Search results for "HORMONE REPLACEMENT THERAPY"

showing 10 items of 72 documents

Metabolic parameters and adipokine profile during GH replacement therapy in children with GH deficiency

2007

Objective: GH replacement therapy in children with GH deficiency (GHD) mainly promotes linear growth. Not only have very few studies fully analyzed the metabolic consequences of GH therapy, but also the question as to whether GH may affect adipokine secretion has been insufficiently investigated. Our aim was to study the effects of GH replacement therapy on auxological data, lipid and glycemic profiles, insulin homeostasis (HOMA-IR) and serum adipokines in children. Methods: This was a 1-year prospective study. Thirty-four GHD children (11.6 ± 2.6 years) and thirty healthy matched controls were enrolled. Children affected by GHD were studied both before beginning continuous GH replacement t…

LeptinMalemedicine.medical_specialtyTime FactorsAdolescentHormone Replacement TherapyEndocrinology Diabetes and Metabolismmedicine.medical_treatmentAdipokineBiologyBody Mass IndexSettore MED/13 - Endocrinologiachemistry.chemical_compoundEndocrinologyInternal medicinemedicineHumansInsulinInsulin-Like Growth Factor IChildGrowth DisordersTriglyceridesGlycemicmedicine.diagnostic_testAdiponectinHuman Growth HormoneCholesterolLeptinInsulinBody WeightCholesterol LDLGeneral MedicineBody HeightTreatment OutcomeEndocrinologychemistryTransgender hormone therapyFemaleAdiponectinLipid profileGH METABOLISM ADIPOKINESEuropean Journal of Endocrinology
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Early menopause is associated with lack of response to antiviral therapy in women with chronic hepatitis C.

2011

Background & Aims Chronic hepatitis C (CHC) and liver fibrosis progress more rapidly in men and menopausal women than in women of reproductive age. We investigated the associations among menopause, sustained virologic response (SVR), and liver damage in patients with CHC. Methods We performed a prospective study of 1000 consecutive, treatment-naive patients 18 years of age and older with compensated liver disease from CHC. Liver biopsy samples were analyzed (for fibrosis, inflammation, and steatosis) before patients received standard antiviral therapy. From women (n = 442), we collected data on the presence, type, and timing of menopause; associated hormone and metabolic features; serum lev…

Liver CirrhosisMaleTime Factorsmedicine.medical_treatmentBiopsyMenopause PrematuremenopauseHepacivirusmedicine.disease_causeGastroenterologySeverity of Illness IndexRisk FactorsOdds RatioProspective StudiesTreatment FailureProspective cohort studymedicine.diagnostic_testGastroenterologyAge FactorsHormone replacement therapy (menopause)Hepatitis CMiddle AgedViral LoadImmunohistochemistryMenopauseItalyLiver biopsyRNA ViralFemaleInflammation Mediatorshcv svr menopauseViral loadAdultmedicine.medical_specialtyantiviral therapy; menopause; prognostic factors; hcv therapyGenotypeHepatitis C virusAntiviral AgentsRisk AssessmentSex FactorsInternal medicinehcvmedicineHumanshcv; ifn; menopauseHepatologybusiness.industryInterleukin-6Tumor Necrosis Factor-alphaOdds ratioHepatitis C Chronicmedicine.diseaseifnEndocrinologyLogistic ModelsbusinessBiomarkersGastroenterology
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Sex Differences in Age-Associated Type 2 Diabetes in Rats—Role of Estrogens and Oxidative Stress

2019

Females live longer than males, and the estrogens are one of the reasons for this difference. We reported some years ago that estrogens are able to protect rats against oxidative stress, by inducing antioxidant genes. Type 2 diabetes is an age-associated disease in which oxidative stress is involved, and moreover, some studies show that the prevalence is higher in men than in women, and therefore there are sex-associated differences. Thus, the aim of this study was to evaluate the role of estrogens in protecting against oxidative stress in type 2 diabetic males and females. For this purpose, we used Goto-Kakizaki rats, which develop type 2 diabetes with age. We found that female diabetic ra…

Male0301 basic medicineAgingmedicine.medical_specialtyArticle SubjectHormone Replacement Therapymedicine.drug_classmedicine.medical_treatmentGlucose uptakeType 2 diabetes030204 cardiovascular system & hematologymedicine.disease_causeBiochemistry03 medical and health sciences0302 clinical medicineLipid oxidationInternal medicineDiabetes mellitusmedicineAnimalsMetabolomicsRats Wistarlcsh:QH573-671Sex Characteristicsbusiness.industrylcsh:CytologyEstrogensHormone replacement therapy (menopause)Cell BiologyGeneral Medicinemedicine.diseaseMitochondriaOxidative StressGlucose030104 developmental biologyEndocrinologyDiabetes Mellitus Type 2Organ SpecificityEstrogenOvariectomized ratFemalebusinessOxidative stressResearch ArticleOxidative Medicine and Cellular Longevity
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Associations between cognitive performance and the rehabilitation, medical care and social support provided to French children with Prader-Willi synd…

2020

International audience; Prader-Willi syndrome (PWS) is a rare genetic neurodevelopmental disorder with a characteristic behavioural phenotype. A multidisciplinary approach to care is required to prevent multiple medical complications in individuals affected by PWS. The aim of this study was to describe the rehabilitation, medical care, educational and social support provided to school-aged French PWS patients with varying neuropsychological profiles. Data were obtained from a French multicentre study that included patients aged 4-20 years with diverse genetic syndromes. Nineteen PWS subjects with a mean age of 9.2 years were included. The mean full-scale intellectual quotient (IQ) was 58 (W…

Male0301 basic medicinemedicine.medical_treatmentIntellectual disabilityMESH: CognitionCBCL030105 genetics & heredityCognitionMultidisciplinary approachMESH: ChildIntellectual disabilityMedicineChildGenetics (clinical)RehabilitationMESH: Hormone Replacement TherapyNeurological RehabilitationNeuropsychologyWechsler Adult Intelligence ScaleGeneral Medicine3. Good healthMESH: Young AdultChild PreschoolEducation SpecialFemaleFrancePrader-Willi SyndromeOccupational therapymedicine.medical_specialtyAdolescentHormone Replacement TherapyMESH: Social Support[SDV.GEN.GH] Life Sciences [q-bio]/Genetics/Human geneticsYoung Adult03 medical and health sciencesSocial supportMESH: Neurological RehabilitationGeneticsHumansPsychiatryMESH: AdolescentMESH: Humansbusiness.industryMESH: Child PreschoolSocial Supportmedicine.diseaseMESH: MaleMESH: FrancePatient care management030104 developmental biologyMESH: Education Special[SDV.GEN.GH]Life Sciences [q-bio]/Genetics/Human geneticsMESH: Prader-Willi SyndromebusinessMESH: FemaleEuropean Journal of Medical Genetics
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Aromatase Inhibitors Plus Weight Loss Improves the Hormonal Profile of Obese Hypogonadal Men Without Causing Major Side Effects

2020

Objective: In obese men, the increased expression of the aromatase enzyme in adipose tissue leads to high conversion of androgens to estrogens contributing to hypogonadotropic hypogonadism (HHG). Our objective is to evaluate efficacy and safety of weight loss (WL) plus aromatase inhibitor (AI) therapy in severely obese men with HHG. We hypothesize that AI+WL will be more effective as compared to WL alone in improving the hormonal profile, thus muscle strength and symptoms of HHG (primary outcomes), with no significant adverse effects on lean mass, metabolic profile, and bone mineral density (secondary outcomes).Design: Randomized double-blind placebo-controlled pilot trial.Methods: Twenty-t…

Male0301 basic medicineobesityBone densityEndocrinology Diabetes and MetabolismPilot Projectslcsh:Diseases of the endocrine glands. Clinical endocrinologyaromatase inhibitorsEndocrinology0302 clinical medicineBone DensityWeight lossMedicineTestosteroneTestosteroneBone mineralEstradiolMiddle AgedPrognosisClinical TrialAndrogensMetabolomemedicine.symptombone microarchitecturemedicine.drugAdultmedicine.medical_specialtyHormone Replacement Therapymedicine.drug_classAnastrozole030209 endocrinology & metabolismsex hormonesBone and Bones03 medical and health sciencesDouble-Blind MethodHypogonadotropic hypogonadismInternal medicineWeight LossHumanshypogonadismMuscle StrengthAgedbody compositionlcsh:RC648-665Aromatase inhibitorbusiness.industrymedicine.disease030104 developmental biologyEndocrinologyLean body massbusinessBiomarkersFollow-Up StudiesFrontiers in Endocrinology
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A Pooled Analysis of Reproductive Factors, Exogenous Hormone Use, and Risk of Multiple Myeloma among Women in the International Multiple Myeloma Cons…

2015

Abstract Background: Female sex hormones are known to have immunomodulatory effects. Therefore, reproductive factors and exogenous hormone use could influence the risk of multiple myeloma in women. However, the role of hormonal factors in multiple myeloma etiology remains unclear because previous investigations were underpowered to detect modest associations. Methods: We conducted a pooled analysis of seven case–control studies included in the International Multiple Myeloma Consortium, with individual data on reproductive factors and exogenous hormone use from 1,072 female cases and 3,541 female controls. Study-specific odds ratios and corresponding 95% confidence intervals (CI) were estima…

MaleEpidemiologymedicine.medical_treatmentPhysiology[ SDV.CAN ] Life Sciences [q-bio]/Cancer0302 clinical medicineRisk FactorsOdds RatioGonadal030212 general & internal medicineReproductive HistoryMultiple myelomaIncidenceMiddle AgedStatisticalPrognosis3. Good healthPostmenopauseContraceptionOncologyTransgender hormone therapy030220 oncology & carcinogenesisMeta-analysisRegression AnalysisFemaleMultiple MyelomaFactor AnalysisAdultHormone Replacement Therapy[SDV.CAN]Life Sciences [q-bio]/CancerArticle03 medical and health sciencesYoung AdultMeta-Analysis as TopicReproductive factors exogenous hormone use and risk of multiple myelomamedicineConfidence IntervalsHumansNeoplasm StagingSteroid Hormonesbusiness.industryCase-control studyOdds ratiomedicine.diseaseHormonesLogistic ModelsHormonal contraceptionCase-Control StudiesSample SizeImmunologyHormone therapybusinessHormoneFollow-Up StudiesMeta-Analysis
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Hormone replacement therapy enhances IGF-1 signaling in skeletal muscle by diminishing miR-182 and miR-223 expressions : a study on postmenopausal mo…

2014

MiRNAs are fine-tuning modifiers of skeletal muscle regulation, but knowledge of their hormonal control is lacking. We used a co-twin case-control study design, that is, monozygotic postmenopausal twin pairs discordant for estrogen-based hormone replacement therapy (HRT) to explore estrogen-dependent skeletal muscle regulation via miRNAs. MiRNA profiles were determined from vastus lateralis muscle of nine healthy 54-62-years-old monozygotic female twin pairs discordant for HRT (median 7 years). MCF-7 cells, human myoblast cultures and mouse muscle experiments were used to confirm estrogen's causal role on the expression of specific miRNAs, their target mRNAs and proteins and finally the act…

MaleMICRORNASMonozygotic twinmenopausePATHWAYMice0302 clinical medicineMyocyteInsulin-Like Growth Factor IIN-VIVO0303 health sciencesphosphorylationAge FactorsBREAST-CANCER CELLSWOMENMiddle Aged3142 Public health care science environmental and occupational healthPostmenopauseESTROGENmedicine.anatomical_structureMCF-7 CellsmTORGROWTHFemaleAUTOPHAGYMESSENGER-RNASignal TransductionIGF-1 receptormedicine.medical_specialtyHormone Replacement Therapymedicine.drug_classmiR-142-3pBiology03 medical and health sciencesInternal medicinemicroRNAmedicineAnimalsHumansMuscle SkeletalProtein kinase BPI3K/AKT/mTOR pathwayAged030304 developmental biologyAKTagingSkeletal muscleOriginal ArticlesTwins MonozygoticCell BiologyAKT; FOXO3A; IGF-1 signaling; IGF-1R; aging; mTOR; menopause; miR-142-3p; miR-182; miR-223; phosphorylationmiR-223EndocrinologyEstrogenCase-Control StudiesmiR-1823121 General medicine internal medicine and other clinical medicineFOXO3AIGF-1 signalingIGF-1R030217 neurology & neurosurgeryHUMAN LONGEVITYHormone
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High prevalence of hypovitaminosis D in Sicilian children affected by growth hormone deficiency and its improvement after 12 months of replacement tr…

2014

PURPOSE: Although the correlation between vitamin D and growth hormone (GH)-insulin-like growth factor 1 (IGF1) axis is documented, as of date, few and conflicting studies have prospectively analyzed vitamin D before and after GH treatment. Our aim was to evaluate as to how the condition of GH deficiency (GHD) or GH treatment influences vitamin D in children. METHODS: Eighty Sicilian GHD children (M/F 58/22; mean age 10.3 years), grouped according to the season of evaluation in group A (June-September; 41 children) and group B (November-February; 39 children), were evaluated at baseline and after 12 months of GH treatment. RESULTS: Twenty-eight children (35 %) were vitamin D insufficient an…

Malemedicine.medical_specialtyAdolescentHormone Replacement TherapyEndocrinology Diabetes and Metabolismmedicine.medical_treatmentComorbidityGrowth hormoneGrowth hormone deficiencySettore MED/13 - EndocrinologiaYoung AdultEndocrinologyHypovitaminosisInternal medicinemedicineVitamin D and neurologyPrevalenceHumansChildDwarfism Pituitaryvitamin D growth hormoneHigh prevalencebusiness.industryHuman Growth HormoneGrowth factorInfantmedicine.diseaseVitamin D DeficiencyEndocrinologyTreatment OutcomeChild PreschoolGh treatmentFemalebusinessGH Deficiency
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Growth hormone replacement therapy prevents sarcopenia by a dual mechanism: improvement of protein balance and of antioxidant defenses.

2013

The aim of our study was to elucidate the role of growth hormone (GH) replacement therapy in three of the main mechanisms involved in sarcopenia: alterations in mitochondrial biogenesis, increase in oxidative stress, and alterations in protein balance. We used young and old Wistar rats that received either placebo or low doses of GH to reach normal insulin-like growth factor-1 values observed in the young group. We found an increase in lean body mass and plasma and hepatic insulin-like growth factor-1 levels in the old animals treated with GH. We also found a lowering of age-associated oxidative damage and an induction of antioxidant enzymes in the skeletal muscle of the treated animals. GH…

Malemedicine.medical_specialtyAgingSarcopeniaIGF-1. Mitochondrial biogenesis Myostatin p70S6KHormone Replacement TherapyMyostatinProtein degradationmedicine.disease_causeAntioxidantsInternal medicineMedicineAnimalsRats WistarMuscle Skeletalbiologybusiness.industryProtein turnoverSkeletal muscleProteinsmedicine.diseaseMitochondria MuscleRatsSomatropinEndocrinologymedicine.anatomical_structureMitochondrial biogenesisSarcopeniaGrowth Hormonebiology.proteinBody CompositionGeriatrics and GerontologybusinessOxidative stressThe journals of gerontology. Series A, Biological sciences and medical sciences
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Comparison between euglycemic hyperinsulinemic clamp and surrogate indices of insulin sensitivity in children with growth hormone deficiency

2018

Objective: Data about the impact of growth hormone treatment (GHT) on insulin sensitivity in children are quite controversial, due to the different surrogate indices that have been used. Design: We evaluated insulin sensitivity through the euglycemic hyperinsulinemic clamp, considered the gold standard technique, in 23 children affected by growth hormone deficiency (GHD) at baseline and after 12. months of GHT and in 12 controls with short stature at baseline, and we compared the clamp-derived index (M-value) with the most commonly used surrogate index of insulin sensitivity, as ISI Matsuda, and with circulating plasma markers of insulin sensitivity, as adiponectin and resistin levels. Resu…

Malemedicine.medical_specialtyHormone Replacement TherapyEndocrinology Diabetes and Metabolism030209 endocrinology & metabolismShort statureGrowth hormone deficiencySettore MED/13 - Endocrinologia03 medical and health sciences0302 clinical medicineEndocrinologyInternal medicineGlucose IntolerancemedicineHumansResistinProspective StudiesChildGrowth DisordersEuglycemic hyperinsulinemic clampAdiponectinHuman Growth Hormonebusiness.industryGrowth hormone deficiency; Hyperinsulinemic clamp; Insulin sensitivity; Endocrinology Diabetes and Metabolism; EndocrinologyInsulin sensitivityHyperinsulinemic clampGold standard (test)Glucose Tolerance TestPrognosismedicine.diseaseInsulin sensitivityGrowth hormone treatmentEndocrinologyCase-Control Studies030220 oncology & carcinogenesisGlucose Clamp TechniqueFemaleResistinGrowth hormone deficiencyAdiponectinInsulin Resistancemedicine.symptombusinessBiomarkersFollow-Up Studies
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