Search results for "Guanidines"

showing 10 items of 30 documents

Responses to histamine and selective H2-receptor agonists in lung parenchymal strips from normal and sensitized guinea-pigs.

1989

Histamine produces concentration-dependent contractions of lung parenchyma strips obtained from normal and sensitized guinea-pigs. The responsiveness of the sensitized lung strips to histamine was significantly increased compared to normal tissues. Clemizole (0.1 microM) was equally effective as an H1-antagonist in normal (dose-ratio 9.12) and sensitized (dose-ratio 9.77) tissues. The concentration-response curves to histamine were displaced to the left by cimetidine (0.1 microM to 0.1 mM) with similar dose-ratios in normal and sensitized tissues. Cimetidine enhanced maximal responses to histamine only in normal lung strips. The effects of submaximal equieffective concentrations of histamin…

Malemedicine.medical_specialtyImmunologyGuinea PigsToxicologyGuanidineschemistry.chemical_compoundImpromidineHistamine H2 receptorDimapritInternal medicineParenchymamedicineAnimalsPharmacology (medical)Receptors Histamine H2CimetidineLungPharmacologyLungDose-Response Relationship Drugbusiness.industryMethylhistaminesImidazolesIsoproterenolThioureaMuscle SmoothImpromidineDimapritClemizolemedicine.anatomical_structureEndocrinologychemistryBenzimidazolesbusinessCimetidineHistaminemedicine.drugHistamineMuscle ContractionAgents and actions
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In vivo studies on the antiandrogenic effects of cimetidine versus cyproterone acetate in rats

1981

To investigate the antiandrogenic action of cimetidine in vivo, prostatic androgen uptake and metabolism, spermatogenesis, morphology of the prostate and testes, and plasma hormone levels were studied using Sprague-Dawley rats, and the results were compared with the effects of cyproterone acetate or castration. Cimetidine and cyproterone acetate caused significant reduction in the weights of the ventral prostate and testes. The changes of ventral prostate were accompanied by a dose-related epithelial atrophy. No adverse effect on spermatogenesis was observed after treatment with cimetidine at daily doses of 50 mg/kg or cyproterone acetate of 10 mg/kg. Although cimetidine treatment induced a…

Malemedicine.medical_specialtymedicine.drug_classUrologyPharmacologyGuanidineschemistry.chemical_compoundIn vivoProstateInternal medicineTestismedicineAnimalsTestosteroneCastrationCyproteroneCimetidineCyproterone AcetateSpermatogenesisTestosteroneProstateCyproterone acetateAndrogen AntagonistsRats Inbred StrainsOrgan SizeLuteinizing HormoneAndrogenRatsCastrationEndocrinologymedicine.anatomical_structureOncologychemistryFollicle Stimulating HormoneCimetidineSpermatogenesismedicine.drugThe Prostate
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Cu(II) complexes with a sulfonamide derived from benzoguanamine. Oxidative cleavage of DNA in the presence of H2O2 and ascorbate

2004

Reaction between benzoguanamine (2,4-diamino-6-phenyl-1,3,5-triazine) and 2-mesitylenesulfonyl chloride leads to formation of a sulfonamide able to form two mononuclear Cu(II) complexes with a CuL(2) stoichiometry. The local environment of the metal cation is a distorted octahedron, with two ligands and two solvent molecules; both complexes crystallize in the monoclinic structure, space group P2(1), with Z=2. In the presence of ascorbate/H(2)O(2,) the two complexes significantly cleavage double-strand pUC18 DNA plasmid. Both complexes exhibit more nuclease efficiency that the copper phenantroline complex. From scavenging reactive oxygen studies we conclude that the hydroxyl radical and a si…

Models MolecularDNA damagechemistry.chemical_elementAscorbic AcidCrystallography X-RayCleavage (embryo)PhotochemistryGuanidinesBiochemistryPeroxideMedicinal chemistryInorganic ChemistryMetalchemistry.chemical_compoundOrganometallic CompoundsMoleculechemistry.chemical_classificationSulfonamidesMolecular StructureTriazinesDNAHydrogen PeroxideCopperSulfonamidechemistryvisual_artvisual_art.visual_art_mediumHydroxyl radicalOxidation-ReductionCopperJournal of Inorganic Biochemistry
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A new approach to inhibit human β-tryptase by protein surface binding of four-armed peptide ligands with two different sets of arms

2013

A series of six new tetravalent ligands (1-6) with two different sets of arms bind to the surface of β-tryptase, a tetrameric enzyme with an A(2)B(2) arrangement of its four monomers and two different binding sites on its protein surface (as suggested by a docking study). Besides proteinogenic amino acids also the guanidiniocarbonyl pyrrole cation (abbreviated as GCP), as an artificial arginine analog, was introduced into the arms of the ligands to investigate its influence on protein surface binding and enzyme inhibition. Furthermore, four ligands (7-10) with four identical arms also containing the GCP group were additionally synthesized to study the influence of the GCP moiety on the inhi…

Models MolecularSerine Proteinase InhibitorsArginineStereochemistrySurface PropertiesChemieLigandsBiochemistryGuanidineschemistry.chemical_compoundStructure-Activity RelationshipMoietyHumansPyrrolesAmino Acid SequencePhysical and Theoretical ChemistryBinding sitechemistry.chemical_classificationBinding SitesMolecular StructureOrganic ChemistryLigand (biochemistry)Combinatorial chemistryAmino acidEnzymeMonomerchemistryDocking (molecular)TryptasesPeptidesBiologie
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Experimental inhibition of nitric oxide increases Plasmodium relictum (lineage SGS1) parasitaemia.

2012

7 pages; International audience; Malaria is a widespread vector-borne disease infecting a wide range of terrestrial vertebrates including reptiles, birds and mammals. In addition to being one of the most deadly infectious diseases for humans, malaria is a threat to wildlife. The host immune system represents the main defence against malaria parasites. Identifying the immune effectors involved in malaria resistance has therefore become a major focus of research. However, this has mostly involved humans and animal models (rodents) and how the immune system regulates malaria progression in non-model organisms has been largely ignored. The aim of the present study was to investigate the role of…

PlasmodiumCanariesNitric Oxide Synthase Type IIDiseaseParasitemia[ SDV.IMM.IA ] Life Sciences [q-bio]/Immunology/Adaptive immunologyGuanidinesImmune defencechemistry.chemical_compound0302 clinical medicineImmunopathology[ SDV.EE.IEO ] Life Sciences [q-bio]/Ecology environment/SymbiosisEnzyme InhibitorsExperimental infection0303 health sciencesbiologyGeneral Medicine3. Good healthNitric oxide synthaseInfectious Diseases[SDV.IMM.IA]Life Sciences [q-bio]/Immunology/Adaptive immunologyAvian malariaSparrows[ SDV.MP.PAR ] Life Sciences [q-bio]/Microbiology and Parasitology/ParasitologyMalaria Avian030231 tropical medicineImmunologyPlasmodium relictum lineage SGS1ImmunopathologyNitric oxide03 medical and health sciencesImmune systemAvian malariaparasitic diseasesmedicineAnimals[SDV.MP.PAR]Life Sciences [q-bio]/Microbiology and Parasitology/Parasitology030304 developmental biology[ SDE.BE ] Environmental Sciences/Biodiversity and EcologyNitric oxidemedicine.diseasebiology.organism_classificationPlasmodium relictumchemistryImmunologybiology.proteinParasitology[SDE.BE]Environmental Sciences/Biodiversity and EcologyMalaria[SDV.EE.IEO]Life Sciences [q-bio]/Ecology environment/Symbiosis
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Involvement of NO in contact hypersensitivity.

1998

The NO synthases (NOS) generate NO from L-arginine. High concentrations of NO have been shown to be responsible for tissue injury and cell death, while low concentrations of NO induce vasodilatation and other signaling effects. We have investigated the involvement of NO in contact hypersensitivity (CHS) reactions. CHS induced by treatment of BALB/c mice with the contact allergen 2,4-dinitrofluorobenzene (DNFB) was significantly reduced by the NOS inhibitor N-methyl-L-arginine (L-NMA), but not by the stereoisomer D-NMA, as shown by reduced ear swelling responses and evaluation of ear tissue sections. The CHS response was also reduced by aminoguanidine, which is known to preferentially inhibi…

Programmed cell deathLangerhans cellArginineInjections IntradermalT-LymphocytesImmunologyNitric Oxide Synthase Type IIBiologyArginineDermatitis ContactNitric OxideGuanidineschemistry.chemical_compoundMicemedicineImmunology and AllergyAnimalsRNA MessengerEnzyme InhibitorsSkinMice Inbred BALB Cintegumentary systemEpidermis (botany)Histocompatibility Antigens Class IIGeneral MedicineAllergensMolecular biologyPimagedineNitric oxide synthasemedicine.anatomical_structurechemistryLangerhans Cellsbiology.proteinDinitrofluorobenzeneSignal transductionNitric Oxide SynthaseKeratinocyteHaptensInternational immunology
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Synthetic and theoretical studies of novel ring closure and ring opening reactions

2006

Ring closure and ring opening reactions are in many cases useful synthetic procedures in organic chemistry. They allow the preparation of complex molecules with high stereoselectivity and good yields. Mechanistic and theoretical studies have been carried out on the transformation of 2-aminopyrimidines into imidazo[1,2-c]pyrimidines and guanidines, respectively, through ring closure and ring opening reactions, as well as the transamidation reactions through the ring closure and ring opening of guanidine derivatives, which constitute novel synthetic methods. Sepulveda Arques, Jose, Jose.Sepulveda@uv.es

Transamidation reactionUNESCO::QUÍMICAMichael addition:QUÍMICA::Química orgánica [UNESCO]Ring openingUNESCO::QUÍMICA::Química orgánicaImidazopyrimidines ; Guanidines ; Michael addition ; Ring closure ; Ring opening ; Transamidation reactionGuanidines:QUÍMICA [UNESCO]Ring closureImidazopyrimidines
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A minireview on NHE1 inhibitors. A rediscovered hope in oncohematology.

2015

Background: Na+/H+ exchanger-1 (NHE-1) is involved in pH regulation and is up-regulated in different malignancies. Activation of NHE-1 is one way for allowing cells to avoid intracellular acidification and protect them against apoptosis. Inhibitors of NHE-1 are able to decrease intracellular pH and induce apoptosis. Some statins can also act by partial inhibition of NHE-1. This review presents progress in understanding the mechanisms of action of these inhibitors, connections with certain genetic mutations and acquired treatment resistance, as well as new patents on them. Methods: A MEDLINE search for original and review articles using key terms, Na+/H+ exchanger, leukemia, cariporide, and …

lovastatinlcsh:MedicineApoptosisPharmacologyGuanidinesAmiloridep-glycoproteinhemic and lymphatic diseasesDrug InteractionsSulfonesCation Transport ProteinsSodium-Hydrogen Exchanger 1leukemiaMyeloid leukemiaHydrogen-Ion ConcentrationSorafenibUp-RegulationLeukemiaLeukemia Myeloid AcuteImatinib MesylateSignal transductionTyrosine kinasemedicine.drugSignal TransductionSorafenibNiacinamideisoprenylationSodium-Hydrogen Exchangersbcr/ablAntineoplastic AgentsGenes ablGeneral Biochemistry Genetics and Molecular BiologystatinsPatents as TopicCell Line TumorLeukemia Myelogenous Chronic BCR-ABL PositivemedicineHumansProtein Kinase Inhibitorscariporidena+/h+ exchangerTumor hypoxiabusiness.industryPhenylurea Compoundslcsh:ROsmolar Concentrationintracellular phmedicine.diseaseImatinib mesylatefms-Like Tyrosine Kinase 3Fms-Like Tyrosine Kinase 3MutationCancer researchTumor Hypoxiaflt3/itdHydroxymethylglutaryl-CoA Reductase InhibitorsbusinessHeme Oxygenase-1DNA DamageBiomedical papers of the Medical Faculty of the University Palacky, Olomouc, Czechoslovakia
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Pharmacological characterization and autoradiographic localization of histamine H2 receptors in human brain identified with [125I]iodoaminopotentidin…

1992

125I-Aminopotentidine (125I-APT), a reversible probe of high specific radioactivity and high affinity and selectivity for the H2 receptor, was used to characterize and localize this histamine receptor subtype in human brain samples obtained at autopsy. On membranes of human caudate nucleus, specific 125I-APT binding at equilibrium revealed a single component, with a dissociation constant of 0.3 nM and maximal capacity of about 100 fmol/mg of protein. At 0.2 nM, 125I-APT specific binding, as defined with tiotidine, an H2-receptor antagonist chemically unrelated to iodoaminopotentidine, represented 40-50% of the total. Specific 125I-APT binding was inhibited by a series of typical H2-receptor…

medicine.medical_specialtyHistamine H1 receptorHippocampal formationBiologyBiochemistryGuanidinesIodine RadioisotopesCellular and Molecular Neurosciencechemistry.chemical_compoundHistamine receptorHistamine H2 receptorInternal medicinemedicineHumansReceptors Histamine H2Tissue DistributionReceptorHistaminergicBrainHuman brainEndocrinologymedicine.anatomical_structurechemistryHistamine H2 AntagonistsAutoradiographyHistamineJournal of neurochemistry
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Sodium-hydrogen exchange inhibition: novel strategy to prevent myocardial injury following ischemia and reperfusion.

1999

Activation of Na+/H+ exchange and subsequent calcium overload in cardiac myocytes appear to play an important role in myocardial tissue injury following ischemia and reperfusion. Results of several in vitro studies in isolated myocytes and heart preparations and in vivo studies in pigs and rats have suggested that inhibition of Na+/H+ exchange is an effective means to prevent lethal reperfusion injury, arrhythmia, and improve myocardial contractile dysfunction. In patients with acute myocardial infarction (MI), any preventive agent is administered immediately before or shortly after reperfusion, rather than before the occurrence of coronary occlusion. The direct interventional approach to t…

medicine.medical_specialtySodium-Hydrogen Exchangersmedicine.medical_treatmentPremedicationIschemiaMyocardial InfarctionMyocardial Reperfusion InjuryPilot ProjectsGuanidineschemistry.chemical_compoundInternal medicineAngioplastyLactate dehydrogenasemedicineAnimalsHumansMyocardial infarctionSulfonesAngioplasty Balloon CoronaryInfusions IntravenousCariporidebiologybusiness.industrymedicine.diseaseRatschemistryCoronary occlusionAnesthesiaCardiologybiology.proteinCreatine kinaseCardiology and Cardiovascular MedicinebusinessReperfusion injuryAnti-Arrhythmia AgentsThe American journal of cardiology
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