Search results for "Growth factor"

showing 10 items of 1300 documents

Regulated segregation of kinase Dyrk1A during asymmetric neural stem cell division is critical for EGFR-mediated biased signaling.

2010

SummaryStem cell division can result in two sibling cells exhibiting differential mitogenic and self-renewing potential. Here, we present evidence that the dual-specificity kinase Dyrk1A is part of a molecular pathway involved in the regulation of biased epidermal growth factor receptor (EGFR) signaling in the progeny of dividing neural stem cells (NSC) of the adult subependymal zone (SEZ). We show that EGFR asymmetry requires regulated sorting and that a normal Dyrk1a dosage is required to sustain EGFR in the two daughters of a symmetrically dividing progenitor. Dyrk1A is symmetrically or asymmetrically distributed during mitosis, and biochemical analyses indicate that it prevents endocyto…

Cell divisionMitosisProtein Serine-Threonine KinasesMiceNeural Stem CellsCell MovementGeneticsSubependymal zoneAnimalsHumansEpidermal growth factor receptorPhosphorylationMitosisProgenitorAdaptor Proteins Signal TransducingbiologyProtein StabilityIntracellular Signaling Peptides and ProteinsMembrane ProteinsCell BiologyProtein-Tyrosine KinasesSTEMCELLNeural stem cellCell biologyErbB ReceptorsStem cell divisionCancer researchbiology.proteinMolecular MedicineSignal transductionCell DivisionSignal TransductionCell stem cell
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Vascular niche factor PEDF modulates Notch-dependent stemness in the adult subependymal zone.

2009

We sought to address the fundamental question of how stem cell microenvironments can regulate self-renewal. We found that Notch was active in astroglia-like neural stem cells (NSCs), but not in transit-amplifying progenitors of the murine subependymal zone, and that the level of Notch transcriptional activity correlated with self-renewal and multipotency. Moreover, dividing NSCs appeared to balance renewal with commitment via controlled segregation of Notch activity, leading to biased expression of known (Hes1) and previously unknown (Egfr) Notch target genes in daughter cells. Pigment epithelium-derived factor (PEDF) enhanced Notch-dependent transcription in cells with low Notch signaling,…

Cell divisionTranscription GeneticNotch signaling pathwayGene ExpressionBiologyMicePEDFEpendymaSubependymal zoneBasic Helix-Loop-Helix Transcription FactorsAnimalsNuclear Receptor Co-Repressor 1Nerve Growth FactorsProgenitor cellHES1Receptor Notch1Eye ProteinsCells CulturedSerpinsHomeodomain ProteinsNeuronsTranscription Factor HES-1General NeuroscienceAge FactorsTranscription Factor RelACell DifferentiationNeural stem cellErbB ReceptorsAdult Stem CellsTranscription Factor HES-1NeuroscienceSignal TransductionNature neuroscience
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Synthesis of insulin and its effects in Y79 human retinoblastoma cells

1994

This paper demonstrates that Y79 human retinoblastoma cells contain immunoreactive insulin (IRI) and release it in the medium. Cells cultured either in suspension or in monolayer showed a similar content of IRI. Moreover, in both conditions, IRI concentration was higher in cells cultured in serum-supplemented medium rather than in serum-free medium. Retinoblastoma cells are capable of synthesizing insulin. This was demonstrated by incubating Y79 cells with [3H]leucine. The synthesized radioactive insulin was separated and assayed by means of a HPLC procedure described in this paper. Both cell growth and [3H]thymidine and [3H]uridine incorporation into acid-insoluble fraction was reduced (-7…

Cell divisionmedicine.medical_treatmentBiologyCellular and Molecular Neurosciencechemistry.chemical_compoundLeucinemedicineTumor Cells CulturedHumansInsulinRNA NeoplasmCycloheximideInsulin-Like Growth Factor IChromatography High Pressure LiquidCell growthInsulinGrowth factorEye NeoplasmsRetinoblastomaDNA Neoplasmretinoblastoma cellsSensory SystemsUridineIn vitroOphthalmologychemistryBiochemistryCell cultureThymidineCell Division
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Human Hsp10 and Early Pregnancy Factor (EPF) and their relationship and involvement in cancer and immunity: current knowledge and perspectives.

2009

This article is about Hsp10 and its intracellular and extracellular forms focusing on the relationship of the latter with Early Pregnancy Factor and on their roles in cancer and immunity. Cellular physiology and survival are finely regulated and depend on the correct functioning of the entire set of proteins. Misfolded or unfolded proteins can cause deleterious effects and even cell death. The chaperonins Hsp10 and Hsp60 act together inside the mitochondria to assist protein folding. Recent studies demonstrated that these proteins have other roles inside and outside the cell, either together or independently of each other. For example, Hsp10 was found increased in the cytosol of different t…

Cell physiologyHsp10 tumor immunity chaperonins early pregnancy factor developmentProgrammed cell deathProtein Foldingmedicine.medical_treatmentBiologyPregnancy ProteinsGeneral Biochemistry Genetics and Molecular BiologyAutoimmune DiseasesImmune systemImmunityNeoplasmsExtracellularmedicineChaperonin 10Suppressor Factors ImmunologicHumansGeneral Pharmacology Toxicology and PharmaceuticsSettore BIO/16 - Anatomia UmanaGrowth factorGeneral MedicineCell biologyMitochondriaProtein TransportHSP60IntracellularLife sciences
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Oncogenic extracellular HSP70 disrupts the gap-junctional coupling between capillary cells

2015

// Dominique Thuringer 1 , Kevin Berthenet 1 , Laurent Cronier 2 , Gaetan Jego 1,3 , Eric Solary 4 , Carmen Garrido 1,3,5 1 INSERM, U866, Faculty of Medecine, Dijon, France 2 CNRS ERL7368, STIM Lab, University of Poitiers, Poitiers, France 3 University of Burgundy, Dijon, France 4 INSERM, U1009, Institut Gustave Roussy, Villejuif, France 5 CGFL, BP77980 21000 Dijon, France Correspondence to: Dominique Thuringer, email: // Keywords : HSP, Cx43, pannexin, Ca 2+ oscillations, ATP release Received : January 30, 2015 Accepted : February 17, 2015 Published : March 10, 2015 Abstract High levels of circulating heat shock protein 70 (HSP70) are detected in many cancers. In order to explore the effec…

Cell signalingPannexinBiologyMolecular biologyCx43Cell biologyATP releaseTransactivationCa2+ oscillationsOncologypannexinExtracellularbiology.proteinHSPPhosphorylationEpidermal growth factor receptorReceptorIntracellularResearch Paper
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A specific CD4 epitope bound by tregalizumab mediates activation of regulatory T cells by a unique signaling pathway

2014

CD4(+)CD25(+) regulatory T cells (Tregs) represent a specialized subpopulation of T cells, which are essential for maintaining peripheral tolerance and preventing autoimmunity. The immunomodulatory effects of Tregs depend on their activation status. Here we show that, in contrast to conventional anti-CD4 monoclonal antibodies (mAbs), the humanized CD4-specific monoclonal antibody tregalizumab (BT-061) is able to selectively activate the suppressive properties of Tregs in vitro. BT-061 activates Tregs by binding to CD4 and activation of signaling downstream pathways. The specific functionality of BT-061 may be explained by the recognition of a unique, conformational epitope on domain 2 of th…

Cell signalingProtein Conformationmedicine.drug_classMolecular Sequence DataImmunologyAntibodies Monoclonal HumanizedCrystallography X-RayLymphocyte ActivationMonoclonal antibodyT-Lymphocytes RegulatoryEpitopeT-Lymphocyte SubsetsTransforming Growth Factor betamedicineHumansImmunology and Allergyddc:610Amino Acid SequenceIL-2 receptorPhosphorylationCells CulturedbiologyInterleukin-2 Receptor alpha SubunitAntibodies MonoclonalPeripheral toleranceCell BiologyTransforming growth factor betaMolecular biologyCell biologyCD4 Antigensbiology.proteinEpitopes B-LymphocyteSignal transductionImmunosuppressive AgentsProtein BindingSignal TransductionConformational epitopeImmunology & Cell Biology
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TGF-β superfamily signaling is essential for tooth and hair morphogenesis and differentiation

2007

Members of the transforming growth factor beta (TGF-beta) superfamily of signaling molecules are involved in the regulation of many developmental processes that involve the interaction between mesenchymal and epithelial tissues. Smad7 is a potent inhibitor of many members of the TGF-beta family, notably TGF-beta and activin. In this study, we show that embryonic overexpression of Smad7 in stratified epithelia using a keratin 5 promoter, results in severe morphogenetic defects in skin and teeth and leads to embryonic and perinatal lethality. To further analyze the functions of Smad7 in epithelial tissues of adult mice, we used an expression system that allowed a controlled overexpression of …

Cell signalingmedicine.medical_specialtyHistologyMorphogenesisEmbryonic DevelopmentMice TransgenicNerve Tissue ProteinsBiologySmad7 ProteinPathology and Forensic MedicineNestinMice03 medical and health sciences0302 clinical medicineIntermediate Filament ProteinsGenes ReporterTransforming Growth Factor betaInternal medicineMorphogenesismedicineAnimalsHumansTransgenes030304 developmental biology0303 health sciencesR-SMADIntegrasesintegumentary systemTooth Abnormalities[SDV.BBM.BM]Life Sciences [q-bio]/Biochemistry Molecular Biology/Molecular biologyCell DifferentiationCell BiologyGeneral MedicineHair follicleSurvival AnalysisCell biologyKeratin 5Endocrinologymedicine.anatomical_structureGene Expression RegulationLac OperonTransforming growth factor beta 3030220 oncology & carcinogenesisRabbitsAmeloblastToothHairSignal TransductionTransforming growth factorEuropean Journal of Cell Biology
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Alternative splicing products of the tenascin gene distinguish rat liver fat storing cells from arterial smooth muscle cells and skin fibroblasts

1992

Abstract Fat storing-(Ito-)cells (FSC) transform into a myofibroblast-like cell type during liver fibrogenesis. A similar development can be observed in cell culture. At the moment, a definite marker to differentiate transformed FSC from smooth muscle cells (SMC) is not available. We recently found that FSC, SMC and skin fibroblasts (SF) synthesize tenascin, a novel matrix protein. As it is reported that various tissues express different tenascin forms by the mechanism of alternative pre-mRNA splicing, we analyzed the tenascin transcripts in these cell types. Total RNA extracted from cultured FSC, SMC and SF, analyzed by Northern blot hybridization, showed a 7.2 kb transcript in FSC, a 8.7 …

Cell typeCell Adhesion Molecules NeuronalRNA SplicingMolecular Sequence DataBiophysicsGene ExpressionTenascinBiochemistryExtracellular matrixTransforming Growth Factor betaGene expressionAnimalsRNA MessengerNorthern blotMolecular BiologyExtracellular Matrix ProteinsMessenger RNABase SequencebiologyAlternative splicingCell DifferentiationMuscle SmoothRats Inbred StrainsTenascinCell BiologyFibroblastsmusculoskeletal systemMolecular biologyFibronectinsRatsCytoskeletal ProteinsAdipose TissueOligodeoxyribonucleotidesRNA splicingbiology.proteinBiochemical and Biophysical Research Communications
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Multiple signal transduction pathways regulate clusterin (gp 80) gene expression in MDCK cells

1996

ABSTRACT Clusterin (gp 80, apolipoprotein J, TRPM-2) is a widely expressed multifunctional glycoprotein. Its demonstrated and proposed functions include the transport of lipids and membrane fragments, the inhibition of the cytolytic action of the terminal complement complex and the modulation of cell—cell interactions. The expression of the gene is enhanced during tissue injury and remodelling and by hormone-withdrawal-induced apoptosis of prostate and mammary cells. We show here that, in the kidney-derived epithelial cell line MDCK, clusterin mRNA is repressed by glucocorticoids and by progesterone. Treatment with epidermal growth factor also represses clusterin gene expression in MDCK cel…

Cell typeTranscription GeneticKidneyDexamethasoneEpitheliumCell LineAlkaloidsDogsEndocrinologyEpidermal growth factor1-Methyl-3-isobutylxanthineGene expressionCyclic AMPAnimalsRNA MessengerEnzyme InhibitorsAldosteroneMolecular BiologyProgesteroneProtein Kinase CProtein kinase CGlycoproteinsBenzophenanthridinesMessenger RNAEpidermal Growth FactorClusterinbiologyChemistryMolecular biologyeye diseasesPhenanthridinesCell biologyKineticsClusterinCell culturebiology.proteinTetradecanoylphorbol Acetatesense organsSignal transductionMolecular ChaperonesSignal TransductionJournal of Molecular Endocrinology
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The effect of cadmium on brain cells in culture

2009

Cadmium is a long-living heavy metal, abundantly present in the environment, which accumulates in the body. In this study, we investigated the effects of cadmium on the expression of molecular chaperones, and of certain cell-specific proteins, in a variety of brain cell types in culture, namely primary cultures of rat cortical neurons and astrocytes, a brain capillary endothelial cell line (RB4E.B cells), and pheochromocytoma cells (PC12), induced or not to differentiate by NGF treatment. The metal induces a dose-dependent increase of Hsp70 in all cell types. Responses to the metal are cell-specific in the case of Hsc70 and Hsp90: i) in astrocytes, as well as in PC12 cells, cadmium has no s…

Cell typecadmium brain cells molecular chaperones PIPPinCell SurvivalCellBlotting Westernchemistry.chemical_elementNerve Tissue ProteinsBiologyPC12 CellsSettore BIO/10 - BiochimicaNerve Growth FactorGeneticsmedicineAnimalsCytoskeletonCell ShapeCells CulturedFluorescent DyesCerebral CortexNeuronsCadmiumBrainEndothelial CellsRNA-Binding ProteinsCell DifferentiationGeneral MedicineCell cycleMolecular biologyHsp70Cell biologyRatsEndothelial stem cellmedicine.anatomical_structurechemistryApoptosisAstrocytesCadmiumMolecular Chaperones
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