Search results for "Glucose Metabolism"

showing 10 items of 45 documents

Reduction in insulin sensitivity and inadequate β-cell capacity to counteract the increase in insulin resistance in children with idiopathic growth h…

2014

Purpose: To evaluate the performance of various indexes of insulin sensitivity and secretion and to identify the most useful indicator of deterioration of glucose metabolism in a cohort of children with growth hormone (GH) deficiency (GHD) during GH treatment. Methods: In 73 GHD children (55 M, 18 F; mean age 10.5 years) at baseline and after 12 months of treatment, we evaluated a number of surrogate indexes of insulin secretion and sensitivity. In a subgroup of 11 children we also performed an euglycemic hyperinsulinemic clamp. Results: After 12 months, a significant increase in fasting glucose (p < 0.001) and HbA1c levels (p < 0.001) was documented, despite all children remained with a no…

Blood GlucoseMalemedicine.medical_specialtyAdolescentHormone Replacement TherapyEndocrinology Diabetes and Metabolismmedicine.medical_treatmentChildren; Glucose metabolism; Growth hormone treatment; Insulin secretion; Insulin sensitivity; Endocrinology Diabetes and Metabolism; EndocrinologyHypopituitarismCarbohydrate metabolismHypopituitarismSettore MED/13 - EndocrinologiaEndocrinologyInsulin resistanceInsulin-Secreting CellsInternal medicinemedicineHormone replacement therapy (male-to-female)HumansInsulinGrowth hormone treatmentChildDwarfism PituitaryChildrenGlucose metabolismGlucose tolerance testmedicine.diagnostic_testHuman Growth Hormonebusiness.industryInsulin secretionInsulinGlucose Tolerance TestInsulin sensitivitymedicine.diseaseGrowth hormone treatmentTreatment OutcomeEndocrinologyChild PreschoolConcomitantFemaleInsulin Resistancebusiness
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p63 Isoforms Regulate Metabolism of Cancer Stem Cells

2014

p63 is an important regulator of epithelial development expressed in different variants containing (TA) or lacking (ΔN) the N-terminal transactivation domain. The different isoforms regulate stem-cell renewal and differentiation as well as cell senescence. Several studies indicate that p63 isoforms also play a role in cancer development; however, very little is known about the role played by p63 in regulating the cancer stem phenotype. Here we investigate the cellular signals regulated by TAp63 and ΔNp63 in a model of epithelial cancer stem cells. To this end, we used colon cancer stem cells, overexpressing either TAp63 or ΔNp63 isoforms, to carry out a proteomic study by chemical-labeling …

Gene isoformProteomicsProteomeRegulatorBiologyProteomicsBiochemistryTransactivationCancer stem cellmedicineHumansMetabolomicsProtein IsoformsProtein Interaction MapsSettore BIO/10 - BIOCHIMICAp63 colon cancer stem cells proteomics stable isotope dimethyl labeling glucose metabolismSettore BIO/12Tumor Suppressor ProteinsCancerGeneral Chemistrymedicine.diseasePhenotypePeptide FragmentsCell biologyIsotope LabelingNeoplastic Stem CellsStem cellSignal TransductionTranscription Factors
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Genomic and Metabolomic Profile Associated to Clustering of Cardio-Metabolic Risk Factors

2016

Background To identify metabolomic and genomic markers associated with the presence of clustering of cardiometabolic risk factors (CMRFs) from a general population. Methods and Findings One thousand five hundred and two subjects, Caucasian, > 18 years, representative of the general population, were included. Blood pressure measurement, anthropometric parameters and metabolic markers were measured. Subjects were grouped according the number of CMRFs (Group 1: <2; Group 2: 2; Group 3: 3 or more CMRFs). Using SNPlex, 1251 SNPs potentially associated to clustering of three or more CMRFs were analyzed. Serum metabolomic profile was assessed by 1H NMR spectra using a Brucker Advance DRX 600 spect…

Male0301 basic medicineHeredityPhysiologylcsh:MedicinePhysiologyBlood Pressure030204 cardiovascular system & hematologyBioinformaticsBiochemistry0302 clinical medicineGlucose MetabolismRisk FactorsPolymorphism (computer science)GenotypeMedicine and Health SciencesMetaboliteslcsh:Scienceeducation.field_of_studyMultidisciplinaryFatty AcidsGenomicsMiddle AgedLipidsGenetic MappingPhysiological ParametersCardiovascular DiseasesPhysical SciencesCarbohydrate MetabolismResearch ArticleAdultGenetic MarkersGenotypePermutationFADS2PopulationVariant GenotypesSingle-nucleotide polymorphismBiologyPolymorphism Single Nucleotide03 medical and health sciencesMetabolomicsMetabolic DiseasesGeneticsmedicineHumansMetabolomicsGenetic Predisposition to DiseaseObesityeducationGenotypingAgedDiscrete Mathematicslcsh:RBody WeightBiology and Life SciencesLipid Metabolismmedicine.diseaseObesityMetabolism030104 developmental biologyCombinatoricslcsh:QMathematicsPLOS ONE
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The desert gerbil Psammomys obesus as a model for metformin-sensitive nutritional type 2 diabetes to protect hepatocellular metabolic damage: Impact …

2017

Introduction While metformin (MET) is the most widely prescribed antidiabetic drug worldwide, its beneficial effects in Psammomys obesus (P. obesus), a rodent model that mimics most of the metabolic features of human diabetes, have not been explored thoroughly. Here, we sought to investigate whether MET might improve insulin sensitivity, glucose homeostasis, lipid profile as well as cellular redox and energy balance in P. obesus maintained on a high energy diet (HED). Materials and methods P. obesus gerbils were randomly assigned to receive either a natural diet (ND) consisting of halophytic plants (control group) or a HED (diabetic group) for a period of 24 weeks. MET (50 mg/kg per os) was…

Male0301 basic medicinePhysiologymedicine.medical_treatment[SDV]Life Sciences [q-bio]Body-WeightRespiratory chainlcsh:MedicineMitochondria LiverBiochemistrychemistry.chemical_compoundLiver Parenchymal-CellsEndocrinologyGlucose MetabolismAnimal CellsKetogenesisMedicine and Health SciencesElectrochemistryGlucose homeostasisGut Microbiotalcsh:ScienceEnergy-Producing OrganellesComputingMilieux_MISCELLANEOUS2. Zero hungerMultidisciplinaryOrganic CompoundsMonosaccharidesFatty AcidsChemical ReactionsLipidsMetforminMitochondria3. Good healthChemistryPhysiological ParametersLiverPhysical SciencesCarbohydrate MetabolismCellular Structures and OrganellesCellular TypesAnatomyOxidation-ReductionResearch Articlemedicine.medical_specialtyIsolated Rat HepatocytesEndocrine DisordersCarbohydratesBioenergeticsBiologyCarbohydrate metabolism03 medical and health sciencesInsulin resistanceInternal medicineFood-IntakeDiabetes MellitusmedicineAnimalsHypoglycemic AgentsObesityRespiratory-Chain[ SDV ] Life Sciences [q-bio]Fatty acid metabolismInsulinBody WeightOrganic Chemistrylcsh:RChemical CompoundsGluconeogenesisBiology and Life SciencesCell Biologymedicine.diseaseGlucose-6-Phosphate HydrolysisDisease Models AnimalGlucoseMetabolism030104 developmental biologyEndocrinologyDiabetes Mellitus Type 2GluconeogenesischemistryMetabolic DisordersHepatocyteslcsh:QInsulin ResistanceGerbillinaeGlucose-ProductionFatty-Acid-MetabolismOxidation-Reduction Reactions
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Glucagon-like peptide-2 treatment improves glucose dysmetabolism in mice fed a high fat diet

2016

Previous studies suggested that endogenous glucagon-like peptide 2 (GLP-2) is dispensable for the regulation of glucose homeostasis under normal conditions, while it can play a beneficial role in obesity conditions. The purpose of the present study was to investigate whether chronic treatment with Gly2-GLP-2, a stable analogue of GLP-2, can have an impact on glycaemic and lipid control in mice fed a high-fat diet (HFD), an animal model of human obesity and insulin resistance. HFD mice were treated once a day with Gly2-GLP-2 for 4 weeks. Body weight, food intake, fasting glucose, intraperitoneal glucose tolerance, insulin-induced glucose clearance, glucose-stimulated insulin secretion, β-cel…

Male0301 basic medicinemedicine.medical_specialtyEndocrinology Diabetes and MetabolismDrug Evaluation PreclinicalMicrovesicular SteatosisCarbohydrate metabolismDiet High-FatSettore BIO/09 - FisiologiaRandom Allocation03 medical and health sciencesEndocrinologyInsulin resistanceInternal medicineDiabetes mellitusGlucagon-Like Peptide 2medicineAnimalsGlucose homeostasisObesityPancreasPancreatic islets.Glucose Metabolism Disordersbusiness.industrydigestive oral and skin physiologyInsulin resistancemedicine.diseaseGlucagon-like peptide-2LipidsObesityMice Inbred C57BL030104 developmental biologyEndocrinologyLiverPeptidesbusinessGLP-2Dyslipidemia
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Common variants of the liver fatty acid binding protein gene influence the risk of type 2 diabetes and insulin resistance in Spanish population.

2012

Journal Article; Research Support, Non-U.S. Gov't; SUMMARY The main objective was to evaluate the association between SNPs and haplotypes of the FABP1-4 genes and type 2 diabetes, as well as its interaction with fat intake, in one general Spanish population. The association was replicated in a second population in which HOMA index was also evaluated. METHODS 1217 unrelated individuals were selected from a population-based study [Hortega study: 605 women; mean age 54 y; 7.8% with type 2 diabetes]. The replication population included 805 subjects from Segovia, a neighboring region of Spain (446 females; mean age 52 y; 10.3% with type 2 diabetes). DM2 mellitus was defined in a similar way in b…

MaleAnatomy and PhysiologyEspañaDiabetes Mellitus Tipo 2:Named Groups::Persons::Age Groups::Adult::Middle Aged [Medical Subject Headings]Type 2 diabetesResistencia a la InsulinaVariación Genética:Analytical Diagnostic and Therapeutic Techniques and Equipment::Investigative Techniques::Epidemiologic Methods::Statistics as Topic::Probability::Risk::Risk Factors [Medical Subject Headings]:Phenomena and Processes::Genetic Phenomena::Genotype::Haplotypes [Medical Subject Headings]:Organisms::Eukaryota::Animals::Chordata::Vertebrates::Mammals::Primates::Haplorhini::Catarrhini::Hominidae::Humans [Medical Subject Headings]EndocrinologyPolymorphism (computer science)Risk FactorsAnálisis de RegresiónFactores de Riesgo:Geographicals::Geographic Locations::Europe::Spain [Medical Subject Headings]Geneticseducation.field_of_studyMultidisciplinaryAdultoQRMiddle AgedCardiovascular Diseases:Diseases::Nutritional and Metabolic Diseases::Metabolic Diseases::Glucose Metabolism Disorders::Diabetes Mellitus::Diabetes Mellitus Type 2 [Medical Subject Headings]Regression AnalysisMedicineFemale:Phenomena and Processes::Genetic Phenomena::Genotype [Medical Subject Headings]:Phenomena and Processes::Genetic Phenomena::Genetic Variation [Medical Subject Headings]Fatty Acid Binding Protein 3Research ArticleAdult:Phenomena and Processes::Genetic Phenomena::Genetic Variation::Mutation [Medical Subject Headings]GenotypeSciencePopulation:Check Tags::Male [Medical Subject Headings]Single-nucleotide polymorphismEndocrine SystemBiologyFatty Acid-Binding ProteinsPolymorphism Single Nucleotide:Diseases::Nutritional and Metabolic Diseases::Metabolic Diseases::Glucose Metabolism Disorders::Hyperinsulinism::Insulin Resistance [Medical Subject Headings]Insulin resistanceGenetic variation:Named Groups::Persons::Age Groups::Adult [Medical Subject Headings]medicineGeneticsHumans:Named Groups::Persons::Age Groups::Adult::Aged [Medical Subject Headings]education:Diseases::Cardiovascular Diseases [Medical Subject Headings]Biology:Phenomena and Processes::Genetic Phenomena::Genetic Structures::Genome::Genome Components::Genes::Alleles [Medical Subject Headings]AllelesAgedDiabetic Endocrinology:Analytical Diagnostic and Therapeutic Techniques and Equipment::Investigative Techniques::Epidemiologic Methods::Statistics as Topic::Regression Analysis [Medical Subject Headings]:Phenomena and Processes::Genetic Phenomena::Genetic Variation::Polymorphism Genetic::Polymorphism Single Nucleotide [Medical Subject Headings]Polymorphism GeneticEndocrine PhysiologyHaplotypeGenetic Variation:Phenomena and Processes::Genetic Phenomena::Genetic Variation::Polymorphism Genetic [Medical Subject Headings]medicine.diseaseObesity:Chemicals and Drugs::Amino Acids Peptides and Proteins::Proteins::Carrier Proteins::Fatty Acid-Binding Proteins [Medical Subject Headings]:Check Tags::Female [Medical Subject Headings]Diabetes Mellitus Type 2HaplotypesSpainMetabolic DisordersMutationInsulin ResistancePopulation GeneticsPloS one
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Effect of aerobic exercise and low carbohydrate diet on pre-diabetic non-alcoholic fatty liver disease in postmenopausal women and middle aged men - …

2014

Background Pre-diabetes and non-alcoholic fatty liver disease (NAFLD) are associated with an unhealthy lifestyle and pose extremely high costs to the healthcare system. In this study, we aim to explore whether individualized aerobic exercise (AEx) and low carbohydrate diet (LCh) intervention affect hepatic fat content (HFC) in pre-diabetes via modification of gut microbiota composition and other post-interventional effects. Methods/design A 6-month randomized intervention with 6-month follow-up is conducted from January 2013 to December 2015. The target sample size for intervention is 200 postmenopausal women and middle-aged men aged 50–65 year-old with pre-diabetes and NAFLD. The qualified…

MaleHealth BehaviorPATHOGENESISPhysiologyGut floralaw.inventionImpaired glucose toleranceDiet Carbohydrate-RestrictedStudy ProtocolRandomized controlled trialNon-alcoholic Fatty Liver DiseaselawSurveys and QuestionnairesEPIDEMIOLOGYGlucose metabolismbiologyMicrobiotaFatty liverMiddle AgedPostmenopauseLiverResearch DesignMetabonomicsOBESITYBody CompositionFemaleLIFE-STYLELiver fat contentHumanmedicine.medical_specialtyGut microbiotaCarbohydrate metabolismCHINAPrediabetic StateIMPAIRED GLUCOSE-TOLERANCEDiabetes mellitusInternal medicineDietary CarbohydratesmedicineHumansAerobic exerciseExerciseLife StyleAgedbusiness.industryPublic Health Environmental and Occupational HealthDIABETES-MELLITUSFeeding BehaviorADULTSmedicine.diseasebiology.organism_classificationObesityGastrointestinal TractClinical settingMICEEndocrinologyLipid metabolismDietary SupplementsbusinessBMC Public Health
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Co-localisation of hypoxia and perfusion markers with parameters of glucose metabolism in human squamous cell carcinoma (hSCC) xenografts

2009

Purpose: To examine relationships between tumour hypoxia, perfusion and metabolic microenvironment at themicroregional level in three different human squamous cell carcinomas (hSCC). Materials and methods: Nude mice bearing FaDu, UT-SCC-15, and UT-SCC-5 hSCC were injected with pimonidazole hypoxia and Hoechst perfusion markers. Bioluminescence imaging was used to determine spatial distribution of glucose and lactate content in serial tumour sections. Metabolite levels were grouped in 10 concentration ranges. Images were co-registered and at each concentration range the proportion of area stained for pimonidazole and Hoechst was determinedin 11–13 tumours per tumour line. Results: The spatia…

MaleRadiation-Sensitizing AgentsPathologymedicine.medical_specialtyMetaboliteglucose metabolismTransplantation HeterologousCellMice NudeBiologyCarbohydrate metabolismperfusionbiological imagingMicechemistry.chemical_compoundhuman tumour xenograftsCell Line TumorBiomarkers TumormedicineCo localisationAnimalsHumansPimonidazoleBioluminescence imagingRadiology Nuclear Medicine and imagingLactic AcidHypoxiatumour micromilieuFluorescent DyesRadiological and Ultrasound TechnologyHypoxia (medical)Glucosemedicine.anatomical_structureMicroscopy Fluorescencechemistrypimonidazole hypoxiaNitroimidazolesCarcinoma Squamous CellBenzimidazolesFemalemedicine.symptomPerfusionNeoplasm Transplantation
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Glycemic Dysregulations Are Associated With Worsening Cognitive Function in Older Participants at High Risk of Cardiovascular Disease: Two-Year Follo…

2021

The authors wish to thank the PREDIMED-Plus participants and staff for their engagement, as well as to the primary care centers involved in the study. We also thank the Cerca Programme of the Generalitat de Catalunya, and the CIBEROBN, CIBERESP and CIBERDEM initiatives of Instituto de Salud Carlos III in Spain, and the collaborators in the PRIME consortium for helpful input. The first version of the present article has been published in the public repository Research Square as a preprint publication of our work (44). We particularly thank Stephanie Nishi for her assistance with manuscript language revision.

Malemedicine.medical_specialtyCognition disordersEndocrinology Diabetes and Metabolism030209 endocrinology & metabolismGlycemic ControlprediabetesType 2 diabetesdiabetes durationTrastorns de la cognicióDiseases of the endocrine glands. Clinical endocrinology03 medical and health scienceschemistry.chemical_compoundCognitionEndocrinology0302 clinical medicineInsulin resistanceinsulin resistanceDiabetes mellitusInternal medicinemedicineHumansHypoglycemic AgentsCognitive DysfunctionProspective StudiesPrediabetesglycated (glycosylated) hemoglobinCognitive declinecognitive functionAgedGlucose Metabolism DisordersOriginal ResearchGlycemicDiabetisbusiness.industryMalalties cardiovascularsDiabetesMiddle AgedRC648-665medicine.disease3. Good healthCardiovascular diseaseschemistryFemaletype 2 diabetesGlycated hemoglobinMetabolic syndromebusiness030217 neurology & neurosurgery
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The Low Energy-Coupling Respiration in Zymomonas mobilis Accelerates Flux in the Entner-Doudoroff Pathway.

2015

Performing oxidative phosphorylation is the primary role of respiratory chain both in bacteria and eukaryotes. Yet, the branched respiratory chains of prokaryotes contain alternative, low energy-coupling electron pathways, which serve for functions other than oxidative ATP generation (like those of respiratory protection, adaptation to low-oxygen media, redox balancing, etc.), some of which are still poorly understood. We here demonstrate that withdrawal of reducing equivalents by the energetically uncoupled respiratory chain of the bacterium Zymomonas mobilis accelerates its fermentative catabolism, increasing the glucose consumption rate. This is in contrast to what has been observed in o…

Metabolic Processes0301 basic medicineRespiratory chainlcsh:MedicineBiochemistryOxidative PhosphorylationGlucose Metabolismlcsh:ScienceZymomonasMultidisciplinarybiologyOrganic CompoundsSimulation and ModelingMonosaccharidesChemical ReactionsCatabolismAerobiosisEnzymesChemistryBiochemistryPhysical SciencesCarbohydrate MetabolismOxidoreductasesOxidation-ReductionResearch Article030106 microbiologyCarbohydratesAcetaldehydeOxidative phosphorylationResearch and Analysis MethodsZymomonas mobilisElectron Transport03 medical and health sciencesOxidationEntner–Doudoroff pathwayDehydrogenasesOrganic Chemistrylcsh:RChemical CompoundsBiology and Life SciencesProteinsNADbiology.organism_classificationElectron transport chainKineticsGlucoseMetabolismFermentationEnzymologyFermentationlcsh:QFlux (metabolism)BacteriaPLoS ONE
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