Search results for "HEPATITIS B"

showing 10 items of 451 documents

Pilot study of natural human interleukin-2 in patients with chronic hepatitis B

1993

Ten patients with chronic hepatitis B received increasing doses of nIL-2 (30 000 U, 100 000 U, 300 000 U, 1.0 million U) subcutaneously in a phase I trial. Each dose was applied once per week over 3 weeks. Serum samples were taken before and 2, 12, 24, 48 and 72 h after the first application of each dose level. Serum concentrations of interleukin-1 (IL-1), IL-2, IL-6, interferon-alfa (IFN- α ), IFN- γ , tumor necrosis factor- α (TNF- α ) and GM-CSF as well as the cytokine-dependent serum components neopterin, beta-2-microglobulin (B2M), C-reactive protein (CPR), soluble IL-2-receptor (sIL-2R) and 2′-5′-oligoadenylate synthetase (2–5 OA) were assayed using ELISAs and RIAs. None of the sample…

HepatitisInterleukin 2Hepatitis B virusChemotherapyHepatologybusiness.industrymedicine.medical_treatmentNeopterinPharmacologyHepatitis Bmedicine.diseasemedicine.disease_causechemistry.chemical_compoundCytokineHBeAgchemistryImmunologymedicinebusinessmedicine.drugJournal of Hepatology
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AB0438 Safety of tnf blockers in case of non-alcoholic fatty liver disease and cirrhosisa systematic review

2018

Background The management of inflammatory rheumatisms and psoriasis has largely evolved over the last 15 years with the emergence of biotherapies whose main adverse effect is the increased infection risk. The prevalence of metabolic syndrome is increasing and has been estimated at 30% in patients with rheumatoid arthritis with an excess of 45% compared to healthy subjects. One of the major complications of the metabolic syndrome is the Non Alcoholic Fatty Liver Disease (NAFLD), which prevalence is 25% in the global population, and 30% in a cohort of patients with rheumatoid arthritis. The main complication of NAFLD is the development of cirrhosis, known to increase infectious risk. Surprisi…

Hepatitismedicine.medical_specialtyCirrhosisbusiness.industryAutoimmune hepatitisHepatitis Bmedicine.diseaseGastroenterologyInfliximabEtanerceptInternal medicinemedicineAdalimumabLiver functionbusinessmedicine.drugRheumatoid arthritis – biological DMARDs
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Heterozygosity for the hemochromatosis gene in liver diseases - prevalence and effects on liver histology

2000

:Background/Aims: The effect of heterozygosity for the C282Y mutation in the HFE hemochromatosis gene on iron accumulation and disease progression in liver disease patients is unclear. Methods: We investigated the prevalence of this mutation in 531 patients and 205 healthy controls. In addition, we assessed the influence of the mutation on liver histology in 34 C282Y heterozygous and 124 age-, sex- and disease-matched controls without the mutation using the modified HAI and Chevallier score. Results: The highest prevalence of the C282Y mutation was observed in patients with autoimmune hepatitis (17.2%, p<0.01) compared to 6.4% in healthy controls. Heterozygotes with hepatitis C and B virus …

Hepatitismedicine.medical_specialtyHepatologybusiness.industryAutoimmune hepatitisHepatitis CHepatitis BCompound heterozygositymedicine.diseaseGastroenterologyLiver diseasePrimary biliary cirrhosisInternal medicineImmunologymedicinebusinessHemochromatosisLiver
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Guía de tratamiento de la infección por virus de la hepatitis C. Asociación Española para el Estudio del Hígado (AEEH)

2018

Hepatitismedicine.medical_specialtyHepatologybusiness.industrymedicine.medical_treatmentGastroenterologySalvage therapyHepatitis CHepatitis BLiver transplantationmedicine.diseaseGastroenterology03 medical and health sciences0302 clinical medicinePharmacotherapy030220 oncology & carcinogenesisInternal medicinemedicineCoinfection030211 gastroenterology & hepatologybusinessGastroenterología y Hepatología
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The asialoglycoprotein receptor mediates hepatic binding and uptake of natural hepatitis B virus particles derived from viraemic carriers.

1994

As a putative mechanism of hepatitis B virus (HBV) uptake into hepatocytes the interaction between HBV and the hepatic, human-derived asialoglycoprotein receptor (ASGPR) was investigated. Sera from patients with different variations of hepatitis B surface antigen-(HBsAg) positive chronic hepatitis, HBV particles isolated from HBV carriers with high-titre viraemia and commercial HBsAg served as sources of HBV. ASGPR was affinity-purified from human liver. HBV that had bound to isolated ASGPR was either detected by radio-immunoassay using solid-phase bound ASGPR or enzyme immunoassay with biotin-ASGPR bound to immobilized HBV. Furthermore, binding and uptake of purified, 125I-labelled HBV par…

HepatoblastomaHBsAgHepatitis B virusCarcinoma HepatocellularAsialoglycoproteinsReceptors Cell SurfaceAsialoglycoprotein Receptormedicine.disease_causeBinding CompetitiveVirusVirologymedicineTumor Cells CulturedHumansHepatitis B e AntigensViremiaBinding siteHepatitis B virusCOS cellsHepatitis B Surface AntigensbiologyCell MembraneLiver Neoplasmsvirus diseasesBlood ProteinsHepatitis Bmedicine.diseaseHepatitis BVirologyMolecular biologydigestive system diseasesLiverAcute DiseaseCarrier StateChronic Diseasebiology.proteinReceptors VirusAsialoglycoprotein receptorAntibodyThe Journal of general virology
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Epidemiology, risk factors, and natural history of hepatocellular carcinoma

2002

The incidence of hepatocellular carcinoma is increasing in many countries. The estimated number of new cases annually is over 500,000, and the yearly incidence comprises between 2.5 and 7% of patients with liver cirrhosis. The incidence varies between different geographic areas, being higher in developing areas; males are predominantly affected, with a 2:3 male/female ratio. The heterogeneous geographic distribution reflects the epidemiologic impact of the main etiologic factors and environmental risk, which are the hepatitis B (HBV) and hepatitis C (HCV) viruses. The percentage of cases of hepatocellular carcinoma attributable to HBV worldwide is 52.3% and is higher in Asia where the serop…

Hepatocellular carcinomaLiver cirrhosisHepatitis BHepatitis C
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HCV/HBV coinfection: The dark side of DAAs treatment?

2016

HepatologyCoinfectionbusiness.industryHIV InfectionsHepatitis CHepatitis BHepatitis Bmedicine.diseaseAntiviral AgentsHepatitis CVirology03 medical and health sciences0302 clinical medicine030220 oncology & carcinogenesismedicineCoinfectionHumans030211 gastroenterology & hepatologybusinessLiver International
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Corrigendum to “Premature ovarian senescence and a high miscarriage rate impair fertility in women with HCV” [J Hepatol 68 (2018) 33–41](S01688278173…

2018

It has come to our attention that the PITER framework investigator, Alessandro Federico, was incorrectly listed as F. Alessandro in the original manuscript. Please note that the correct name of this author is Alessandro Federico (2nd University of Naples). The correct list of PITER investigators is in the footnote below.

HepatologyHepatitis B; EASL guidelines; Treatment; Interferon; Entecavir; Tenofovir; TAF; HBsAg; Hepatocellular carcinoma; HBV DNA; HBV reactivation; Mother to child transmissionHepatocellular carcinomaHBV reactivationEASL guidelinesHepatitis BEntecavirTreatmentHBsAgTAFHBV DNAMother to child transmissionInterferonTenofovirEASL guideline
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Reactivación de hepatitis B en paciente con hepatitis crónica C tratado con antivirales de acción directa

2018

Hepatologybusiness.industryGastroenterologyHepatitis CHepatitis Bmedicine.diseaseDIRECT ACTING ANTIVIRALSVirology03 medical and health sciences0302 clinical medicinePharmacotherapyChronic hepatitis030220 oncology & carcinogenesismedicine030211 gastroenterology & hepatologybusinessViral loadGastroenterología y Hepatología
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How long to treat chronic hepatitis B virus infection with lamivudine?

2000

Hepatologybusiness.industryLamivudineVirologyVirusHepatitis B ChronicTreatment OutcomeChronic hepatitisLamivudineHumansReverse Transcriptase InhibitorsMedicinebusinessRetrospective Studiesmedicine.drugJournal of Hepatology
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