was discovered first in this tumor entity. D2HG is generated in large amounts due to various “gain-of–function” mutations in the isocitrate dehydrogenases IDH-1 and IDH-2. Meanwhile, D2HG has been detected in se…

0301 basic medicineCancer ResearchPathologymedicine.medical_specialtyMetabolite610 MedizinBiology03 medical and health scienceschemistry.chemical_compoundmedicineBioluminescence imagingBioluminescenceOligodendroglial TumorOriginal Researchddc:610D-2 hydroxyglutarateglioblastomaMyeloid leukemiaCancerACUTE MYELOID-LEUKEMIA; ISOCITRATE DEHYDROGENASE 1; IDH2 MUTATIONS; CHROMATOGRAPHY/MASS SPECTROMETRY; MAGNETIC-RESONANCE; 2-HYDROXYGLUTARATE; CANCER; GLIOMAS; L-2-HYDROXYGLUTARATE; METABOLITES; D-2 hydroxyglutarate; IDH mutations; bioluminescence imaging; oncometabolite; glioblastomabioluminescence imagingIDH mutationsmedicine.diseaseoncometaboliteLymphoma030104 developmental biologychemistryOncologyChondrosarcoma
researchProduct

Exosomal Chaperones and miRNAs in Gliomagenesis: State-of-Art and Theranostics Perspectives

2018

Gliomas have poor prognosis no matter the treatment applied, remaining an unmet clinical need. As background for a substantial change in this situation, this review will focus on the following points: (i) the steady progress in establishing the role of molecular chaperones in carcinogenesis; (ii) the recent advances in the knowledge of miRNAs in regulating gene expression, including genes involved in carcinogenesis and genes encoding chaperones; and (iii) the findings about exosomes and their cargo released by tumor cells. We would like to trigger a discussion about the involvement of exosomal chaperones and miRNAs in gliomagenesis. Chaperones may be either targets for therapy, due to their…

0301 basic medicineMolecular ChaperoneCellReviewmedicine.disease_causelcsh:ChemistryGene expressiontheranostic toolslcsh:QH301-705.5SpectroscopyChaperone GeneSettore MED/27 - Neurochirurgiamolecular chaperonesGliomaGeneral MedicineHsp60Extracellular MatrixComputer Science ApplicationsCell Transformation Neoplasticmedicine.anatomical_structuregliomas; molecular chaperones; Hsps (Heat shock proteins); Hsp60; miRNA; exosomes; extracellular vesicles; theranostic toolsextracellular vesiclesHumanexosomesBiologyCatalysisInorganic Chemistry03 medical and health sciencesGliomamicroRNAmedicineAnimalsHumansHsps (Heat shock proteins)Physical and Theoretical ChemistryMolecular BiologyGenemiRNAAnimalSettore BIO/16 - Anatomia UmanaOrganic ChemistryBiological Transportmedicine.diseaseMicrovesiclesExosomegliomasMicroRNAs030104 developmental biologylcsh:Biology (General)lcsh:QD1-999Cancer researchextracellular vesicleTheranostic toolCarcinogenesisInternational Journal of Molecular Sciences
researchProduct

Establishment and Preliminary Characterization of Three Astrocytic Cells Lines Obtained from Primary Rat Astrocytes by Sub-Cloning.

2020

Gliomas are complex and heterogeneous tumors that originate from the glial cells of the brain. The malignant cells undergo deep modifications of their metabolism, and acquire the capacity to invade the brain parenchyma and to induce epigenetic modifications in the other brain cell types. In spite of the efforts made to define the pathology at the molecular level, and to set novel approaches to reach the infiltrating cells, gliomas are still fatal. In order to gain a better knowledge of the cellular events that accompany astrocyte transformation, we developed three increasingly transformed astrocyte cell lines, starting from primary rat cortical astrocytes, and analyzed them at the cytogenet…

0301 basic medicinelcsh:QH426-470Somatic cellPrimary Cell CultureArticle03 medical and health sciencesCytogenetics0302 clinical medicineGliomaSettore BIO/10 - BiochimicaParenchymaGeneticsmedicineAnimalsEpigeneticsSettore BIO/06 - Anatomia Comparata E CitologiaGenetics (clinical)Cell Line TransformedCloningbiologymedicine.diseaseCell biologyClone CellsRatsgliomaslinker histone H1.0lcsh:GeneticsSettore BIO/18 - Geneticaastrocyte cell lines030104 developmental biologymedicine.anatomical_structureHistoneepigenetic alterationsCell culture030220 oncology & carcinogenesisAstrocytesbiology.proteinAstrocyteGenes
researchProduct

Brain Mapping-Aided SupraTotal Resection (SpTR) of Brain Tumors: The Role of Brain Connectivity

2021

Brain gliomas require a deep knowledge of their effects on brain connectivity. Understanding the complex relationship between tumor and functional brain is the preliminary and fundamental step for the subsequent surgery. The extent of resection (EOR) is an independent variable of surgical effectiveness and it correlates with the overall survival. Until now, great efforts have been made to achieve gross total resection (GTR) as the standard of care of brain tumor patients. However, high and low-grade gliomas have an infiltrative behavior and peritumoral white matter is often infiltrated by tumoral cells. According to these evidences, many efforts have been made to push the boundary of the re…

Cancer ResearchConnectomicsmedicine.medical_specialtyMini Reviewlow-grade gliomasBrain tumorbrain connectomelcsh:RC254-282Brain mappingResectionLesionWhite matterNeuroimagingmedicineBrain connectome Supratotal resection Brain mapping Brain tumor Connectomics Extent of resection High-grade gliomas Low-grade GliomasconnectomicsBrain GliomaSettore MED/27 - Neurochirurgiabusiness.industryAstrocytomalcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensmedicine.diseaseextent of resectionmedicine.anatomical_structureOncologySuprtotal resectionbrain mappingRadiologymedicine.symptomsupratotal resectionbusinesshigh-grade gliomasbrain tumorMRI
researchProduct

HGG-03. THE GLYCOSPHINGOLIPIDS METABOLISM IS A NOVEL TARGET IN H3K27M-MUTANT DIFFUSE MIDLINE GLIOMA

2021

Abstract H3K27M-mutant diffuse midline glioma (H3K27M-mutant DMG) is a rare malignant brain tumour entity in children and adults with a median overall survival of around 12 months. Genomic and proteomic analysis may help to identify new target structures, however not all relevant targets are covered by these analyses. Glycosphingolipids and particularly gangliosides play a major role in brain development and have been involved in the pathology of brain tumours. The conversion of ceramide to glucosylceramide via glucosylceramide synthase (GCS) is one of the first steps in the synthesis of glycosphingolipids. Therefore, targeting GCS will inhibit their synthesis. Here we analysed the glycosph…

Cancer ResearchMutationBrain developmentmedicine.diagnostic_testChemistryCell growthMutantMetabolismEliglustat tartratemedicine.diseasemedicine.disease_causeFlow cytometryOncologyGliomamedicineCancer researchAcademicSubjects/MED00300AcademicSubjects/MED00310High Grade GliomasNeurology (clinical)Neuro-Oncology
researchProduct

P08.76 Anti-EGFL7 treatment as an add-on for glioma therapy

2016

Glioblastoma multiforme (GBM) is one the most common and malignant forms of brain tumors. The median survival time of patients diagnosed with primary GBM is around 15 months. In spite of multimodal treatments GBMs remain essentially incurable. Therefore, alternative treatments targeting previously unexplored aspects of GBMs are required. However, the molecular mechanisms underlying the formation of brain tumors have only partially been defined. Especially Notch, a conserved developmental pathway, is promising in terms of GBM formation, as components of this signaling cascade are aberrantly expressed in gliomas. Studies reported that the inhibition of Notch decreased glioma cell proliferatio…

Cancer ResearchText miningOncologybusiness.industryP08 Glioblastom and Anaplastic gliomasGliomaCancer researchMedicineEGFL7Neurology (clinical)businessmedicine.diseasenervous system diseases
researchProduct

Prognostic Implications of the Complement Protein C1q in Gliomas

2019

The contribution of the complement system in the pathophysiology of brain cancers has been recently considered in light of its well-known involvement in carcinogenesis. Complement system represents an important component of the inflammatory response, which acts as a functional bridge between the innate and adaptive immune response. C1q, the first recognition subcomponent of the complement classical pathway, has recently been shown to be involved in a range of pathophysiological functions that are not dependent on complement activation. C1q is expressed in the microenvironment of various types of human tumors, including melanoma, prostate, mesothelioma, and ovarian cancers, where it can exer…

Male0301 basic medicinemedicine.disease_causePathogenesisbioinformatics analysis; C1q complement; gliomas; prognostic significance of C1q; survival probability0302 clinical medicinegliomaTumor MicroenvironmentImmunology and AllergyComplement C1qbioinformatics analysiOriginal ResearchSettore MED/27 - NeurochirurgiaBrain NeoplasmsMelanomaBioinformatics analysiGliomaPrognosisAcquired immune systemNeoplasm ProteinsGene Expression Regulation NeoplasticBioinformatics analysisFemalePrognostic significance of C1q.Databases Nucleic Acidlcsh:Immunologic diseases. Allergybioinformatics analysisImmunologyprognostic significance of C1qBiology03 medical and health sciencesClassical complement pathwayC1q complementGliomaBiomarkers TumormedicineHumanssurvival probabilitySurvival probabilityGliomasC1q complementComplement C1qmedicine.diseaseComplement systemgliomas030104 developmental biologyCancer researchlcsh:RC581-607Carcinogenesis030215 immunologyFrontiers in Immunology
researchProduct

With a Little Help from My Friends: The Role of Intraoperative Fluorescent Dyes in the Surgical Management of High-Grade Gliomas

2018

High-grade gliomas (HGGs) are the most frequent primary malignant brain tumors in adults, which lead to death within two years of diagnosis. Maximal safe resection of malignant gliomas as the first step of multimodal therapy is an accepted goal in malignant glioma surgery. Gross total resection has an important role in improving overall survival (OS) and progression-free survival (PFS), but identification of tumor borders is particularly difficult in HGGS. For this reason, imaging adjuncts, such as 5-aminolevulinic acid (5-ALA) or fluorescein sodium (FS) have been proposed as superior strategies for better defining the limits of surgical resection for HGG. 5-aminolevulinic acid (5-ALA) is i…

Oncologymedicine.medical_specialtyReviewlcsh:RC321-571Resection03 medical and health scienceschemistry.chemical_compoundHigh-grade glioma0302 clinical medicineGliomaInternal medicineYELLOW 560 filterMedicinefluorescein sodiumastrocytomalcsh:Neurosciences. Biological psychiatry. NeuropsychiatryProtoporphyrin IXSettore MED/27 - Neurochirurgiabusiness.industryGeneral NeuroscienceglioblastomaAstrocytomaMultimodal therapymedicine.diseaseFluorescenceextent of resectionchemistry5-aminolevulinic acid030220 oncology & carcinogenesisPrimary Malignant Brain TumorsSodium fluoresceinbusinesshigh-grade gliomas030217 neurology & neurosurgeryBrain Sciences
researchProduct

Chaperonology: The Third Eye on Brain Gliomas

2018

The European Organization for Research and Treatment of Cancer/National Cancer Institute of Canada Phase III trial has validated as a current regimen for high-grade gliomas (HGG) a maximal safe surgical resection followed by radiotherapy with concurrent temozolamide. However, it is essential to balance maximal tumor resection with preservation of the patient&rsquo

Oncologymedicine.medical_specialtymedicine.medical_treatmentArticlelcsh:RC321-571Third eye03 medical and health sciences0302 clinical medicineHigh-grade gliomaInternal medicinemedicineSurvival ratelcsh:Neurosciences. Biological psychiatry. NeuropsychiatrychaperonotherapychaperonologyNeuroscience (all)neuroimagingHeat shock proteinbusiness.industrySettore BIO/16 - Anatomia UmanaSettore MED/27 - NeurochirurgiaGeneral NeuroscienceChaperonology; Chaperonotherapy; Heat shock proteins; High-grade gliomas; Molecular chaperones; Neuroimaging; Neuromonitoring; Neuroscience (all)molecular chaperonesCancermedicine.diseaseBrain gliomas3. Good healthBrain diseaseNatural historyRadiation therapyRegimen030220 oncology & carcinogenesisheat shock proteinsMolecular chaperonebusinesshigh-grade gliomas030217 neurology & neurosurgeryneuromonitoringBrain Sciences
researchProduct