Search results for "HLA-B27"

showing 6 items of 26 documents

HLA-B27-restricted cytotoxic T lymphocyte responses to arthritogenic enterobacteria or self-antigens are dominated by closely related TCRBV gene segm…

1996

Identification of the T-cell receptors (TCR) used by synovial cytotoxic T lymphocytes (CTL) of patients with reactive arthritis (ReA) may be crucial to better understanding the pathogenetic mechanism underlying the HLA-B27 association of spondylarthropathies. The authors, therefore, sequenced 25 TCRB chains from HLA-B27-restricted CD8+ CTL clones and two clonal lines specific for self- or Yersinia enterocolitica antigen isolated from synovial fluids of 3 HLA-B27+ patients with ReA and PBL of one healthy HLA-B27+ individual. Fourteen non-HLA-B27-restricted CTL served as controls. Both autoreactive and Y. enterocolitica specific HLA-B27-restricted CTL used a highly limited set of VB genes wit…

musculoskeletal diseasesAdultMaleSalmonella typhimuriumYersinia InfectionsReceptors Antigen T-Cell alpha-betaImmunologyMolecular Sequence Datachemical and pharmacologic phenomenaChlamydia trachomatisBiologyCD8-Positive T-LymphocytesArthritis ReactiveAutoantigensPolymerase Chain ReactionProhibitinsSynovial FluidCytotoxic T cellHumansAmino Acid SequenceGene Rearrangement beta-Chain T-Cell Antigen Receptorskin and connective tissue diseasesReceptorSpondylarthropathiesGeneHLA-B27 AntigenYersinia enterocoliticaHLA-B27Antigens BacterialT-cell receptorhemic and immune systemsGeneral MedicineDNAChlamydia InfectionsCTL*ImmunologySalmonella InfectionsCD8T-Lymphocytes CytotoxicScandinavian journal of immunology
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Bacteria-specific cytotoxic CD8+ T cells: a missing link in the pathogenesis of the HLA-B27-associated spondylarthropathies.

1994

The term seronegative spondylarthropathies is used for an entity of rheumatic syndromes of peripheral joints and the spine (ankylosing spondylitis, reactive arthritis, Reiter's syndrome, arthritis in psoriasis and in inflammatory bowel disease) which are strongly associated with the MHC class I molecule HLA-B27. However, the mechanisms whereby HLA-B27 confers disease susceptibility have so far remained unknown. There is strong evidence that gut inflammation and infection with gram-negative bacteria play a role in the induction of B27-associated disease. HLA-B27, like other MHC class I molecules, physiologically binds antigenic peptides in its binding groove and presents them to CD8+ T lymph…

musculoskeletal diseasesCytotoxicity ImmunologicAnkylosisEpitopeEpitopesAntigenEnterobacteriaceaeMHC class IMedicineCytotoxic T cellAnimalsHumansSpondylarthropathiesHLA-B27 AntigenHLA-B27Antigens Bacterialbiologybusiness.industryArthritisSynovial MembraneGeneral MedicineDisease Models AnimalImmunologybiology.proteinBacterial antigenbusinessCD8Protein BindingT-Lymphocytes CytotoxicAnnals of medicine
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Conserved TCR β chain usage in reactive arthritis; evidence for selection by a putative HLA-B27-associated autoantigen

2002

Previous work suggested that expanded CD8+ T-cell clones in the synovial fluid (SF) of HLA-B27+ patients with reactive arthritis (ReA) preferentially use the T-cell receptor variable region (TCRBV) 1, similar CDR3 sequences, and joining region (BJ) 2S3. To determine the range of conservation and disease-specificity of CDR3-sequences, we analyzed the TCRBV1-J2S3 repertoire from 33 healthy HLA-B27+ individuals, patients with various types of spondyloarthropathies (SpA), and with rheumatoid arthritis (RA) by CDR3-spectratyping. After collection and database submission of all available TCRB-CDR3 from HLA-B27-restricted or SpA-derived T cells, we systematically screened the entire human sequence…

musculoskeletal diseasesGeneticsHLA-B27T cellImmunologyT-cell receptorArthritisGeneral MedicineBiologymedicine.disease_causemedicine.diseaseBiochemistryConserved sequenceAutoimmunitymedicine.anatomical_structureAntigenGeneticsmedicineImmunology and Allergyskin and connective tissue diseasesCD8Tissue Antigens
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Enterobacterial Antigens with Tropism for Joint Structures and HLA-B27=Restricted Cytotoxic T-Cells in Reactive Arthritis

1995

In reactive arthritis (ReA), sterile synovitis is an immunological sequela following gastrointestinal or urogenital infection with facultatively intracellular bacteria (Yersinia, Salmonella, Shigella, Chlamydia). It is widely accepted now that the development of arthritis is closely related to the persistance of bacteria or bacterial antigens in extraarticular mucosal or lymphoid tissues (i.e. gut mucosa, gut associated lymphoid tissue, genitourinary mucosa); however, it is still unclear which host mechanisms are responsible for the poorer elimination of arthritis-causing microorganisms in those ReA patients. Bacterial components are also camed to the joints where they can be demonstrated i…

musculoskeletal diseasesGut-associated lymphoid tissueImmunologyYersiniaArthritis ReactiveTropismMicrobiologyImmune systemEnterobacteriaceaeRheumatologyAntigenSynovitismedicineAnimalsHumansImmunology and AllergySynovial fluidHLA-B27 AntigenAntigens BacterialbiologyGeneral Medicinebiology.organism_classificationmedicine.diseasemedicine.anatomical_structureImmunologyJointsBacterial antigenSynovial membraneT-Lymphocytes CytotoxicScandinavian Journal of Rheumatology
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Non-conventional forms of HLA-B27 are expressed in spondyloarthritis joints and gut tissue

2016

Objectives Human leukocyte antigen (HLA)-B27 (B27) is the strongest genetic factor associated with development of Ankylosing Spondylitis and other spondyloarthropathies (SpA), yet the role it plays in disease pathogenesis remains unclear. We investigated the expression of potentially pathogenic non-conventional heavy chain forms (NC) of B27 in synovial and intestinal tissues obtained from SpA patients. We also determined the presence of NC-B27 in joints, lymphoid and gastrointestinal tissue from B27 transgenic (TG1) rats with M.tuberculosis-induced SpA. Methods Expression of NC-B27 in human SpA joints and gut and in (21-3 × 283-2)F1 HLA-B27/Huβ2m rat tissue was determined by immunohistochem…

musculoskeletal diseasesalpha-DefensinsHLA-B27 transgenic rat modelGastrointestinal DiseasesCD8 AntigensImmunologyGene ExpressionArticleSpondyloarthropathieAnimalsHumansHLA class I free-heavy chainImmunology and AllergySpondylitis AnkylosingSpondyloarthropathiesskin and connective tissue diseasesHLA-B27 AntigenHLA-B27CD11 AntigensHistocompatibility Antigens Class ISynovial MembraneReceptors KIR3DL2Arthritis ExperimentalR1HLA class I free-heavy chainsRatsDisease Models AnimalSettore MED/16 - ReumatologiaHLA class I free-heavy chains; HLA-B27; HLA-B27 transgenic rat model; Spondyloarthropathies; Immunology and Allergy; ImmunologyBone RemodelingRats Transgenic
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Arthritis Associated with Ulcerative Colitis: Our Experience in Palermo

2009

To The Editor: Ulcerative colitis (UC) is one of the inflammatory bowel diseases, which affects the small intestine and colon [1]. The pathophysiology of UC is not known [2, 3]. The association between arthritis and UC is well known. The authors report the frequency of arthritis in 53 Sicilian patients with UC. The diagnosis of UC was made according to criteria described by Schachter and Kirsner [4]. The involvement of sacroilitic joints was assessed radiologically according to Bennett and Burch grade (0=normal joint, 1=suspicious sacroiliitis 2=abnormal joint with sclerosis and/or erosions, 3=unequivocally abnormal with erosions, sclerosis, widening or narrowing or partly ankylosed, 4=tota…

musculoskeletal diseasesmedicine.medical_specialtylcsh:MedicineArthritisLetter to EditorGastroenterologyInternal medicineSettore MED/33 - Malattie Apparato LocomotoremedicineAnkylosisRheumatoid factorSpondylitisHLA-B27Inflammatory bowel diseases Arthritis spondylitis ulcerative colitisbusiness.industryArthritislcsh:RSacroiliitisGeneral MedicineInflammatory Bowel Diseasesmedicine.diseaseUlcerative colitisSurgerymedicine.anatomical_structureUlcerative colitisAnklebusinessSpondylitisLibyan Journal of Medicine
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