Search results for "Growth factor"

showing 10 items of 1300 documents

Mesenchymal Stem Cells Improve Motor Functions and Decrease Neurodegeneration in Ataxic Mice

2014

The main objective of this work is to demonstrate the feasibility of using bone marrow-derived stem cells in treating a neurodegenerative disorder such as Friedreich's ataxia. In this disease, the dorsal root ganglia of the spinal cord are the first to degenerate. Two groups of mice were injected intrathecally with mesenchymal stem cells isolated from either wild-type or Fxntm1Mkn/Tg(FXN)YG8Pook (YG8) mice. As a result, both groups presented improved motor skills compared to nontreated mice. Also, frataxin expression was increased in the dorsal root ganglia of the treated groups, along with lower expression of the apoptotic markers analyzed. Furthermore, the injected stem cells expressed th…

Malemedicine.medical_specialtyAtaxiaCellular differentiationGene ExpressionBone Marrow CellsMice TransgenicMotor ActivityMesenchymal Stem Cell TransplantationTransplantation AutologousMiceGlutathione Peroxidase GPX1Neurotrophin 3Internal medicineGanglia SpinalIron-Binding ProteinsDrug DiscoverymedicineGeneticsAnimalsTransplantation HomologousNerve Growth FactorsMolecular BiologyInjections SpinalPharmacologyGlutathione PeroxidasebiologyBrain-Derived Neurotrophic FactorMesenchymal stem cellCell DifferentiationMesenchymal Stem CellsAnatomySpinal cordCatalaseDisease Models AnimalEndocrinologymedicine.anatomical_structureFriedreich AtaxiaFrataxinbiology.proteinMolecular MedicineOriginal ArticleFemaleBone marrowmedicine.symptomStem cellAdult stem cell
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Liver-specific p38α deficiency causes reduced cell growth and cytokinesis failure during chronic biliary cirrhosis in mice

2012

p38α mitogen-activated protein kinases (MAPK) may be essential in the up-regulation of proinflammatory cytokines and can be activated by transforming growth factor β, tumor necrosis factor-α, interleukin-1β, and oxidative stress. p38 MAPK activation results in hepatocyte growth arrest, whereas increased proliferation has been considered a hallmark of p38α-deficient cells. Our aim was to assess the role of p38α in the progression of biliary cirrhosis induced by chronic cholestasis as an experimental model of chronic inflammation associated with hepatocyte proliferation, apoptosis, oxidative stress, and fibrogenesis. Cholestasis was induced in wildtype and liver-specific p38α knockout mice by…

Malemedicine.medical_specialtyBiliary cirrhosisMAP Kinase Kinase 2ApoptosisBiologymedicine.disease_causeProinflammatory cytokineMitogen-Activated Protein Kinase 14MiceCholestasisInternal medicinemedicineAnimalsCyclin D1Cyclin B1Cell ProliferationCytokinesisMice KnockoutHepatologyLiver Cirrhosis BiliaryHepatologymedicine.diseaseMice Inbred C57BLSurvival RateDisease Models AnimalOxidative Stressmedicine.anatomical_structureEndocrinologyLiverHepatocyteChronic DiseaseDisease ProgressionHepatocytesTumor necrosis factor alphaOxidative stressSignal TransductionTransforming growth factorHepatology
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Common Genetic Variation in Relation to Brachial Vascular Dimensions and Flow-Mediated Vasodilation

2019

Common Genetic Variation in Relation to Brachial Vascular Dimensions and Flow-Mediated Vasodilation

Malemedicine.medical_specialtyBrachial ArteryVasodilationGenome-wide association studyPolymorphism Single NucleotideArticleInternal medicinemedicine.arteryGenetic variationmedicineHumanscardiovascular diseasesBrachial arteryCation Transport ProteinsUltrasonographybusiness.industryGenetic VariationGeneral MedicineMethyltransferasesta3121Middle Agedbody regionsVasodilationInsulin-Like Growth Factor Binding Protein 3PhenotypeCardiovascular DiseasesGenetic Locicardiovascular systemCardiologyMedical geneticsDilation (morphology)Femalebusinesscirculatory and respiratory physiologyFlow-Mediated Vasodilation
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Aflibercept in Combination With FOLFIRI as First-line Chemotherapy in Patients With Metastatic Colorectal Cancer (mCRC): A Phase II Study (FFCD 1302)

2020

Abstract Background FOLFIRI (irinotecan, 5-fluorouracil, and leucovorin) + aflibercept improves median overall survival (OS) and progression-free survival (PFS) in patients with previously treated metastatic colorectal cancer (mCRC). Our aim was to investigate efficacy and tolerability of this combination in the first line. Patients and Methods Patients with untreated documented mCRC received aflibercept plus FOLFIRI every 14 days until progression or unacceptable toxicity in an open, phase II single-arm, multicenter trial. The primary endpoint was the 6-month PFS rate. Secondary endpoints were OS and tolerability. A 2-step Simon design was used with H0: 55% and H1= 75%. Data were analyzed …

Malemedicine.medical_specialtyColorectal cancerRecombinant Fusion Proteins[SDV]Life Sciences [q-bio]LeucovorinPhases of clinical researchGastroenterology03 medical and health sciences0302 clinical medicineInternal medicineMulticenter trialAntineoplastic Combined Chemotherapy ProtocolsClinical endpointmedicineHumansComputingMilieux_MISCELLANEOUSAgedAfliberceptAged 80 and overDose-Response Relationship Drugbusiness.industryGastroenterologyMiddle Agedmedicine.diseaseProgression-Free Survival3. Good healthIrinotecanReceptors Vascular Endothelial Growth FactorOncologyTolerability030220 oncology & carcinogenesisFOLFIRICamptothecinFemale030211 gastroenterology & hepatologyFluorouracilColorectal NeoplasmsbusinessFollow-Up Studiesmedicine.drug
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Osteogenesis imperfecta: a clinical study of the first ten years of life.

1992

One hundred twenty-seven children with osteogenesis imperfecta (O.I.) were studied during the first 10 years of life. According to Sillence, 40 patients were assigned to type I, 39 to type III, and 48 to type IV O.I. Centiles for height, weight, and the annual number of fractures could be established for the different types of O.I. The development of the skeletal changes could be documented for the different forms of the disease. At birth, the skeletal changes were significantly more severe in type III than in type IV patients. During the first 10 years of life the number of fractures, extent of skeletal deformities, and growth retardation did not differ between types III and IV. Only fract…

Malemedicine.medical_specialtyDentinogenesis imperfectaEndocrinology Diabetes and MetabolismPoison controlShort statureBone and BonesClinical studyFractures BoneEndocrinologymedicineHumansOrthopedics and Sports MedicineLongitudinal StudiesInsulin-Like Growth Factor IChildHemihypertrophyBone Developmentbusiness.industryBody WeightInfant NewbornInfantOsteogenesis Imperfectamedicine.diseaseBody HeightSurgeryRadiographyScoliosisOsteogenesis imperfectaMotor SkillsChild PreschoolOrthopedic surgeryKidney stonesFemalemedicine.symptombusinessCalcified tissue international
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Relationship between transforming growth factor beta1 and progression of hypertensive renal disease.

2002

In this study the role of circulating transforming growth factor beta1 (TGFbeta1) on progression of renal hypertensive disease has been investigated. Fifty consecutive outpatients with essential hypertension were enrolled and divided into three groups, according to their urinary albumin excretion (UAE). Group A comprised 10 hypertensives with UAEor=20 mg/24 h (normoalbuminuric group); Group B included 21 hypertensives with UAE20300 mg/24 h (microalbuminuric group); Group C encompassed 19 hypertensives with UAEor= 300 mg/24 h (proteinuric group). In all patients UAE by immunonephelometric assay, circulating TGFbeta1 by a solid phase specific sandwich ELISA technique, BUN and creatinine by ro…

Malemedicine.medical_specialtyDiastoleLeft ventricular hypertrophyEssential hypertensionStatistics NonparametricMuscle hypertrophyExcretionTransforming Growth Factor beta1chemistry.chemical_compoundTransforming Growth Factor betaInternal medicineInternal MedicinemedicinePrevalenceAlbuminuriaHumansInterventricular septumCreatinineEjection fractionbusiness.industryMiddle Agedmedicine.diseasemedicine.anatomical_structureEndocrinologyHypertension RenovascularchemistryEchocardiographyCardiologyDisease ProgressionRegression AnalysisFemaleHypertrophy Left VentricularbusinessBiomarkersJournal of human hypertension
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Associations of IGF-1 and Adrenal Androgens with Cognition in Childhood

2019

<b><i>Background:</i></b> Little is known about the association between adrenarche and cognition in general populations of children. We therefore studied the associations of dehydroepiandrosterone sulfate (DHEAS), androstenedione (A4), testosterone, insulin-like growth factor-1 (IGF-1), and adrenarche with cognition among prepubertal children. <b><i>Methods:</i></b> These cross-sectional analyses are based on baseline data of the Physical Activity and Nutrition in Children Study. A total of 387 children (183 girls, 204 boys) were included in the analyses. Raven’s Coloured Progressive Matrices (CPM) score was used to assess nonverbal reasoning.…

Malemedicine.medical_specialtyEndocrinology Diabetes and Metabolism030209 endocrinology & metabolism03 medical and health scienceschemistry.chemical_compoundCognition0302 clinical medicineEndocrinologyDehydroepiandrosterone sulfateRaven's Progressive MatricesParental educationInternal medicineHumansMedicineTestosteroneAndrostenedioneInsulin-Like Growth Factor IChildTestosterone030219 obstetrics & reproductive medicinebusiness.industryAdrenarcheAge FactorsAndrostenedioneCognitionDehydroepiandrosteroneBaseline dataEndocrinologychemistryPediatrics Perinatology and Child HealthFemalebusinessHormone Research in Paediatrics
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Effect of circulating growth hormone on muscle IGF-I protein concentration in female mice with growth hormone receptor gene disruption.

2009

Growth hormone (GH) is a potent secretague for circulating insulin-like growth factor-I (IGF-I). The purpose of this study was to examine the effect of circulating GH on muscle IGF-I protein expression using GH transgenic animal models. Three different models were used: mice that overexpress bovine GH (bGH; n=10), mice without a functional GH receptor (GHR-/-; n=10), and wildtype mice (n=10). All mice were 16-week old females and each group differed in their basic phenotypic characteristics. Immediately after euthanization the triceps surae muscle group (soleus, plantaris, and gastrocnemius muscles) was removed. IGF-I was extracted from the muscle with an acid-ethanol solution (12.5% 2N hyd…

Malemedicine.medical_specialtyEndocrinology Diabetes and MetabolismTransgenemedicine.medical_treatmentMice TransgenicGrowth hormone receptorBiologyMiceEndocrinologyTriceps surae muscleInternal medicinemedicineAnimalsInsulin-Like Growth Factor IReceptorMuscle SkeletalInsulin-like growth factor 1 receptorMice KnockoutGrowth factorRadioimmunoassayReceptors SomatotropinMice Inbred C57BLEndocrinologyGrowth HormoneCattleFemaleHormoneGrowth hormoneIGF research : official journal of the Growth Hormone Research Society and the International IGF Research Society
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Vitamin D status is linked to biomarkers of oxidative stress, inflammation, and endothelial activation in obese children.

2012

To examine vitamin D, parathyroid hormone, and serum calcium-phosphorus levels relationships to biomarkers of oxidative/nitrosative stress, inflammation, and endothelial activation, potential contributors for vascular complications in obese children.Cross-sectional clinical study of 66 obese Caucasian children aged 7 to 14 years. Cardiovascular risk factors were assessed. Malondialdehyde and myeloperoxidase as measures of oxidative stress, and plasma nitrite+nitrate, urinary nitrate, and 3-nitrotyrosine as markers of nitrosative stress were measured. Adipocytokines, inflammatory molecules (high-sensitivity C-reactive protein, interleukin-6, and tumor necrosis factor-α), endothelial activati…

Malemedicine.medical_specialtyEndotheliumAdolescentNitrogenParathyroid hormoneInflammationmedicine.disease_causeEndothelial activationchemistry.chemical_compoundRisk FactorsInternal medicinemedicineVitamin D and neurologyHumansObesityVitamin DChildInflammationbusiness.industryPhosphorusMalondialdehydeVascular endothelial growth factorOxidative Stressmedicine.anatomical_structureEndocrinologyCross-Sectional StudieschemistryCardiovascular DiseasesParathyroid HormonePediatrics Perinatology and Child HealthCalciumFemaleEndothelium Vascularmedicine.symptombusinessOxidative stressBiomarkersThe Journal of pediatrics
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β-Adrenoceptors differentially regulate vascular tone and angiogenesis of rat aorta via ERK1/2 and p38

2014

β-Adrenoceptors (β-ARs) modulate ERK1/2 and p38 in different cells, but little is known about the contribution of these signaling pathways to the function of β-ARs in vascular tissue. Immunoblotting analysis of rat aortic rings, primary endothelial (ECs) and smooth muscle cells (SMCs) isolated from aorta showed that β-AR stimulation with isoprenaline activated p38 in aortic rings and in both cultured cell types, whereas it had a dual effect on ERK1/2 phosphorylation, decreasing it in ECs while increasing it in SMCs. These effects were reversed by propranolol, which by itself increased p-ERK1/2 in ECs. Isoprenaline β-AR mediated vasodilation of aortic rings was potentiated by the ERK1/2 inhi…

Malemedicine.medical_specialtyEndotheliumPhysiologyAngiogenesisVasodilator AgentsAdrenergic beta-AntagonistsMyocytes Smooth MuscleNeovascularization PhysiologicAorta ThoracicStimulationVasodilationFibroblast growth factorp38 Mitogen-Activated Protein KinasesMuscle Smooth VascularInternal medicineIsoprenalinemedicine.arteryReceptors Adrenergic betamedicineAnimalsHumansRats WistarMitogen-Activated Protein Kinase 1PharmacologyMatrigelAortaMitogen-Activated Protein Kinase 3business.industryAdrenergic beta-AgonistsPropranololRatsVasodilationHEK293 Cellsmedicine.anatomical_structureEndocrinologycardiovascular systemMolecular MedicineEndothelium Vascularbusinessmedicine.drugVascular Pharmacology
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