Search results for "HORMONES"

showing 10 items of 1169 documents

TSH/IGF-1 Receptor Cross-Talk Rapidly Activates Extracellular Signal-Regulated Kinases in Multiple Cell Types

2017

We previously showed that thyrotropin (TSH)/insulinlike growth factor (IGF)-1 receptor cross-talk appears to be involved in Graves’ orbitopathy (GO) pathogenesis and upregulation of thyroid-specific genes in human thyrocytes. In orbital fibroblasts from GO patients, coadministration of TSH and IGF-1 induces synergistic increases in hyaluronan secretion. In human thyrocytes, TSH plus IGF-1 synergistically increased expression of the sodium-iodide symporter that appeared to involve ERK1/2 activation. However, the details of ERK1/2 activation were not known, nor was whether ERK1/2 was involved in this synergism in other cell types. Using primary cultures of GO fibroblasts (GOFs) and human thyr…

0301 basic medicinemedicine.medical_specialtyCell typeendocrine systemendocrine system diseasesMAP Kinase Signaling SystemThyrotropinPertussis toxinReceptor IGF Type 103 medical and health sciencesEndocrinologyDownregulation and upregulationInternal medicinemedicineHumansSecretionHyaluronic AcidInsulin-Like Growth Factor IPhosphorylationReceptorResearch ArticlesMitogen-Activated Protein Kinase 1Mitogen-Activated Protein Kinase 3SymportersChemistryHEK 293 cellsReceptors ThyrotropinReceptor Cross-TalkFibroblastsSignalingGraves Ophthalmopathy030104 developmental biologyEndocrinologyHEK293 CellsThyroid Epithelial CellsSymporterPhosphorylationhormones hormone substitutes and hormone antagonistsEndocrinology
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Obesity as a Risk Factor for Alzheimer’s Disease: Implication of Leptin and Glutamate

2019

Obesity is known to induce leptin and insulin resistance. Leptin is a peptide hormone synthesized in adipose tissue that mainly regulates food intake. It has been shown that insulin stimulates the production of leptin when adipocytes are exposed to glucose to encourage satiety; while leptin, via a negative feedback, decreases the insulin release and enhances tissue sensitivity to it, leading to glucose uptake for energy utilization or storage. Therefore, resistance to insulin is closely related to leptin resistance. Obesity in middle age has also been related to Alzheimer’s disease (AD). In recent years, the relation between impaired leptin signaling pathway and the onset of AD has been stu…

0301 basic medicinemedicine.medical_specialtyMini Reviewmedicine.medical_treatmentGlucose uptakeExcitotoxicityAdipose tissuemedicine.disease_causelcsh:RC321-57103 medical and health sciences0302 clinical medicineInsulin resistanceInternal medicinemedicineoverweightleptin-resistanceReceptorlcsh:Neurosciences. Biological psychiatry. Neuropsychiatrybusiness.industryGeneral NeuroscienceInsulinLeptindigestive oral and skin physiologyGlutamate receptormedicine.disease030104 developmental biologyEndocrinologyLTPbusinessexcitotoxicity030217 neurology & neurosurgeryhormones hormone substitutes and hormone antagonistsNeurosciencedementiaFrontiers in Neuroscience
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The Adverse Effects of Environmental Noise Exposure on Oxidative Stress and Cardiovascular Risk

2018

Abstract Epidemiological studies have provided evidence that traffic noise exposure is linked to cardiovascular diseases such as arterial hypertension, myocardial infarction, and stroke. Noise is a nonspecific stressor that activates the autonomous nervous system and endocrine signaling. According to the noise reaction model introduced by Babisch and colleagues, chronic low levels of noise can cause so-called nonauditory effects, such as disturbances of activity, sleep, and communication, which can trigger a number of emotional responses, including annoyance and subsequent stress. Chronic stress in turn is associated with cardiovascular risk factors, comprising increased blood pressure and …

0301 basic medicinemedicine.medical_specialtyPhysiologyClinical Biochemistrytraffic noise exposure030204 cardiovascular system & hematologyenvironmental risk factorsmedicine.disease_causeBiochemistryendothelial dysfunctionCoronary artery disease03 medical and health sciences0302 clinical medicinestress hormonesComprehensive Invited ReviewRisk FactorsInternal medicineDiabetes mellitusmedicineHumansChronic stressMyocardial infarctionEndothelial dysfunctionMolecular BiologyStrokeGeneral Environmental Sciencebusiness.industryaircraft noise exposureEnvironmental ExposureCell Biologymedicine.diseaseStrokeOxidative Stress030104 developmental biologyBlood pressureCardiovascular DiseasesHypertensionCardiologyGeneral Earth and Planetary SciencesEnvironmental PollutantsNoiseReactive Oxygen SpeciesbusinessOxidative stressAntioxidants & Redox Signaling
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Regular Intake of Pistachio Mitigates the Deleterious Effects of a High Fat-Diet in the Brain of Obese Mice

2020

Obesity has been associated with neurodegeneration and cognitive dysfunctions. Recent data showed that pistachio consumption is able to prevent and ameliorate dyslipidemia, hepatic steatosis, systemic and adipose tissue inflammation in mice fed a high-fat diet (HFD). The present study investigated the neuroprotective effects of pistachio intake in HFD mice. Three groups of mice were fed a standard diet (STD), HFD, or HFD supplemented with pistachio (HFD-P) for 16 weeks. Metabolic parameters (oxidative stress, apoptosis, and mitochondrial dysfunction) were analyzed by using specific assays and biomarkers. The pistachio diet significantly reduced the serum levels of triglycerides and choleste…

0301 basic medicinemedicine.medical_specialtyobesityPhysiologyClinical BiochemistryAdipose tissuepistachiomedicine.disease_causeBiochemistryArticleSuperoxide dismutase03 medical and health sciences0302 clinical medicineInsulin resistanceInternal medicinemedicineoxidative stressMolecular Biologychemistry.chemical_classificationReactive oxygen speciesoxidative strebiologybusiness.industrylcsh:RM1-950digestive oral and skin physiologyneurodegenerationfood and beveragesnutritional and metabolic diseasesCell Biologymedicine.diseaseHeme oxygenaselcsh:Therapeutics. Pharmacology030104 developmental biologyEndocrinologychemistrybiology.proteinlipids (amino acids peptides and proteins)HFDSteatosisbusiness030217 neurology & neurosurgeryDyslipidemiaOxidative stresshormones hormone substitutes and hormone antagonists
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Nutrition, obesity and hormones

2018

Obesity is a chronic pathological condition with a multifactorial aetiology, characterised by an excessive body fat accumulation with multiple organ-specific consequences. Emerging evidence highlights that obesity appears to be associated with multiple alterations in the endocrine system. However, the mechanisms underlying the interactions between obesity and this system remain still controversial. This review discusses the impact of obesity on various endocrine systems and, in particular, would provide a general overview on the biochemical changes that may occur in each of these axes in association with obesity.

0301 basic medicinemedicine.medical_specialtyobesityPlant ScienceGrowth hormoneSettore BIO/09 - FisiologiaGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciencesInternal medicinemedicineEndocrine systemlcsh:QH301-705.5thyroid hormonesbusiness.industryBiochemistry (medical)medicine.diseaseObesityThyroid hormone030104 developmental biologyEndocrinologyendocrine systemnutritionlcsh:Biology (General)Thyroid hormonesgrowth hormoneSettore BIO/14 - FarmacologiabusinessHormoneJournal of Biological Research
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Does sex hormone-binding globulin cause insulin resistance during pubertal growth?

2019

Background The directional influences between serum sex hormone-binding globulin (SHBG), adiposity and insulin resistance during pubertal growth remain unclear. The aim of this study was to investigate bidirectional associations between SHBG and insulin resistance (HOMA-IR) and adiposity from childhood to early adulthood. Methods Participants were 396 healthy girls measured at baseline (age 11.2 years) and at 1, 2, 4 and 7.5 years. Serum concentrations of estradiol, testosterone and SHBG were determined by ELISA, glucose and insulin by enzymatic photometry, insulin-like growth factor 1 (IGF-1) by time-resolved fluoroimmunoassays, whole-body fat mass by dual-energy X-ray absorptiometry and …

0301 basic medicinemedicine.medical_specialtypubertyGlobulinEndocrinology Diabetes and Metabolismmedicine.medical_treatment030209 endocrinology & metabolismlcsh:Diseases of the endocrine glands. Clinical endocrinology03 medical and health sciences0302 clinical medicineEndocrinologyInsulin resistanceSex hormone-binding globulinInternal medicineinsulin resistanceInternal Medicinemedicinesex hormone-binding globulinkehonkoostumussukupuolihormonitadipositylcsh:RC648-665biologybusiness.industryResearchInsulinmenarcheConfoundinginsuliiniresistenssimurrosikämedicine.diseasetytöt030104 developmental biologyEndocrinologyglobuliinitHomeostatic model assessmentMenarchebiology.proteinbusinesshormones hormone substitutes and hormone antagonistsEarly pubertyEndocrine Connections
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Transient hypothyroidism during lactation alters the development of the corpus callosum in rats. An in vivo magnetic resonance image and electron mic…

2020

Magnetic resonance imaging (MRI) data of children with late diagnosed congenital hypothyroidism and cognitive alterations such as abnormal verbal memory processing suggest altered telencephalic commissural connections. The corpus callosum (CC) is the major inter-hemispheric commissure that contra-laterally connects neocortical areas. However, in late diagnosed neonates with congenital hypothyroidism, the possible effect of early transient and chronic postnatal hypothyroidism still remains unknown. We have studied the development of the anterior, middle and posterior CC, using in vivo MRI and electron microscopy in hypothyroid and control male rats. Four groups of methimazole (MMI) treated r…

0301 basic medicineneocortical developmentmedicine.medical_specialtyNeuroscience (miscellaneous)autismattention deficit/hyperactivity disorderCorpus callosumNerve conduction velocitylcsh:RC321-571lcsh:QM1-695law.invention03 medical and health sciencesCellular and Molecular Neuroscience0302 clinical medicineIn vivolawInternal medicineLactationmedicinelcsh:Neurosciences. Biological psychiatry. NeuropsychiatryOriginal Researchthyroid hormonesiodine dietmedicine.diagnostic_testbusiness.industrycongenital hypothyroidismpsychiatric diseasesMagnetic resonance imaginglcsh:Human anatomyCommissuremedicine.diseaseCongenital hypothyroidismNeuroanatomy030104 developmental biologyEndocrinologymedicine.anatomical_structureAnatomyElectron microscopebusiness030217 neurology & neurosurgery
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Genomic and non-genomic mechanisms of action of thyroid hormones and their catabolite 3,5-diiodo-l-thyronine in Mammals

2020

Since the realization that the cellular homologs of a gene found in the retrovirus that contributes to erythroblastosis in birds (v-erbA), i.e. the proto-oncogene c-erbA encodes the nuclear receptors for thyroid hormones (THs), most of the interest for THs focalized on their ability to control gene transcription. It was found, indeed, that, by regulating gene expression in many tissues, these hormones could mediate critical events both in development and in adult organisms. Among their effects, much attention was given to their ability to increase energy expenditure, and they were early proposed as anti-obesity drugs. However, their clinical use has been strongly challenged by the concomita…

0301 basic medicinenonalcoholic fatty liver diseaseobesityDiiodothyroninesEndogenyReviewthyroid hormone metabolism and transportMitochondrionmedicine.disease_causeProto-Oncogene Maslcsh:Chemistry0302 clinical medicineTranscription (biology)Settore BIO/10 - BiochimicaGene expressionSettore BIO/06 - Anatomia Comparata E CitologiaSettore MED/49 - Scienze Tecniche Dietetiche Applicatelcsh:QH301-705.5SpectroscopyMammalsReceptors Thyroid Hormonehepatic steatosisthyroid hormone mechanisms of actionGeneral Medicineresistance to thyroid hormones (RTH)Computer Science ApplicationsCell biology35-diiodo-L-thyronineThyroid Hormones030209 endocrinology & metabolismBiologyIodide PeroxidaseCatalysisInorganic Chemistry03 medical and health sciencesmedicineAnimalsHumansPhysical and Theoretical ChemistryMolecular BiologyGeneOrganic ChemistryBiological TransportLipid Metabolismhepatic steatosi030104 developmental biologyNuclear receptorlcsh:Biology (General)lcsh:QD1-999MutationBasal MetabolismLipid PeroxidationOxidative stressHormone
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Human POMC processing in vitro and in vivo revealed by quantitative peptidomics

2018

ABSTRACTHuman obesity can result from the aberrant production or processing of proopiomelanocortin (POMC) in hypothalamic neurons, but it is unclear which human POMC-derived peptides are most relevant to body weight regulation. To address this question, we analysed both hypothalamic neurons derived from human pluripotent stem cells (hPSCs) and primary human hypothalamic tissue using quantitative liquid chromatography tandem mass spectroscopy (LC-MS/MS). In both in vitro- and in vivo-derived samples, we found that POMC was processed into β-melanocyte stimulating hormone (β-MSH), whose existence in the human brain has been controversial. β-MSH and desacetyl α-MSH (d-α-MSH) were produced at ro…

0303 health sciencesendocrine systembiologyintegumentary systemChemistryHuman brainIn vitroCell biology03 medical and health sciences0302 clinical medicinemedicine.anatomical_structureProopiomelanocortinAcetylationIn vivomedicinebiology.proteinSecretionInduced pluripotent stem cell030217 neurology & neurosurgeryhormones hormone substitutes and hormone antagonists030304 developmental biologyHormone
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Estrogen Regulates the Satellite Cell Compartment in Females

2018

SUMMARYSkeletal muscle mass, strength, and regenerative capacity decline with age, with many measures showing greater deterioration in females about the time estrogen levels decrease at menopause. Here we show that maintenance of muscle stem cells, satellite cells, as well as self-renewal and differentiation into muscle fibers, are severely compromised by estrogen deficiency. Mechanistically, by hormone replacement, use of a selective estrogen-receptor modulator (bazedoxifene), and conditional estrogen receptor knockout, we implicate 17β-estradiol and satellite cell expression of estrogen receptorα(ERα) and show that estrogen signaling through this receptor is necessary to prevent apoptosis…

0303 health sciencesmedicine.medical_specialtymedicine.drug_classCellEstrogen receptorSkeletal muscleBiologymedicine.diseaseBazedoxifeneMenopause03 medical and health sciences0302 clinical medicinemedicine.anatomical_structureEndocrinologyApoptosisEstrogenInternal medicinemedicineStem cellReceptorEstrogen receptor alpha030217 neurology & neurosurgeryhormones hormone substitutes and hormone antagonists030304 developmental biologymedicine.drugSSRN Electronic Journal
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